ライフサイエンスコーパス: emboli

1 ds individual platelets or larger fragments (emboli).

2 ons (deep venous thrombosis and/or pulmonary emboli).

3 [47.6%] with no evidence of acute pulmonary emboli).

4 Four patients had pulmonary emboli.

5 red with only 6.0 for those without cerebral emboli.

6 ompared with 17.9 for those without cerebral emboli.

7 patients (15%) were found to have pulmonary emboli.

8 constituents of venous thrombi and pulmonary emboli.

9 ctions had highest rates (0.7%) of pulmonary emboli.

10 arterial hypoxemia and risk for paradoxical emboli.

11 nous thromboses, and 9 (0.6%) were pulmonary emboli.

12 by administration of three large autologous emboli.

13 ed the numbers of shed cells, fragments, and emboli.

14 s may have deep vein thrombosis or recurrent emboli.

15 een (4.0%) of the 403 patients had pulmonary emboli.

16 eak off and are carried in the blood flow as emboli.

17 al pulmonary arteries and detection of small emboli.

18 al pulmonary arteries and detection of small emboli.

19 cially in instances of isolated subsegmental emboli.

20 excluded the outflow graft as the source of emboli.

21 ging have enabled visualization of pulmonary emboli.

22 mboli and lower sensitivity for subsegmental emboli.

23 and displayed rapid resolution of pulmonary emboli.

24 ibasilar lung nodules consistent with septic emboli.

25 g the risk of stroke associated with retinal emboli.

26 ct, glaucoma, and even retinal and choroidal emboli.

27 tumorigenic and did not form lymphovascular emboli.

28 lation in patients with presumed paradoxical emboli.

29 the rabbits were radiographed to locate the emboli.

30 utic modality in the prevention of pulmonary emboli.

31 ur deep venous thrombosis, and two pulmonary emboli.

32 tic events, including 4 with fatal pulmonary emboli.

33 eral emboli, and four (14%) had only central emboli.

34 Six patients (1%) developed pulmonary emboli.

35 sured in anesthetized ferrets with pulmonary emboli.

36 ties were seen in all animals with pulmonary emboli.

37 e were highly suggestive of septic pulmonary emboli.

38 n perfusion defects are generally not due to emboli.

39 xtremity thrombosis, and recurrent pulmonary emboli.

40 e, transient ischemic attack, and peripheral emboli.

41 ent with the diagnosis of multiple pulmonary emboli.

42 osis developed with two sustaining pulmonary emboli.

43 ude strokes, pulmonary emboli and peripheral emboli.

44 ol the ventricular rate and prevent systemic emboli.

45 inal artery occlusion and multiple choroidal emboli.

46 ults from incomplete resolution of pulmonary emboli.

47 ggested as a cause for asymptomatic cerebral emboli.

48 o and in vivo with the formation of platelet emboli.

49 counting confirmed the location of the lung emboli.

50 onfirm in vivo the location of the pulmonary emboli.

51 ged mediastinal lymph nodes but no pulmonary emboli.

52 ossible late relapses mainly due to cerebral emboli.

53 CT angiogram shows no evidence of pulmonary emboli.

54 by 2050, along with the numbers of systemic emboli.

55 ght the importance of subsegmental pulmonary emboli.

56 ntestinal toxicities, and nonfatal pulmonary emboli.

57 of 8), hypoxemia (2 of 8), septic pulmonary emboli (1 of 8), septic shock (2 of 8), neutrophilia (5

58 patients, with 112 ischemic strokes/systemic emboli (1.3%) and 323 major bleeds (3.8%) recorded.

59 (20.6%); 7 patients with nonfatal pulmonary emboli (20.6%); and 9 patients (26.5%) who died, possibl

60 t skin manifestations, particularly cerebral emboli (32.8% vs 18.4%, P = .01), without increased mort

61 1 [IQR: 0 to 1]; p = 0.08), total volume of emboli (45 [IQR: 0 to 175] mm(3) vs. 33 [IQR: 0 to 133]

62 ions were associated with more extracerebral emboli (75.0% vs 31.8%, P = .02).

63 eye movements returned normal and choroidal emboli absorbed almost completely.

64 luate the incidence of asymptomatic cerebral emboli (ACE) during ablation of atrial fibrillation (AF)

65 ded stroke, myocardial infarction, pulmonary emboli, adult respiratory distress syndrome, and repeat

66 BP8 PET to detect source thrombi and culprit emboli after deep vein thrombosis and pulmonary embolism

67 inately PVC ablations) experienced new brain emboli after the procedure.

68 table adhesion and/or survival of metastatic emboli after tumor cell intravasation.

69 ce, elevated FVIII stabilized thrombi (fewer emboli) after short injury, but it had no effect after l

70 (2)AP(13-24) peptide into existing pulmonary emboli also declined significantly over time.

71 No pulmonary emboli, alterations in oxygen saturation, or hemodynamic

72 All 5 lobar emboli and 16 of 17 segmental emboli identified on stand

73 Section thickness was 1.25 mm for pulmonary emboli and 5 mm for deep venous thrombosis.

74 patients who presented with septic pulmonary emboli and community-acquired methicillin-resistant Stap

75 Fibrin emboli and focal hypoperfusion may explain the developme

76 Patients presenting with systemic emboli and found to have mobile aortic atheroma on trans

77 e most common causes of death were pulmonary emboli and infectious bronchopneumonia.

78 His condition was complicated by pulmonary emboli and lobar infarction, all contributing to rapid d

79 ted high sensitivity for lobar and segmental emboli and lower sensitivity for subsegmental emboli.

80 on frequencies of ischemic strokes/systemic emboli and major bleeds in atrial fibrillation patients.

81 oiety may improve the detection of pulmonary emboli and other clinically important fixed intravascula

82 mboembolic events include strokes, pulmonary emboli and peripheral emboli.

83 Anti-CR1/tPA accelerated lysis of pulmonary emboli and prevented stable occlusive carotid arterial t

84 The relationship between cerebral emboli and progression of dementia was analyzed using lo

85 ors and stroke, associations between retinal emboli and renal disease and function remain unclear.

86 lmonary vein ablation, leading to septic air emboli and requiring urgent cardiac surgery.

87 osed the vascular network to the risk of gas emboli and the spread of gas between xylem conduits, thu

88 ve the formation of tumor-platelet-leukocyte emboli and their interactions with the endothelium of di

89 P8 PET imaging detected the location of lung emboli and venous thrombi after DVT-PE, revealing signif

90 (82%) had concomitant central and peripheral emboli, and four (14%) had only central emboli.

91 n lowers the composite of stroke or systemic emboli, and ischemic stroke versus rivaroxaban.

92 were less prone to produce lethal pulmonary emboli, and larger numbers were recovered in heart and k

93 (ie, risk of cardioembolic stroke, systemic emboli, and new atrial fibrillation [AF] during follow-u

94 hniques have been used to identify pulmonary emboli, and no single technique has been shown to be sup

95 lipid composition of lungs positive for fat emboli, and those negative for emboli in Physeter macroc

96 Retinal emboli are associated with the presence of carotid arter

97 periodic shallow-depth activity and why gas emboli are found in animals driven to surface precipitou

98 knowledge, population-based data on retinal emboli are limited in Asia.

99 Bilateral retinal emboli are rare, although multiple emboli in a single ey

100 Arterial air emboli arising from severe pulmonary injury can cause is

101 ers and is supported by the demonstration of emboli as the most common cause of stroke in these patie

102 However, anticoagulation may help prevent emboli, as it does for patients who have thrombi in othe

103 inolytic molecules in experimental pulmonary emboli, assessed the contribution of alpha2-antiplasmin

104 period for indications other than pulmonary emboli assessment were identified.

105 were positive for 1 patient, who had septic emboli associated with Staphylococcus aureus bacteremia.

106 Forty-three patients (21%) had pulmonary emboli at CT.

107 lation is associated with a risk of cerebral emboli attributed to cardioversions and numerous ablatio

108 In 31 patients suspected of pulmonary emboli but without these confounding factors, the five D

109 causing heart attacks, strokes, or pulmonary emboli, but the origin of these mechanical properties is

110 Low-dose r-tPA alone did not dissolve emboli, but was synergistic with alpha2-antiplasmin inac

111 The sensitivity (correct identification of emboli by all three reviewers) of DSA was 92% and of CFA

112 Dissolution of pulmonary emboli by alpha2-antiplasmin inactivation alone was comp

113 lasticity involving the rapid envelopment of emboli by endothelial membrane projections that subseque

114 wed for the presence or absence of pulmonary emboli by three independent reviewers who were unaware o

115 Asymptomatic circulating emboli can be detected by transcranial Doppler ultrasoun

116 Retinal arteriolar emboli can be found in approximately 1% of adults more t

117 The data show that air emboli can be generated from this pump and the rate at w

118 invasion, local dissemination, intravascular emboli, circulating tumor cell clusters, and micrometast

119 Because emboli consist mainly of thrombus, anticoagulants are li

120 All thrombi and emboli contained few biconcave red blood cells but many

121 TTO and emboli correlated with TATs.

122 iption of how the composition of thrombi and emboli depends on their vascular origin and age.

123 ns occurred in the TOF group, with pulmonary emboli despite prophylaxis and an unrecognized cerebrova

124 post-palpation release of circulating tumor emboli detected 2-3 min after palpation (P<0.01) by huma

125 The number of emboli detected by transcranial Doppler within 3 hours o

126 stenting with a device to capture and remove emboli ("embolic protection") is an effective alternativ

127 Cerebral emboli entering the middle cerebral arteries were counte

128 evastating complication of ophthalmic artery emboli following Calcium Hydroxylapatite filler injectio

129 d for the formation of thrombi and pulmonary emboli following intravascular injection of collagen.

130 a dramatic increase in metastatic pulmonary emboli following palpation of the primary tumor.

131 ral killer (NK) cell clearance of tumor cell emboli following surgery is thought to be vital in preve

132 Carotid Artery Implant for Trapping Upstream Emboli for Preventing Stroke in Atrial Fibrillation Pati

133 Carotid Artery Implant for Trapping Upstream Emboli for Preventing Stroke in Atrial Fibrillation Pati

134 Spheroidgenesis and lymphovascular emboli formation are the direct result of calpain-mediat

135 s, whereas a relatively even distribution of emboli formation was seen during cryoballoon ablations.

136 than in wild-type mice despite a far greater emboli formation.

137 ftment, cell dispersal to ectopic sites, and emboli formation.

138 t PECAM-1 null mice; and (4) lysed pulmonary emboli formed subsequently more effectively than lmw-scu

139 all AF-related incident strokes and systemic emboli from 2002 to 2012 in the Oxford Vascular Study (O

140 all AF-related incident strokes and systemic emboli from 2002 to 2012 in the Oxford Vascular Study.

141 While tumor emboli from invasive primary cardiac lymphomas have been

142 sed risk of stroke due almost exclusively to emboli from left atrial appendage (LAA) thrombi.

143 procedures of our model, including preparing emboli from rat donors.

144 otid arteries (CCAs) was designed to capture emboli >1.4 mm in diameter.

145 However, 48-h-old pulmonary emboli had lysed and were seen neither by radiography no

146 ic link between aortic atheroma and systemic emboli has been described both in pathologic and transes

147 These emboli have a unique microscopic appearance of compact c

148 iphius cavirostris (two species in which fat emboli have been described).

149 rding accurate diagnosis of small peripheral emboli have so far prevented unanimous acceptance of CT

150 of all-cause mortality, stroke and systemic emboli, heart failure admission, cardiac transplantation

151 All 5 lobar emboli and 16 of 17 segmental emboli identified on standard angiograms were also ident

152 e prevalence of and risk factors for retinal emboli in a large, contemporary, multiethnic Asian popul

153 l retinal emboli are rare, although multiple emboli in a single eye may be seen in up to one third of

154 or fluorescein isothiocyanate-fibrin labeled emboli in anesthetized mice.

155 dependent reviewers who recorded presence of emboli in categorized pulmonary artery anatomic territor

156 t MUC1 in the passive dissemination of tumor emboli in IBC.

157 Available data suggest that retinal emboli in otherwise asymptomatic people are associated w

158 identify potential cardiovascular sources of emboli in patients who have had strokes.

159 n) for the prevention of stroke and systemic emboli in patients with atrial fibrillation.

160 opathy, verrucous endocarditis, and arterial emboli in patients with cancer, often occurring with muc

161 llation (AF) is a frequent source of cardiac emboli in patients with ischemic stroke.

162 role to reduce the incidence of cardiogenic emboli in patients with mechanical cardiac valves or non

163 rin anticoagulation on the risk of recurrent emboli in patients with mobile aortic atheroma.

164 gy to prevent recurrent presumed paradoxical emboli in patients with patent foramen ovale is unknown.

165 itive for fat emboli, and those negative for emboli in Physeter macrocephalus and Ziphius cavirostris

166 FO is an alternative therapy for paradoxical emboli in selected patients.

167 ARY-X, which manifests florid lymphovascular emboli in severe combined immunodeficient/nude mice.

168 re a clinically relevant source of pulmonary emboli in some patients with cardiovascular implantable

169 tomography imaging to capture the spread of emboli in stems of three drought-resistant angiosperm tr

170 Clopidogrel and Aspirin for Reduction of Emboli in Symptomatic Carotid Stenosis (CARESS) is a ran

171 ht to facilitate formation of platelet-tumor emboli in the circulation, which in turn allows sequestr

172 However, there were still multiple emboli in the conjunctival and retinal arteries.

173 imab reduced the occurrence of platelet-rich emboli in the filters from 42% to 7% (P<0.01).

174 A strong increase in MK nuclei emboli in the lung, in WT and Myh9(-/-) mice, indicates

175 e relative risk of those patients having >20 emboli in the postoperative period (odds ratio, 10.23; 9

176 linical trials on the inhibition of cerebral emboli in the prevention and treatment of dementia are n

177 etastases in the lung and formation of tumor emboli in the pulmonary arteries.

178 and identify infection sources or bacterial emboli in the rest of the body.

179 ve found an increased incidence of recurrent emboli in these patients.

180 l CT image interpretation, and patients with emboli in these small vessels may have deep vein thrombo

181 This model exhibited lymphovascular emboli in vivo and corresponding spheroids in vitro.

182 raft-generated spheroids with lymphovascular emboli in vivo with both structures demonstrating E-cad

183 assess its potential for imaging thrombi and emboli in vivo.

184 tem assembled at the site of acute pulmonary emboli in vivo.

185 intracranial haemorrhage, and three systemic emboli) in people assigned to aspirin (yearly risk 1.8%v

186 ablation experienced a total of 16 cerebral emboli, in comparison with zero patients undergoing righ

187 s, factors associated with prevalent retinal emboli included older age (per 5-year increase; odds rat

188 The frequency of retinal emboli increases with age and are more common in men tha

189 Pressure also deformed the shed emboli, increasing their maximum major axis.

190 tive sequelae which may be due to hypoxemia, emboli, inflammation, drug toxicity, and/or other etiolo

191 ted that spontaneous showering of tumor cell emboli into the lung was robust, regardless of animal ge

192 o introduce large blood clots or macroscopic emboli into the middle cerebral artery (MCA) via an indw

193 T image interpretation, and all had multiple emboli involving at least the lobar arteries.

194 Prevention of stroke and systemic emboli is paramount in the management of atrial fibrilla

195 d treatment of isolated peripheral pulmonary emboli is uncertain.

196 Paradoxical emboli lead to strokes and cerebral abscesses, and these

197 cute pulmonary embolism was induced by large emboli made from clotting of autologous blood.

198 they would suggest that persons with retinal emboli may require both general cardiovascular and renal

199 This Notch 3 addiction of lymphovascular emboli might be exploited in future therapeutic strategi

200 The composition of pulmonary emboli mirrored the most distal part of venous thrombi f

201 Spontaneous cerebral emboli occur frequently in patients with Alzheimer's dis

202 four immediate procedural complications: Air emboli occurred in two patients, and prolonged bleeding

203 Y-X also was exhibited by the lymphovascular emboli of human IBC cases independent of their molecular

204 The lymphovascular emboli of human IBC exhibited dual N3icd and ALDH1 immun

205 unolocalized to the pulmonary lymphovascular emboli of MARY-X and caused their dissolution.

206 authors investigated the effect of cerebral emboli on cognitive and functional decline in both Alzhe

207 The proportion of patients with new cerebral emboli on MRI did not differ between bivalirudin and hep

208 d clinically with stroke had evidence of new emboli on MRI.

209 was proportion of patients with new cerebral emboli on MRI.

210 tion of implantable catheter (eight), septic emboli (one)], gastrointestinal tract [12; colitis (seve

211 ses, mainly atherosclerosis, but also septic emboli or blood disorders.

212 in, kidney, and spleen showed no evidence of emboli or infarct.

213 i were venous (12 patients, 7 with pulmonary emboli) or arterial (4 patients) or both.

214 nitive functioning in patients with cerebral emboli over 2 years, with a mean increase in score of 15

215 lowing embolization to calculate the dose of emboli (P(50) in mg) that produces neurological deficits

216 maging test that could locate both pulmonary emboli (PE) and their source, active deep venous thrombi

217 ed to the detection of unsuspected pulmonary emboli (PE) on routine cancer staging scans.

218 A rate of 8 or more emboli per hour was predictive of embolic stroke (P = 0.

219 Groups were similar for median number of emboli per patient (1 [interquartile range (IQR): 0 to 3

220 Pulmonary emboli (PEs) and lower extremity deep vein thrombi (DVTs

221 5 versus 11.4 ng/g; P=0.0002), in plaques of emboli-positive patients (22.7 versus 13.5 ng/g; P=0.003

222 Spontaneous cerebral emboli predict more rapid progression of dementia over 2

223 annual rate of ischemic strokes and systemic emboli prevented by warfarin minus intracranial hemorrha

224 Modifications of TAVR, emboli-prevention devices, and better intraprocedural ph

225 (Carotid Artery Stenting With Emboli Protection Surveillance-Post-Marketing Study [CAS

226 The CASES-PMS (Carotid Artery Stenting With Emboli Protection Surveillance-Post-Marketing Study) mul

227 the Carotid WALLSTENT plus FilterWire EX/EZ Emboli Protection System (Boston Scientific, Natick, Mas

228 d to undergo carotid-artery stenting with an emboli-protection device (cumulative incidence, 12.2 per

229 ho underwent carotid artery stenting with an emboli-protection device and those who underwent endarte

230 d carotid artery stenting with the use of an emboli-protection device as compared with endarterectomy

231 Stenting with the use of an emboli-protection device is a less invasive revasculariz

232 d trial, carotid stenting with the use of an emboli-protection device is not inferior to carotid enda

233 ditions, carotid stenting with the use of an emboli-protection device is not inferior to carotid enda

234 g carotid-artery stenting with the use of an emboli-protection device to endarterectomy in 334 patien

235 the Saphenous Vein Graft Angioplasty Free of Emboli Randomized (SAFER) trial.

236 rences among arterial and venous thrombi and emboli related to their origin, destination and duration

237 in source and predominant procedural step of emboli remain unclear.

238 y for all patients with asymptomatic retinal emboli remains uncertain.

239 atic deep vein thrombosis and four pulmonary emboli, resulting in an overall venous thromboembolism r

240 Tumor cell fragments, single cells, and emboli shed from the footpad tumor were easily distingui

241 with serious complications such as cerebral emboli, stroke, and migraine with aura.

242 The Asymptomatic Carotid Emboli Study (ACES) was a prospective observational stud

243 formation of microthrombi around tumor cell emboli (TCE), thereby inhibiting natural killer (NK) cel

244 rombosed central vein, resulted in pulmonary emboli that passed through dilated intrapulmonary vessel

245 cases had three biopsies showing cholesterol emboli, the first of which was a postreperfusion (immedi

246 dentified 88 individuals (0.9%) with retinal emboli; the overall person-specific, age-standardized pr

247 ic system assembles at the site of pulmonary emboli, thrombus dissolution is halted by alpha2-antipla

248 ndence at 90 days, mortality at 90 days, and emboli to new territory were also similar among groups.

249 0 days, symptomatic intracranial hemorrhage, emboli to new territory, and vasospasm were compared.

250 s-linking both caused experimental pulmonary emboli to resist endogenous and TPA-induced fibrinolysis

251 te to the nose bridge can result in arterial emboli to the ophthalmic system with optic nerve, retina

252 tion of an endothelial opening through which emboli translocated into the perivascular parenchyma.

253 Tumor emboli trapped in the vessels of lungs and lymph nodes a

254 Missed emboli typically were solitary and involved smaller arte

255 s on its deformation and possible release of emboli under different hemodynamic conditions.

256 of arterial and venous thrombi and pulmonary emboli using high-resolution scanning electron microscop

257 er, who retrospectively recorded presence of emboli using the same format; these results served as th

258 an (0.68; 0.55-0.83), but increased systemic emboli versus rivaroxaban (3.86; 1.17-12.75).

259 mbolism, in which alveolar gas or venous gas emboli (via cardiac shunts or via pulmonary vessels) are

260 ific, age-standardized prevalence of retinal emboli was 0.75% (95% CI, 0.60-0.95), with the highest p

261 Age-standardized prevalence of retinal emboli was calculated using the 2010 Singapore adult pop

262 mg/kg) and the percent (mean+/-SD) lysis of emboli was determined: (1) control, normal factor XIIIa

263 Angiographic evidence of pulmonary emboli was found in 34% (35/ 103) of patients.

264 Lysis of experimental pulmonary emboli was measured 4 hours after embolization in anesth

265 images were interpreted, and the location of emboli was recorded.

266 chamber, was found to be possible while the emboli was still small.

267 The probable source of the cholesterol emboli was the recipient in six cases and the donor in o

268 Interestingly, large emboli were also produced in GP V(-/-) mice, but not in

269 phic detection rates of potential sources of emboli were ascertained by doing a systematic review of

270 Interventions or Retinal emboli were ascertained from retinal photographs obtaine

271 Biofilm emboli were characterized by their size and their suscep

272 Locations and ratings of emboli were compared with reference standard CT images b

273 independent radiologists, and all pulmonary emboli were confirmed by a panel of three thoracic radio

274 Pulmonary emboli were created by injecting thrombi into the inferi

275 Spontaneous cerebral emboli were detected in 63 (44%) dementia patients, 36 (

276 aged endothelial surface, and small cerebral emboli were detected in vivo by PET imaging.

277 A total of 48 emboli were detected with CT.

278 ase in score of -3.8 for patients in whom no emboli were detected.

279 Central emboli were evident in 27 (96%) of these patients; 23 (8

280 Instead, emboli were found to translocate outside the vessel lume

281 Four (25%) of the 16 patients with emboli were identified at initial clinical CT image inte

282 of 3 major Asian ethnic populations, retinal emboli were most commonly seen in Indian persons and ass

283 Gas emboli were obvious by ultrasound within 15 minutes afte

284 Pulmonary emboli were present in 13 (16%) of 80 patients.

285 Large gas emboli were produced within the vasculature in the rat c

286 83 incident ischemic strokes and 71 systemic emboli were related to AF, of which 272 (59.9%) occurred

287 Unlike Fg-/- mice, the emboli were relatively small and released from the top o

288 Incidental pulmonary emboli were seen in 16 (4%) oncology patients but were i

289 low-up, all patients with reported pulmonary emboli were treated.

290 The structures within all of the thrombi and emboli were very tightly packed, in contrast to clots fo

291 events (deep venous thromboses or pulmonary emboli) were documented and confirmed prospectively.

292 All clots, including 24-h-old pulmonary emboli, were delineated.

293 rdial infarction, or acute peripheral artery emboli, were determined as well as major bleeding events

294 Six (60%) of 10 patients with emboli who underwent any lower extremity imaging had dee

295 ted faster decline in patients with cerebral emboli, with a mean increase in score of 12.0 in these p

296 e rapid deterioration in those with cerebral emboli, with a mean increase in score of 59.0 in these p

297 r the accurate diagnosis of small peripheral emboli, with a reported miss rate of up to 30% with sing

298 inoma (IBC) is characterized by florid tumor emboli within lymphovascular spaces called lymphovascula

299 inoma (IBC) is characterized by florid tumor emboli within lymphovascular spaces termed lymphovascula

300 t vaporization to noninvasively generate gas emboli within vasculature.