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1 ane (TMPD) causes lupus and the formation of ectopic lymphoid tissue.
2 ested that class switching occurs within the ectopic lymphoid tissue.
3 ll as lambda-light chains accumulated within ectopic lymphoid tissue.
4  T cells are critical for the development of ectopic lymphoid tissues.
5 rating T and B lymphocytes were found within ectopic lymphoid tissue, activation-induced cytidine dea
6                              The presence of ectopic lymphoid tissue alone was insufficient, as an an
7                           The latter home to ectopic lymphoid tissue and continue to produce autoanti
8   These B cell aggregates resembled tertiary ectopic lymphoid tissue and may thus provide a local env
9 e undergoing primary immunization implicates ectopic lymphoid tissue as a site where Ag-specific humo
10 phoid organogenesis and in the generation of ectopic lymphoid tissue at sites of chronic inflammation
11 y plasma cells/plasmablasts recruited to the ectopic lymphoid tissue by CXCR4.
12 ed lupus is associated with the formation of ectopic lymphoid tissue containing activated dendritic c
13                                              Ectopic lymphoid tissue containing B cells forms in the
14 tosus, these studies suggest that IFN-I from ectopic lymphoid tissue could play a role in the pathoge
15 erapies and identification of leptomeningeal ectopic lymphoid tissue (ELT) in patients with multiple
16                                              Ectopic lymphoid tissue formation is associated with man
17                                     Although ectopic lymphoid tissue formation is associated with man
18 ticle, we present a novel model of inducible ectopic lymphoid tissue formation, breach of humoral sel
19                                              Ectopic lymphoid tissue forming germinal centers suggest
20                                 We show that ectopic lymphoid tissue induced by TMPD not only resembl
21                                        Thus, ectopic lymphoid tissue induced by TMPD recapitulates ma
22 ike disease with autoantibody production and ectopic lymphoid tissue (lipogranuloma) formation.
23                    Individual collections of ectopic lymphoid tissue ("lipogranulomas") from the same
24 mocyanin (NP-KLH) and NP-OVA develops within ectopic lymphoid tissue ("lipogranulomas") induced by te
25 ronic inflammation promotes the formation of ectopic lymphoid tissue morphologically resembling secon
26 ot known whether Ab responses develop within ectopic lymphoid tissue or if B cells only secondarily m
27                                              Ectopic lymphoid tissue, such as bronchus-associated lym
28 understand the organizing principles of this ectopic lymphoid tissue, the cellular components contrib
29 local CXCL13 expression and the formation of ectopic lymphoid tissues, the loss of CXCL13 in the lung
30     To better understand the genesis of such ectopic lymphoid tissue, this study investigated whether
31 nic antibodies may be generated in tertiary (ectopic) lymphoid tissues (tLTs).
32 e number of anti-NP-secreting B cells in the ectopic lymphoid tissue was greatly enhanced by immuniza
33  the meninges and the formation of meningeal ectopic lymphoid tissue was partially dependent on the e
34 ation are encountered in the synovium, where ectopic lymphoid tissue with germinal centers is formed.