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1 ructure and function across broad classes of drug receptors.
2 cellular signal transduction and targeted as drug receptors.
3 inding can yield important information about drug-receptor and drug-drug interactions for a wide rang
4 ctural foundation for future optimization of drug-receptor binding and unbinding rates.
5 hich may be used for direct determination of drug-receptor binding interactions and for the rapid on-
6                                              Drug-receptor binding interactions of four agonists, ACh
7                       From DNA base pairs to drug-receptor binding, hydrogen (H-)bonding and aromatic
8 ane protein, was responsible for the altered drug-receptor binding.
9 on can be partially decoupled by varying the drug-receptor dissociation rate constant, k(off), and th
10 ould also aid drug discovery by establishing drug/receptor engagement.
11            These fundamental correlations on drug-receptor H-bond interactions may be generally usefu
12 r receptor exclusively activated by designer drug) receptor hM4Di in sensorimotor cortex and AAV-expr
13 r receptor exclusively activated by designer drugs) receptor hM4di abrogated spontaneous functional r
14 ropose that HERG inactivation stabilizes the drug-receptor interaction during membrane depolarization
15 nt for activity, but its precise role in the drug-receptor interaction has not been specifically inve
16 esses its receptor in the open pore, and the drug-receptor interaction is then stabilized by inactiva
17 e molecular mechanism of this double-faceted drug-receptor interaction may help in designing new ther
18                                              Drug-receptor interaction theory predicts that proportio
19 and in determining important elements of the drug-receptor interaction.
20 mbrane receptors provide valuable models for drug-receptor interactions across many important classes
21                                     Modeling drug-receptor interactions at the 3D level offers consid
22           This effect of a third molecule on drug-receptor interactions cannot be studied using tradi
23 ding sites, bound conformations and specific drug-receptor interactions for several allosteric modula
24  unnatural amino acid incorporation to probe drug-receptor interactions in functional G protein-coupl
25 d with studies using rabbit aorta to examine drug-receptor interactions in vascular smooth muscle.
26 d as an important factor in protein folding, drug-receptor interactions, and catalyst selectivities.
27 cokinetics, blood-brain barrier penetration, drug-receptor interactions, heavy metal toxicity, pharma
28 variation may be secondary to differences in drug-receptor interactions, patient's compliance and tol
29 he understanding of the structural bases for drug-receptor interactions.
30 alignments from SQ are consistent with known drug-receptor interactions.
31                                              Drug receptor kinetics is as a key component in drug dis
32  signaling in the translational control of a drug receptor KOR, which involves the mediator of netrin
33 ontributions of F1764 and Y1771 to a complex drug receptor site in the pore of Na+ channels.
34 residues that are analogous to the mammalian drug receptor site, and the lateral pore fenestrations.
35  receptors exclusively activated by designer drugs) receptors, we enhanced Gi/o- or Gs-protein-mediat