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1 thelium and the cranial neural crest-derived ectomesenchyme.
2 thin the embryonic craniofacial ectoderm and ectomesenchyme.
3 ity at later phases commits the cells to the ectomesenchyme.
4  is required for the subsequent formation of ectomesenchyme.
5 ved from two embryonic sources: neural crest ectomesenchyme and ectodermal epithelium.
6 d ray-finned fish lacked the ability to form ectomesenchyme and its derivatives.
7  Clim-2 is particularly pronounced in facial ectomesenchyme and limb bud mesenchyme in association wi
8 est that can be reprogrammed into migrating 'ectomesenchyme' by the targeted misexpression of Twist (
9                  In mouse, prior to E10, all ectomesenchyme cells in the mandibular arch are equally
10 sults in reprogramming of this population of ectomesenchyme cells into chondrocytes.
11                          In mammals, rostral ectomesenchyme cells of the mandibular arch give rise to
12      Significantly, however, the response of ectomesenchyme cells to epithelial regulatory signals wa
13 cal interactions between oral epithelium and ectomesenchyme culminating in the formation of mineraliz
14  nr2f2;nr2f5 double mutants, but the initial ectomesenchyme delay persisted in both.
15 ces a diverse array of cell types, including ectomesenchyme derivatives that elaborate the vertebrate
16  restricted expression of the genes in molar ectomesenchyme derived from cranial neural crest cells p
17 n mediating BMP signaling in the CNC-derived ectomesenchyme during early stages of tooth development
18  Dlx5 and Dlx6 expression in the distal arch ectomesenchyme following Dlx5- and Dlx6-mediated inducti
19 est specifier genes and upregulate so-called ectomesenchyme genes that are characteristic of skeletal
20 utants show marked delays in upregulation of ectomesenchyme genes, such as dlx2a, prrx1a, prrx1b, sox
21 ration of cranial neural crest (CNC)-derived ectomesenchyme in the mandibular primordium where intram
22 idated, the early patterning signals driving ectomesenchyme into a tongue lineage are largely unknown
23 lly restricted to early neural crest and non-ectomesenchyme lineages.
24  family, Lhx6 and Lhx7, are expressed in the ectomesenchyme of the maxillary and mandibular processes
25 roper patterning of the neural crest-derived ectomesenchyme of the pharyngeal arches, from which many
26 , whereas cranial neural crest cells acquire ectomesenchyme potential dependent on activation of the
27 tiation stage, when the neural crest-derived ectomesenchyme predominates in the salivary mesenchyme t
28 ear receptors as intrinsic activators of the ectomesenchyme program: zebrafish nr2f5 single and nr2f2
29 e, ultimately allowing the rostral growth of ectomesenchyme that now characterizes gnathostome head d
30 gnals rely on Smad-dependent pathways in the ectomesenchyme to mediate epithelial-mesenchymal interac
31 y require some prespecification of maxillary ectomesenchyme to restrict the instructive influence of
32 e recovery but does not explain the impaired ectomesenchyme transition.
33  epithelium and mesenchyme and show that the ectomesenchyme underlying the maxillary molar epithelium
34  the pharyngeal arches, proliferation as the ectomesenchyme within the arches, and differentiation in
35 rises from cranial neural crest cell-derived ectomesenchyme within the mandibular portion of the firs