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1 okinetic profile, with a fast onset and slow elimination phase.
2  malaria elimination programs are in the pre-elimination phase.
3 nce in southern Thailand, an area in the pre-elimination phase.
4 sh compartments remained constant during the elimination phase.
5  lung but is redundant in the early adaptive elimination phase.
6 rt-up phase for elimination (1987-1994); the elimination phase (1995-2002); and the postelimination p
7     The presence of a second peak during the elimination phase as well as a high concentration of NSC
8 e elimination of thiacloprid, independent of elimination phase duration, exposure concentrations, and
9 ion half-life, 1 hour) and a relatively slow elimination phase (elimination half-life, 27 hours).
10 pattern was observed with an initially quick elimination phase following slower clearance afterward.
11 ed to a greater than 30-fold increase in the elimination phase half-life for these particles.
12 mice for an extended period of time, with an elimination phase half-life of almost 2 d.
13 hat evolved in the Americas during 3 measles elimination phases (ie, preelimination, elimination, and
14 ment failure at the end of the triamcinolone elimination phase in the salmeterol-minus group was 4.3
15 n earthworms were detected at the end of the elimination phase, indicating that not all zwitterionic/
16 loid, T, and NK cells are key players in the elimination phase of cancer immunoediting, also referred
17  roles either in MCA tumorigenesis or in the elimination phase of cancer immunoediting, did not play
18 in contrast to the hypothesis that an early "elimination phase" of cancer immunosurveillance is event
19 -bearing mice compared to control, while the elimination-phase (slow-phase) half-life remains compara
20 2)alpha], 0.5 hours) followed by a prolonged elimination phase (t(1/2)beta, 30 hours).
21    However, because of a very slow secondary elimination phase, the FAEEs were found to persist in th
22                               The red marrow elimination phase was statistically slower in patients w
23 ning to the visceral leishmaniasis (VL) post-elimination phase where robust surveillance is critical
24 iciency exponentially increased in the early elimination phase while passive tumor targeting linearly
25  rapid distribution phase followed by a slow elimination phase with a t1/2gamma of approximately 130