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1 s of drugs of abuse and, possibly, increased drug seeking behavior.
2 ing the released glutamate from synapses and drug seeking behavior.
3 ssociated cues can lead to drug cravings and drug seeking behavior.
4 heroin dependence and their association with drug seeking behavior.
5 y contribute to cue-induced drug craving and drug-seeking behavior.
6 indings that have important implications for drug-seeking behavior.
7 this crucial circuitry to promote compulsive drug-seeking behavior.
8 icantly enhanced resistance to extinction of drug-seeking behavior.
9 inhibition on sensitized behavior to foster drug-seeking behavior.
10 tal cellular phenomenon driving pathological drug-seeking behavior.
11 aminergic neurons involved in motivation and drug-seeking behavior.
12 hereas "novelty-seeking" predicts compulsive drug-seeking behavior.
13 t/orx has been implicated in food reward and drug-seeking behavior.
14 accumbens (NAc) exerts powerful control over drug-seeking behavior.
15 lves, which is an effect that could underlie drug-seeking behavior.
16 symptoms of cocaine addiction is compulsive drug-seeking behavior.
17 se, such as ethanol, can trigger craving and drug-seeking behavior.
18 and glutamatergic systems may play a role in drug-seeking behavior.
19 e ventral prefrontal cortex had no effect on drug-seeking behavior.
20 including physical dependence and compulsive drug-seeking behavior.
21 rtant for maintaining prolonged sequences of drug-seeking behavior.
22 ns underlies cocaine-primed reinstatement of drug-seeking behavior.
23 ere inactivation of only the cBLA attenuated drug-seeking behavior.
24 geneous than commonly thought for regulating drug-seeking behavior.
25 rding effects of cocaine and cocaine-induced drug-seeking behavior.
26 io-sexual context appears to enhance further drug-seeking behavior.
27 actions between these factors for motivating drug-seeking behavior.
28 series circuit that mediates cocaine-induced drug-seeking behavior.
29 t also play a role in drug reinforcement and drug-seeking behavior.
30 by inducing a craving response that prompts drug-seeking behavior.
31 e transmission in cocaine-induced relapse to drug-seeking behavior.
32 mediator of cocaine-induced reinstatement of drug-seeking behavior.
33 d of abstinence had significantly heightened drug-seeking behavior.
34 rsus non-drug context that was predictive of drug-seeking behavior.
35 nter-imposed abstinence to induce heightened drug-seeking behavior.
36 programming of brain circuitry that leads to drug-seeking behavior.
37 iated cues during abstinent reinstatement of drug-seeking behavior.
38 d individuals with low DT exhibit heightened drug-seeking behavior.
39 nhibition of this pathway blunts cue-induced drug-seeking behavior.
40 d of abstinence had significantly heightened drug-seeking behavior.
41 and (heroin + cue)-induced reinstatement of drug-seeking behavior.
42 avior and may have a role in the etiology of drug-seeking behavior.
43 e manipulates striatal circuits to reinforce drug-seeking behavior.
44 1- and D2-MSNs to limit distinct triggers of drug-seeking behavior.
45 eries of tasks to evaluate goal-directed and drug-seeking behavior.
46 d heroin self-administration and cue-induced drug-seeking behavior.
47 th the ability of a drug-paired cue to drive drug-seeking behavior.
48 ired CSs intact and able to continue driving drug-seeking behavior.
49 It also failed to induce reinstatement of drug-seeking behavior.
50 hin the DG that could directly contribute to drug-seeking behavior.
51 ivational states that instigate and maintain drug-seeking behavior.
52 ian memories that can precipitate relapse to drug-seeking behavior.
53 Addiction involves an inability to control drug-seeking behavior.
54 discover new therapeutic candidates to treat drug-seeking behavior.
55 drug users, and are thought to facilitate a drug-seeking behavior.
56 d effects on an addict's emotional state and drug-seeking behavior.
57 ortex (mPFC) is implicated in the relapse of drug-seeking behavior.
58 muli, such cues can instigate and invigorate drug-seeking behavior.
59 t stable rates of responding and conditioned drug-seeking behavior.
60 C) that promote behavioral sensitization and drug-seeking behavior.
61 e memory processes involved in extinction of drug-seeking behavior.
62 ecific HDAC is involved in the extinction of drug-seeking behavior.
63 The ventral pallidum (VP) is necessary for drug-seeking behavior.
64 craving") that instigates and/or invigorates drug-seeking behavior.
65 ological responses, craving, withdrawal, and drug-seeking behavior.
66 the study of anxiogenesis and stress-induced drug-seeking behavior.
67 ility of drug-predictive cues to precipitate drug-seeking behavior.
68 utamate signaling plays an essential role in drug-seeking behavior.
69 sducing stimuli into salient cues that drive drug-seeking behavior.
70 tic disorders or predispose an individual to drug-seeking behavior.
71 activation in drug-induced reinstatement of drug-seeking behavior.
72 d to cocaine-conditioned cues and relapse to drug seeking behaviors.
73 fic behavioral modifications associated with drug seeking behaviors.
74 tress modulation, depression, and relapse to drug-seeking behaviors.
75 considerations when designing experiments on drug-seeking behaviors.
76 in dopamine-mediated craving and relapse to drug-seeking behaviors.
77 caine experience were used to predict future drug-seeking behaviors.
78 n opioid withdrawal, sleep disturbances, and drug-seeking behaviors.
79 robiological substrates that drive food- and drug-seeking behaviors.
80 hich may contribute to maladaptive food- and drug-seeking behaviors.
81 maladaptive plasticity that drives food- and drug-seeking behaviors.
82 med in regions involved in the extinction of drug-seeking behaviors.
83 in experience-dependent neuroplasticity and drug-seeking behaviors.
84 differences in extinction and incubation of drug-seeking behaviors.
85 g modulator that potentially plays a role in drug-seeking behaviors.
86 at regulates orexin neuronal activity during drug-seeking behaviors.
87 modulating gene expression that may subserve drug-seeking behaviors.
88 onergic state underlying depression-like and drug-seeking behaviors.
89 l raphe nucleus that mediates depressive and drug-seeking behaviors.
90 ed memories.SIGNIFICANCE STATEMENT Continued drug-seeking behavior, a defining characteristic of coca
91 d subsequent enhanced locomotor response and drug seeking behavior after repeated cocaine administrat
92 ished efficacy of the cocaine S(D) to elicit drug-seeking behavior after 4 months of abstinence paral
93 e with the ability of multiple cues to drive drug-seeking behavior after just one reactivation and tr
94 he accumulation of CP-AMPARs and cue-induced drug-seeking behavior after prolonged abstinence from co
95 efeat (ISD), a stress protocol that promotes drug-seeking behavior, alters intertemporal decision-mak
96 t stimulus-control over both drug taking and drug seeking behavior and are difficult to extinguish.
98 iver of neurobehavioral pathology related to drug-seeking behavior and behavioral updating, thus iden
99 exerts modulatory control over cue-motivated drug-seeking behavior and downstream neuroimmune functio
100 with OCT3 mediates corticosterone effects on drug-seeking behavior and establish OCT3 function as an
101 e neuroadaptations underlying stress-induced drug-seeking behavior and may be useful in the treatment
102 tagonist, on cocaine-primed reinstatement of drug-seeking behavior and on cocaine-induced changes in
103 al justice system often results from illegal drug-seeking behavior and participation in illegal activ
104 cleus accumbens (NAc) facilitate conditioned drug-seeking behavior and primarily originate from media
106 opeptide released into the VTA that promotes drug-seeking behaviors and potentiates excitatory synapt
108 ession, is well known to increase relapse of drug seeking behavior, and can adversely impact health.
111 egions associated with reward processing and drug-seeking behavior, and recent evidence suggests that
112 iously drug-associated stimuli on subsequent drug-seeking behavior, and the NMDA subtype of glutamate
117 ketamine doses used were capable of inducing drug-seeking behaviors as measured by place preference c
119 footshock stress did not by itself reinstate drug-seeking behavior but potentiated reinstatement in r
120 hippocampal neurogenesis and drug-taking or drug-seeking behaviors, but the lack of a causative link
121 xciting new possibility is the extinction of drug-seeking behavior by manipulation of epigenetic mech
122 ests that VNS consolidates the extinction of drug-seeking behavior by reversing drug-induced changes
125 In the present study, the extent to which drug-seeking behavior could be subject to these differen
126 e-associated cue was sufficient to reinstate drug-seeking behavior, despite the continued presence of
130 tly extinguished and spontaneous recovery of drug-seeking behavior following presentation of previous
131 n about the role that astrocytes may play in drug-seeking behavior for commonly abused substances.
134 a mean of preventing the "high" and reducing drug-seeking behavior has become a major strategy in med
137 established, the neurophysiological bases of drug-seeking behavior have yet to be fully elucidated.
138 s been shown to facilitate the extinction of drug-seeking behavior in a manner resistant to reinstate
139 (2) priming- or cue-induced reinstatement of drug-seeking behavior in abstinent subjects (models of r
143 demonstrated that 1 significantly attenuates drug-seeking behavior in an animal model of cocaine rela
144 potent of the analogues successfully reduced drug-seeking behavior in an animal model of drug-relapse
145 hanges in reward circuit activity that drive drug-seeking behavior in animal models of addiction.
147 ntal cues, suggesting that cues that trigger drug-seeking behavior in nonhuman primates do not cause
150 hat this disconnection selectively decreased drug-seeking behavior in rats extensively trained under
151 ions on the maintenance and reinstatement of drug-seeking behavior in rats trained to self-administer
155 we tested whether an estrogen could augment drug-seeking behavior in response to an ordinarily subth
156 nd memory reactivation-dependent decrease in drug-seeking behavior in the cocaine-predictive context
157 ions and in the mediation of drug-taking and drug-seeking behaviors in animal models of addiction.
160 on of only the rBLA blocked reinstatement of drug-seeking behavior induced by cocaine-associated cues
161 BLA and cBLA attenuated the reinstatement of drug-seeking behavior induced by cocaine-associated cues
165 ms by which stress triggers reinstatement of drug-seeking behaviors is particularly pertinent to nico
166 oned behaviors, such as conditioned fear and drug-seeking behavior, is a process of active learning,
167 blockade of MORs in the VTA counteracted two drug-seeking behaviors, locomotor activity and place pre
168 Furthermore, dCA3 inhibition attenuated drug-seeking behavior (non-reinforced lever presses) sel
169 e primary targets for cocaine, the continued drug-seeking behavior of transporter knock-out mice sugg
171 session, a drug-priming injection reinstated drug-seeking behavior only in rats that in the past had
172 (vGluT2) neurons have contrasting effects on drug-seeking behavior, our data may indicate a complex r
173 via their metabolites, are key regulators of drug-seeking behaviors, positioning the microbiome as a
174 ears to contribute both to the initiation of drug-seeking behavior (pre-lever press phasic neuronal r
175 kg, i.p.) alone were sufficient to reinstate drug-seeking behavior, pretreatment with E2 potentiated
176 comotor-activating effects of cocaine and on drug-seeking behavior, rats receiving methyl supplementa
178 e brain regions can trigger reinstatement of drug-seeking behavior.SIGNIFICANCE STATEMENT It is well
179 tly reduced previously acquired instrumental drug-seeking behavior that depends on drug-associated cu
180 pproach to facilitate learned suppression of drug-seeking behavior that may aid drug abstinence.
181 sions profoundly impaired the acquisition of drug-seeking behavior that was maintained by drug-associ
182 Pavlovian and operant conditioning influence drug-seeking behavior, the role of rapid dopamine signal
183 ssential for cocaine self-administration and drug-seeking behavior, there is limited knowledge of the
187 cellular dopamine during extended periods of drug-seeking behavior triggered by a visual cue was dete
188 pear to selectively and dissociably regulate drug-seeking behavior under conditions of cocaine abstin
189 Drugs of abuse are able to elicit compulsive drug-seeking behaviors upon repeated administration, whi
190 cannabis, on heroin self-administration and drug-seeking behavior using an experimental rat model.
191 of GRIA1, a glutamatergic gene implicated in drug-seeking behavior, verified the increased enrichment
192 ia and hyperkatifeia, which drive pronounced drug-seeking behavior via processes of negative reinforc
195 During subsequent reinstatement sessions, drug-seeking behavior was assessed after noncontingent p
200 rimed, but not cue-induced, reinstatement of drug-seeking behavior, whereas inhibition of SNr GABA ne
201 ration, and cocaine-induced reinstatement of drug-seeking behavior, whereas R-MOD inhibited cocaine-i