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1 olved in transcription, DNA repair, and cell division.
2 ce of genes involved in bacterial growth and division.
3 n of enzymes involved in cell elongation and division.
4 curate alignment and segregation during cell division.
5 e segregation, and it ultimately delays cell division.
6 standing the coordination of cell growth and division.
7 to the spatial and temporal control of cell division.
8 lasmic reorganization, before the first cell division.
9 r to progression to metaphase and subsequent division.
10 latory step in the process of bacterial cell division.
11 ed transmembrane proteins essential for cell division.
12 nome in S phase and segregate it during cell division.
13 d previously unknown modes of bacterial cell division.
14 ication and segregation with cell growth and division.
15 be attributed to apoptosis and reduced cell division.
16 ing bacterial cell shape and permitting cell division.
17 is regulated by a balance between fusion and division.
18 iably to the two daughter cells through cell division.
19 e role of Nek8445 in regulating Giardia cell division.
20 for protein import, peroxisomal growth, and division.
21 egulated in order to support cell growth and division.
22 ylation, is stably propagated during mitotic division.
23 that are asymmetrically retained during cell division.
24 rate (Ser) function redundantly in the third division.
25 the bacteria, in particular to assist their division.
26 estrating chromosome segregation during cell division.
27 3 (ARC3), a crucial regulator of chloroplast division.
28 nregulated were critical for cell growth and division.
29 by favouring symmetric over asymmetric cell division.
30 targeted to growing cell plates during cell division.
31 n synthase relocation to midcell during cell division.
32 genome to the daughter cells following cell division.
33 bled at the future division site during cell division.
34 through asymmetric segregation during active division.
35 lobus cells undergo a robust and symmetrical division.
36 simultaneously imaging the cells to measure division.
37 more biomass than larger cells prior to cell division.
38 d space and could play similar roles in cell division.
39 n activating some of the key players in cell division.
40 spindle, which then guides the axis of cell division.
41 ptal PG synthesis and regulation during cell division.
42 chromosome segregation at the first meiotic division.
43 cal adhesion formation and turnover and cell division.
44 tion theory and the structural model of cell division.
45 hey were detected at the midbody during cell division.
46 to control the metabolism, growth, and cell division.
47 tone to bacterial cell wall biosynthesis and division.
48 ocytes progressing through the first meiotic division.
49 fe including cell development, movement, and division.
50 y macrophages and were sites of ongoing cell division.
51 separates daughter cells and completes cell division.
52 nerating two distinct cells through one cell division.
53 synthesis in ovococcal bacteria during cell division.
54 l that undergoes predominant asymmetric cell division.
55 ntrations of key metabolites needed for cell division.
56 factors are operating in the context of cell division.
57 mmetries featured in self-renewing stem cell divisions.
58 neurogenic progenitors arise from asymmetric divisions.
59 D) plane through a series of asymmetric cell divisions.
60 ring mitotic rather than during meiotic cell divisions.
61 -wide whose fluctuations persist for several divisions.
62 ssumption that germline mutations track cell divisions.
63 enishment divisions and consuming neurogenic divisions.
64 with planar-oriented symmetric self-renewal divisions.
65 uplication during synchronous embryonic cell divisions.
66 molting, and the proper pattern of seam-cell divisions.
67 Notch signaling between the second and third divisions.
68 memory in individual cells through multiple divisions.
69 s that are distinct from cell elongation and division?
72 ose that SlGBP1 acts as an inhibitor of cell division, a function conserved with the human hGBP-1 pro
73 e layered diagrammatic representation of CNS divisions, according to their arrangement in correspondi
74 h may underlie the drastic reduction in cell division activity in both shoot and root apical meristem
75 transcriptional programs that can drive the division and (trans-)differentiation of non-beta cells t
77 not negatively impact the synchrony of cell division and cell cycle progression during diel growth.
80 ily known for its essential activity in cell division and cortical granule exocytosis, in development
82 of a "transition zone" boundary between the division and elongation zones is critical for efficient
88 e segregation is essential for faithful cell division and if not maintained results in defective cell
91 h requires not only tight regulation of cell division and metabolism, but also concerted control of t
97 ignal, and consequently limits aberrant cell division and potential cellular transformation through a
98 the synthesis of the procuticle during cell division and provide a template for further cutin deposi
100 r model established the timing of QC and CEI division and suggests that SHR repression of QC division
101 otubule network integrity and dynamics, cell division and survival, with biological response on the t
104 signaling pathway regulating formative root divisions and provides a framework to explore this pathw
106 ects of motility, membrane trafficking, cell division, and death), but others are more unique feature
107 ociated with compartments - collocalization, division, and merging - which are commonly thought to pl
108 s, the protein number partitioning upon cell division, and the modeling of cell communication (juxtac
110 f the spindle is crucial for the proper cell division, and two centrosomes in animal cells naturally
115 y in time - and suggested crowding from cell division at the apical surface drives basalward motion.
116 trization events of normally asymmetric cell divisions at the fourth larval stage, leading to the ret
117 e. induction of cell death and inhibition of division, at a single-cell level are used as metrics to
119 plied in specifying and reorienting the cell division axis, but how general such reorientation events
120 eems to contribute both to PG elongation and division biosynthetic complexes based on its localizatio
121 regulation of genes involved in mitotic cell division but an up-regulation of genes involved in apopt
122 ing protein 3 (PBP3/FtsI) initiate polarized division, but the process arrests at an early stage of d
123 mbrane constriction is coordinated with cell division by active mobilisation-and-capture of Pal at di
124 clin induced pavement cells to re-enter cell division by establishing mitotic microtubule arrays.
125 tubules formed by Drp1 promote mitochondrial division by facilitating ER-mitochondria interactions.
127 ebral bridge (PB), the upper (CBU) and lower division (CBL) of the central body and a pair of globula
128 he integrity of the cell wall, disrupts cell division, cell separation, and impairs the fitness of th
131 ggest that CDKA1 may promote a switch into a division-competent state, and E2F/Dp1 may promote mainte
133 coextensive model has been proposed in which division-coupled conversion of NSCs into differentiated
135 bition of Rac family small GTPase 1 and cell division cycle 42 activation, as well as downstream intr
136 t cancer cell cycle, is associated with Cell Division Cycle 6 (CDC6), Cyclin-dependent kinase 2 (CDK2
138 hase-promoting complex (APC/C) bound to Cell division cycle protein 20 (CDC20), and ends upon mitotic
139 impacts of the cht7 mutation during the cell division cycle under nutrient deficiency in light-dark s
140 ersistence is characterized by a halt in the division cycle, aberrant morphology, and, in the case of
142 parasites have a life cycle with unique cell-division cycles, and a repertoire of divergent CDKs and
144 ision and suggests that SHR repression of QC division depends on formation of the SHR homodimer.
148 Dw2 modulates endomembrane function and cell division during sorghum internode growth, providing insi
152 variety of cellular processes including cell division, endosomal vesicle trafficking, and viral buddi
153 on, resulted in increased cardiomyocyte cell division, enhanced cardiac function, and improved long-t
154 ptions are thought to originate from meiotic division errors in the female germline, quantitative stu
156 n excellent model for understanding how cell division, expansion and differentiation are coordinated
159 a role for the G-proteins in regulating GSC division frequency, RNA-i against seven out of 35 G-prot
161 during cell elongation (RodA-PBP2) and cell division (FtsW-FtsI), and we are still uncovering the im
162 eration pathways, indicating a role for cell division genes in somatic evolution in healthy skin.
163 e MTOC inheritance patterns during stem cell division have been associated with accelerated cellular
170 ction with septation is critical to faithful division in Gram-negative bacteria and vital to the barr
172 deleting OCRL on endocytic traffic and cell division in newly created human PT CRISPR/Cas9 OCRL knoc
173 ferentially inherited during asymmetric cell division in organisms ranging from yeast to humans.
174 uard, that adjusts the balance of fusion and division in response to increased mitochondrial connecti
175 Here, in exploring the mechanism of cell division in S. acidocaldarius, we identify a role for th
176 lower developmental stages by promoting cell division in the distal and ventral portion of the limb.
177 t class II TCP genes also contribute to cell division in the leaf, the gynoecium and the ovules in A.
180 X3/NS1 does not traffic, and stimulates cell divisions in the same cells in which it is transcribed.
181 l ASC induction requires at least eight cell divisions in vivo, with BLIMP-1 being required for diffe
182 rs an innate immune response and drives cell division independently of known density-dependent prolif
185 The inheritance of regulators through cell division is a key deterministic force, but identifying i
189 cell division occurs as per the Adder model (division is triggered upon adding a fixed size from birt
192 on of plant cells, the control of plant cell division leading to nodule development, autoregulation o
193 BC1D15-NuMA1 association impaired asymmetric division machinery by hijacking NuMA from LGN binding, t
194 we reconstituted part of the bacterial cell division machinery using its purified components FtsZ, F
197 Brady Jr." from the "Psychotic Disorders Division, McLean Hospital, Harvard Medical School, Belmo
199 ere, we report a dual-comb Vernier frequency division method to vastly reduce the required electrical
201 Partitioning of this local pool at each cell division modulates nuclear growth kinetics and dictates
202 25 bps) prototype and an 8-channel frequency-division multiplexed (0.5 m x 1,000 bps) prototype are d
203 ze for excessively large cells; and (2) cell division occurs as per the Adder model (division is trig
206 we sought to systematically characterize the division of labor between YAP and TAZ in non-small cell
207 e implications for both fork restart and the division of labor during leading-strand synthesis genera
210 w-celled haploid structure that orchestrates division of labor to coordinate successful interaction w
211 ganization, cells in each state exhibit true division of labor, providing growth/survival advantages
213 present an updated freshwater biogeographic division of mainland Southeast Asia and describe 12 spec
215 distinction is reflected in a medial-lateral division of prefrontal cortex - with lateral frontal pol
217 D-19 first wave for the 9 departments in the Division of Surgery at The University of Texas MD Anders
218 ivision of the central body (CBU), the lower division of the central body (CBL), and the paired nodul
219 ses the protocerebral bridge (PB), the upper division of the central body (CBU), the lower division o
221 ange more, and the typical structure shows a division of the society in upper and lower classes.
222 encoding of predator fear in the dorsomedial division of the ventromedial hypothalamus (VMHdm) and th
223 eadfins (Percomorphacea: Polynemidae) is the division of their pectoral fin into an upper, unmodified
224 ) by simultaneously measuring metabolism and division of thousands of individual Saccharomyces cerevi
225 gic therapy and regularly followed up at the divisions of dermatology of several main hospitals in th
226 1 in controlling the length of terminal cell divisions of outer radial glial (oRG) progenitors, sugge
228 idence for altered activity within the major divisions of the prefrontal cortex, including orbitofron
229 le-cell measurements of bacterial growth and division often relied on testing preconceived models of
231 sterior to anterior, dictates the pattern of divisions over the FSC domain, promotes more posterior F
234 stent with their distinct roles in polarized division, peptidoglycan organization is different in cel
237 m concentration increases transiently at the division plane just before cleavage furrow ingression, s
238 initial cell and the angle of the transition division plane, which sufficiently distinguished a gamet
240 divisions that coincide with different cell division planes and growth directions enable the develop
241 When PLK4 is chemically inhibited, cell division proceeds without centrosome duplication, genera
244 pon the polymerization of the bacterial cell division protein FtsZ (a homolog of tubulin); 5) is comp
245 ts have implicated a conserved subcomplex of division proteins composed of FtsQ, FtsL, and FtsB (FtsQ
246 erochromatin can be transmitted through cell division provided the counteracting demethylase Epe1 is
247 aried by hospital referral region and Census Division (range: 18.6% [East South Central] to 39.1% [We
248 suggests that a 10% change in phytoplankton division rate may be associated with a 50% reduction in
249 These differences are related to the cell division rate of the studied cells and thus suggest an i
250 nmental properties controlling phytoplankton division rates (e.g., nutrients and light), as they omit
251 wound-induced cell expansion and restorative division rates in a dose-dependent manner, leading to tu
253 ing cell number regulation that acts on cell division rates such that the number of cells in the tiss
254 ccurring during periods of declining modeled division rates, an observation that highlights the impor
256 d division, cell-fate decision in the second division requires a lower level of Suppressor of Hairles
258 stems belonging to the resistance-nodulation-division (RND) superfamily are ubiquitous in Gram-negati
259 by efflux pumps of the Resistance-Nodulation-Division (RND) superfamily of proteins creating an effic
268 lapping functions with co-regulators of cell division such as the cohesin subunits SMC1, SMC3, NIPBL
269 PARC6), another key regulator of chloroplast division, suggesting a role of OR(His) in competing with
271 opagated during "tandem duplication," a cell division that remodels the parental cell into two daught
273 ely studied, how cell shape changes and cell divisions that occur concurrently in epithelia influence
276 lls, we found that cell growth is coupled to division through a sizer operating largely in the G1 pha
277 regulation of SHR transcription modulate the division timing of two different specialized cell types
280 ells the capacity to undergo periclinal cell division to repopulate the vascular stem cell pool.
281 hat human OPCs undergo consecutive symmetric divisions to exponentially increase the progenitor pool
283 can aid in diverse processes, including cell division, transcription regulation, and cell signaling.
284 ounding and cortical stiffening promote cell division under confined conditions that are similar to t
285 n caudate nucleus and putamen-a striatal sub-division unique to primates-with both dopamine and serot
289 ith MDH, tested and confirmed at our allergy division, were retrospectively evaluated in terms of cli
290 tomatal development requires asymmetric cell division, where the nucleus moves to the division site b
291 nherited by outer daughter cells during cell division, where they stabilize the cortex to promote api
292 proteins in cells underlies asymmetric cell division, which is an important driver of development an
293 in 2 (PBP2) are unable to initiate polarized division, while cells treated with inhibitors that preve
294 ' are induced upon normal and cancerous cell division, while the 'differentiation-tRNAs' are active i
296 ential to couple genome duplication and cell division with the establishment and maintenance of cellu
297 ting a fixed fate imbalance of self-renewing divisions with an ever-decreasing proliferation rate.
298 ut also by limited numbers of symmetric cell divisions with some characteristics reminiscent of inter
299 increased the proportion of planar-oriented divisions, without effecting SC viability, fibronectin d
300 thesis was that the cells preparing for cell division would consume more energy and metabolites as bu