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1 ad cycle 1 dose-limiting toxicity (pulmonary embolus).
2 thmia, symptomatic hypotension, or pulmonary embolus).
3 osis type (deep vein thrombosis or pulmonary embolus).
4 ients (17%) had a possible cardiac source of embolus.
5 and 1 eventually developed a major pulmonary embolus.
6 age younger than 65 years and no more than 1 embolus.
7 n conduit/right atrial thrombus or pulmonary embolus.
8 No patient developed a pulmonary embolus.
9 and its 360 degrees distribution around the embolus.
10 ent as a result of an asymptomatic pulmonary embolus.
11 leak, small bowel obstruction, and pulmonary embolus.
12 during treatment as a result of a pulmonary embolus.
13 cyst and accounts for the compactness of the embolus.
14 3% of patients having a stroke or peripheral embolus.
15 ent required surgery for a delayed popliteal embolus.
16 echniques permit direct visualization of the embolus.
17 cause serious conditions due to paradoxical embolus.
18 gnant patients suspected of having pulmonary embolus.
19 ient ischemic attack, and 1 had a peripheral embolus.
20 as a result of sepsis and one of a pulmonary embolus.
21 g on the discrimination between contrast and embolus.
23 fic diseases, RRs were as follows: pulmonary embolus 1.87 (95% CI 1.13-3.07, p=0.01 [including 0.2% m
24 Grade 4 adverse events included pulmonary embolus (1), vomiting and constipation (1), and proteinu
25 ndrome (8.0% vs 9.2% vs 15.8%), or pulmonary embolus (1.9% vs 2.4% vs 6.8%) and to require intubation
26 common serious adverse events were pulmonary embolus (11 [1.6%] in the albumin group vs 8 [1.2%] in t
27 cerning CTPE findings: (1) bilateral central embolus, (2) right ventricle-to-left ventricle ratio gre
28 mA; 0.35-second tube rotation) and pulmonary embolus (64 sections at 1.25 mm, 140 kVp, 645 mA, 0.5-se
29 tients, V-P scintigrams predicted unilateral embolus; 64 patients underwent pulmonary angiography of
30 the proportion of patients with new cerebral embolus after TAVR and to investigate whether parenteral
31 (VTE) (deep venous thrombosis and pulmonary embolus) after splenectomy was 4.3% compared with 1.7% i
33 n thrombosis but a reduced rate of pulmonary embolus and heparin-induced thrombocytopenia among criti
34 ings were driven by lower rates of pulmonary embolus and heparin-induced thrombocytopenia and corresp
35 enerates the formation of the lymphovascular embolus and is responsible for its unique properties of
36 le-out' for myocardial infarction, pulmonary embolus, and aortic dissection, with an eye to improving
39 ombus, ischemic stroke, or systemic arterial embolus; and nonsystemic TEC, defined as Fontan conduit/
41 nique properties of the lymphovascular tumor embolus are poorly understood largely because of the abs
42 iagnosis in 15 cases, with the most proximal embolus at the segmental level in four cases and at the
43 re calculated for each MR technique on a per-embolus basis, and 95% confidence intervals were calcula
45 evaluation, daily transcranial Doppler with embolus detection studies, and neurological examinations
46 l charts were reviewed for demographic data, embolus detection, and outcomes up to 2 years after the
47 in silico trials to relate lesion volume to embolus diameter and calculate probabilistic lesion over
48 e including 3D information, identifying that embolus diameter could be determined from infarct volume
50 lpha2-antiplasmin inactivation, causing more embolus dissolution than clinical-dose r-tPA alone (P<0.
51 ditionally, we demonstrate how the resulting embolus distribution patterns compare and correlate with
52 mbosis not otherwise specified, or pulmonary embolus during the study period, and who had been treate
53 silico model can generate deep insights into embolus dynamics which is not otherwise available from s
58 was complicated by another saddle pulmonary embolus, heparin-induced thrombocytopenia, and COVID-19
59 us studies, demonstrated that the tumor cell embolus (IBC spheroid) forms on the basis of an intact a
60 roaches (H-2K(d)-E-cad), that the tumor cell embolus (IBC spheroid) forms on the basis of an intact a
62 umonia in 21, Hemothorax in 12 and pulmonary embolus in 8.Median/5-year survival were 20.7 months/17.
65 an extra breast cancer,stroke, or pulmonary embolus in about 6 per 1000 users aged 50-59 and 12 per
69 intracranial haemorrhages, and one systemic embolus) in people assigned to warfarin and 48 primary e
70 e of infection, venous thrombosis, pulmonary embolus, inability to aspirate blood, mechanical failure
73 e, comorbidity, and surgical risk (pulmonary embolus, leak, death, incisional hernia) than in other p
74 mproved understanding of embolic events, and embolus movement to the brain, is critical to develop te
76 of platelet accumulation at the site of the embolus occluding the MCA and within downstream cerebral
77 in PAI-1 immunoreactivity at the site of the embolus occluding the MCA were detected 1 h (n=7) and 4
81 o received tofersen, one died from pulmonary embolus on day 137, and one from respiratory failure on
82 ations OR 0.72 (95% CI 0.55-0.93), pulmonary embolus OR 0.29 (95% CI 0.11-0.73), pneumonia OR 0.66 (9
83 or laceration (APL), postoperative pulmonary embolus or deep venous thrombosis (PEDVT), foreign body
85 ncluding TNM staging, metastasis, and cancer embolus; Overall, this study suggested that tagSNPs rs11
86 roke, transient ischemic attack, or systemic embolus (p = 0.014) were positive predictors of warfarin
87 n thrombosis (DVT) alone in 49.7%, pulmonary embolus (PE) alone in 25.4%, DVT plus PE in 13.6%, arter
90 xtender rounds reduces the risk of pulmonary embolus, pneumonia, and death when compared to chart doc
91 e adverse events (including death, pulmonary embolus, pneumonia, deep wound infection, and acute myoc
92 clinical scenarios: postoperative pulmonary embolus, pneumothorax, myocardial infarction, gastrointe
94 dies examined deep vein thrombosis/pulmonary embolus prevention (42%) or venous ulceration (25%).
96 of detached biofilm particles depends on the embolus size and could be attributed to nutrient-limited
98 d be determined from infarct volume and that embolus size is critically important to the resting plac
99 3 days after surgery, following a pulmonary embolus (spleen weight 500 g, mortality 1/108, 0.9%).
100 Major complications (reintubation, pulmonary embolus, stroke, renal failure, coma, cardiac arrest, my
101 allows more confident detection of pulmonary embolus than does CFA, with no loss in diagnostic accura
102 culty with rapidly adequately diagnosing the embolus, the lack of good data supporting the use of thr
103 bedside echocardiogram suggesting pulmonary embolus, thrombolytic therapy was administered during ca
106 ined) were cellulitis (two cases), pulmonary embolus (two cases), and deep-vein thrombosis (three cas
107 oses (acute myocardial infarction, pulmonary embolus, upper gastrointestinal bleed), and comorbiditie
112 gnant patients suspected of having pulmonary embolus, with considerable variability in their policies