ライフサイエンスコーパス: electrographic

1 in 296 infants (33.9%) and was predominantly electrographic.

2 in 43 (10.3%) and were associated with other electrographic abnormalities previously reported to indi

3 We investigated electrographic activities and neural dynamics leading to

4 Hippocampal high-frequency electrographic activity (HFOs) represents one of the maj

5 d LDH measurements were well correlated with electrographic activity and neuron counts, respectively.

6 lation requires systems that detect abnormal electrographic activity and provide stimulation (closed

7 The identification of abnormal electrographic activity is important in a wide range of

8 A normative electrographic activity map could be a powerful resource

9 ot coupled to the production of pathological electrographic activity nor were they due to cell death.

10 The specific electrographic activity responsible for seizure-induced

11 Mutants exhibit spontaneous abnormal electrographic activity, hyperactivity and convulsive be

12 s in the appearance of cortical epileptiform electrographic activity, increases of seizure duration a

13 evels and new onset seizure (clinical and/or electrographic) among patients who received low (500 mg

14 BA(A) receptors is sufficient to elicit both electrographic and behavioral correlates of seizures in

15 after seizure onset strongly suppresses both electrographic and behavioral seizures induced by kainic

16 Flupirtine was also effective in arresting electrographic and behavioral seizures when administered

17 tish phenotype exhibit spontaneous recurrent electrographic and behavioral seizures.

18 time was sufficient to immediately interrupt electrographic and behavioral seizures.

19 Treating electrographic and clinical seizures with currently used

20 ular (AV) node reentry tachycardia: combined electrographic and radiographic features.

21 er observed in sham-injured animals and have electrographic appearance similar to the onset of obviou

22 ese findings reveal gene-linked quantitative electrographic biomarkers free from epileptiform activit

23 Electrographic biomarkers improve post-stroke epilepsy p

24 minant features and can be used as potential electrographic biomarkers in epilepsy detection research

25 ese findings reveal gene-linked quantitative electrographic biomarkers in the absence of epileptiform

26 Electrographic biomarkers, including epileptiform activi

27 Similarly, varying electrographic brain activities from different brain reg

28 thdrawal cycles reduced behavioral (HIC) and electrographic (BSE) signs of seizure activity in a dose

29 e response that did not have a corresponding electrographic change on electroencephalogram consistent

30 However, the abrupt electrographic changes and large brief cardio-respirator

31 lar system ranging from transient and benign electrographic changes to myocardial injury, cardiomyopa

32 In all 21 patients with stored electrographic data and appropriate interventions, the i

33 Connectome-based analyses of electrographic data from stereo electroencephalography (

34 We present the clinical and electrographic data of 17 patients with reading-induced

35 ion of medial septal GABAergic neurons, upon electrographic detection of spontaneous hippocampal seiz

36 tients and outpatients suggest that abnormal electrographic discharges can be detected before there i

37 ies, including staring-freezing episodes and electrographic (EEG) seizures.

38 Ripples are brief high-frequency electrographic events with important roles in episodic m

39 Recent intracranial electrographic evidence has suggested that seizure-onset

40 d with seizure duration (P = 0.001) and with electrographic evidence of seizure spread to the contral

41 Correlations between electrographic features and mortality and modified Ranki

42 d provide novel information that traditional electrographic features do not.

43 xplored the biomarker potential of different electrographic features in five models of PIE.

44 gut dysmotility and a movement disorder) and electrographic features including hypsarrhythmia (associ

45 This study suggests that etiology and electrographic findings are associated with ASM treatmen

46 n associated with preserved volition despite electrographic generalization and uniform average oscill

47 are extended periods without behavioural or electrographic ictal events.

48 sleep also showed a higher concordance with electrographic ictal onset zone from scalp EEG recording

49 entified in 296 newborns (33.9%), being only electrographic in 213 (71.9%) and clinical followed by e

50 quency and onset, with a higher tendency for electrographic inter-ictal spikes and beta- and gamma-fr

51 Similarly, action potentials recorded during electrographic interictal activity in the 'high [K+]o' m

52 rictal epileptiform discharges (IEDs) are an electrographic manifestation of excessive hypersynchroni

53 recordings have recently discovered that an electrographic measure of epileptogenicity, interictal e

54 quent seizure was assessed by behavioral and electrographic measures.

55 on partially testing this hypothesis at the electrographic network level within 81 individuals with

56 tracranial monitoring offers a view into the electrographic networks that organize around and in resp

57 treatment in adulthood were able to correct electrographic, neuroanatomical and synaptic alterations

58 In control rats, bFGF strongly enhanced the electrographic NMDA responses.

59 Seizures during cEEG were purely electrographic (no detectable clinical correlate) in the

60 Electrographic-only seizures were present in 9 of 32 sub

61 epileptic treatment, started after the first electrographic or clinical seizure, or preventively when

62 , were defined based on the evolution of the electrographic pattern and clinical semiology.

63 c computational model closely replicates the electrographic pattern of a typical human focal seizure

64 ork mechanisms underlying these two distinct electrographic patterns might be helpful in designing di

65 he brain can trigger seizures with different electrographic patterns.

66 To determine whether nonconvulsive electrographic post-traumatic seizures result in increas

67 Adult cats were implanted for electrographic recording and with bipolar unilateral sti

68 th LAS were defined by a complete absence of electrographic recording by a circular mapping catheter

69 network connectivity matrix from non-seizure electrographic recordings of patients and use these conn

70 tion of seizure-onset zones using only brief electrographic recordings to reduce patient morbidity an

71 ts and addresses both clinical remission and electrographic resolution of hypsarrhythmia.

72 Concurrently, the electrographic responses to NMDA and clonidine were pote

73 We used intracranial electrographic resting-state and neurostimulation record

74 nd 12 hours during rewarming for evidence of electrographic seizure activity by 2 central amplitude-i

75 stimulation of the trigeminal nerve reduced electrographic seizure activity by up to 78%, and bilate

76 , Lis1+/- hippocampi are prone to interictal electrographic seizure activity in an elevated [K(+)](o)

77 he induction of epileptogenesis by prolonged electrographic seizure activity induced through low-Mg2+

78 study of newborns with HIE treated with TH, electrographic seizure activity occurred in 296 infants

79 al uncoupling of neonatal seizures refers to electrographic seizure activity that is not clinically m

80 nobarbital was most effective in suppressing electrographic seizure activity, but MK-801 had a slight

81 erm memory loss, insomnia, and temporal lobe electrographic seizure activity.

82 monitoring revealed repetitive temporal lobe electrographic seizure activity.

83 and behavioral seizures without accompanying electrographic seizure activity.

84 scribe how to use transcranial US to disrupt electrographic seizure activity.

85 ostfertilization confirmed the occurrence of electrographic seizure activity; seizure-like behaviors

86 mice, CYM2503 increased the latency to first electrographic seizure and decreased the total time in s

87 Additionally, to characterize electrographic seizure and hyperexcitable pattern predic

88 peritoneally, increased the latency to first electrographic seizure and the latency to first stage 3

89 primary efficacy endpoint was a reduction in electrographic seizure burden of more than 80% without t

90 (NeuroPace, Inc.) is a chronic, closed-loop electrographic seizure detection and stimulation system.

91 amic measurement of [Ca2+]i during prolonged electrographic seizure discharges in an in vitro SE mode

92 nd induces myoclonic behavioral seizures and electrographic seizure discharges in the BLA and hippoca

93 First, we demonstrate that electrographic seizure events, induced by pentylenetetra

94 Randomization was to an electrographic seizure group (ESG) in which seizures det

95 'Preictal' (30 s immediately preceding the electrographic seizure onset) and ictal phases, 'ictal-o

96 atients, starting between 98 and 14 s before electrographic seizure onset, and the maps had a degree

97 he SOZ and spread to non-SOZ channels before electrographic seizure onset.

98 cell death and its relationship to specific electrographic seizure patterns in a rat model of focall

99 id onset and end of deficits associated with electrographic seizure start and end.

100 and the secondary outcome of clinical and/or electrographic seizure, were assessed using multivariabl

101 s, and did not differ until the onset of the electrographic seizure.

102 CSF lymphocytic pleocytosis (all 4 tested), electrographic seizures (3 of 4 tested), and striking MR

103 atients were required to have had 20 or more electrographic seizures (development cohort) or self-rep

104 indings in 12 of 14 patients (86%) including electrographic seizures (n = 10) and acute changes relat

105 ateralized rhythmic delta activity predicted electrographic seizures (odds ratio, 6.24; CI, 1.49-26.0

106 ions (OR, 3.24; 95% CI, 1.31-8.00; P = .01), electrographic seizures (OR, 2.85; 95% CI, 1.13-7.19; P

107 rden of ictal-interictal patterns, including electrographic seizures after moderate-to-severe traumat

108 patients with neurological injury to detect electrographic seizures and clinically important changes

109 We aimed to determine whether electrographic seizures and electrographic status epilep

110 Electrographic seizures and electrographic status epilep

111 Dlx1 mutant mice show generalized electrographic seizures and histological evidence of sei

112 ausing mutation into mouse brain resulted in electrographic seizures and impaired hemispheric archite

113 e associated with a high prevalence (75%) of electrographic seizures and might serve as an early pred

114 pear normal histologically, show spontaneous electrographic seizures and reduced power of gamma oscil

115 ation of the epileptogenic region terminates electrographic seizures and reduces the frequency of cli

116 grafting markedly reduced the occurrence of electrographic seizures and restored behavioral deficits

117 This triggered prolonged electrographic seizures and select bilateral neuronal de

118 v1.1 expression and reduced the incidence of electrographic seizures and sudden unexpected death in e

119 of WT SST interneurons resulted in prolonged electrographic seizures and was accompanied by SST hyper

120 Electrographic seizures are frequent in lobar intraparen

121 Electrographic seizures are seizures that are evident on

122 eely moving rats induced both behavioral and electrographic seizures as well as cytotoxicity.

123 e in vivo characterization and rescue of the electrographic seizures associated with TSC.

124 The mice also display clinical and electrographic seizures both spontaneously and with phys

125 f multidien period length, self-reported and electrographic seizures consistently occurred during the

126 In patients with electrographic seizures detected by ICE, scalp EEG demon

127 ept 28, 2013, we screened 30 infants who had electrographic seizures due to hypoxic ischaemic encepha

128 ars was significantly higher in infants with electrographic seizures during rewarming (relative risk

129 Findings that higher odds of electrographic seizures during rewarming are associated

130 The primary outcome was the occurrence of electrographic seizures during rewarming initiated at 72

131 More infants had electrographic seizures during the rewarming epoch compa

132 in unanesthetized mice, low-stage, clinical electrographic seizures had minimal effect on dendritic

133 Most electrographic seizures have no associated clinical chan

134 zations occurred in 161/180 patients and 238 electrographic seizures in 14/180.

135 42%) had seizures, which were categorized as electrographic seizures in 41 (20.5%) and electrographic

136 severe ictal-interictal continuum including electrographic seizures in four (2.6%).

137 eo-EEG confirmed the presence of spontaneous electrographic seizures in Lis1 mutant mice.

138 n hippocampal slices in vitro and attenuated electrographic seizures in neonatal rats in vivo.

139 , as well as high rates of identification of electrographic seizures in patients with unexplained acu

140 Continuous electroencephalography may detect electrographic seizures in some subjects.

141 a shorter horizon of 1 h, possible only for electrographic seizures in the development cohort, showe

142 e revealed abnormal epileptic discharges and electrographic seizures in three of six homozygotes.

143 ons, as well as resistance to behavioral and electrographic seizures in vivo.

144 )](o), 4-aminopyridine, and bicuculline, and electrographic seizures induced by high [K(+)](o) in CA3

145 Studies have shown that a high burden of electrographic seizures is associated with worsened clin

146 ognition and rapid treatment of clinical and electrographic seizures is important during acute illnes

147 E characterized by continuous behavioral and electrographic seizures lasting for hours.

148 who had hypoxic ischaemic encephalopathy and electrographic seizures not responding to a loading-dose

149 Electrographic seizures occurred in 17% of lobar and 5%

150 The effect of electrographic seizures on outcome is the focus of activ

151 re used to evaluate the associations between electrographic seizures or electrographic status epilept

152 Thalamic involvement did not affect electrographic seizures or hyperexcitable patterns in ei

153 were observed in anesthetized animals during electrographic seizures over a 3-hour period.

154 poral lobe involvement, hematoma volume, and electrographic seizures predicted poor outcome in lobar

155 ent, intraparenchymal hemorrhage volume, and electrographic seizures predicted poor outcome.

156 frequency of the spontaneous behavioral and electrographic seizures progressively increased over tim

157 ine whether identification and management of electrographic seizures reduces secondary brain injury a

158 hy recordings with stimulations delivered to electrographic seizures was 24.8%, 1.2%, 7.6% and 8.8%.

159 e was decreased, and the average duration of electrographic seizures was longer in Kchip2(-/-) mice c

160 Electrographic seizures were identified in 296 newborns

161 idence interval 3.7, 80; p < 0.001), whereas electrographic seizures were not associated with an incr

162 Spontaneous, synchronized spikes and electrographic seizures with behavioral accompaniments w

163 zure counts, (2) cEEG-based hourly counts of electrographic seizures, and (3) detections of intericta

164 cal acute symptomatic seizures, 107 (9%) had electrographic seizures, and 364 (31%) had EAs; 532 (45%

165 res, 96% (103 of 107) and 95% (61 of 64) for electrographic seizures, and 64% (233 of 364) and 48% (1

166 Mutant animals developed spontaneous electrographic seizures, as well as long-term deficits i

167 patterns can be identified that, similar to electrographic seizures, cause brain tissue hypoxia, a m

168 variable regression identified predictors of electrographic seizures, hyperexcitable patterns, and po

169 Electrographic status epilepticus, but not electrographic seizures, is associated with mortality an

170 halographs were scored as 1) no seizures, 2) electrographic seizures, or 3) electrographic status epi

171 ure episode when there were no behavioral or electrographic seizures, we found enhanced spontaneous a

172 tal-interictal continuum patterns, including electrographic seizures, were associated with clinical m

173 ta activity were six times as likely to have electrographic seizures, which were associated with 5.47

174 in the duration and frequency of spontaneous electrographic seizures.

175 ation, as well as spontaneous behavioral and electrographic seizures.

176 etworks have a marked propensity to generate electrographic seizures.

177 n dentate granule cells, which in turn cause electrographic seizures.

178 creased neural activity and have spontaneous electrographic seizures.

179 animals markedly decreases the frequency of electrographic seizures.

180 hemic encephalopathy (HIE) and 147 (84%) had electrographic seizures.

181 s and any lateralized periodic discharges or electrographic seizures.

182 ctroencephalography is widely used to detect electrographic seizures.

183 to soman developed behavioral expression of electrographic seizures.

184 ole) that inhibits convulsive behaviours and electrographic seizures.

185 es, single-trial visual evoked responses and electrographic seizures.

186 aphy (aEEG) has facilitated the detection of electrographic seizures; however, whether these seizures

187 nd encompass a range of techniques including electrographic signal analysis, dynamical systems modeli

188 very of visual function, including increased electrographic signaling and endogenous 11-cis-retinal p

189 ical evaluation of epileptogenicity based on electrographic signatures in intracerebral electroenceph

190 e different patient-specific aetiologies and electrographic signatures, our model suggests that dynam

191 s commonly associated with seizures and with electrographic spike-waves.

192 ociations between electrographic seizures or electrographic status epilepticus and mortality or short

193 etermine whether electrographic seizures and electrographic status epilepticus are associated with hi

194 Electrographic seizures and electrographic status epilepticus are common in critical

195 as electrographic seizures in 41 (20.5%) and electrographic status epilepticus in 43 (21.5%).

196 In multivariable analysis, electrographic status epilepticus was associated with an

197 Electrographic status epilepticus, but not electrographi

198 seizures, 2) electrographic seizures, or 3) electrographic status epilepticus.

199 A pilot electrographic study using subcutaneous electrodes revea

200 Early treatment, rather than electrographic suppression of SE, correlated with preven

201 e many noninvasive methods and in particular electrographic techniques, have been developed to reduce

202 The currently used electrographic terminology, including J-point elevation,

203 y Hospital) database containing clinical and electrographic variables on 5427 continuous EEG sessions

204 Similarities to patients included electrographic waveform patterns at seizure onset, varia

205 ure rate and relative severity (clinical and electrographic) were performed, and differences were ass