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1 The PAC count was quantified by 24-hour electrocardiography.
2 yocardial Infarction (TIMI) risk scores, and electrocardiography.
3 rs; 54% women) who underwent routine 12-lead electrocardiography.
4 py as well as echocardiography and conscious electrocardiography.
5 ST-segment analysis using continuous 12-lead electrocardiography.
6 ease, regional wall motion, and quantitative electrocardiography.
7 cytes and idioventricular rhythms by in vivo electrocardiography.
8 riability was measured by 24-hour ambulatory electrocardiography.
9 ography, electrocardiography, and ambulatory electrocardiography.
10 ngina can start stress testing with exercise electrocardiography.
11 om concomitant LVH was found through resting electrocardiography.
12 hoven and Lewis dominated the early years of electrocardiography.
13 re normal, and ischemic changes were seen on electrocardiography.
14 3 respondents underwent echocardiography and electrocardiography.
15 duration of < or = 110 ms on routine 12-lead electrocardiography.
16 es the sensitivity and specificity of stress electrocardiography.
17 of ST-segment elevation on postresuscitation electrocardiography.
18 ce potentials defines the forward problem of electrocardiography.
19 th a right bundle branch block morphology by electrocardiography.
20 terdam, the Netherlands) and high-resolution electrocardiography (1.6 kHz in orthogonal XYZ leads) wa
21 of creatine kinase (CK-MB) elevation (daily electrocardiography; 16 serial CK-MB measurements); or 2
22 ral negative T waves and low QRS voltages on electrocardiography (33%); ventricular arrhythmias (82%)
23 initial testing costs were $174 for exercise electrocardiography; $404 for CTA; $501 to $514 for phar
25 1 [-$4991 to -$4969]; P < .001) and exercise electrocardiography (-$7449 [-$7452 to -$7444]; P < .001
28 ught to characterize the value of AI-enabled electrocardiography (AI-ECG) as a predictor of future AF
33 neously, fNIRS, EEG, electromyography (EMG), electrocardiography and behavioral measures were assesse
35 iver transplantation on disease progression, electrocardiography and Doppler echocardiography were pe
36 icion, because the initial clues provided by electrocardiography and echocardiography might not be ty
37 cting data on physical examination findings, electrocardiography and echocardiography results, fluid
44 lts suggest that electrical abnormalities on electrocardiography and Holter monitoring precede detect
45 ith electrical abnormalities on the basis of electrocardiography and Holter monitoring, of whom 20 (4
49 raphy, magnetic resonance imaging, and fetal electrocardiography and magnetocardiography, are availab
53 echocardiography, immediate neonatal surface electrocardiography and postnatal transesophageal electr
55 diagnostic test evidence (including exercise electrocardiography and stress echocardiography and sing
57 low-up, 30 mo); 36 of them agreed to undergo electrocardiography and transthoracic echocardiography.
58 ding written consent, each patient underwent electrocardiography and transthoracic echocardiography.V
60 rhythmogenesis following SE using continuous electrocardiography and video electroencephalography (vE
61 patients with both electrical abnormalities (electrocardiography and/or Holter monitoring) and abnorm
62 e (finger photoplethysmography), heart rate (electrocardiography) and MSNA (microneurography) were as
63 lysis In Myocardial Infarction (TIMI) score, electrocardiography, and 0 + 2 h values of laboratory tr
64 termined by imaging studies, signal-averaged electrocardiography, and 24-h ambulatory electrocardiogr
66 atients were followed with echocardiography, electrocardiography, and ambulatory electrocardiography.
68 armacokinetics, pharmacodynamics, continuous electrocardiography, and clinical events were assessed.
72 noninvasively captured by echocardiography, electrocardiography, and magnetic resonance microscopy i
73 s, fasting glucose and insulin measurements, electrocardiography, and monitoring of vital signs and b
74 flow, thoracoabdominal bands, body position, electrocardiography, and oxygen saturation (n = 136); or
75 y, physical examination, laboratory testing, electrocardiography, and plain x-ray imaging may often s
76 en present with nonspecific symptoms, normal electrocardiography, and preserved left ventricular ejec
78 panel consisting of pulse oximetry, 12-lead electrocardiography, and serum troponin T would have pro
80 troponin I, without new ischemic changes on electrocardiography, and who had no cardiovascular compl
81 se using ankle and brachial blood pressures; electrocardiography; and assessments of microalbuminuria
82 rea nitrogen, creatinine, and glucose tests; electrocardiography; and portable chest radiography).
83 The automated multitest laboratory provides electrocardiography, anthropometry, chest and breast x-r
84 First, history, physical examination, and electrocardiography are the core of the syncope workup (
87 vity (SGNA), vagal nerve activity (VNA), and electrocardiography before and after pacing-induced CHF.
90 ardiography), heart rate variability (Holter electrocardiography), body composition (dual-energy x-ra
91 patients referred specifically for exercise electrocardiography, both abnormal heart rate recovery a
92 e, or orthostatic hypotension should receive electrocardiography but do not otherwise require immedia
93 us), has distinct repolarization patterns on electrocardiography, but it is not known whether the gen
94 aging, radionuclide angiography and exercise electrocardiography, but its influence on exercise echoc
95 including Rose angina questionnaire, 12-lead electrocardiography, C-reactive protein, and calculation
96 ing, two-dimensional echocardiography [2DE], electrocardiography, cardiac magnetic resonance imaging)
97 using Cox regression, composed by age, sex, electrocardiography, cardiovascular risk factors, LVEF,
99 compared with those of coronary angiography, electrocardiography, cine MRI, and creatine kinase measu
100 estimated how accurately resting or exercise electrocardiography classified participants into high-,
101 emental cost-effectiveness ratio of exercise electrocardiography compared with no testing was $57,700
102 eded by a standard battery of medical tests (electrocardiography, complete blood count, and measureme
107 ) from the Common Standards for Quantitative Electrocardiography database were used to generate deriv
108 he cumulative number of 7 traits (changes on electrocardiography; decreased LV systolic, diastolic di
115 ptal wall thickening, and fibrosis, although electrocardiography displayed a left axis shift of mean
116 New-onset AF was assessed and confirmed by electrocardiography during a mean follow-up of 4.7 years
117 -two patients with ICDs underwent ambulatory electrocardiography during a mental stress protocol, 3 m
118 icial intelligence (AI) algorithm applied to electrocardiography during sinus rhythm has recently bee
123 w evidence on the benefits of screening with electrocardiography (ECG) in asymptomatic adults to redu
126 iagnosis of myocardial infarction (MI) using electrocardiography (ECG) is the cornerstone of effectiv
128 led transthoracic echocardiography (TTE) and electrocardiography (ECG) may provide a scalable strateg
129 r abnormalities by using resting or exercise electrocardiography (ECG) might help identify persons wh
130 t to measure the impact of pre-hospital (PH) electrocardiography (ECG) on scene-to-hospital time for
131 n, a telemedicine visit was initiated and an electrocardiography (ECG) patch was mailed to the partic
133 onstrated late potentials on signal-averaged electrocardiography (ECG) recorded in patients with BrS.
138 ate myocardial relaxation using clinical and electrocardiography (ECG) variables as a first step in t
139 se were studied by vectorcardiography (VCG), electrocardiography (ECG), and coronary arteriography.
140 mutation carriers (n = 84) were evaluated by electrocardiography (ECG), Holter monitoring, late-enhan
143 ned by cardiac magnetic resonance (CMR) and electrocardiography (ECG), with incident atrial fibrilla
144 rly stage cardiac autonomic dysfunction with electrocardiography (ECG)-based measures in MetS subject
150 to monitor HR and cardiac electrophysiology [electrocardiography (ECG)] were exposed once by whole-bo
151 ined after standard tests, including 24-hour electrocardiography [ECG]), to undergo additional noninv
153 a physical examination, a chest radiograph, electrocardiography, echocardiography with detailed exam
154 nd April 2013 underwent clinical assessment, electrocardiography, echocardiography, and biomarker mea
155 oatrial node preparation, telemetric in vivo electrocardiography, echocardiography, and in vivo elect
156 cardiac amyloidosis at a single center with electrocardiography, echocardiography, and laboratory te
157 nal vascular photography, micro-albuminuria, electrocardiography, echocardiography, and plasma B-type
160 extracellular volume fraction may complement electrocardiography, echocardiography, cardiopulmonary e
161 age, 57 years; 59% women) by use of 5 tests (electrocardiography, echocardiography, carotid ultrasoun
162 cipants (mean age 58 years; 59% women) using electrocardiography, echocardiography, carotid ultrasoun
163 assessed by recording adverse events and by electrocardiography, echocardiography, haematological te
164 trio with HCM underwent clinical evaluation, electrocardiography, echocardiography, magnetic resonanc
166 ation (SaO2), intra-arterial blood pressure, electrocardiography (EKG), and transesophageal echocardi
167 and carotid sinus massage during continuous electrocardiography, electroencephalography and blood pr
168 dated sheep with tracheostomies monitored by electrocardiography, electroencephalography, arterial li
169 After an initial evaluation consisting of electrocardiography, electrolytes, blood urea nitrogen,
170 entral venous pressure, chest wall movement, electrocardiography, electromyography, electroencephalog
171 d by discharge summary documentation, enzyme/electrocardiography evidence of AMI, inpatient ICD-9 cod
173 Study subjects underwent 48-h ambulatory electrocardiography, fasting blood tests, and clinical e
175 ital signs, clinical laboratory findings, or electrocardiography findings in any of the treatment gro
178 ocardiography is more accurate than exercise electrocardiography for the identification of CAD in wom
180 mo of MPS--who were referred for rest-stress electrocardiography-gated 99mTc-sestamibi MPS with AC we
181 (MF) registration algorithms were applied to electrocardiography-gated and dual-gated data, creating
182 atients underwent contrast material-enhanced electrocardiography-gated cardiac multidetector CT.
183 on-counting CT scanner using a retrospective electrocardiography-gated contrast-enhanced UHR scanning
184 d selected studies that compared prospective electrocardiography-gated coronary CT angiography with c
185 rd-approved, HIPAA-compliant study, thoracic electrocardiography-gated dual-source multidetector CT a
186 ition, and 4-bin respiration-gated and 8-bin electrocardiography-gated images (32 bins in total) usin
187 n reconstruction (HD*PET): ungated and 8-bin electrocardiography-gated images using 5-min acquisition
188 eformable, motion-coherent modeling based on electrocardiography-gated multidetector computed tomogra
191 -replenishment MCE, standard (99m)Tc-labeled electrocardiography-gated SPECT, and quantitative CA wit
192 m SPECT with both attenuation correction and electrocardiography-gating were used as a diagnostic tes
193 ent, and carotid artery ultrasound, exercise electrocardiography has been proposed as a screening too
195 ardial infarction or evidence of ischemia on electrocardiography (hazard ratio, 0.13; 95% CI, 0.04 to
196 accuracy could be inferred: signal-averaged electrocardiography; heart rate variability; severe vent
197 bining state-of-the-art cardiac imaging with electrocardiography, histopathology, and molecular analy
198 ho were randomly assigned to 24-h ambulatory electrocardiography (Holter) monitoring and who had a no
200 nsional and M-mode echocardiography, surface electrocardiography, Holter monitoring and exercise test
201 undergo another examination, which included electrocardiography, Holter monitoring, echocardiography
202 ands with sinus bradycardia were examined by electrocardiography, Holter recording, exercise stress t
203 rognostic value of preoperative quantitative electrocardiography in patients who underwent isolated c
204 ena as "Luciani periods." With the advent of electrocardiography in the early 20th century, this form
210 ng to multidisciplinary clinical evaluation, electrocardiography, laboratory test, echocardiography,
211 objective normalization of echocardiography, electrocardiography, laboratory testing, graded exercise
212 grade flow was 180 +/- 67 min; a median of 5 electrocardiography leads showed ST-segment deviation (q
213 severe ventricular arrhythmia on ambulatory electrocardiography; left ventricular ejection fraction;
214 These findings suggest that quantitative electrocardiography may be valuable for risk stratificat
215 al intelligence-enabled echocardiography and electrocardiography may enable scalable risk stratificat
216 ntal regions' spontaneous activation, and an electrocardiography measure of PNS (high frequency heart
219 m-limited exercise tests were performed with electrocardiography (n = 300) and thallium scintigraphy
221 phy (CTA) or to functional testing (exercise electrocardiography, nuclear stress testing, or stress e
222 mly assigned to functional testing (exercise electrocardiography, nuclear stress, or stress echocardi
223 tery disease were randomized to FT (exercise electrocardiography, nuclear stress, or stress echocardi
225 rent arrhythmia per current guidelines using electrocardiography or ambulatory monitor recording and
230 graphy (CTA) or functional testing (exercise electrocardiography or nuclear stress testing) from 2009
231 his findings before the benefit of clinical electrocardiography or the discovery of the sinoatrial a
232 , premature atrial complexes on preoperative electrocardiography (OR, 2.1 [CI, 1.3 to 3.4]), American
234 ith incident left ventricular hypertrophy by electrocardiography over 6 years (eg, adjusted hazard ra
236 output (acetylene rebreathing), heart rate (electrocardiography), oxygen uptake (Douglas bag techniq
237 t was used to monitor pulse, blood pressure, electrocardiography, oxygen uptake, carbon dioxide outpu
240 f rest echocardiography (p = 0.79), exercise electrocardiography (p = 0.38) or exercise echocardiogra
242 to assess the ability of computer-simulated electrocardiography parameters to predict clinical outco
245 , variably including electroencephalography, electrocardiography, plethysmography, mechanical ventila
247 ariate analysis model, clinical and exercise electrocardiography predictors of cardiac events were ag
248 fe support should include diagnostic 12-lead electrocardiography programs as one of their services.
249 trophysiological changes were assessed using electrocardiography (QRS duration), vectorcardiography (
250 history and underwent physical examination, electrocardiography, quality of life, and laboratory ass
252 atio after adjustment for age, sex, exercise electrocardiography result, and secondary prevention med
255 rdiac arrest victims with a first registered electrocardiography rhythm of ventricular fibrillation o
256 pare T-wave alternans (TWA), signal-averaged electrocardiography (SAECG) and programmed ventricular s
257 t electrocardiography (ECG), signal-averaged electrocardiography (SAECG), echocardiography, cardiac m
263 nin I assay if the CK-MB value is normal and electrocardiography shows ischemic changes; both CK-MB m
264 c screening in adults with resting or stress electrocardiography, stress echocardiography, or myocard
265 atic, low-risk adults with resting or stress electrocardiography, stress echocardiography, or stress
269 without significant ST-segment elevation on electrocardiography, termed NSTE-ACS, account for approx
270 studied, using echocardiography and 12-lead electrocardiography, the phenotypic expression caused by
271 for diabetes and hyperlipidemia, and resting electrocardiography to estimate the patient's probabilit
272 functional MRI experiments with simultaneous electrocardiography to examine regional brain activity a
274 SE (Practical Use of the Latest Standards of Electrocardiography) Trial was a 6-year multisite random
275 y calcium score (CS), traditionally based on electrocardiography-triggered computed tomography (CT),
277 ronary calcium, but it does not suggest that electrocardiography-triggered CT should be replaced by n
278 e correlation in CS between nontriggered and electrocardiography-triggered CT, and to evaluate the pr
280 e3(-/-) mice using real-time qPCR, echo- and electrocardiography, ventricular myocyte patch-clamp, co
281 74 control), the mean (SD) age at diagnostic electrocardiography was 11.0 (9.4) years and 119 (54%) w
282 ill exercise; an abnormal result on exercise electrocardiography was defined by ST segment depression
283 mptomatic prolongation of the QT interval on electrocardiography was identified as the only dose-limi
287 patients who took placebo (P =.89); exercise electrocardiography was positive in 12 patients (11%) wh
289 d ischemia, assessed by echocardiography and electrocardiography, was defined as: 1) development or w
290 analyzed 146 patient-years of vital sign and electrocardiography waveform time series from the bedsid
291 ce imaging, serum biomarker measurement, and electrocardiography were compared across study cohorts.
292 myocardial infarction or ischemic changes on electrocardiography were less frequent in the PCI group
293 y (MSNA), continuous blood pressure (BP) and electrocardiography were measured at baseline, as well a
294 ure, blood pressure, heart rate, and 12-lead electrocardiography were monitored throughout the 24-hr
295 no clinical events due to QT prolongation on electrocardiography were observed, QT prolongation was r
298 re measurements using a sphygmomanometer and electrocardiography, were employed by a physician to per
299 A careful history, physical examination, and electrocardiography will provide a diagnosis or determin