ライフサイエンスコーパス: electrolyte abnormality

1 , cardiac ischemia, catecholamine excess, or electrolyte abnormality.

2 kidney disease, end-stage renal disease, and electrolyte abnormalities.

3 se featuring arterial hypotension along with electrolyte abnormalities.

4 ng the risks of worsening renal function and electrolyte abnormalities.

5 al intravascular volume depletion and marked electrolyte abnormalities.

6 n, rasburicase, and management of associated electrolyte abnormalities.

7 care unit stay, and inotrope use; and fewer electrolyte abnormalities.

8 ctions, portal vein thrombosis, or fluid and electrolyte abnormalities.

9 es included nausea (11%), dehydration (11%), electrolyte abnormality (19%), thrombocytopenia (15%), e

10 Serious adverse events of hypotension, electrolyte abnormalities, acute kidney injury or failur

11 ical assessment, cerebral oxygen saturation, electrolyte abnormalities, adverse events, survival, and

12 dney injury (aHR 1.44, 95% CI 1.41 to 1.47), electrolyte abnormalities (aHR 1.45, 95% CI 1.43 to 1.48

13 ts with hypotension, renal insufficiency, or electrolyte abnormalities, albumin should also be consid

14 tral obesity, hyperglycaemia, dyslipidaemia, electrolyte abnormalities and hypertension.

15 , although patients need to be monitored for electrolyte abnormalities and late toxicities.

16 to RTECs can cause systemic fluid imbalance, electrolyte abnormalities and metabolic waste accumulati

17 Electrolyte abnormalities and other adverse events did n

18 al ventilation and intensive care unit stay, electrolyte abnormalities and repletion doses, duration

19 Treatment directed at correction of electrolyte abnormalities and the underlying cause for t

20 y therapy for congestion, is associated with electrolyte abnormalities and worsening renal function.

21 ious adverse events of hypotension, syncope, electrolyte abnormalities, and acute kidney injury or fa

22 h significantly greater risk of AKI, serious electrolyte abnormalities, and ambulatory hyperkalemia.

23 y cardiac disease, drug-positive urine test, electrolyte abnormalities, and changes in their antiretr

24 tients with abnormal cardiac substrate, with electrolyte abnormalities, and during drug initiation.

25 AEs (hypotension, syncope, bradycardia, AKI, electrolyte abnormalities, and injurious falls).

26 c suction, intravenous fluids, correction of electrolyte abnormalities, and observation.

27 ravascular volume depletion, shock, profound electrolyte abnormalities, and organ dysfunction.

28 on, syncope, fractures, acute kidney injury, electrolyte abnormalities, and primary care attendance w

29 Electrolyte abnormalities are prevalent in patients with

30 Fluid and electrolyte abnormalities are very common in patients wi

31 Specific electrolyte abnormalities associated with a higher risk

32 Both recovered full renal function with no electrolyte abnormalities at the time of discharge.

33 o weight changes, eating disorders may cause electrolyte abnormalities, bradycardia, disturbances in

34 erious adverse events (hypotension, syncope, electrolyte abnormalities, bradycardia, or acute kidney

35 d heart failure (ADHF) can be complicated by electrolyte abnormalities, but the major focus has been

36 Adverse effects include electrolyte abnormalities, cardiac failure, bleeding dia

37 were associated with a preceding outpatient electrolyte abnormality compared with matched controls.

38 es improve along with graft villi formation, electrolyte abnormalities continue, to which FK 506-medi

39 prespecified safety outcome was incidence of electrolyte abnormalities; cost efficacy was defined as

40 Any electrolyte abnormality, defined as abnormal test result

41 rs may be associated with changes in weight, electrolyte abnormalities (eg, hyponatremia, hypokalemia

42 Hyperkalemia is the most frequent electrolyte abnormality found in whole organ transplant

43 ications of hypothermia including shivering, electrolyte abnormalities, hemodynamic changes, arrhythm

44 1.23 [95% CI, 0.76-2.00]), 4.0% vs 2.7% for electrolyte abnormalities (HR, 1.51 [95% CI, 0.99-2.33])

45 Furthermore, clinicians should be aware of electrolyte abnormalities in patients with mitochondrial

46 n College of Cardiology Practice Guidelines, electrolyte abnormalities, including abnormal serum pota

47 isted primarily of reversible hematologic or electrolyte abnormalities, including neutropenic fever i

48 included hypotension, syncope, bradycardia, electrolyte abnormalities, injurious falls, and acute ki

49 tions of eating disorders frequently involve electrolyte abnormalities, it remains unknown whether el

50 te abnormalities, it remains unknown whether electrolyte abnormalities may precede the future diagnos

51 Otherwise unexplained electrolyte abnormalities may serve to identify individu

52 (n = 9 [7.5%]), orthostasis (n = 7 [5.8%]), electrolyte abnormalities (n = 6 [5.0%]), and falls (n =

53 d intravenous fluid hydration, correction of electrolyte abnormalities, nutritional support, and crit

54 pendent predictors of early readmission were electrolyte abnormalities on the day of discharge (odds

55 without structural heart disease, metabolic/electrolyte abnormalities, or the long QT syndrome.

56 t was associated with a greater incidence of electrolyte abnormalities, particularly hypokalemia.

57 The average number of different electrolyte abnormalities per patient ranged from 2.4 to

58 ent, major trauma, hypotension, metabolic or electrolyte abnormalities, renal insufficiency, sepsis,

59 IQR, 4-8] vs 4.0 [IQR, 3-6] days) and higher electrolyte abnormality scores (median, 6 [IQR, 4-7] vs

60 autosomal recessive disease that results in electrolyte abnormalities shortly after birth.

61 s/lethargy, age, ataxia, abdominal pain, and electrolyte abnormalities, significantly influenced indi

62 Hyperkalemia is a common electrolyte abnormality that may be difficult to manage

63 ariants in three families with Gitelman-like electrolyte abnormalities, then investigated 156 familie

64 The median (IQR) time from the earliest electrolyte abnormality to eating disorder diagnosis was

65 Pendrin-knockout mice show no fluid-electrolyte abnormalities under baseline conditions, alt

66 uals with an eating disorder had a preceding electrolyte abnormality vs 7.5% of individuals without a

67 portional hazards regression analysis, serum electrolyte abnormality was an independent predictor of

68 e frequently on the cyclophosphamide arm and electrolyte abnormalities were more common on the CCNU r

69 ll participants improved hypoproteinemia and electrolyte abnormalities with AD treatment alone, witho