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1 ulation frequencies (P < 0.05) and miniature end-plate potential amplitude analysis (P < 0.01) showed
2 ed to 10-30% of normal levels, the miniature end-plate potentials are correspondingly reduced, and th
3 ock owing to temporal summation of prolonged end plate potentials at physiologic rates of stimulation
6 not after a tetanus, increased the speed of end plate potential (EPP) amplitude rundown, and greatly
7 namely, decreased quantal content, decreased end plate potential (EPP) amplitude with prolonged rise
11 ronous ACh release evoked by nerve impulses (end-plate potentials, EPPs) follow a simple binomial dis
13 plitude and decay time constant of miniature end-plate potential (MEPP) and miniature end-plate curre
14 itude but increase in frequency of miniature end-plate potential (mEPP) at the NMJs in Acta1+/Ki mice
15 as the frequency of occurrence of miniature end-plate potentials (MEPP(f)), were evoked by high pota
16 rise and decay time, and, although miniature end plate potential (mEPPs) amplitude and frequency were
18 End-plate potentials (EPPs) and miniature end-plate potentials (MEPPs) were recorded from neuromus
19 t the end-plate border we examined miniature end-plate potentials (MEPPs), sodium current (INa) ampli
20 ionally, miniature end plate potential size, end plate potential size, and quantal content did not di