ライフサイエンスコーパス: eat

1 es of classical aging mutants (daf-2, age-1, eat-2, and daf-16) and animals subjected to RNAi knockdo

2 ed with 56.8%; number needed to treat=5.11), eating disorder symptoms (body shape, weight, eating con

3 ntified 4 moderately heritable (h2 = 36-48%) eating behavior patterns labeled "snacking," "infrequent

4 In addition, a greater eating frequency (>=5 eating occasions/d) was significantly associated with lo

5 , eating when feeling full [2.9% (1.2-4.5)], eating continuously [1.6% (0.1-3.1)], binge eating and l

6 are providers should routinely enquire about eating habits as a component of overall health assessmen

7 namic image processing for simulating actual eating situations.

8 luding substance and behavioural addictions, eating disorders, and attention deficit/hyperactivity di

9 ly regulate fat metabolism without affecting eating behaviors in Caenorhabditis elegans, and identifi

10 zes, and to broaden the focus to include all eating disorder types.

11 control diet typical of what many Americans eat; a diet rich in fruits and vegetables but otherwise

12 An eating performance cycle is defined as the process of ge

13 lity to obesity can be partly mediated by an eating pattern characterized by frequent snacking.

14 te, and energy intake during lunch and in an eating in the absence of hunger task.

15 tonation, and alkoxide addition to form an "-ate" complex.

16 the chicks were less likely to approach and eat prey with high contrast compared to low contrast pat

17 ions, 'Avoidance' (latencies to approach and eat the novel food) did not predict boldness trait, and

18 s correlate with the capacity to migrate and eat bacteria.

19 and "Choose unsaturated oils and spreads and eat in small amounts such as vegetable, rapeseed, olive

20 (acyl chains for phospholipid synthesis) and eat it (degrade acyl chains to acetyl-CoA).

21 bioluminescence using N2 reference worms and eat-2 mutants, and found a nearly 100-fold increase in s

22 153), substance use disorders (N = 131), and eating disorders (N = 14)-who failed to increase their p

23 CI 2-27), insomnia (MD 20, 95% CI 8-32), and eating problems (MD 14, 95% CI 3-24) compared to patient

24 athology, including depression, anxiety, and eating disorders.

25 Energy balance and eating behavior were also normal, even after exposure to

26 bulimia nervosa, binge-eating disorder, and eating disorder not otherwise specified) for probands an

27 patterns (meal timing, eating duration, and eating frequency), and we obtained the daily profile of

28 Six main feeding and eating disorders are now recognised in diagnostic system

29 all conditions categorized under feeding and eating disorders.

30 s a caregiver, and resident age, gender, and eating function.

31 21) controls metabolic organ homeostasis and eating/drinking behavior via FGF receptor 1/Klothobeta (

32 (authorized under conditions for hunting and eating in New Caledonia) we investigated the role of the

33 ood consumption is fundamental for life, and eating disorders often result in devastating or life-thr

34 tastes predicts similar food preferences and eating behavior in later life and is associated with chi

35 ns in symptoms of binge eating, purging, and eating disorders compared with standard care.

36 ed attitudes towards weight, body shape, and eating play a key role in the origin and maintenance of

37 ing for sociodemographics, health status and eating behaviors.

38 ith orofacial function including talking and eating.

39 during an overnight in-laboratory visit and eating was photographically recorded for one week to ass

40 integral component of the cell membrane and 'eat-me' signal recognized by macrophages.

41 intensity; once food was picked up, animals ate for the remainder of the 60-s stimulation period.

42 Formicinae ants, while N. exornata is an ant-eating euryphagous predator catching mainly Myrmicinae a

43 results show that Callilepis species are ant-eating (specialised) stenophagous predators, catching ma

44 , PD (OR: 1.46; 95% CI: 1.23, 1.74), and any eating disorder (OR: 1.32; 95% CI: 1.12, 1.56), but not

45 f BN and PD but not onset of AN, BED, or any eating disorder, and baseline current weight was inverse

46 in standard care, none of whom received any eating disorder treatment during the intervention period

47 ity to burn and the degree to which they are eaten.

48 ly unencumbered homoleptic uranium (IV) aryl-ate species of the form [U(Ar)(6) ](2-) (Ar=Ph, p-tolyl,

49 As eating disorders are under-researched, there is a great

50 "infrequent and unhealthy eating," "avoidant eating," and "emotional and external eating." The highes

51 0.05, 0.23), and always washing hands before eating or preparing foods (aOR: 0.33; 95% CI: 0.12, 0.87

52 a traditional food, the edible fruits being eaten raw and the inflorescences commonly used on sweet

53 These may be beneficial eating strategies, particularly for type 2 diabetic pati

54 ility contributes to the association between eating disorders and schizophrenia.

55 Lower odds of association were found between eating nitrated dry cured meat and other psychiatric dis

56 eating continuously [1.6% (0.1-3.1)], binge eating and loss-of-control eating [8.0% (5.1-11.0) for b

57 s index >=18.5, met criteria for DSM-5 binge eating disorder or bulimia nervosa, had 12 months of con

58 ia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and purging disorder (PD).

59 th a smartphone app, Noom Monitor, for binge eating with or without purging.

60 of-control eating [8.0% (5.1-11.0) for binge eating; 1.6 (-0.1 to 3.3) for loss of control, vs neithe

61 locating food) and maladaptive (e.g., binge eating) ways.

62 ms: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidant-restrictive food intake disord

63 ield greater reductions in symptoms of binge eating, purging, and eating disorders compared with stan

64 tifying genetic variants that regulate binge eating (BE) is critical for understanding the factors th

65 Binge-eating disorder is the most common eating disorder.

66 ary focus on anorexia nervosa (AN) and binge-eating behavior, and encourages further study of all con

67 (n = 13), bulimia nervosa (n = 6), and binge-eating disorder (n = 1), published between January 2003

68 oss the genome of anorexia nervosa and binge-eating disorder patients.

69 uits and neurotransmitters involved in binge-eating disorder pathology and identify RXFP3 as a therap

70 strate for observed sex differences in binge-eating disorder.

71 ating disorders (OED: bulimia nervosa, binge-eating disorder, and eating disorder not otherwise speci

72 ed significant reductions in objective binge-eating days (beta=-0.66, 95% CI=-1.06, -0.25; Cohen's d=

73 VN) is necessary for the expression of binge-eating behavior in female rats.

74 Finally, in a model of binge-eating in female rats, RXFP3 blockade within the PVN pre

75 Using a model of binge-eating, we demonstrated that relaxin-3/RXFP3 signaling i

76 XFP3 blockade within the PVN prevented binge-eating behavior.

77 halamic RLN3/RXFP3 signaling regulates binge-eating behavior.SIGNIFICANCE STATEMENT Binge-eating diso

78 eating behavior.SIGNIFICANCE STATEMENT Binge-eating disorder is the most common eating disorder world

79 f the formation of a chlorine radical-boron 'ate' complex that selectively cleaves sterically unhinde

80 >1 billion years ago and includes the "brain-eating amoeba." Unlike nearly all other known eukaryotic

81 foliage to redress sterol deficits caused by eating sterol-deficient prey.

82 1000 d and adiposity at 6 y is moderated by eating behaviors.

83 te risk score and adiposity was moderated by eating behaviors.

84 The missing glycemic reduction by eating meals ranked according to the discordant CGM was

85 mites enhance beetle reproductive success by eating blowfly eggs.

86 ill react upon consumption of peanut and can eat the food without adverse reactions, known as sensiti

87 ow and told by an experimenter that they can eat it immediately or wait for an unspecified duration o

88 MI and sum of skinfolds (SSF), and candidate eating behavior moderators were portion size, eating rat

89 Carnivorous eating habits are linked to TMAO levels in the animal ki

90 rom these data, we evaluated the chronotype, eating patterns (meal timing, eating duration, and eatin

91 ENT Binge-eating disorder is the most common eating disorder worldwide, affecting women twice as freq

92 Binge-eating disorder is the most common eating disorder.

93 t on the availability of a range of commonly eaten, relatively inexpensive, omega-3-PUFA enriched foo

94 Anorexia nervosa is a complex eating disorder with genetic, metabolic, and psychosocia

95 Compulsive eating behavior is hypothesized to be driven in part by

96 Compulsive eating characterizes many binge-related eating disorders

97 cal behaviours, such as gambling, compulsive eating, shopping, or disinhibited sexual behaviours, whi

98 contribute to the perpetuation of compulsive eating behavior.

99 Measures of compulsive eating severity also directly correlated to leptin, body

100 and DAT systems in this model of compulsive eating.

101 w with palatable diet, a model of compulsive eating.

102 nated by large-eat-small foraging (consumers eating smaller resources) whenever (1) many top consumer

103 (0.1-3.1)], binge eating and loss-of-control eating [8.0% (5.1-11.0) for binge eating; 1.6 (-0.1 to 3

104 ring the efficacy and feasibility of daytime eating as a behavioral modification for real-world condi

105 Overall, an 8-week daytime eating schedule, compared to a delayed eating schedule,

106 the lack of a consistent approach to define eating patterns relative to internal circadian rhythms l

107 A delayed eating schedule is associated with increased risk of obe

108 ytime eating schedule, compared to a delayed eating schedule, promotes weight loss and improvements i

109 age, mouth problems (mouth sores, difficulty eating, dry mouth, bad breath, and/or jaw pain), teeth p

110 t may be useful targets for sleep disorders, eating disorders, or addictive behavior.

111 Such "distracted eating" is associated with increased food intake and ove

112 MF(lim) reached 81 for an adult domestic dog eating a lipid-rich diet.

113 echanism leading to aroma persistence during eating is not fully described.

114 Within Dutch eating habits, satisfying optimization constraints requi

115 ng on the role of characteristics of dynamic eating performance cycles.

116 The dysfunctional eating behavior of VAChTcKO mice was alleviated by donep

117 ine weight suppression to onset risk of each eating disorder controlling for age, dietary restraint,

118 pe of food (solid, liquid), duration of each eating performance cycle, and intake outcome (intake, no

119 Identifying risk factors specific to each eating disorder is critical for advancing etiologic know

120 h increased insulin resistance and emotional eating or disinhibition showed higher brain reactivity t

121 tions, ate faster, and consumed more energy (eating behavior x risk score interactions: P < 0.05).

122 uppresses multiple different pro-engulfment "eat me" signals, including immunoglobulin G (IgG), compl

123 voidant eating," and "emotional and external eating." The highest phenotypic correlation with obesity

124 ortions (5.03 kcal; 0.47, 9.60 kcal), faster eating rates (0.40 g/min; 0.21, 0.59 g/min), and larger

125 standard giving-up-densities, amount of food eaten, we found sharp declines in rodents' perceived pre

126 (Triticum aestivum) provide half of the food eaten by humankind.

127 0.80 to 0.84), and the highest was 3.62 for eating disorders and urogenital conditions (95% CI, 3.11

128 s of color vision diversification with fruit-eating primates.

129 Furthermore, eating the enriched foods resulted in clinically relevan

130 It is known that the mouse-genome, eating-behavior, and exercise-status promotes higher tax

131 In addition, a greater eating frequency (>=5 eating occasions/d) was significan

132 Furthermore, a greater eating frequency was associated with lower fragmentation

133 In general, mice in larger groups ate less but weighed more.

134 He had eaten landlocked ayu fish (Plecoglossus altivelis) the s

135 with ST6 infections were more likely to have eaten polony (a ready-to-eat processed meat) than those

136 When he was 8 years old, he ate a frankfurter containing hypoallergenic cochineal fo

137 their CVD risk status, benefit from healthy eating behaviors and appropriate physical activity.

138 Lifestyle modifications focused on healthy eating and regular exercise are the primary recommendati

139 ly communication via email supported healthy eating, PA, and appropriate weight gain.

140 sms and circuitry underlying non-homeostatic eating.

141 sed principal component analyses to identify eating behavior patterns, twin modeling to decompose cor

142 on of the tetrakis(triphenysiloxide) Pr(III) ate complex, [KPr(OSiPh(3))(4)(THF)(3)], 1-Pr(Ph), with

143 : a monthly timescale with more stability in eating timing than a daily timescale.

144 tics of eating performance cycles, including eating technique (resident-completed, staff-facilitated)

145 Also, individual eating behaviors and nutritional satisfaction are linked

146 Furthermore, stimulation-induced eating is attenuated by dopamine lesions or receptor ant

147 GABA neurons blocked LH stimulation-induced eating, establishing them as a critical downstream circu

148 vement of dopamine in LH stimulation-induced eating.

149 , how these peripheral alterations influence eating is unknown.

150 that genetic risk for obesity may influence eating behaviors that contribute to weight and could be

151 rds small, fast-lived, highly fecund, insect-eating, generalists.

152 governmental Panel on Climate Change (IPCC), eating patterns must change.

153 eral, for people already diagnosed with KOA, eating a diet rich in fruits, vegetables, fish, whole gr

154 and was correlated with the arrival of krill-eating whales.

155 ty is promoted in systems dominated by large-eat-small foraging (consumers eating smaller resources)

156 ions were associated with dominance of large-eat-small foraging in 74 well-resolved (primarily aquati

157 Our results suggest that late eating is associated with cardiometabolic risk factors a

158 haracteristics and behaviors related to late eating may be useful in the development of future interv

159 nd weight loss barriers associated with late eating.

160 RXFP3 as a therapeutic target for binge-like eating disorders.

161 We identified compulsive-like eating behavior in female rats through progressive ratio

162 rentiated rats with the most compulsive-like eating behavior.

163 ctively, this novel model of compulsive-like eating symptoms demonstrates adaptations in insula-ventr

164 eater oro-sensory exposure (OSE) and a lower eating rate (ER).

165 In the injured nerve, macrophages 'eat' apoptotic leukocytes, a process called efferocytosi

166 rons play in habit formation and maladaptive eating.

167 ce were more prone to habits and maladaptive eating.

168 mice was sufficient to phenocopy maladaptive eating behaviors of VAChTcKO mice.

169 ting habits and vulnerability to maladaptive eating in mice.

170 arbling, two major attributes of bovine meat-eating qualities for consumers' satisfaction.

171 with a portable potentiostat after the mild eating procedure.

172 030: 2.5 kg CO2-eq pppd; no target], modeled eating patterns (food-based dietary guidelines; flexitar

173 The role of potentially modifiable eating behaviors in this association is unclear.

174 at translates taste stimulation to motivated eating behavior when hungry may facilitate food avoidanc

175 found that the mitochondrial fusion mutants eat-3 and fzo-1 are more resistant to both heat stress a

176 Although more evidence is needed, eating frequency could be considered for future chrono-n

177 osphatidylserine (PS) represents a neuronal "eat-me" signal involved in microglial-mediated pruning.

178 jects with metabolic syndrome who fasted (no eating or drinking) from dawn to sunset for more than 14

179 revealed that this nucleus gates a stop "no-eat" signal to refrain from feeding when the animal is s

180 ic activation of beta'2 DANs restored normal eating in animals fed high sucrose.

181 phages) and high levels of CD47 (the "do not eat me" signal for macrophages) and PD-L1 (a T-cell inac

182 This study identified the health benefits of eating Blackjack and therefore, the cultivation and cons

183 heme was used to code the characteristics of eating performance cycles, including eating technique (r

184 h we suggest that the social facilitation of eating has evolved as an efficient evolutionary adaptati

185 ope, or the Balkans have a common feature of eating large quantities of fermented foods.

186 The presentation form of eating disorders might vary for men versus women, for ex

187 We found that a history of eating nitrated dry cured meat but not other meat or fis

188 m genomic and neuroimaging investigations of eating disorders in humans presents a rich opportunity t

189 a key role in the origin and maintenance of eating disorders.

190 rich opportunity to sharpen animal models of eating disorders and to identify neural mechanisms that

191 factors-ie, rapidly changing diets, norms of eating, and physical activity patterns-and by broader ec

192 Our results shed light on the relevance of eating frequency as a potential zeitgeber for the circad

193 t and current weight are at greatest risk of eating disorders.

194 Here we examined the stability of eating timing, using both clock hour and relative circad

195 ough day-to-day variability in the timing of eating has poor stability, the timing of eating measured

196 l observations have shown that the timing of eating may be important for health-related outcomes.

197 of eating has poor stability, the timing of eating measured for a week is stable across months withi

198 The timing of eating was stable across months (~ 1-h variation, ICCs r

199 (CBT) has shown efficacy in the treatment of eating disorders.

200 Living primates that often eat leaves, for example, have longer crests and more slo

201 the current obesogenic environment we often eat while electronic devices, such as smart phones, comp

202 rts might therefore benefit from focusing on eating behavior change, particularly in genetically susc

203 goal-directed behaviors and had no impact on eating behavior.

204 ood choice reflects society's moral views on eating right.

205 The patient was advised to only eat well-cooked pork, and has been followed thereafter w

206 ondrial fission (DRP1/drp-1) or fusion (OPA1/eat-3, MFN/fzo-1) genes caused alterations in mitochondr

207 ans, with important implications for optimal eating habits.

208 ell bodies failed to induce food approach or eating.

209 can expect to first get a table, then order, eat, and finally pay the bill.

210 ic carbenes (NHCs) to create organometallic -ate complexes of Au(I) that serve both as WCAs and funct

211 iagnoses for anorexia nervosa (AN) and other eating disorders (OED: bulimia nervosa, binge-eating dis

212 ite when they become depressed, while others eat more.

213 pended may reflect a failure of control over eating behaviour in obesity.

214 In study 1, participants ate (mean +/- SEM) 42 +/- 15 g less in the slow- than in

215 In study 2, participants ate 66 +/- 21 g less in the high- than in the low-OSE co

216 In both studies, participants ate chocolate custard with added caramel sauce (low OSE)

217 e total antioxidant capacity of foods people eat and the photoaging of their skin.

218 e ligands that initiate their phagocytosis ("eat me" signals), while others avoid phagocytosis by dis

219 He started Pica eating disorder (sand and sponge) due to anemia from 5 y

220 Plant-eating dinosaurs evolved varied feeding strategies.

221 icated in broadening the diversity of plants eaten.

222 children who selected larger food portions, ate faster, and consumed more energy (eating behavior x

223 children who selected smaller food portions, ate slower, and consumed less energy, but was positively

224 agocytosis-like mechanism and in the process eat the hepatocyte piece by piece.

225 f food stimuli normally increases to promote eating, yet individuals with anorexia nervosa avoid food

226 to test mediation models between the PRSBMI, eating behavior patterns, and obesity measures.

227 (1.2-4.7), for highest vs lowest quartile], eating fast food [0.5% (0.2-0.7) per meal/wk], eating wh

228 st [OR - 2.145 (95% CI 1.688, 2.725)], quick eating [OR - 1.394 (95% CI 1.091-1.780)] and inadequate

229 collector reactor for irrigation of two raw-eaten vegetables (lettuce and radish) has been investiga

230 sive eating characterizes many binge-related eating disorders, yet its neurobiological basis is poorl

231 rates and is an important survival response: eating the wrong food may be deadly.

232 In particular, time restricted eating (TRE) has been popularized in the diet industry w

233 Time-restricted eating is recommended, whereby intermittent fasting is d

234 Autophagy, self-eating, is a pivotal catabolic mechanism that ensures ho

235 One species, Coccinella septempunctata, eats foliage to redress sterol deficits caused by eating

236 During her short life, she ate aquatic resources, and is related to ancient Beringi

237 Similar eating was induced by stimulation of LH GABA terminals o

238 ating behavior moderators were portion size, eating rate, and energy intake during lunch and in an ea

239 and empty buckets) and 'Eating' (time spent eating food and total intake) did not predict exploratio

240 s of touch was not modulated by subthreshold eating disorder psychopathology.

241 ntly (p < 0.05) during the optimum sweetcorn eating period (23 to 28 DAP) and continued to increase a

242 oncanonical up-regulation of so-called don't eat me molecules on inflamed phagocytes, which reduces t

243 has limited understanding of how the "don't eat me" signal is transduced biochemically.

244 (SIRPalpha) on macrophages to CD47, a "don't eat me" signal on cancer cells, prevents macrophage phag

245 CD47 acts as a "don't eat me" signal that protects cells from phagocytosis by

246 despite their expression of the CD47 "don't eat me" signal.

247 sis by displaying inhibitory ligands ("don't eat me" signals).

248 ticular, cancer cells express CD47, a 'don't eat me' signal that interacts with signal regulatory pro

249 nhibition of the macrophage SIRPalpha 'don't eat me' signal with a SIRPalpha-CD45 RIPR molecule poten

250 es that antagonize the interaction of 'don't eat me' signals with their macrophage-expressed receptor

251 Females that ate plants from the high fertilization treatment laid li

252 75% confidence interval] from the group that ate both control foods, 1.7% [0.7, 2.6]).

253 were more than halved amongst the group that ate both enriched foods.

254 Our findings indicate that eating a healthy diet might reduce the risk of acquired

255 formation of 2 but instead formation of the ate-complex [K(OEt(2))](2)[((tBu)pyrr(2)py)Fe](2) (4).

256 ucose were not differentially altered by the eating schedules.

257 TRE involves confining the eating window to a specified number of hours per day (us

258 s expressed low levels of calreticulin (the "eat me" signal for macrophages) and high levels of CD47

259 exert their influence on body weight through eating-related behaviors.

260 he chronotype, eating patterns (meal timing, eating duration, and eating frequency), and we obtained

261 rmotensive adults were randomly allocated to eat at least three portions/week of omega-3-PUFA enriche

262 A enriched (or control) chicken-meat, and to eat at least three omega-3-PUFA enriched (or control) eg

263 marshmallow available in the future, but to eat the currently attainable marshmallow when the speake

264 r hunger (r = 0.69, P = 0.002) and desire to eat (r = 0.61, P = 0.009), with some tendencies in the w

265 poses that the increased orexigenic drive to eat and the reduced energy expenditure that follow weigh

266 2), which increased their own motivation to eat the food.

267 ransforming taste signals into motivation to eat, the authors compared groups across conditions on bl

268 lved in this form of cognitive motivation to eat.

269 for the mPFC in the cognitive motivation to eat.SIGNIFICANCE STATEMENT Obesity has reached epidemic

270 cues and select when, what, and how much to eat.

271 available, there is rarely any necessity to eat something not tasty.

272 Eight minutes after starting to eat, insulin concentrations increased by 42%-65% in all

273 ing food and beverages and report 'trying to eat more protein'.

274 also a consumer survey on the willingness to eat insects fed with Styrofoam (EPS 80).

275 y disrupt homeostatic mechanisms and lead to eating disorders.

276 ion has been recently proposed as pivotal to eating disorders.

277 14), and the LBP to sCD14 ratio 1 h prior to eating either a high-saturated-fat meal or a high-oleic-

278 for the surface decontamination of ready-to-eat dry-cured ham was studied in two Spanish varieties,

279 ed by food categories (e.g. snacks, ready-to-eat meals, potato and potato-products, bakery and pastry

280 oducts (in savory spreads, cheeses, ready-to-eat meals, soups, and sausages; from 74% [95% CI 69-78]

281 A recall of ready-to-eat processed meat products from this facility was assoc

282 more likely to have eaten polony (a ready-to-eat processed meat) than those with non-ST6 infections (

283 th scanning days) performed a willingness-to-eat task in a separate fMRI measurement.

284 tance, perceived overeating or 'uncontrolled eating' (UE) is the most common obesity-associated perso

285 the nucleus accumbens [NAcc]) and unhealthy eating behaviors and outcomes; however, the mechanisms u

286 olites associated with healthy and unhealthy eating behaviors.

287 abeled "snacking," "infrequent and unhealthy eating," "avoidant eating," and "emotional and external

288 ons, which in turn lead to further unhealthy eating and obesity.

289 mic (LH) GABA neurons induces rapid vigorous eating in sated animals.

290 ogy will transform how we grow food, what we eat, and where we source materials and medicines.

291 They self-reported their height and weight, eating behaviors (15 items), diet quality, and self-meas

292 ating disorder symptoms (body shape, weight, eating concerns, and dietary restraint), and clinical im

293 llows mechanically challenging foods whether eaten often or rarely.

294 Sacoglossans are sea slugs, some of which eat algae, digesting the cells but maintaining functiona

295 d a total esophageal bolus obstruction while eating lunch at the local hospital.

296 less photoaging over 15 years than those who ate foods with low antioxidant capacity.

297 In contrast, individuals who eat fish regularly were considerably less likely to pres

298 to increased glucose levels that occur with eating.

299 atives was compared with individuals without eating disorder diagnoses and their relatives.

300 ting fast food [0.5% (0.2-0.7) per meal/wk], eating when feeling full [2.9% (1.2-4.5)], eating contin

301 For an adult wolf eating a relatively lean meat diet, a BMF(lim) (averaged