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1 was more common in each case group (3.9% in eclamptics, 4.5% in severe preeclamptics, and 4.4% in bo
2 data, and genotyped the variants in 1353 pre-eclamptic and 699 non-pre-eclamptic women to test the as
3 tinguish pre-eclamptic patients from non-pre-eclamptic based on pressure when patients have a track r
7 ein provides a protective effects on the pre-eclamptic models, both to the mother and the fetuses, by
8 ith PD-L1-Fc posed protective effects on pre-eclamptic models, indicating that the use of PD-L1-Fc mi
10 ia, where it is difficult to distinguish pre-eclamptic patients from non-pre-eclamptic based on press
11 have identified Corin gene mutations in pre-eclamptic patients, which decreased corin activity in pr
17 xacerbated further in IUGR, diabetic and pre-eclamptic pregnancies and may also give nitrative stress
18 IR and fetal HLA-C in 484 normal and 254 pre-eclamptic pregnancies at Mulago Hospital, Kampala, Ugand
19 n placental tissue from normotensive and pre-eclamptic pregnancies complicated with fetal growth rest
21 isease and diabetes, while babies from a pre-eclamptic pregnancy have increased risks of preterm birt
22 cytopenia, elevated serum creatinine levels, eclamptic seizure, medically indicated preterm birth, fe
28 idized and total AGT levels in plasma of pre-eclamptic women (n = 17), normotensive-matched controls
29 oportions of oxidized AGT in plasma from pre-eclamptic women compared to matched normotensive pregnan
30 sequenced samples from a hundred Finnish pre-eclamptic women in pools of ten to screen for low-freque
31 riants in 1353 pre-eclamptic and 699 non-pre-eclamptic women to test the association of them with pre
32 omen than in those from non-pregnant and pre-eclamptic women, but no other differences were observed.
33 ated because of placental hypoxia in the pre-eclamptic women, indicating that upstream molecular defe