ライフサイエンスコーパス: electroretinography

1 provement of retinal function (assessed with electroretinography).

2 tive immunohistochemistry) and functionally (electroretinography).

3 imaging, and visual function was assessed by electroretinography.

4 omography, kinetic visual field testing, and electroretinography.

5 erence tomography, and multifocal or pattern electroretinography.

6 ed peripheral retinal function by multifocal electroretinography.

7 n of Akita and control mice was evaluated by electroretinography.

8 kinetic and static perimetry, and full-field electroretinography.

9 lted in functional deficits as determined by electroretinography.

10 fundus ophthalmoscopy, dark adaptometry, and electroretinography.

11 r degeneration was measured by histology and electroretinography.

12 cone survival was assessed by histology and electroretinography.

13 Retinal function was assayed by electroretinography.

14 escued photoreceptor function as measured by electroretinography.

15 inal histology up to 24 months of age and by electroretinography.

16 ull-field electroretinography and multifocal electroretinography.

17 T) or DKO mice, as assessed by histology and electroretinography.

18 ctrometry, and degeneration by histology and electroretinography.

19 ese-enhanced magnetic resonance imaging, and electroretinography.

20 ncluding ophthalmoscopy and full-field flash electroretinography.

21 nohistochemistry, Western blot analysis, and electroretinography.

22 l function was characterized with the use of electroretinography.

23 t hypomorphic and Ret conditional mice using electroretinography.

24 ) measures of central retinal thickness, and electroretinography.

25 isual function in these mice was analyzed by electroretinography.

26 reserves RGC activity as measured by pattern electroretinography.

27 d all had absent rod responses in full field electroretinography.

28 Neural function was assessed with electroretinography.

29 es of mRDH11 disruption were investigated by electroretinography.

30 munohistochemistry, electron microscopy, and electroretinography.

31 othelial cell death, and retinal function by electroretinography.

32 multiple components of retinal function with electroretinography.

33 static contrast sensitivity test; and global electroretinography.

34 Rescue of visual function was confirmed by electroretinography.

35 aphy, mesopic microperimetry, and multifocal electroretinography.

36 nction of USH2A patients was quantified with electroretinography.

37 t time of the N95 wave on results of pattern electroretinography.

38 optical coherence tomography angiography and electroretinography.

39 glaucoma as well as cone-rod dysfunction on electroretinography.

40 icity was evaluated by clinical findings and electroretinography: 30-Hz flicker responses were compar

41 ticularly in areas with decreased multifocal electroretinography amplitude.

42 BEST1 mutations, imaging findings, electroretinography amplitudes, and implicit times.

43 el reduced visual acuity thresholds, reduced electroretinography amplitudes, and thinned the outer pl

44 ptor outer nuclear layer (ONL) thickness and electroretinography amplitudes.

45 ty, biomicroscopy, color fundus photography, electroretinography analysis, and visual-evoked potentia

46 Multifocal electroretinography and chromatic/achromatic contrast se

47 on retinal function and neurodegeneration by electroretinography and detailed morphology.

48 static perimetry, full-field and multifocal electroretinography and electro-oculography.

49 3 expression, mice were analyzed by scotopic electroretinography and fluorescein angiography.

50 Electroretinography and gene expression analyses suggest

51 Full-field electroretinography and histologic examination were used

52 rvival time, visual acuity decline rate, and electroretinography and imaging findings.

53 er retinal function as measured, in vivo, by electroretinography and manganese-enhanced MRI.

54 mprovement observed in this case, multifocal electroretinography and microperimetry indicate that sub

55 and cone function was analyzed by full-field electroretinography and multifocal electroretinography.

56 Further investigations included electroretinography and neuroradiologic imaging.

57 Multifocal electroretinography and ocular coherence tomography are

58 We showed using electroretinography and single cell recordings that the

59 tween photoreceptors and ON-BC neurons using electroretinography and single cell recordings.

60 g rhodopsin signaling normally, as judged by electroretinography and single-cell recording.

61 rked reductions in exocytosis as measured by electroretinography and single-cell recordings.

62 mice were examined by scotopic and photopic electroretinography and then killed for biochemical and

63 phototransduction components was assessed by electroretinography and to couple to vertebrate transduc

64 In vivo electroretinography and transretinal recordings revealed

65 To delineate mechanisms, we used electroretinography and visual evoked potential recordin

66 times after infection, eyes were analyzed by electroretinography and were harvested for quantitation

67 used electrophysiological techniques (i.e., electroretinography) and molecular analyses, this work s

68 se of infection by biomicroscopy, histology, electroretinography, and bacterial and inflammatory cell

69 enetrating electrode arrays, multi-electrode electroretinography, and electromyography, are also viab

70 9 months of age using immunohistochemistry, electroretinography, and fluorescein angiography.

71 A), kinetic and static perimetry, full-field electroretinography, and fundus autofluorescence (FAF).

72 VF), dark-adapted absolute thresholds (DAT), electroretinography, and fundus photography.

73 ce imaging, visual field testing, full-field electroretinography, and genetic testing for inherited r

74 ded toxicology (clinical examination, serial electroretinography, and histopathology) in normal rabbi

75 al retinal examination, noninvasive imaging, electroretinography, and histopathology/immunohistochemi

76 , multifocal electroretinography, full-field electroretinography, and microperimetry.

77 ucted multimodal retinal imaging, full-field electroretinography, and molecular genetic analysis of N

78 At P30, retinal function was measured with electroretinography, and morphologic preservation of the

79 Slit lamp examination (SLE), electroretinography, and myeloperoxidase activity were p

80 ields, optical coherence tomography, pattern electroretinography, and neuro-ophthalmic examinations.

81 s were analyzed by biomicroscopy, histology, electroretinography, and quantitation of bacteria and in

82 Retinal function was assessed by electroretinography, and retinal morphological features

83 Retinal function was assessed by electroretinography, and retinal structure by light micr

84 photoreceptor function was assessed through electroretinography, and survival was documented in morp

85 e scanning laser ophthalmoscopy (AF-SLO) and electroretinography, and the extent of laser-induced CNV

86 nical ophthalmic examinations, neuroimaging, electroretinography, and the results of MMACHC mutation

87 -fluorescence, optical coherence tomography, electroretinography, and ultrasound.

88 py, best-corrected visual acuity, full-field electroretinography, and visual field studies.

89 nfocal and electron microscopy, single-flash electroretinography, and whole-cell patch-clamp recordin

90 Visual acuity, retinal features, electroretinography, and whole-exome sequencing.

91 with fundus autofluorescence and multifocal electroretinography as indicated), will greatly minimize

92 Electroretinography assessed functional recovery after i

93 erical equivalent refraction, visual fields, electroretinography B-wave amplitudes, and qualitative i

94 By electroretinography, b-wave amplitude was reduced by 75%

95 Eyes were analyzed by electroretinography, bacterial quantitation, and antibio

96 in implicit time (IT) assessed by multifocal electroretinography between baseline and at the end of f

97 Patients were also assessed by electroretinography, brain MRI and magnetic resonance sp

98 Electroretinography can be usually be obtained in infant

99 mination, visual acuity (VA), visual fields, electroretinography, color vision testing, and retinal i

100 r initial rate of loss of visual function by electroretinography, compared with eyes without these st

101 etinography findings; quantification of main electroretinography components in all other patients rev

102 The main electroretinography components showed evidence of age-re

103 patients (aged 10-62 years) with full-field electroretinography-confirmed achromatopsia.

104 , and reduced scotopic responses observed on electroretinography consistent with the CRD phenotype of

105 Full-field electroretinography demonstrated a reduced rod response

106 Multifocal electroretinography demonstrated normal amplitude and im

107 Electroretinography demonstrated that cone responses wer

108 also remain functional, albeit with reduced electroretinography (ERG) amplitudes typical of RetGC1-/

109 To determine toxicity, electroretinography (ERG) amplitudes were measured in re

110 tinal tissue as indicated by TUNEL assay and electroretinography (ERG) analysis.

111 Electroretinography (ERG) and both chemical and histolog

112 Functional retinal changes were evaluated by electroretinography (ERG) and by morphologic and ultrast

113 th noninfectious uveitis by using full-field electroretinography (ERG) and correlate the ERG to disea

114 rimetry, fluorescein angiography, full-field electroretinography (ERG) and electro-oculography (EOG),

115 ing, retinal function assessed by full-field electroretinography (ERG) and fundus-controlled perimetr

116 Electroretinography (ERG) and intraocular pressure (IOP)

117 Electroretinography (ERG) and light microscopy were used

118 ete ophthalmic examination, full-field flash electroretinography (ERG) and multifocal ERG, light-adap

119 gating their visual capabilities by means of electroretinography (ERG) and patterned visual evoked po

120 Baseline electroretinography (ERG) and preretinal oxygen (Po(2))

121 Function was assessed with perimetry and electroretinography (ERG) and retinal structure with opt

122 Full-field electroretinography (ERG) and spectral-domain OCT were p

123 Spectral electroretinography (ERG) demonstrated significant impro

124 ship among vector dose, visual function, and electroretinography (ERG) findings.

125 The mice were analyzed by full-field electroretinography (ERG) for light response.

126 microvilli area and an abnormal response on electroretinography (ERG) in UBE3D(+/-) heterozygous mic

127 chanism of confocal IOS, comparative IOS and electroretinography (ERG) measurements were made using n

128 icity was evaluated by clinical findings and electroretinography (ERG) on 244 evaluable injections in

129 By conducting electroretinography (ERG) recordings in live ndufs4 (-/-

130 munoblots, and cone function was analyzed by electroretinography (ERG) recordings.

131 resulted in attenuated scotopic and photopic electroretinography (ERG) responses in mice at 3 months

132 Scotopic electroretinography (ERG) showed a diminished c-wave amp

133 At 7 days after exposure to light electroretinography (ERG) showed that minocycline signif

134 However, electroretinography (ERG) studies in humans and rodents

135 We used electroretinography (ERG) to evaluate retinal function a

136 cy of optical coherence tomography (OCT) and electroretinography (ERG) to monitor pathological and fu

137 Electroretinography (ERG) was performed before surgery a

138 Among mice followed for 8 months, electroretinography (ERG) was performed on both eyes bef

139 Electroretinography (ERG) was performed to evaluate reti

140 ter 10 min systemic hyperoxia; and on day 3, electroretinography (ERG) was performed.

141 Electroretinography (ERG) was used to assess rod and con

142 Strobe flash electroretinography (ERG) was used to examine outer reti

143 Computerized pupillometry and electroretinography (ERG) were performed to assess optic

144 transmission electron microscopy (TEM), and electroretinography (ERG) were used to analyze 6 genotyp

145 g including fundus autofluorescence and OCT, electroretinography (ERG), and both microscopy and molec

146 testing, dark adaptation testing, full-field electroretinography (ERG), and electro-oculography (EOG)

147 handheld tonometer, indirect ophthalmoscopy, electroretinography (ERG), and histology.

148 of bacteria, organ function as determined by electroretinography (ERG), and histopathologic changes w

149 on were documented using fundus photography, electroretinography (ERG), and histopathology.

150 ry, fundus-guided microperimetry, full-field electroretinography (ERG), and multifocal ERG.

151 ry, fundus-guided microperimetry, full-field electroretinography (ERG), and multifocal ERG.

152 Functional abnormalities were assessed by electroretinography (ERG), and neurodegeneration was ass

153 netic perimetry, chromatic static perimetry, electroretinography (ERG), and optical coherence tomogra

154 Retinal function was measured with electroretinography (ERG), and relative content of selec

155 sponses to light were measured by full-field electroretinography (ERG), and retinal tissues were exam

156 Visual acuity (VA), electroretinography (ERG), and spectral-domain optical c

157 nd electron microscopy, immunocytochemistry, electroretinography (ERG), and spectrophotometry.

158 tical coherence tomography (OCT), full-field electroretinography (ERG), electro-oculography (EOG), an

159 for 8 wk and evaluated by AGE fluorescence, electroretinography (ERG), electron microscopy, and micr

160 -LCA (n = 30; ages, 4-55) were studied using electroretinography (ERG), full-field stimulus testing (

161 d reduced retinal thickness were found using electroretinography (ERG), fundus photography (FP), fund

162 Clinical examinations, electroretinography (ERG), histology, and bacterial quan

163 Electroretinography (ERG), histology, light microscopy,

164 ogy, transmission electron microscopy (TEM), electroretinography (ERG), immunohistochemistry, Western

165 rn electroretinography (PERG) and full-field electroretinography (ERG), incorporating international s

166 -) and wild-type (WT) mice were evaluated by electroretinography (ERG), lectin cytochemistry, and cor

167 ctural, and biochemical assessments included electroretinography (ERG), light and electron microscopi

168 age-matched control puppies were studied by electroretinography (ERG), light and electron microscopy

169 of the gene targeted mice were evaluated by electroretinography (ERG), light, and electron microscop

170 almic examinations were performed, including electroretinography (ERG), multifocal ERG (mfERG), perim

171 s autofluorescence (FAF) imaging, full-field electroretinography (ERG), multifocal ERG, and central v

172 ional examinations were performed, including electroretinography (ERG), optical coherence tomography

173 n were determined at multiple time points by electroretinography (ERG), optical coherence tomography

174 -induced retinal degeneration using scotopic electroretinography (ERG), optical coherence tomography

175 Phenotype was assessed with electroretinography (ERG), optical coherence tomography,

176 almic examinations were performed, including electroretinography (ERG), perimetry, optical coherence

177 (NADH/FAD) state by 3D optical cryo-imaging, electroretinography (ERG), spectral-domain optical coher

178 resonance imaging, full-field and multifocal electroretinography (ERG), visual evoked potentials (VEP

179 AF), optical coherence tomography (OCT), and electroretinography (ERG).

180 oldmann visual fields (GVFs), and full-field electroretinography (ERG).

181 s measured by optokinetic tracking (OKT) and electroretinography (ERG).

182 ically, and retinal function was analysed by electroretinography (ERG).

183 of CSNB2 and RP were confirmed by full-field electroretinography (ERG).

184 tudied with optical coherence tomography and electroretinography (ERG).

185 of a and b wave of both were appreciated in electroretinography (ERG).

186 Cone function was determined by electroretinography (ERG).

187 A), visual field sensitivity, and full-field electroretinography (ERG).

188 utant Ush1c, were analyzed by microscopy and electroretinography (ERG).

189 ve impact on visual function, as assessed by electroretinography (ERG).

190 er retinal layers as well as functionally by electroretinography (ERG).

191 e analyzed by optokinetic response (OKR) and electroretinography (ERG).

192 electron microscopy, immunofluorescence, and electroretinography (ERG).

193 ron microscopy, and single-flash and flicker electroretinography (ERG).

194 c perimetry, static chromatic perimetry, and electroretinography (ERG).

195 nd cone function was impaired as assessed by electroretinography (ERG).

196 d the retinal function of these mice through electroretinography (ERG).

197 inal function was assessed by three forms of electroretinography (ERG): slow-sequence multifocal (mf)

198 peak time variability but with pathognomonic electroretinography features.

199 ed microperimetry, full-field and multifocal electroretinography (ffERG and mfERG), spectral-domain o

200 retinal function was studied with full-field electroretinography (ffERG) from 3 months through 2 year

201 hthalmoscopy, fundus photography, full-field electroretinography (ffERG), Goldmann visual fields (VFs

202 almoscopy, Goldmann visual field, full-field electroretinography (ffERG), OCT, and FAF photography.

203 oherence tomography (SD-OCT), and full-field electroretinography (ffERG).

204 E3 gene (n = 38) or by diagnostic full-field electroretinography findings (n = 18).

205 Most electroretinography findings have pathognomonic features

206 Electroretinography findings support the more severe cli

207 Global electroretinography findings were normal.

208 ed on visual acuity, fundus photography, and electroretinography findings.

209 One patient showed undetectable electroretinography findings; quantification of main ele

210 t underwent a complete clinical examination, electroretinography (full field and pattern), visual evo

211 l-domain OCT and OCT angiography, multifocal electroretinography, full-field electroretinography, and

212 dus appearance, visual field, and full-field electroretinography, fundus autofluorescence, and optica

213 phthalmic examinations, including full-field electroretinography, fundus photography, fundus autofluo

214 Multifocal electroretinography had the greatest proportion of posit

215 went complete ocular examination, full-field electroretinography, handheld spectral-domain optical co

216 of endophthalmitis was graded by slit lamp, electroretinography, histological examinations, and dete

217 infection, the EBE incidence was assessed by electroretinography, histology, bacterial counts, and my

218 d a phototransduction deficit as assessed by electroretinography; however, their photoreceptor struct

219 Histology, electroretinography, immunohistochemistry, Western blot

220 dysfunction was provided through multi-focal electroretinography in a subset of such patients.

221 nd maintained visual function as assessed by electroretinography in C57BL/6 mice.

222 unostaining, and measured light responses by electroretinography in mice with targeted disruptions of

223 The role of electroretinography in pediatric practice, and to offer

224 nction because DMOG normalizes the b-wave on electroretinography in wild-type mice.

225 l loss occurred in adult Mfsd2a KO mice, but electroretinography indicated visual function was normal

226 RKL mutations were studied clinically and by electroretinography, kinetic, and chromatic static perim

227 nifest retinal degeneration, as evidenced by electroretinography, light microscopy and pupillometry r

228 trauma and assessed by clinical examination, electroretinography, light microscopy, electron microsco

229 Multifocal electroretinography may be effective in detecting functi

230 the N95 implicit time on results of pattern electroretinography (median, 98.6 milliseconds [95% CI,

231 With appropriate technique, electroretinography methods could be made more widely av

232 coherence tomography (SD-OCT) and multifocal electroretinography (mfERG) along with visual fields.

233 Functional studies by multifocal electroretinography (mfERG) evaluated neurodysfunction,

234 l coherence tomography (OCT), and multifocal electroretinography (mfERG) were performed at baseline a

235 oherence tomography (SD-OCT), and multifocal electroretinography (mfERG) were performed at various in

236 ests consisting of visual fields, multifocal electroretinography (mfERG), and contrast sensitivity we

237 gnosis were followed up including multifocal electroretinography (mfERG), spectral-domain optical coh

238 llary RNFLT, RNFL retardance, and multifocal electroretinography (mfERG).

239 fundus autofluorescence (FAF), or multifocal electroretinography (mfERG).

240 y visual field (HVF) testing, and multifocal electroretinography (mfERG).

241 nt routine examination, including full-field electroretinography, microperimetry, and optical coheren

242 ons included electro-oculography, full-field electroretinography, multifocal electroretinography, spe

243 vision (ie, dark adaptometry, pupillometry, electroretinography, nystagmus, and ambulatory behaviour

244 In vivo electroretinography of Panx1 knockout mice indicated an

245 P measurements were collected daily, whereas electroretinography, optical coherence tomography, and w

246 hy, fundus autofluorescence imaging, sedated electroretinography, optical coherence tomography, genet

247 angiography evidence of active choroiditis, electroretinography parameters indicative of stable or w

248 Pattern electroretinography (PERG) and full-field electroretinog

249 analyses included transient pattern-reversal electroretinography (PERG) and full-field flash ERG, wit

250 Patients underwent pattern electroretinography (PERG), optical coherence tomography

251 One month later, pattern electroretinography (PERG), rate of ATP synthesis, gene

252 Electroretinography phenotype (cone-rod vs rod-cone dysf

253 Objective functional tests, such as electroretinography, provide an alternative to subjectiv

254 ice, and to offer suggestions for successful electroretinography recordings in children.

255 ischemia by histologic analyses and scotopic electroretinography, respectively.

256 d b-wave amplitudes of scotopic and photopic electroretinography responses 4 months after diabetes in

257 The mean change in electroretinography responses was not significantly diff

258 mice, their scotopic, maximal, and photopic electroretinography responses were comparable to those o

259 Full-field electroretinography responses were extinguished in 50% o

260 rescued a- and b-wave amplitudes of scotopic electroretinography responses, compared with vehicle-tre

261 The patients showed normal electroretinography responses, no signs of albinism, and

262 l coherence tomography, and severely reduced electroretinography responses.

263 clinical symptom of night blindness and the electroretinography results suggest a primary rod dysfun

264 (P = 0.02) than those with normal full-field electroretinography results.

265 nction as assessed by VA, visual fields, and electroretinography results; and retinal structural chan

266 omain optical coherence tomography (SD-OCT), electroretinography, retinal morphology, and visual reti

267 -/-) and Oa1(-/-) mice had normal results on electroretinography, retrograde labeling showed a signif

268 Electroretinography revealed a cone-rod pattern of dysfu

269 Electroretinography revealed a rod-cone pattern of dysfu

270 However, twin flash electroretinography revealed a slight delay in recovery

271 Electroretinography revealed severely decreased or non-r

272 Retinal function was evaluated using electroretinography (scotopic, photopic, and pattern).

273 Ganzfeld electroretinography showed faster recovery of retinal fu

274 ailed neuro-ophthalmic examination including electroretinography showed him to have a typical retinal

275 Electroretinography showed mutant mouse eyes had a selec

276 Histology and electroretinography showed no cre-mediated RPE toxicity.

277 ness in chronic autoimmune uveitis mice, and electroretinography showed significantly reduced amplitu

278 Electroretinography showed that scotopic and photopic re

279 Rod function (measured by electroretinography) showed modest but statistically sig

280 , full-field electroretinography, multifocal electroretinography, spectral-domain optical coherence t

281 proportion to the clinical exams, prompting electroretinography testing that revealed an electronega

282 arent to clinical examination and full-field electroretinography testing.

283 emonstrate, using OCT, light microscopy, and electroretinography, that two Sfxn3 (-/-) mouse lines de

284 Here, we used electroretinography to examine the functional role of BK

285 artificial insemination, RNA-sequencing and electroretinography to show that seminal fluid induces a

286 y screens (such as histological analysis and electroretinography) to identify the subset of fish with

287 ography, fundus autofluorescence, multifocal electroretinography, visual fields) and classification o

288 h retinal function tested with OptoMotry and electroretinography was comparable to adult (8-week-old)

289 The high temporal frequency electroretinography was determined mainly by the luminan

290 Simultaneous bilateral dark-adapted electroretinography was performed 2 weeks and 12 weeks a

291 Full-field electroretinography was performed binocularly, using DTL

292 After exposure, electroretinography was performed on mice dark adapted f

293 was performed to assess for cell death, and electroretinography was performed to assess function.

294 Multifocal electroretinography was shown to have a high sensitivity

295 n levels of several phototransduction genes; electroretinography was used to assess quantitatively th

296 -retinylidene-N-retinylethanolamine content; electroretinography was used to measure phototransductio

297 Fundus photography, and electroretinography were performed in 12 patients, and o

298 ure (IOP) tonometry, fundus photography, and electroretinography were performed over multiple time po

299 Microscopy and electroretinography were used to characterize transgenic

300 us photography, fluorescein angiography, and electroretinography were used to evaluate retinal anatom