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1  was more common in each case group (3.9% in eclamptics, 4.5% in severe preeclamptics, and 4.4% in bo
2 data, and genotyped the variants in 1353 pre-eclamptic and 699 non-pre-eclamptic women to test the as
3 tinguish pre-eclamptic patients from non-pre-eclamptic based on pressure when patients have a track r
4  endothelial cells in pregnancy and that pre-eclamptic cells behave differently.
5                   Over half (55.1%) of first eclamptic fits occurred in a health-care facility, with
6                                   In the pre-eclamptic group, blood pressure were correlated with the
7 ein provides a protective effects on the pre-eclamptic models, both to the mother and the fetuses, by
8 ith PD-L1-Fc posed protective effects on pre-eclamptic models, indicating that the use of PD-L1-Fc mi
9 from control (n = 8, 22 to 38 weeks) and pre-eclamptic (n = 9, 24 to 38 weeks) placentas.
10 ia, where it is difficult to distinguish pre-eclamptic patients from non-pre-eclamptic based on press
11  have identified Corin gene mutations in pre-eclamptic patients, which decreased corin activity in pr
12               In culture, the cells from pre-eclamptic placentas failed to properly modulate alpha1 i
13 wever, excessive or chronic ER stress in pre-eclamptic placentas leads to placental dysfunction.
14  in NOx levels in either normotensive or pre-eclamptic placentas.
15  in NOx levels in either normotensive or pre-eclamptic placentas.
16 sosomal dysfunction through ER stress in pre-eclamptic placentas.
17 xacerbated further in IUGR, diabetic and pre-eclamptic pregnancies and may also give nitrative stress
18 IR and fetal HLA-C in 484 normal and 254 pre-eclamptic pregnancies at Mulago Hospital, Kampala, Ugand
19 n placental tissue from normotensive and pre-eclamptic pregnancies complicated with fetal growth rest
20 as reduced about five-fold (p=0.0001) in pre-eclamptic pregnancies.
21 isease and diabetes, while babies from a pre-eclamptic pregnancy have increased risks of preterm birt
22 cytopenia, elevated serum creatinine levels, eclamptic seizure, medically indicated preterm birth, fe
23               The outcome was epileptic (non-eclamptic) seizure captured using diary records.
24 or cells from non-pregnant, pregnant and pre-eclamptic subjects, respectively.
25  % in the non-pregnant and 13.9 % in the pre-eclamptic subjects.
26            The final phenotype, maternal pre-eclamptic syndrome, is further modulated by pre-existing
27 ant (mean, 35 weeks gestation) and (iii) pre-eclamptic women (mean, 36 weeks gestation).
28 idized and total AGT levels in plasma of pre-eclamptic women (n = 17), normotensive-matched controls
29 oportions of oxidized AGT in plasma from pre-eclamptic women compared to matched normotensive pregnan
30 sequenced samples from a hundred Finnish pre-eclamptic women in pools of ten to screen for low-freque
31 riants in 1353 pre-eclamptic and 699 non-pre-eclamptic women to test the association of them with pre
32 omen than in those from non-pregnant and pre-eclamptic women, but no other differences were observed.
33 ated because of placental hypoxia in the pre-eclamptic women, indicating that upstream molecular defe
34 GF expression is altered in placentae of pre-eclamptic women.