ライフサイエンスコーパス: edema

1 or the presence of 3 tomographic features of edema.

2 pillary membrane, allowing the simulation of edema.

3 ion, and in some cases seizures and cerebral edema.

4 OP spikes, and transient microcystic corneal edema.

5 th no evidence of intracranial hemorrhage or edema.

6 ed with microvascular lesions and myocardial edema.

7 akage) and alleviated TNFalpha-induced brain edema.

8 or tenosynovitis, synovitis, and bone marrow edema.

9 rved tails/bodies, reduced eye/head size and edema.

10 visual symptom onset demonstrated optic disc edema.

11 on, characterized by wasting with or without edema.

12 ttae and the presence of clinically definite edema.

13 yes and was bilateral in 82% of infants with edema.

14 ema or predisposing risk factors for corneal edema.

15 ies ranged from cytotoxic edema to vasogenic edema.

16 d chloride flux as the mechanism of neuronal edema.

17 ing to fluid accumulation and development of edema.

18 show defective lymphatic valve formation and edema.

19 morphogenesis results in backflow leading to edema.

20 patients with no history of cystoid macular edema.

21 ent, especially in eyes with uveitic macular edema.

22 hout (n = 8; 53%) associated chronic retinal edema.

23 ale score or development of diabetic macular edema.

24 ep cornea dehydration which leads to corneal edema.

25 l fluid, epiretinal membrane, or optic nerve edema.

26 icular pattern of bullous epithelial corneal edema.

27 t of best-corrected visual acuity or macular edema.

28 ded 2 grade 3 polyneuropathies and 1 grade 3 edema.

29 vitreomacular traction, and cystoid macular edema.

30 iretinal membrane, and 1 eye had optic nerve edema.

31 rence of side effects such as hemorrhage and edema.

32 defined as (0.87 x wall thickness) + (1.97 x edema) + (0.83 x ulceration) + (0.55 x SUVmax ratio) + 1

33 ative complications included cystoid macular edema (10%), corneal decompensation (6%), and choroidal

34 460 +/- 76 mum and 280 +/- 83 mum in severe edema (12%), respectively.

35 posterior synechiae (21.7%), cystoid macular edema (16%), epiretinal membrane (13.2%), glaucoma (11.3

36 (29 eyes, 10%) and transient cystoid macular edema (25 eyes, 8.6%).

37 nges, or both (24 eyes); sequelae of macular edema (3 eyes); blunt trauma (2 eyes); retinal phototoxi

38 occurred in 5.2%, primarily cystoid macular edema (3.7%).

39 f 40; control group, 60 of 100), bone marrow edema (39 of 40 vs 87 of 100), effusion (20 of 40 vs 26

40 (49.3%), neutropenia (47.9%), and peripheral edema (45.1%).

41 Unexpectedly, we found that neuronal edema 48 hours after CCI was associated with reduced cel

42 showed that diffusion-weighted MRI depicted edema 5.1 (P < .001) and 3.5 (P < .001) times greater th

43 51%) developed LEE (new onset, 120; worsened edema, 50).

44 ase (60%), hypoalbuminemia (55%), peripheral edema (51%), and thrombocytopenia (49%).

45 y (74%), vitreous cell (53%), and optic disc edema (52%).

46 of eyes) or pseudophakic or aphakic corneal edema (6% of eyes).

47 For bullous keratoplasty and/or corneal edema, 8.6% and 22% of grafts underwent repeated keratop

48 Swelling of the brain or spinal cord (CNS edema) affects millions of people every year.

49 nal fluid, subretinal hemorrhage, or macular edema after 4 anti-VEGF injections and anti-VEGF-sensiti

50 monstrated improvement in bullous epithelial edema after netarsudil discontinuation.

51 l reaction, capsule edema, and intramuscular edema after total hip arthroplasty at 1.5-T MRI with met

52 vessels, fibrous proliferations, and macular edema, agreement was substantial (weighted kappa, 0.61 t

53 ncluding 3 eyes with transient focal corneal edema and 4 eyes that required Micro-Stent trimming due

54 aive patients with radiation-induced macular edema and a resulting decrease in visual acuity ranging

55 in the detection of wrists with bone marrow edema and a sensitivity of 69% (95% CI: 55%, 81%; 36 of

56 ality, as well as microphthalmia, periocular edema and absence of the anterior chamber of the eye; ad

57 Approximately one third of diabetic macular edema and age-related macular degeneration clinical tria

58 It results in hypotension and tissue edema and contributes to organ dysfunction.

59 st common adverse effects of isradipine were edema and dizziness.

60 nction and elevated parameters of myocardial edema and fibrosis were observed at MRI, which were more

61 on and mitigates confounding factors such as edema and fibrosis.

62 thelial cells, accumulations of macrophages, edema and fibrosis.

63 dual-energy CT in helping detect bone marrow edema and fracture in participants with wrist trauma and

64 The presence of bone marrow edema and fracture was rated per bone.

65 ts had specific transcription patterns, e.g. edema and genes related to cellular invasion, and 10 rad

66 circulatory perfusion and reduced intestinal edema and hemorrhage after BD.

67 mesenteric perfusion and reducing intestinal edema and hemorrhage associated with BD.

68 ung epithelial/endothelial cell dysfunction (edema and hemorrhage).

69 macular edema resolved or until both macular edema and HEs resolved.

70 down attenuated the development of pulmonary edema and improved survival in a mouse model of P aerugi

71 Available evidence suggests that both edema and inadequate immune response resolution may cont

72 interest to prevent the progression of mild edema and might avoid or reduce the use of more invasive

73 The association between edema and mortality was difficult to ascertain from the

74 We hypothesized that edema and myocyte injury would be chronically associated

75 d the central cornea, accompanied by corneal edema and neovascularization.

76 In addition to vision loss caused by macular edema and pathological angiogenesis, DR patients often e

77 s with radiation retinopathy-related macular edema and prevent vision loss through 48 weeks of therap

78 case each of pyrexia, cardiogenic pulmonary edema and pulmonary embolisms.

79 rred primarily owing to persistent/recurrent edema and related foveal damage.

80 with presenting vision in eyes with macular edema and RVO, most eyes treated with ranibizumab achiev

81 ine the ionic mechanisms underlying neuronal edema and their effects on neuronal and network excitabi

82 rtrophic cardiomyopathy and severe pulmonary edema and thrombosis.

83 ssified clinically into early-stage (without edema) and late-stage (with edema) disease.

84 ore, mid-upper arm circumference (MUAC), and edema) and partial (MUAC, edema) definitions of SAM and

85 marker of myelin content, inflammation, and edema), and cerebral blood flow.

86 er arm circumference (MUAC) < 11.5 cm and/or edema, and 1 sachet of RUTF per day for those with MUAC

87 ed rate of neurologic decline, less cerebral edema, and a decrease in microglia activation in compari

88 sly combined, expands the section on macular edema, and adds several characteristics not previously g

89 ion (13%), IOP spikes (6%), hyphema, corneal edema, and BCVA loss (all 4%).

90 in injury, intracranial hemorrhage, cerebral edema, and brain death.

91 reatment with TPPU ameliorates hyperalgesia, edema, and decreases the expression of important pro-inf

92 ty worsened with onset of bullous epithelial edema, and in all cases, visual acuity stabilized or imp

93 The presence of periosteal reaction, capsule edema, and intramuscular edema after total hip arthropla

94 Periosteal reaction, capsule edema, and intramuscular edema differed between the two

95 o the blood-spinal cord barrier, ablated CNS edema, and led to accelerated functional recovery compar

96 and booster pump strains; and infarct size, edema, and microvascular obstruction.

97 Arthralgia, arthritis, edema, and myalgia were more common in adults compared t

98 Plasma amylase activity, pancreatic edema, and myeloperoxidase content in pancreas and lungs

99 icular chamber volumes, function, myocardial edema, and necrosis at each time point using predefined

100 change from baseline, resolution of macular edema, and number of adjunctive treatments.

101 giectasia, dyspigmentation, scar appearance, edema, and pain were detected at low rates, and cosmetic

102 culation is common and includes arrhythmias, edema, and pulmonary arteriovenous malformations (PAVM)

103 lated macular degeneration, diabetic macular edema, and retinal venous occlusive disease.

104 m with greater reductions with LVH, storage, edema, and scar.

105 nical parameters, development and outcome of edema, and survival data were collected.

106 and nonenhancing tumor core, the peritumoral edema, and the contrast-enhancing tumor-were manually la

107 nied by increased uptake by barrier tissues, edema, and ventilation defects-all of which are salient

108 r size>25mm, non-nodular enhancement, breast edema, areola-nipple complex retraction, and lymph-node

109 libercept (IVA) for the treatment of macular edema associated with CRVO based on data from the LEAVO

110 ential target for the treatment of pulmonary edema associated with heart failure generated a novel se

111 Pulmonary edema associated with increased vascular permeability is

112 vere neurotoxicity, including fatal cerebral edema associated with T cell infiltration into the brain

113 The presence of LGE without edema at 6-month CMR is associated with worse prognosis,

114 RR 1.34; 95% C.I. 1.02-1.75), and cysts with edema at baseline (RR 1.39; 95% C.I. 1.05-1.85).

115 ted eyes had investigator-determined macular edema at month 36 compared with sham-treated eyes (33.3%

116 ation (jugular venous distention, rales, and edema) at baseline in the TOPCAT trial (Treatment of Pre

117 ng in enhanced vascular permeability, tissue edema, augmented leukocyte adhesion, platelet aggregatio

118 can be reliably estimated when only MUAC and edema-based data are available.

119 The MUAC and edema-based definition of AM did not lead to different $

120 therapy for center-involved diabetic macular edema (CI-DME) in a 2-year randomized clinical trial.

121 Center-involved diabetic macular edema (ci-DME) is a major cause of vision loss.

122 tients with center-involved diabetic macular edema (CI-DME) with good visual acuity (VA) represent a

123 After surgical treatment, the stromal edema cleared completely in all 3 infants, leaving behin

124 Corneal edema cleared in 12/30 eyes and cleared with scar in 18/

125 Cystoid macular edema (CME) before intraocular surgery was not encounter

126 .9 mm vs. 3.2 mm; P = 0.01), cystoid macular edema (CME) involving the foveola (30% vs. 70%; P = 0.00

127 rected visual acuity (BCVA), cystoid macular edema (CME), and the highest fluorescein angiography (FA

128 iretinal membrane (ERM), and cystoid macular edema (CME), were analyzed.

129 rhage, infectious keratitis, cystoid macular edema [CME], retinal detachment [RD], or RD surgery) fol

130 reatment goal was absence of MRI bone marrow edema combined with clinical remission, defined as DAS28

131 ed inside the esophageal lumen produced mild edema compared with RFA (13 [12-14] applications) which

132 ive DR (PDR), clinically significant macular edema (CSME), diabetic macular edema (DME), or ocular su

133 and Snellen measurements, amount of retinal edema (CST) did: increased CST correlated with increased

134 anatomic variables such as extent of retinal edema (CST) may have a meaningful impact on the anticipa

135 ites, pleural or pericardial effusions, skin edema, cystic hygroma, increased nuchal translucency, or

136 erence (MUAC), and edema) and partial (MUAC, edema) definitions of SAM and MAM.

137 Conclusion Lower-extremity edema developed in more than 50% of study patients who u

138 entual stroke damage, yet our concept of how edema develops is incomplete and treatment options remai

139 eatment for persistent HEs in the absence of edema did not result in better vision.

140 l reaction, capsule edema, and intramuscular edema differed between the two groups (P < .001 for each

141 lary dilatation and congestion, interstitial edema, diffuse alveolar damage, and microthrombosis.

142 y-stage (without edema) and late-stage (with edema) disease.

143 l photocoagulation rates in diabetic macular edema (DME) eyes did not significantly differ from 474/1

144 and previously treated (PT) diabetic macular edema (DME) eyes in a real-life setting.

145 ular thickness in eyes with diabetic macular edema (DME) in clinical trials.

146 Diabetic macular edema (DME) is a leading cause of vision loss in diabeti

147 For example, diabetic macular edema (DME) is a leading cause of vision loss in patient

148 Diabetic macular edema (DME) is the most common cause of vision loss in d

149 ars, absence of preexisting diabetic macular edema (DME) or postvitrectomy persistent cystoid macular

150 Patients with diabetic macular edema (DME) received three intravitreal injections of 0.

151 Diabetic macular edema (DME) remains a leading cause of vision loss world

152 n-perfusion areas (NPAs) in diabetic macular edema (DME) using two different Optical Coherence Tomogr

153 icant macular edema (CSME), diabetic macular edema (DME), or ocular surgery.

154 e majority of patients with diabetic macular edema (DME), the most common cause of vision loss in wor

155 ibizumab and bevacizumab in diabetic macular edema (DME).

156 cular degeneration (AMD) or diabetic macular edema (DME).

157 anibizumab in patients with diabetic macular edema (DME).

158 onotherapy for treatment of diabetic macular edema (DME).

159 ) and treatment response in diabetic macular edema (DME).

160 -threatening complications (diabetic macular edema [DME] and proliferative diabetic retinopathy [PDR]

161 , diabetic retinopathy (DR; diabetic macular edema, DME), or noninfectious uveitis (NIU).

162 ctions are a mandatory treatment for macular edema due to nAMD, DME and RVO.

163 n blood volume, heart rate, respiration, and edema during pregnancy may lead to increased risk of adv

164 ons such as severe hyperkalemia or pulmonary edema) during severe AKI allowed many patients to escape

165 COVID-19 patients can present with pulmonary edema early in disease.

166 targeting emphysema, cardiomegaly, hernias, edema, effusions, atelectasis, masses, and nodules.

167 ective for the treatment of diabetic macular edema, even in refractory cases that have failed to resp

168 lash hypertrichosis, eyelid erythema, eyelid edema, eyelid hyperpigmentation, high upper eyelid creas

169 To investigate the diffusion of edema factor (EF) and lethal factor (LF), we use sensiti

170 ranslocates either its lethal factor (LF) or edema factor (EF) into the host cell.

171 tigen (PA) plus either lethal factor (LF) or edema factor (EF), respectively, play an important yet i

172 , improved long-term motor behavior, reduced edema formation as assessed by magnetic resonance imagin

173 essure mediates capillary stress failure and edema formation secondary to calpain-induced disruption

174 ersistent lung inflammation and protein-rich edema formation, the hallmarks of acute respiratory dist

175 n the lungs with neutrophil infiltration and edema, further confirmed as consolidations visualized by

176 e, retinal detachment, retinal tear, macular edema, glaucoma, or choroidal detachment-and used adjust

177 s with glaucoma (IOP >= 21 mmHg with corneal edema, Haabs striae, optic nerve cupping or buphthalmos)

178 n the aftermath of TBI and that treatment of edema has the potential to reverse these effects.

179 Recently, high T2 (edema) has been observed in the basal inferolateral wall

180 ibrillation, contusion, diarrhea, peripheral edema, hemorrhage, muscle spasms, and pneumonia, as well

181 th no evidence of intracranial hemorrhage or edema.Her subsequent hospital course was complicated by

182 pening, weeping eyes, crusty eyelids, eyelid edema, highly vascularized cornea and tarsal plates (TPs

183 of diffuse alveolar damage (DAD), including edema, hyaline membranes, and proliferation of pneumocyt

184 all cases, the reticular bullous epithelial edema improved or resolved after discontinuation of neta

185 At presentation, there was corneal edema in 30 eyes (45.4%), enlarged corneal diameter in 3

186 CMR abnormalities (infarction or myocardial edema in a coronary territory) was present in 53.4% (62/

187 ular pattern of reticular bullous epithelial edema in a series of patients treated with netarsudil on

188 ia is a key contributing factor for cerebral edema in acute liver failure.

189 In the treatment of macular edema in CRVO, IVB yields the best cost utility among th

190 The extent of diurnal variation in corneal edema in Fuchs dystrophy is unknown.

191 , 99%; 682 of 696 bones) in the detection of edema in individual bones.

192 Edema in OCT resolved completely at the end of follow-up

193 ering the risk of life-threatening pulmonary edema in PVOD, if treated by conventional pulmonary hype

194 of 4 of 5 patients had a history of corneal edema in the affected eye, and the fifth patient had ris

195 Previous work suggests that interstitial edema in the heart can acutely promote ventricular arrhy

196 es, in order to lower the risk for vasogenic edemas in the clinic.

197 loss of ZO-1 junctional contacts and corneal edema, in female than male mice, characteristic of late-

198 e fifth patient had risk factors for corneal edema including a history of anterior uveitis and an ant

199 ALT1(PD/PD) mice are protected from vascular edema induced by either passive cutaneous anaphylaxis or

200 endpoints were: hemispheric space-modifying edema, infarct growth between per-occlusion and 24 h MRI

201 ancreatic activation of trypsinogen and more edema, infiltration of lung and pancreas by inflammatory

202 important physiological processes including edema, inflammation, and tumor lipid heterogeneity in pr

203 Diabetic macular edema is a major complication of diabetes resulting in l

204 Edema is an important target for clinical intervention a

205 p to study the crucial role of astrocytes in edema, ischemia, glioma progression, stroke, and epileps

206 I findings (meniscus, cartilage, bone marrow edema, joint effusion, ligaments, tendons) were examined

207 (T1 and T2 relaxation times, ECV, myocardial edema, late gadolinium enhancement [LGE], and myocardial

208 Lower-extremity edema (LEE) is an underreported complication of TIPS cre

209 ia; thrombocytosis; leukocytosis; fever; leg edema; lower Barthel Index (BI) score; immobility; pares

210 atic system with resolution of the lymphatic edema, marked improvement in his pulmonary function test

211 cyst-like or multiloculated shape, vasogenic edema, mass effect, and size greater than 3cm.

212 and ellipsoid zone (EZ) integrity in macular edema (ME) patients.

213 b to ranibizumab in the treatment of macular edema (ME) resulting from retinal vein occlusion (RVO).

214 occlusion disease (BRVO) cases with macular edema (ME).

215 noninfectious uveitis complicated by macular edema (ME).

216 hibit inflammation in a contact irritant ear edema mouse model.

217 the modified Radiographic Assessment of Lung Edema (mRALE) score.

218 biological activities in the victim such as edema, myotoxicity and cytotoxicity, contributing to the

219 in all FECD corneas with clinically definite edema (n = 15), in >=81% of FECD corneas suspicious for

220 % of FECD corneas suspicious for subclinical edema (n = 16), in <=42% of FECD corneas deemed not to h

221 in <=42% of FECD corneas deemed not to have edema (n = 62), and in <=5% of normal corneas (n = 74).

222 foetuses like haemorrhages, diffuse cerebral edema, necrotizing encephalitis and decreased bone size

223 Thus, our understanding of poststroke edema needs to be revised, and these findings could prov

224 eas TRALI presents as pulmonary permeability edema (noncardiogenic).

225 ed an increase in excitability when neuronal edema normalized by 1 week after CCI.

226 sease, reversible, hypoxia-induced cytotoxic edema occurred predominantly in the white matter in chil

227 h a 2-day history of progressively worsening edema of her face and lower extremities.

228 in ASM, and it prevented the constant major edema of OC.

229 linded trial involving patients with chronic edema of the leg and cellulitis, compression therapy res

230 trial, we assigned participants with chronic edema of the leg and recurrent cellulitis, in a 1:1 rati

231 Chronic edema of the leg is a risk factor for cellulitis.

232 specificity in the detection of bone marrow edema of the wrist.

233 , optic neuropathy presented with optic disc edema, often associated with uveitis, retinitis, or both

234 Adverse events included rearrest, pulmonary edema on chest x-ray, acute renal dysfunction, bleeding

235 Cardiac MRI showed diffuse myocardial edema on T2 short tau inversion-recovery sequences and n

236 lammation and rapid development of pulmonary edema on thoracic images, coronary artery aneurysms, and

237 rogression (new onset of clinically definite edema or >=5% increase in CCT) or intervention by endoth

238 entity that is commonly misdiagnosed as disc edema or choroidal neovascularization.

239 8-month small and medium joint pain, without edema or erythema.

240 Most patients had a history of corneal edema or predisposing risk factors for corneal edema.

241 tec, Dublin, CA) OCTA images with no macular edema or significant motion artifact were acquired 5 tim

242 ssociated with postoperative cystoid macular edema (OR = 0.36, 95% CI: 0.14-0.91, P = 0.031) and more

243 of the left breast with no skin retraction, edema, or erythema.

244 ry in response to changes in myocyte volume, edema, or fibrosis.

245 with different subtypes of diabetic macular edema over a 6-month follow-up period.

246 he foveola (30% vs. 70%; P = 0.001), retinal edema overlying the lesion (39% vs. 61%; P = 0.003), ret

247 or postvitrectomy persistent cystoid macular edema (P < .05).

248 Eyelid edema (P = .04) and tearing (P = .02) were more signific

249 mortality can occur due to a combination of edema, pneumonitis and fibrosis.

250 Eye pain, eyelid edema, poor presenting visual acuity, larger corneal ulc

251 weight regain included: younger age, venous edema, poorer physical function, and more depressive sym

252 Lower-extremity edema portends worse survival.

253 Poststroke brain edema predicts the severity of eventual stroke damage, y

254 e nanoparticles leads to resolution of brain edema, protection of axons in hippocampus region, and my

255 ced CCC phosphorylation, attenuates cerebral edema, protects against brain damage, and improves outco

256 s in zebrafish causes glomerular injury with edema, proteinuria and structural changes of the glomeru

257 ies this protection, with attenuated retinal edema, reduced inflammation and preserved neuroretinal m

258 Cystoid macular edema refractory to carbonic anhydrase inhibitor therapy

259 ive macular degeneration or diabetic macular edema requiring bilateral anti-vascular endothelial grow

260 aged, and two had symptoms from perilesional edema requiring radiosurgery, but all three patients rem

261 .27) over the first 4 hours, indicating that edema resolution steepened the central posterior cornea.

262 ranibizumab injections either until macular edema resolved or until both macular edema and HEs resol

263 and alteration in tissue water distribution (edema), respectively, were assessed with a chemical shif

264 .7% and 13.0% vs. 27.3% for BCVA and macular edema, respectively).

265 development that may be impacted by macular edema, ROP, or both.

266 e (mFS), Subarachnoid Hemorrhage Early Brain Edema Score) (P < 0.05).

267 inner nuclear layer thickness increased with edema severity: 183 +/- 36 mum and 51 +/- 27 mum in mild

268 associations between plasmin (ogen) uria and edema status as well as eGFR.

269 Edema stemming from leaky blood vessels is common in eye

270 Traumatic brain injury (TBI) causes brain edema that induces increased intracranial pressure and d

271 system, resulting in acute lung inflammatory edema; the renin-angiotensin system, promoting cardiovas

272 is a viable strategy for development of CNS edema therapies.

273 -induced vascular permeability and pulmonary edema through the modulation of actin cytoskeleton dynam

274 tion of vascular leak and consequent cardiac edema to the genesis of atrial arrhythmias remains unkno

275 Abnormalities ranged from cytotoxic edema to vasogenic edema.

276 brain tissue-in terms of changes in ICP and edema-to a growing tumor.

277 alveolar-capillary barrier dysfunction, and edema toxin may significantly exacerbate pulmonary patho

278 Anthrax lethal toxin and edema toxin, which are composed of protective antigen (P

279 Lethal and edema toxins are critical virulence factors of Bacillus

280 when comparing the structures of lethal and edema toxins.

281 Treatment with mannitol, a first-line edema treatment in clinical practice, was also associate

282 LEE was defined as new-onset or worsened edema up to 1 year after TIPS creation.

283 the severity of liver dysfunction and brain edema was attenuated by recAP, associated with reduction

284 Macular edema was defined as a center macular thickness (CMT) >=

285 %; specificity, 90%; accuracy, 86%); capsule edema was found in 33 of 40 (infection group) and five o

286 city, 95%; accuracy, 91%); and intramuscular edema was found in 38 of 40 (infection group) and 14 of

287 risk for the development of cystoid macular edema was found to be associated with recurrence in the

288 Macular edema was present in 60% of eyes and was bilateral in 82

289 In 4 of 6 episodes, corneal edema was present, typically in the corneal stroma at th

290 The reference standard for bone marrow edema was the combined reading of MRI scans.

291 with ACIOL, complications of cystoid macular edema were higher in 10-0 polypropylene iris-sutured PCI

292 odes of reticular bullous epithelial corneal edema were identified in 5 eyes of 5 patients treated wi

293 ia, anaphylactic reaction, and angioneurotic edema were only recorded in 2.2%, 2.4%, and 1.2%, respec

294 NTE (shrinkage, induration, telangiectasia, edema) were 2.12 (95% CI, 1.55 to 2.89; P < .001) for 30

295 cant increases in T(2), a possible marker of edema, were found in multiple regions.

296 rized by pulmonary hydrostatic (cardiogenic) edema, whereas TRALI presents as pulmonary permeability

297 d VE-cadherin phosphorylation could suppress edema while leaving other VEGFR2-dependent functions int

298 ged 18 years and older with diabetic macular edema who received intravitreal injections of bevacizuma

299 nduced vasodilatation, vascular leakage, and edema, with fewer adverse effects.

300 ation of retinitis, vitritis, and optic disc edema without optic nerve enhancement should prompt sero