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1 re more likely to have mature cervixes (less ectopy).
2 ection was not independently associated with ectopy.
3 recovery time; and (c) inducible ventricular ectopy.
4 -wave segment changes or complex ventricular ectopy.
5 is associated with more frequent ventricular ectopy.
6 iated with a higher frequency of ventricular ectopy.
7 ated with an increased burden of ventricular ectopy.
8 entricular tachycardia, and supraventricular ectopy.
9 SCD and 14 with frequent/complex ventricular ectopy.
10 ntified sites initiating pulmonary vein (PV) ectopy.
11 ived to adulthood with increased ventricular ectopy.
12 ous dissociated PV rhythm and ADO-induced PV ectopy.
13 yopathy with frequent or complex ventricular ectopy.
14 tricular tachycardia or frequent ventricular ectopy.
15 rsus 14.5% (p=0.002) for complex ventricular ectopy.
16 sponse, heart rate recovery, and ventricular ectopy.
17 ith low atrial voltage and nonpulmonary vein ectopies.
18 tricular arrhythmia observed (0 indicates no ectopy; 1, isolated premature ventricular beats; 2, bige
19 %] vs 14 of 94 [15%]; P < .001), ventricular ectopy (14 of 33 [42%] vs 15 of 70 [21%]; P = .03), and
20  in trials requiring evidence of ventricular ectopy (25%) than in the remaining trials (10%).
21  status, LVEF decrement per 10%, ventricular ectopy ( 500 in 24 hours) and myocardial fibrosis detect
22 r than patients without adenosine-induced PV ectopy (63% versus 76% at 1 year; log rank, 0.014).
23 ular (P = 0.033) and ventricular (P = 0.026) ectopy across all cats.
24                             Increased atrial ectopy (AE) increases the risk of atrial fibrillation (A
25 pose of the study was to determine if atrial ectopy (AE) or atrial arrhythmias during exercise are pr
26 s remained associated with lower ventricular ectopy after cardiovascular comorbidities were controlle
27             We hypothesized that ventricular ectopy after exercise (i.e., during the recovery phase)
28 iency virus (HIV) infection, associated with ectopy among adolescent girls aged 12-20 years who were
29 acids may be associated with low ventricular ectopy among AMI patients.
30 n n-3 fatty acid consumption and ventricular ectopy among AMI patients.
31 the effects of caffeinated coffee on cardiac ectopy and arrhythmias, daily step counts, sleep minutes
32  been used to guide treatment of ventricular ectopy and arrhythmias.
33  profile of ventricular and supraventricular ectopy and bradyarrhythmia on ambulatory 24-h Holter ECG
34 nd is associated with reversible ventricular ectopy and DCM.
35 This prospective study used AECG to evaluate ectopy and HRV in subclinical HCM cats compared to healt
36 ainide has been shown to depress ventricular ectopy and improve exercise capacity in patients with An
37                    We examined the extent of ectopy and metaplastic activity as risks for HPV16 acqui
38 analysis of MKLT and describe the effects of ectopy and slow changes in cardiac cycles on the disturb
39 t of ablation was absence of frequent atrial ectopy and spontaneous AF during isoproterenol infusion
40 nts and produced pause-dependent ventricular ectopy and sustained ventricular tachycardia after acute
41 -25(-/-) mice exhibited more frequent atrial ectopy and were also more susceptible to pacing-induced
42 ts of amount of exposed columnar epithelium (ectopy) and age on the presence of alpha9 or alpha3/alph
43                The AP defects, cardiomyocyte ectopy, and AF caused by TBX5 deficiency were rescued by
44 ad atrial fibrillation, frequent ventricular ectopy, and nonsustained ventricular tachycardia, respec
45 es and ARIs during sinus rhythm, ventricular ectopy, and premature stimulation (r=0.72, slope=-0.76,
46 s, abnormal heart rate recovery, ventricular ectopy, and ST-segment abnormalities.
47 entricular tachycardia, frequent ventricular ectopy, and/or bigeminy and optical mapping demonstrated
48  wall motion abnormalities; RV outflow tract ectopy; and exercise-induced T-wave pseudonormalization.
49 c metaplasia rather than the sheer extent of ectopy appears to increase risk for incident HPV16 in he
50 he acute phase of myocardial ischemia, focal ectopies arising from this location, and including both
51 ocardial infarction (MI) may be triggered by ectopy arising from Purkinje fibers.
52 n of ventricular tachycardia and ventricular ectopy arising in a PAP has a high success rate.
53 ntified shock lead impedance and ventricular ectopy as significantly associated with increased risk o
54                    All patients had frequent ectopy at baseline with a median PVC count of 7275 (rang
55             After adjustment for ventricular ectopy at rest and during exercise, peak oxygen uptake,
56  acid) was associated with lower ventricular ectopy (beta = -0.35, P = 0.011), and this effect remain
57 ulated metaplastic rate as the difference in ectopy between visits.
58                   An increase in ventricular ectopy burden was associated with progressively lower ev
59 ot be restricted to the generation of atrial ectopy but extends to the development of atrial remodeli
60 ll appreciated is whether simple ventricular ectopy can result in cardiomyopathy.
61     In this study, we describe murine thymic ectopy, cervical thymic tissue that possesses the same g
62                                  Ventricular ectopy could be induced by isoproterenol-challenge in is
63                                              Ectopy data for 189 and 92 HIV-positive and -negative ad
64 ta-blocker significantly reduced ventricular ectopy during exercise compared with placebo plus beta-b
65 1 to 1.9; P=0.003), but frequent ventricular ectopy during exercise did not (adjusted hazard ratio, 1
66                         Frequent ventricular ectopy during exercise predicted an increased risk of de
67 10 to 1.97; p = 0.0089), whereas ventricular ectopy during exercise was not predictive of death in th
68 ue to patient factors--excessive ventricular ectopy during exercise, unsustained MTWA, or failure to
69 reased risk of death better than ventricular ectopy during exercise.
70  among patients without frequent ventricular ectopy during exercise; hazard ratio, 1.8; 95 percent co
71                         Frequent ventricular ectopy during recovery after exercise is a better predic
72                           Severe ventricular ectopy during recovery after exercise is predictive of i
73                         Although ventricular ectopy during recovery after exercise predicts death in
74  confounding variables, frequent ventricular ectopy during recovery predicted an increased risk of de
75 er potential confounders, severe ventricular ectopy during recovery remained predictive of death (adj
76 5 to 2.1; P<0.001), but frequent ventricular ectopy during recovery was a stronger predictor (11 perc
77                           Severe ventricular ectopy during recovery was associated with an increased
78 23 patients, 140 (7%) had severe ventricular ectopy during recovery.
79 ormal QT interval, and displayed ventricular ectopy during stress testing consistent with CPVT.
80                               ADO induced PV ectopy during the early phase of ADO effect only in 12 P
81 ify the implications of adenosine-induced PV ectopy for atrial fibrillation (AF) recurrence after PV
82                                  Heart rate, ectopy frequency and complexity and HRV parameters, incl
83 f a new aortic gradient and caused excessive ectopy from balloon movement in the left ventricular out
84 elayed the development of spontaneous atrial ectopy, fully prevented sAF, suppressed atrial dilation,
85 1 partner in the past 6 months, and cervical ectopy greater than 25%.
86  was frequent (43% with at least ventricular ectopy &gt;=5%), most often moderate (ventricular tachycard
87                                              Ectopy (&gt;/= 1000 premature ventricular complexes/24 hour
88                              Although thymic ectopy has long been recognized in humans, the functiona
89  three mechanisms of arrhythmogenesis: focal ectopy, heart block, and reentry.
90 ated by a spontaneous LBLPS-like sequence of ectopy; however, CRDS remains difficult to recognize cli
91 ort the prognostic importance of ventricular ectopy immediately after exercise, when reactivation of
92  220 electrically silent PVs, ADO induced PV ectopy in 28 (13%) veins.
93                    Adenosine can also induce ectopy in electrically silent PVs after isolation, possi
94                   However, ADO can induce PV ectopy in electrically silent PVs in a manner not closel
95 rognostic importance of frequent ventricular ectopy in recovery after exercise among patients with sy
96 eart rate recovery, and frequent ventricular ectopy in recovery.
97 ition characterized by prominent ventricular ectopy in response to catecholamine stress, which can be
98           Repetitive monomorphic ventricular ectopy (in the absence of sustained ventricular tachycar
99 ars) with repetitive monomorphic ventricular ectopy, including 8 patients (30%) with depressed ventri
100                        Different patterns of ectopy, including isolated PVCs, bigeminy, trigeminy, an
101 sity to CDA rendered the atria vulnerable to ectopy-induced arrhythmia.
102                                Patients with ectopy-induced cardiomyopathy are significantly older th
103                  In this milieu, spontaneous ectopy initiated AF.
104 r tachyarrhythmias and other benign forms of ectopy: inpatient versus outpatient.
105                         Adenosine-induced PV ectopy is a predictor of recurrent AF after PV isolation
106                    Very frequent ventricular ectopy may also result in a cardiomyopathy in a minority
107 high-resolution photograph of the cervix for ectopy measurement were collected.
108 tive use to be associated with the amount of ectopy, multivariate logistic regression analysis showed
109 tachycardia (n=33, 25 patients), ventricular ectopy (n=10, 8 patients), and ventricular tachycardia (
110 ricular tachycardia, and complex ventricular ectopy (nonsustained ventricular tachycardia or bigeminy
111                         Frequent ventricular ectopy occurred only during exercise in 945 patients (3
112  an increased risk of death than ventricular ectopy occurring only during exercise.
113 tor, with more partners associated with less ectopy (odds ratio, 0.47; 95% confidence interval, 0.22-
114 ar cell models suggested that rate-dependent ectopy of Purkinje fiber origin is the predominant ventr
115                     Lown grade 4 ventricular ectopy on ambulatory ECG was present in three patients i
116  (RCA) perfusion area and showed ventricular ectopy on electrocardiogram (ECG) at rest that diminishe
117          Eight of 9 patients had ventricular ectopy on exercise stress testing.
118 cardiomyopathy or heart failure, ventricular ectopy or nonsustained ventricular tachycardia, and lowe
119 g arrhythmia patients, 6 had frequent atrial ectopy or tachycardia, 86 had paroxysmal AF, 39 had pers
120 s not different than in patients with atrial ectopy or tachycardia.
121 almost twice the odds of complex ventricular ectopy (OR, 1.74; 95% CI, 1.11-2.74).
122 rethral valves, neurogenic bladder, ureteral ectopy, or bladder exstrophy.
123                     In 2 patients, posterior ectopy organized to subsequently initiate isthmus-depend
124 n abnormalities in regions where ventricular ectopy originates.
125 uggests a possible oligogenic origin for the ectopies originating from Purkinje fibres.
126 p between repetitive monomorphic ventricular ectopy originating from the right ventricular outflow tr
127 xcluding myocardial fibrosis and ventricular ectopy owing to case censoring).
128                 Exercise-induced ventricular ectopy predicts an increased risk of death in population
129 ar tachycardia (VT) and frequent ventricular ectopy (premature ventricular contractions [PVCs] >10/h)
130                                              Ectopy recurred but was ultimately resolved by ablation
131 -perfused hearts, we demonstrated that focal ectopies require, in the normal mouse heart, the simulta
132  ablation of the focal source of ventricular ectopy results in normalization of left ventricular func
133            Most participants had ventricular ectopy, supraventricular tachycardia, and supraventricul
134 displayed 3-fold higher rates of ventricular ectopy than Casq2+/+ mice (n=31; P<0.05).
135 inus rhythm, pacing, and AF initiation by PV ectopy that are determined largely by the functional pro
136 ing isoproterenol infusion identified atrial ectopy that initiated AF and the presence of inducible A
137     Although the most common sites of atrial ectopy that trigger atrial fibrillation (AF) are in or a
138       Of the several measures of ventricular ectopy that were univariate predictors, the frequency of
139 ion of extrasystoles and the factors causing ectopies to be arrhythmia triggers during myocardial isc
140 of extrasystoles, and the properties leading ectopies to become arrhythmia triggers (topology), in th
141 other and fetus; these may range from benign ectopy to life-threatening arrhythmias.
142  occur during pregnancy, ranging from benign ectopy to life-threatening arrhythmias.
143 r arrhythmic behavior, ranging from frequent ectopy to pacing-induced ventricular tachycardia/ventric
144 ependent progression from spontaneous atrial ectopy to paroxysmal and eventually long-lasting AF.
145 ependent progression from spontaneous atrial ectopy to paroxysmal and finally long-lasting AF.
146 lt T-wave alternans (MTWA) test, when due to ectopy, unsustained MTWA, or low exercise heart rate (HR
147 with bileaflet prolapse, complex ventricular ectopy (VE), and abnormal T waves comprise the recently
148 uent association of more serious ventricular ectopy, VT, with lower [K+]e concentrations supports the
149                           Severe ventricular ectopy was defined as the presence of ventricular triple
150                         Frequent ventricular ectopy was defined by the presence of seven or more vent
151  multivariate analysis, adenosine-induced PV ectopy was found to be the only independent predictor of
152                             Exercise-induced ectopy was not associated with survival.
153                                     However, ectopy was not significant, whether measured before or c
154                                  Ventricular ectopy was present only in MyBP-C(t/t) mice during ambul
155                                The burden of ectopy was quantified through 24-hour Holter monitoring.
156                                              Ectopy was quantitatively measured on colpophotographs.
157            For each isolated PV, dissociated ectopy was recorded and ADO was administered.
158                         Adenosine-induced PV ectopy was seen in 45 (30%) patients, and dormant conduc
159           However, the burden of ventricular ectopy was similar in patients with (17,859+/-13,488 ect
160                         NSVT and ventricular ectopy were common early after TPVR but were infrequent
161 solation of 270 PVs, 50 PVs with dissociated ectopy were identified.
162            Older age and decreasing cervical ectopy were independently positively associated with hav
163  of adenosine-induced PV reconnection and PV ectopy were recorded.
164  AF among patients with adenosine-induced PV ectopy were significantly lower than patients without ad
165 longside the presence of complex ventricular ectopy, which remains the most robust predictor of morta
166 Prospective Randomized Evaluation of Cardiac Ectopy with Dobutamine or Nesiritide Therapy) trial were
167                 The timing of ADO-induced PV ectopy with respect to ADO effects on heart rate varied.

 
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