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1 y obvious signs of seizure activity on scalp electroencephalogram.
2 ings with foramen ovale electrodes and scalp electroencephalogram.
3 d spectral peaks differing from the baseline electroencephalogram.
4 in functional magnetic resonance imaging and electroencephalogram.
5 eneralized spike and wave discharges seen on electroencephalogram.
6 tures that contribute minimally to the scalp electroencephalogram.
7 fter cardiac arrest and preceded isoelectric electroencephalogram.
8 e performed a spectral analysis of the sleep electroencephalogram.
9 he theta rhythm, as shown on the hippocampal electroencephalogram.
10 signal strength and map dissimilarity of the electroencephalogram.
11 early visual cortex as measured by the human electroencephalogram.
12 eration of a local field potential (LFP) and electroencephalogram.
13 le for everyday monitoring than utilizing an electroencephalogram.
14 ed evoked responses relative to pre-stimulus electroencephalogram.
15 oach to measure mnemonic hidden states in an electroencephalogram.
16 n electrographic signatures in intracerebral electroencephalograms.
17 mes of brain probed by clinical intracranial electroencephalograms.
18 ation again, this time applied to observers' electroencephalogram activity, we established where and
24 t cortical hyper excitability as measured by electroencephalogram and auditory-evoked potentials.
25 uced status epilepticus was characterized by electroencephalogram and behavior in GluA1 knockout mice
26 tinct brain state characterized by activated electroencephalogram and complete skeletal muscle paraly
29 CI device integrates wearable, wireless, dry electroencephalogram and electrooculogram systems and a
31 lunteers for the acquisition of simultaneous electroencephalogram and functional magnetic resonance i
32 ls debated the issues on the accuracy of the electroencephalogram and its place, the absolute need fo
33 amma during fast stopping and recorded scalp electroencephalogram and local field potentials from dee
36 llations measured by local field potentials, electroencephalograms and magnetoencephalograms exhibit
37 s were assessed using 5-minute resting-state electroencephalograms and parallel electrocardiograms.
39 and subthalamic nucleus along with cortical electroencephalograms and were compared to recordings fr
41 pilepsy localization from seizure semiology, electroencephalogram, and magnetic resonance imaging.
42 in responses were recorded with high-density electroencephalogram, and sources of event-related poten
43 tivity and prolonged propagation time on the electroencephalogram, and the absence of metabolic dysfu
44 unction of pyramidal cell activity, with the electroencephalogram approximated by the sum of populati
46 erized by spike-wave discharges (SWD) in the electroencephalogram, arises from aberrations within the
47 coma after cardiac arrest, including visual electroencephalogram assessment by trained electroenceph
48 evelops during SRSE despite seizure control (electroencephalogram background suppression with anesthe
49 nt effects on state consolidation and/or the electroencephalogram but had no effect on total wake.
50 by video observation of each animal and the electroencephalogram by an automated seizure detection p
53 r 2 are suspected to be epileptogenic and if electroencephalogram changes are equivocal or discordant
54 Gaining a better understanding of sleep and electroencephalogram changes in patients with Huntington
56 mesial temporal lobe seizures based on scalp electroencephalogram coherence features, lends weight to
57 istic regression classifiers that used scalp electroencephalogram coherence properties as input featu
59 aneous circulation, initial rhythm, combined electroencephalogram/CT findings, Charlson Comorbidity I
60 address this by analyzing human intracranial electroencephalogram data recorded during 2 associative
62 relationship between cascades of activity in electroencephalogram data, cognitive state, and reaction
65 chanisms, and the low-frequency power in the electroencephalogram (delta power) during non-rapid eye
66 hypothesis that a protocol incorporating the electroencephalogram-derived bispectral index (BIS) is s
68 make no recommendation concerning the use of electroencephalogram-derived parameters as a measure of
69 A detailed history, physical examination, electroencephalogram, developmental evaluation, Autism D
72 closely parallel those observed in the human electroencephalogram during propofol-induced unconscious
73 by measuring the level of randomness in the electroencephalogram during the prestimulus baseline per
74 edominately display bilaterally synchronized electroencephalogram (EEG) activity during slow-wave sle
75 k, many mouse models for AD exhibit abnormal electroencephalogram (EEG) activity in addition to the e
76 of arousal: sleep and wakefulness, cortical electroencephalogram (EEG) activity, acetylcholine (ACh)
78 medullary reticular formation, and implanted electroencephalogram (EEG) and ECG recording electrodes.
79 t budgerigars (Melopsittacus undulatus) with electroencephalogram (EEG) and electrooculogram (EOG) el
82 continuous-valued neural recordings like the electroencephalogram (EEG) and local field potential (LF
83 itoring the patient's brain activity with an electroencephalogram (EEG) and manually titrating the an
86 al activity, because these components of the electroencephalogram (EEG) are sensitive to basal forebr
88 s, and slow-wave sleep with interhemispheric electroencephalogram (EEG) asymmetry, resembling the uni
91 e highly synchronous across the scalp in the electroencephalogram (EEG) but have low spatial coherenc
94 respond to sleep deprivation, the strongest electroencephalogram (EEG) correlate of sleep pressure,
95 the clinical, psychometric, and wake-/sleep-electroencephalogram (EEG) correlates of induced hyperam
101 in alpha- and beta-range oscillations in the electroencephalogram (EEG) during observation of reachin
102 tral slow oscillations recorded in the scalp electroencephalogram (EEG) during rapid eye movement (RE
103 ance, brain biomarkers, and abnormalities in electroencephalogram (EEG) during the perioperative peri
105 ate the acute effects of alcohol on cortical electroencephalogram (EEG) in adolescent (P36) and adult
111 The signature of slow-wave sleep in the electroencephalogram (EEG) is large-amplitude fluctuatio
113 y review the principles underlying processed electroencephalogram (EEG) monitors and recent studies v
114 lue of HFOs for developing epilepsy in scalp electroencephalogram (EEG) of children after a first unp
115 ent decline in the slow wave (delta, 1-4 Hz) electroencephalogram (EEG) of nonrapid eye movement (NRE
117 taneous, ChR2, or forepaw stimulation-evoked electroencephalogram (EEG) or local field potential (LFP
118 locked signals superimposed upon the ongoing electroencephalogram (EEG) or result from phase-alignmen
119 eration depends upon theta rhythm, a 6-10 Hz electroencephalogram (EEG) oscillation that is modulated
121 ected by frontal-midline theta-band (4-8 Hz) electroencephalogram (EEG) oscillations, strengthen the
123 n comparable measurement scales, we recorded electroencephalogram (EEG) over medial frontal cortex of
124 l population are linked to variations in the electroencephalogram (EEG) over motor, pre-motor cortex
125 frog (Babina daunchina) using the optimized electroencephalogram (EEG) paradigm of mismatch negativi
126 EM sleep by an "activated," low-voltage fast electroencephalogram (EEG) paradoxically similar to that
127 es of unknown etiology with a characteristic electroencephalogram (EEG) pattern and developmental reg
128 al isolation on sleep, wakefulness and delta electroencephalogram (EEG) power during non-rapid eye mo
130 ergic neurons on the sleep-wake behavior and electroencephalogram (EEG) power spectrum using the phar
135 in the cerebral cortex fire irregularly and electroencephalogram (EEG) recordings display low-amplit
136 e simultaneous human intrathalamic and scalp electroencephalogram (EEG) recordings from eight volunte
137 ng an array of 12 behavioral assessments and electroencephalogram (EEG) recordings on freely-moving m
138 study was to use oscillatory changes in the electroencephalogram (EEG) related to informative cue pr
141 ormal baseline and social interaction-evoked electroencephalogram (EEG) signals, and an altered compo
142 The slow (<1 Hz) rhythm, the most important electroencephalogram (EEG) signature of non-rapid eye mo
144 global sleep-wake history, and reflected in electroencephalogram (EEG) slow wave activity (SWA, 0.5-
145 investigating characteristics of individual electroencephalogram (EEG) slow waves in young and elder
146 sleep homeostasis: slow-wave sleep (SWS) and electroencephalogram (EEG) slow-wave activity in non-rap
147 is work uses an innovative method to analyze electroencephalogram (EEG) spectral frequencies within s
148 acological treatments in rats to study which electroencephalogram (EEG) spectral properties are assoc
151 ation approach for the analysis of the human electroencephalogram (EEG) to decode choice outcomes in
152 attention has turned to assessments based on electroencephalogram (EEG) to evaluate subtle post-concu
154 als that dietary CAF does not alter baseline electroencephalogram (EEG) total power, but significantl
161 uring somatosensory evoked potentials (SEP), electroencephalogram (EEG), direct current (DC) potentia
162 t (NREM) sleep are characterized by distinct electroencephalogram (EEG), electromyogram (EMG), and au
163 = 6) were surgically implanted to record the electroencephalogram (EEG), electromyogram, and locomoto
164 f human activity, biological signals such as Electroencephalogram (EEG), Electrooculogram (EOG), Elec
165 rized by fast, desynchronized rhythms in the electroencephalogram (EEG), hippocampal theta activity,
166 This tuning was significantly weaker in electroencephalogram (EEG), suggesting that ECoG is more
167 2 female macaque monkeys, and also recorded electroencephalogram (EEG), while they viewed a variety
169 istence of a slow wave sleep (SWS)-promoting/electroencephalogram (EEG)-synchronizing center in the m
180 ry 6-12 months until they had an isoelectric electroencephalogram (EEG, attesting to a vegetative sta
181 ated markers of consciousness extracted from electroencephalograms (EEG), we computed autonomic cardi
182 (PB) complex in regulating electrocortical (electroencephalogram [EEG]) and behavioral arousal: lesi
183 tween manifest variables of the brain (e.g., electroencephalogram [EEG], functional MRI [fMRI]) and m
184 e recorded local field potentials (LFPs) and electroencephalograms (EEGs) from contralateral cortex.
189 electrodes, frontofrontal and frontoparietal electroencephalogram electrodes and then recorded sleep/
190 selective N170 component at occipitotemporal electroencephalogram electrodes, which was still present
191 ype mice were surgically implanted to record electroencephalogram, electromyogram, locomotor activity
192 our previous examination of these mice using electroencephalogram/electromyogram (EEG/EMG) monitoring
193 hat affect sleep and wakefulness by using an electroencephalogram/electromyogram-based screen of rand
194 l Evoked Potential (VEP), a component of the electroencephalogram elicited by visual stimuli, and cog
195 t 12 and 24 hours after cardiac arrest using electroencephalogram epochs and outcome labels as inputs
199 developed spontaneous and recurring abnormal electroencephalogram events consistent with progressive
201 utine clinical interpretation of these scalp electroencephalograms failed to identify any of the scal
202 statistical association between quantitative electroencephalogram features and neurologic outcome cha
203 ts for an expanded selection of quantitative electroencephalogram features and whether accounting for
205 ogic outcome (good vs poor) and quantitative electroencephalogram features in 12-hour intervals using
207 essive diffuse sensory motor axonopathy, and electroencephalogram findings progressed from generalize
210 ally, as the disease progressed, an abnormal electroencephalogram gamma activity (30-40 Hz) emerged i
211 erogeneous patterns, along with intracranial electroencephalogram gamma power changes, several minute
212 tral index (BIS), developed from a processed electroencephalogram, has been reported to decrease the
214 igns of early severe anoxic changes on CT or electroencephalogram, higher creatinine levels and recei
215 milarity analysis (RSA) [17] to intracranial electroencephalogram (iEEG) data from ten presurgical ep
217 n of the progression of changes in sleep and electroencephalogram in Huntington's disease has never b
220 e contingent negative variation (CNV) in the electroencephalogram, is the signature of the subjective
222 ng, electroencephalogram or continuous video electroencephalogram, lumbar puncture, and genetic testi
223 ilepsy is localized through different modes (electroencephalogram, magnetic resonance imaging, etc) t
224 ene carriers suggest that alterations in the electroencephalogram may reflect underlying neuronal dys
225 everely decreased brain wave activity on the electroencephalogram, may be unintentionally induced by
227 omatic injector combined with 24-h video and electroencephalogram monitoring demonstrated significant
233 from the mesial temporal lobe based on scalp electroencephalogram network connectivity measures.
235 cidence of epileptiform abnormalities in the electroencephalogram of people with focal epilepsy.
237 alysis performed with patients without early electroencephalogram or CT changes still revealed better
241 atio, 7.11; 95% CI, 5.01-10.08), unfavorable electroencephalogram patterns (false-positive rate, 0.07
242 gical outcome of low-voltage and isoelectric electroencephalogram patterns 24 hrs after resuscitation
244 poor outcome of low-voltage and isoelectric electroencephalogram patterns was 68% (confidence interv
248 rease in slow-wave sleep, decreases in delta electroencephalogram power and evoked delta activity, an
250 in the subthalamic nucleus (STN) to frontal electroencephalograms preceded the onset and followed th
251 rol without suppression-burst or isoelectric electroencephalogram predicted good functional recovery
254 epresentational similarity analysis to human electroencephalograms, recorded while female and male pa
255 Z) performed an auditory oddball task during electroencephalogram recording before and after auditory
260 Chemogenetic activation experiments and electroencephalogram recordings pointed to glutamatergic
266 seizures vs two or fewer, 1.08, 1.05-1.11), electroencephalogram results (epileptiform abnormality v
268 ical outcome showed continuous, diffuse slow electroencephalogram rhythms, whereas this was never obs
269 by using trial-by-trial oscillations in the electroencephalogram's alpha band (8-14 Hz) collected fr
271 ity, as assessed by quantitative analysis of electroencephalogram signal, and ischemic neuronal injur
273 e we combined PET measures of Abeta and tau, electroencephalogram sleep recordings, and retrospective
278 ayed robust in vivo activity in a sleep-wake electroencephalogram (sw-EEG) assay consistent with mGlu
279 Here, we used a specially designed dual-electroencephalogram system and the conceptual framework
280 Of the 26 patients studied, 5 patients had electroencephalograms that showed periodic discharges co
282 This was followed by a change in the child's electroencephalogram to the chaotic pattern of hypsarrhy
283 at this knowledge can guide their use of the electroencephalogram to track more accurately the brain
292 neonatal epilepsy with suppression bursts on electroencephalogram, we have expanded the phenotypic sp