1 erwent detailed neurological examination and
electrodiagnostic and genetic testing for the major know
2 ndrome) has been advanced primarily based on
electrodiagnostic and limited pathological data, but rem
3 Antemortem
electrodiagnostics and radiologic imaging indicated a di
4 We evaluated the clinical,
electrodiagnostic,
and histopathological features of 14
5 vated serum creatine kinase level, myopathic
electrodiagnostic changes, brain MRI with cobblestone co
6 Furthermore, the traditional
electrodiagnostic classification into axonal and demyeli
7 osis of GBS subtype if the proposed modified
electrodiagnostic criteria are used.
8 The
electrodiagnostic criteria can be supplemented with addi
9 of neurological examinations and genetic and
electrodiagnostic data of confirmed sensory polyneuropat
10 pective study for all patients who underwent
electrodiagnostic (
EDX) evaluation in Israel's Sheba Med
11 wer-extremity sensory and motor deficits and
electrodiagnostic evidence of a length-dependent mixed d
12 that best distinguish between patients with
electrodiagnostic evidence of CTS and patients without i
13 We provide
electrodiagnostic evidence that combined ONTT regimen-co
14 Electrodiagnostic examination showed severely low compou
15 s or small series; however, its clinical and
electrodiagnostic features have not been well characteri
16 We report the clinical and
electrodiagnostic findings in 49 affected family members
17 Electrodiagnostic findings were consistent with a necrot
18 Electrodiagnostic findings were consistent with bilatera
19 tion, retinal nerve fiber layer thinning, or
electrodiagnostic findings.
20 The clinical and
electrodiagnostic hallmarks suggest selective motor fibe
21 ripheral nerve injury relies on clinical and
electrodiagnostic information, supplemented by intraoper
22 tertiary ophthalmology center with access to
electrodiagnostic services from February 18, 2009, throu
23 Electrodiagnostic studies and a sural nerve biopsy showe
24 Electrodiagnostic studies are an important early step in
25 Electrodiagnostic studies evaluating for disorders of ne
26 FAF in 61 patients, OCT in 58 patients, and
electrodiagnostic studies in 36 patients.
27 We emphasize the use of
electrodiagnostic studies to investigate patients with b
28 magnetic resonance imaging, ultrasonography,
electrodiagnostic studies, and muscle biopsies (n = 3).
29 An
electrodiagnostic study including electromyography of th
30 erve fibre density, which was independent of
electrodiagnostic test severity.
31 ailed ophthalmic examination, accompanied by
electrodiagnostic testing (EDT) and dysmorphologic asses
32 diagnosis of PTS was confirmed by using both
electrodiagnostic testing and 3.0-T MR neurography.
33 Electrodiagnostic testing and magnetic resonance imaging
34 objective methods for assessment, including
electrodiagnostic testing and nerve imaging, provide add
35 Serial
electrodiagnostic testing demonstrated a rapidly progres
36 hic scans were abnormal in all patients, and
electrodiagnostic testing revealed rod and cone dysfunct
37 Electrodiagnostic testing showed a motor and sensory axo
38 Neurological examination and
electrodiagnostic testing showed gait-difficulties, abse
39 An approach to laboratory testing,
electrodiagnostic testing, and imaging to exclude diseas
40 ther compressive neuropathies should undergo
electrodiagnostic testing, which is approximately more t
41 idence of spinal cord motor neuron injury on
electrodiagnostic testing.
42 n maneuvers were independently compared with
electrodiagnostic testing.
43 ic assessment, including retinal imaging and
electrodiagnostic testing.
44 Electrodiagnostic tests and magnetic resonance imaging o
45 In contrast, expensive
electrodiagnostic tests and magnetic resonance imaging r
46 Moreover, the role of
electrodiagnostic tests needs to be further defined, and