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1 d-type lysozyme with a higher catalytic rate double deletion mutant.
2 with a single deletion in speA or speC and a double deletion mutant.
3 effect is abolished in the N- and C-terminal double deletion mutant.
4 (SRPK1) complements the growth defect of the double-deletion mutant.
5 o create diploid strains that are homozygous double-deletion mutants.
6                                      Using a double-deletion mutant (12203) of CPXV, we show that dir
7                       The DeltaptlEDeltaptlD double deletion mutant accumulated 12, the substrate for
8                                          The double deletion mutant also exhibited substantially redu
9                                The nhaA nhaB double deletion mutant also showed inhibited growth in a
10                                              Double deletion mutants also display phenotypes associat
11                The colonies of the rbmC bap1 double deletion mutant and bap1 single deletion mutant e
12 nvolved construction of isogenic single- and double-deletion mutants and inoculation of 9-day-old F4a
13 80% of the wild-type apoA-I control value by double deletion mutants apoA-I[delta(1-41)delta(185-243)
14                           Expression of this double-deletion mutant as a glutathione S-transferase fu
15  from the expected sickness or health of the double-deletion mutant as compared to its constituent si
16                               We find that a double deletion mutant (Delta ces1 Delta ces4 ) grows at
17 d to plasma and wild-type apoA-I, and in the double deletion mutants, Delta(1-41, 185-243) and Delta(
18 t activate NF-kappaB in cells expressing the double deletion mutant Delta133-224/Delta372-419.
19 f a moderate magnitude were recovered by the double deletion mutant Delta671-690/Delta720-765.
20                                            A double deletion mutant (DeltasisA DeltasisB) resulted in
21              RPA0376 and RPA2838 single- and double-deletion mutants did not differ in longevity from
22 ast to the single mutants, the robA and nudG double deletion mutant exhibits a severe nud phenotype a
23 ane profiling of cells infected with an HCMV double-deletion mutant for UL148 and UL148D with restore
24 f NPC1, ncr-1 and ncr-2, and an ncr-2; ncr-1 double deletion mutant forms dauer larvae constitutively
25 e largely unchanged total sterol levels, the double deletion mutant (hms1-1Delta hms1-2Delta) exhibit
26 ored the survival of the Y. pestis nhaA nhaB double deletion mutant in blood.
27 ular lysophospholipase activity, a plb1/plb2 double deletion mutant is completely devoid of lysophosp
28 ts display wild-type conidial germination, a double-deletion mutant is delayed in germination in resp
29                                              Double deletion mutants lacking pgpA and pgpB are viable
30 osystem I-less background strain, and also a double-deletion mutant lacking both plastocyanin and cyt
31                                          The double deletion mutant of apoA-I had reduced thermal and
32 m, the endotoxicity and CAMP resistance of a double deletion mutant of lpxE-eptA was similar to that
33            The WT apoA-I, amino, carboxy and double deletion mutants of apoA-I showed differences in
34 ss-linking of WT apoA-I, amino-terminal, and double deletion mutants of apoA-I to ABCA1 showed simila
35                           We made single and double deletion mutants of ccmK3 and ccmK4 in Synechococ
36 y analysis of gene expression in single- and double-deletion mutants of each protein to identify thei
37                          Cultures of the p53 double deletion mutant ovarian cell line SK-OV-3 were in
38 ith a T3SS1 deletion mutant or a T3SS1 T3SS2 double deletion mutant produced no mortality.
39 nal populations with minimal toxicity, using double-deletion-mutant rabies viruses.
40       Analyses of ert-m and ant1 single- and double-deletion mutants suggest Ant1 as a closely linked
41 or the wild-type RNA is 60 degreesC; for the double-deletion mutant U2653Delta/C2667Delta it is 65 de
42                                The resulting double deletion mutant virus strain, dl5-29, fails to fo
43                   Furthermore, the rbmC bap1 double deletion mutant was unable to form biofilms at th
44                                  A menF dhbC double-deletion mutant was both MK and DHB deficient.
45 ence in a mouse peritonitis model, whereas a double-deletion mutant was highly attenuated (P<.004) ve
46 ccharomyces cerevisiae) vps23Delta bro1Delta double-deletion mutant, we showed that the Vps23p ESCRT-
47 an rescue the lethal phenotype of a min slmA double deletion mutant, which we think is due to the eli
48                                          The double deletion mutant with deletions in both copies of
49                      The construction of the double deletion mutant without Arg208His209 and its NMR