ライフサイエンスコーパス: end product

1 FE to cis-1,2-difluoroethene (cis-DFE) as an end product.

2 t this is a true intermediate and not a dead-end product.

3 oduction of simple nitriles as the bioactive end product.

4 ) Z(2) H, and sulfate was the sole metabolic end product.

5 l expertise for performance and detection of end product.

6 total glucose, generating UDP-GlcNAc as the end product.

7 alase system, which produces d-lactate as an end product.

8 ribute to the synthesis of at least this one end product.

9 that thiosulfate is an additional metabolic end product.

10 ified strombine as coral's main fermentative end product.

11 and 10,11-dihydroxy carbamazepine as a dead-end product.

12 affording 1,4-ketocarbonyls as a value-added end product.

13 ies: exposure, execution, adverse event, and end-product.

14 ygen by producing ethanol as their metabolic end-product.

15 cessing and reduces the quality of the sugar end-product.

16 fied as both intermediates and apparent dead-end products.

17 sform specific central metabolites into many end products.

18 y drying processes and quality attributes of end products.

19 ough the production of different biochemical end products.

20 iochemical pathway without alteration of the end products.

21 ssion of the receptor for advanced glycation end products.

22 reducing the formation of advanced glycation end products.

23 clusters to any of 1172 clusters with known end products.

24 s (HAAs) as major reaction intermediates and end products.

25 fications that are called advanced glycation end products.

26 genes and subsequent accumulation of pathway end products.

27 , and rerouting carbon flux toward desirable end products.

28 pathways to meet metabolic demand for their end products.

29 s to biologically and environmentally benign end products.

30 challenges and assess the quality of traded end-products.

31 n of phenolic acids and methyl-donors in the end-products.

32 th glycolaldehyde-derived advanced glycation end-products (0, 50, 100, and 200 mug/ml) for 48 hours i

33 noic acid, results in the accumulation of an end product, 2-(omega-carboxyethyl)pyrrole (CEP), which

34 etabolites [10-fold for (S)-reticuline], and end products (30-fold for codeine).

35 d thiols can undergo isomerization to a dead-end product (a 4-methylcoumarin-3-yl thioether) upon pho

36 Because advanced glycation-end products accumulate throughout life, we investigated

37 nsferases to gain insight into the extent of end product accumulation and possible feedback inhibitio

38 Tissue advanced glycation end product (AGE) accumulation has been proposed as a ma

39 fficient synthesis of the advanced glycation end product (AGE) methylglyoxal-derived imidazolium cros

40 Advanced glycation end products (AGE) accumulate in diabetic patients and a

41 o determine the effect of advanced glycation end products (AGE) in the presence and absence of Porphy

42 Advanced glycation end-product (AGE) formation is increased in diabetes.

43 howed reduced fluorescence advanced glycated end-products (AGE) and a reduced amount of alpha-dicarbo

44 Advanced glycation end-products (AGE) are reactive metabolites produced as

45 nyls and the receptor for advanced glycation end products (AGER).

46 The receptor of advanced glycation end products, AGER (previously known as RAGE), interfere

47 Advanced glycation end products (AGEs) accumulate in T2DM, resulting in inf

48 Advanced glycation end products (AGEs) accumulate in tissues with age and i

49 Advanced glycation end products (AGEs) accumulated during long-term hypergl

50 tion of total fluorescent advanced glycation end products (AGEs) and Amadori adducts were determined.

51 digestibility, release of advanced glycation end products (AGEs) and antioxidant capacity of puffed c

52 osis include formation of advanced glycation end products (AGEs) and reactive oxygen species (ROS), b

53 Advanced glycation end products (AGEs) are a heterogeneous group of compoun

54 on cascade, the so-called advanced glycation end products (AGEs) are formed, including proteins with

55 pothesized that levels of advanced glycation end products (AGEs) are higher in the gingival crevicula

56 Advanced glycation end products (AGEs) are involved in the inflammatory pro

57 predominant precursor for advanced glycation end products (AGEs) due to its protein glycation reactio

58 perglycemic conditions or advanced glycation end products (AGEs) ex vivo resulted in significant abno

59 rs methylglyoxal (MG) and advanced glycation end products (AGEs) formation in cancer cells.

60 The consumption of advanced glycation end products (AGEs) has increased because of modern food

61 l data suggest a role for advanced glycation end products (AGEs) in cardiovascular disease (CVD), par

62 Presence of advanced glycation end products (AGEs) in the heart induces a proinflammato

63 F) noninvasively measures advanced glycation end products (AGEs) in the skin and is a risk indicator

64 nsumption of diets low in advanced glycation end products (AGEs) on cardiometabolic parameters are co

65 Six skin collagen advanced glycation end products (AGEs) originally measured near to the time

66 Advanced glycation end products (AGEs) promote adverse health effects and m

67 s in the oxidative stress-advanced glycation end products (AGEs) receptor for AGEs (RAGE) pathway, an

68 alpha-dicarbonyls) yield advanced glycation end products (AGEs) that can alter the structures and fu

69 examined associations of advanced glycation end products (AGEs) with renal function loss (RFL) and i

70 Advanced glycation end products (AGEs), a heterogeneous group of compounds

71 n results in formation of advanced glycation end products (AGEs), also originating from alpha-dicarbo

72 wo decades, many types of advanced glycation end products (AGEs), formed through the reactions of an

73 e canonical RAGE ligands, Advanced Glycation End products (AGEs), HMGB1, and S100 proteins, have all

74 nent subpathways included advanced glycation end products (AGEs), phosphatidylcholines, sphingolipids

75 of RAGE ligands including advanced glycation end products (AGEs), S100 proteins, and HMGB1.

76 atoes on the formation of advanced glycation end products (AGEs), the activity of angiotensin-convert

77 predominant precursor of advanced glycation end products (AGEs), which result in protein dysfunction

78 uced diabetic mice and on advanced glycation end products (AGEs)-induced H9c2 cardiomyocytes.

79 to altered production of advanced glycation end products (AGEs).

80 teins, yielding early and advanced glycation end products (AGEs).

81 tein products (AOPPs) and advanced glycation end products (AGEs).

82 where proteins accumulate advanced glycated end products (AGEs).

83 tion through formation of advanced glycation end products (AGEs).

84 Advanced glycation end-products (AGEs) accumulate during prolonged hypergly

85 ki'), on the formation of advanced glycation end-products (AGEs) and the activity of angiotenisn-conv

86 Advanced glycation end-products (AGEs) are also present in foods.

87 Resulting advanced glycation end-products (AGEs) contribute to pathogenesis of diabet

88 formation of fluorescent advanced glycation end-products (AGEs) in vitro of raw and roasted buckwhea

89 , heat processes generate Advanced Glycation End-products (AGEs), including N(epsilon)-carboxymethyll

90 ctivates the formation of advanced glycation end-products (AGEs).

91 formation of fluorescent advanced glycation end-products (AGEs).

92 due to an accumulation of advanced glycation end-products (AGEs).

93 For investigating the advanced glycation end-product-albumin (AGE-albumin) from activated macroph

94 signaling, accumulation of advanced glycated end-products, altered autophagy, changes in myocardial s

95 cted between days 16 and 21 for fermentative end-product analysis and 16S ribosomal RNA bacterial gen

96 d a significant decrease in advance glycated end products and protection against glycoxidation-induce

97 Higher levels of advanced glycation-end products and reduced glyoxalase-1 activity and gluta

98 lial injury (receptor for advanced glycation end products and surfactant protein D) and endothelial i

99 Complement activation end products and their receptors mediate cell-cell inter

100 eventing the formation of advanced glycation end products and therefore potentially mitigate the diab

101 c; only two showed accumulation of anaerobic end products and three exhibited the classical heat shoc

102 rization is crucial to define the quality of end products and to describe the evolution of processes

103 Lower receptor for advanced glycation end products and tumor necrosis factor-alpha (TNF-alpha)

104 absence of an exogenous supply of chorismate end-products and present biochemical evidence suggesting

105 hydroxymethylfurfural and advanced glycation end products) and microbiological safety and sensory tes

106 ntent (i.e. less elastin, advanced glycation end-products) and increase in conduit artery diameter ar

107 phosphates, endothelin-1, advanced glycation end-products, and asymmetric dimethylarginine), can be i

108 form of the receptor for advanced glycation end-products, and safety.

109 (EcoRV and HincII) that also generate blunt-ended products, and to a structurally distinct enzyme (t

110 n plants, and thus, besides being the actual end-product, antibodies targeting endogenous host protei

111 y, as secondary metabolites and fermentation end products are absorbed either by the epithelial layer

112 progenitor sgRNAs, whereas the mature sgRNA end products are resistant.

113 These end products are transported into the cell where a beta-

114 -2 and RAGE (receptor for advanced glycation end products) are not involved.

115 multiligand receptor for advanced glycation end products as receptors during MRP8-mediated effects o

116 using a video-based assessment tool, and an end product assessment tool.

117 lowing mineralization and advanced glycation end product-associated modification.

118 0A8/A9-RAGE (receptor for advanced glycation end products) axis could represent a viable approach to

119 y identified receptor for advanced glycation end products-binding protein levels only showed a signif

120 Fibrin thus is not an inert end product but partakes in further thrombus growth.

121 eptor that interacts with advanced glycation end products, but also with C3a, CpG DNA oligonucleotide

122 quire several months to identify a metabolic end product by biosynthetic pathway screens, but this ti

123 stages (raw, roasted, and winnowed side and end products) by high-performance liquid chromatography

124 lites in blood and is a precursor as well as end product central to numerous important metabolic path

125 f reduced accumulation of advanced glycation end products compared with the strut interior.

126 es between subunits, followed by disassembly end-products consisting of highly curved oligomers and 1

127 Fibrin, the coagulation end product, consolidates the platelet plug at sites of

128 ptor and the receptor for advanced glycation-end products, cross the blood-brain-barrier and reach ne

129 the formation of dietary advanced glycation end-products (d-AGEs), dicarbonyls and acrylamide.

130 etion of receptor for advanced glycosylation end product decreased UVB-induced resistance to apoptosi

131 of TLR4- and receptor for advanced glycation end products-dependent signaling.

132 ate (MEP) pathway of isoprenoid synthesis by end products dimethylallyl diphosphate (DMADP) and isope

133 fibroblasts treated with advanced glycation end-products display reduced ATP production, electron tr

134 Our findings suggest that advanced glycation end-products disrupt tendon fibroblast homeostasis and m

135 We argue that the 'biodegradable' end-product does not necessarily degrade once emitted to

136 d that ribitol, considered to be a metabolic end-product, enhances matriglycan expression in dystroph

137 Fermentation end-products, extent of feed degradation and composition

138 ch as O-GlcNAcylation and advanced glycation end product formation.

139 non-fermentable pentoses inhibit growth and end-product formation during fermentation of cellulose-d

140 in a variety of organic compounds, including end products from the degradation of influent substrates

141 complexes, this is the first case where the end products from these reactions could be characterized

142 cumulating intermediates but not the pathway end product fumigaclavine C also were less virulent than

143 zymes studied caused significantly different end product functionality, presumably due to the differe

144 d the chemical structure of the biosynthetic end-product, has become a cornerstone of knowledge-based

145 Advanced glycation end products have been implicated in the pathogenesis of

146 odynamic power, and the benign nature of its end products have raised interest in oxidases and oxygen

147 ors 2 and 4, receptor for advanced glycation end-products, high-mobility group box 1, uric acid, IL-3

148 The major end product in human and porcine epidermis is a trihydro

149 ative chemical reactions to produce the same end products in the presence and absence of oxygen.

150 levated levels of lipoxidation and glycation end products in the primary olfactory system, protein ca

151 oxide (N2O) is considered an intermediate or end-product in denitrification pathways.

152 Fermentation end products, in particular the short-chain fatty acid (

153 erglycemia and exposure to advanced glycated end products inactivated MFG-E8, recognizing a key mecha

154 ment cellulosic biomass to formate and other end products, including CO2 This organism lacks formate

155 ATP fuels the removal of metabolic end-products, including H(+) ions that profoundly modula

156 lIPMS3 (Solyc08g014230) encodes a functional end product inhibition-insensitive version of the commit

157 by consolidating process steps and reducing end-product inhibition of enzymes compared with separate

158 In an example of end-product inhibition, cholesterol accelerates the prot

159 n-6, soluble receptor for advanced glycation end products, interleukin-1ra, tumor necrosis factor alp

160 ies of both the starting fibroblasts and the end product, iPSCs, and are also of paramount importance

161 Receptor for advanced glycation end products is targeted by FBXO10 for ubiquitination an

162 The export of low-molecular-weight catabolic end-products is facilitated by polytopic transmembrane p

163 O), a major precursor for advanced glycation end products, is increased in diabetes.

164 epressed by inhibitors of advanced glycation end products, L-type calcium channels, protein kinase C,

165 The method of end-products labeling also generates a cluster of isomer

166 ress, and accumulation of advanced glycation end products, leading to altered bone metabolism, struct

167 form of the receptor for advanced glycation end-products levels in the sevoflurane group, compared w

168 cemia causes receptor for advanced glycation end products-mediated epigenetic modification of naive T

169 a significant increase in advanced glycation-end product-modified proteins in the myocardium of old m

170 oform of the Receptor for Advanced Glycation End-products (nRAGE) in DSB-repair.

171 Lactate is the end product of aerobic glycolysis, a unique form of meta

172 is a significant, although underappreciated end product of choline and amino acid oxidation.

173 We propose that the end product of chromophore bleaching in rod photorecepto

174 We recently found that the end product of docosahexaenoic acid (DHA) oxidation, 2-(

175 Supplementation of palmitate, the end product of FASN catalysis, rescued cancer cells from

176 Lactate is an end product of glucose metabolism, which serves metaboli

177 Restoring pyruvate levels, the end product of glycolysis, preserved DeltaPsim and preve

178 iation of SMSCs and found that pyruvate, the end product of glycolysis, stimulates their differentiat

179 cetate production derived from pyruvate, the end product of glycolysis.

180 f gut bacteria that catabolize galactose, an end product of lactose metabolism, and of bacteria that

181 nic nitrate was once considered an oxidation end product of nitric oxide metabolism with little biolo

182 Urea is the major end product of nitrogen metabolism in humans, which is e

183 ructures in both functional proteins and the end product of pathologic protein misfolding.

184 However, sulfate, the end product of sulfur-containing amino acids (SAAs), con

185 ation to intracellular proteins utilizes the end product of the nutrient sensing hexosamine biosynthe

186 N2, the end product of this metabolism, is produced from the oxi

187 The end product of this pathway - cortisol - is secreted in

188 Since, hydrogen peroxide is the oxidative end product of uric acid (UA) by uricase, an efficient a

189 e that these immature particles are not dead-end products of assembly, but progress into mature 30S s

190 nase PHEOPHORBIDE a OXYGENASE (PAO), are the end products of chlorophyll degradation.

191 Bile acids are important end products of cholesterol metabolism.

192 -read sequencing technologies to investigate end products of de novo chromosome 17p11.2 rearrangement

193 Polymorphism observed within the aggregation end products of fibrils are known to arise due to micros

194 tricarboxylic acid (TCA) cycle converts the end products of glycolysis and fatty acid beta-oxidation

195 e, histidine, and methionine; (iv) catabolic end products of lignin (pyruvate and oxaloacetate) must

196 expression of proinflammatory cytokines, and end products of lipid oxidation had a synergistic effect

197 he utilization of metabolic intermediates or end products of metabolic pathways as substrates for enz

198 cetylneuraminic acid (Neu5Ac), which are the end products of sialidase activity.

199 o significantly enhance flux toward specific end products of the sterol pathway.

200 nce alkali-metal compounds are often not the end products of these applications, their roles are rare

201 The end-product of ameloblast activity is a marvel of struct

202 As the end-product of CMVMC, a reduced set of relevant and non-

203 Carbon monoxide (CO), an end-product of heme oxygenase (HO)-1 activity, can confe

204 cinoembryonic antigen (CEA) by measuring the end-product of immunoassay performed on magnetic particl

205 actosyldiacylglycerol (18:3-16:3-MGDG) as an end-product of MDA incorporation.

206 Lipofuscin is a nondegradable end-product of oxidative stress; its cerebral presence r

207 Serum uric acid is the end-product of purine metabolism and at high levels is a

208 ersions because CO(2) is an intermediate and end-product of the digestion process and modifies the ca

209 (2020) identify two end-products of bacterial fermentation that regulate int

210 Elevations in glucocorticoids, the end-products of HPA activation, play roles in adaptive a

211 Our study identified the novel end-products of lipid peroxidation, accumulating in circ

212 compounds formed during roasting, defined as end-products of Maillard reaction.

213 may involve the effect of advanced glycation end products on DC migration.

214 cleaves the crosslinks of advanced glycation end products on the extracellular matrix.

215 investigate the impact of advanced glycation end-products on tendon fibroblasts to further our mechan

216 either on the detection of relatively stable end products or on the use of synthetic probes, and they

217 ve suggested that BOXes (bilirubin oxidation end products), originating from released heme surroundin

218 vage soluble receptor for advanced glycation end-products, plasma interleukin-6, and monocyte chemota

219 s) and RAGE (receptor for advanced glycation end-products) play a critical role in metabolic pathways

220 t-chain fatty acids (SCFAs) are fermentation end products produced by the intestinal microbiota and h

221 Production of distinct end product profiles from different substrates (GPP vers

222 ) can engage receptor for advanced glycation end product (RAGE) to direct monocytes to a proinflammat

223 eceptor, receptor for advanced glycosylation end product (RAGE), exhibited decreased expression of bo

224 DNA through receptor for advanced glycation end products (RAGE) and induces production of type I int

225 ytes through receptor for advanced glycation end products (RAGE) and Toll-like receptor 2, leading to

226 The receptor for advanced glycation end products (RAGE) and Toll-like receptor 4 (TLR4) is i

227 The receptor for advanced glycan end products (RAGE) has been identified as a susceptibil

228 role of the receptor for advanced glycation end products (RAGE) in neuroinflammation, neurodegenerat

229 ted that the receptor for advanced glycation end products (RAGE) is a critical molecule in the pathog

230 The receptor for advanced glycation end products (RAGE) is a highly expressed cell membrane

231 The receptor for advanced glycation end products (RAGE) is a multiligand transmembrane recep

232 The receptor for advanced glycation end products (RAGE) is a pattern recognition receptor ca

233 The receptor for advanced glycation end products (RAGE) is a pattern recognition receptor th

234 The receptor for advanced glycation end products (RAGE) is highly expressed in human and mur

235 The receptor for advanced glycation end products (RAGE) is highly expressed in various cance

236 However, in Receptors for Advanced Glycation End Products (RAGE) knockout mice after postnatal day 3,

237 egulation of receptor for advanced glycation end products (RAGE) messenger RNA, but not toll-like rec

238 act with the receptor for advanced glycation end products (RAGE) on hepatic Kupffer cells, resulting

239 binds to the receptor for advanced glycation end products (RAGE) on Kupffer cells, ultimately leading

240 The receptor for advanced glycation end products (RAGE) plays a key role in mammal physiolog

241 or (TLR) and receptor for advanced glycation end products (RAGE) signals.

242 suppress the receptor for advanced glycation end products (RAGE) via nuclear factor erythroid-2-relat

243 The receptor for advanced glycation end products (RAGE), a cell membrane receptor, recognize

244 tor protein, Receptor for Advanced Glycation End products (RAGE), has been extensively studied, there

245 (TLR)2, the receptor for advanced glycation end products (RAGE), myeloid differentiation primary res

246 or 4 (TLR4), receptor for advanced glycation end products (RAGE), p-ERK1/2, nuclear NF-kappaB p65, an

247 y binding to receptor for advanced glycation end products (RAGE).

248 GB1) and its receptor for advanced glycation end products (RAGE).

249 gene for the receptor for advanced glycation end-products (RAGE) are associated with an increased inc

250 The receptor for advanced glycation end-products (RAGE) is a multiligand pattern recognition

251 xpression of receptor for advanced glycation end-products (RAGE) is suggested to play a crucial role

252 LR4) and the receptor for advanced glycation end-products (RAGE) revealed the involvement of alarmins

253 ading to the receptor for advanced glycation end-products (RAGE) shedding into soluble and nuclear fo

254 doplanin and receptor for advanced glycation end-products (RAGE), and most cells were negative for al

255 binds to the receptor for advanced glycation end-products (RAGE).

256 through the receptor for advanced glycation end-products (RAGE; ie, its receptor), are involved in f

257 y mediators (receptor for advanced glycation end products [RAGE], MPO, uteroglobin/CC-10); between gr

258 ant pathways included advanced glycosylation end product receptor signaling and telomere maintenance

259 ctive allenic polyketides and citreodiols as end products, respectively.

260 iption error rate estimated from mistakes in end product RNAs is 10-3-10-5.

261 idely depending, among other factors, on the end product's location in the pathway, which determines

262 nuria, and suppression of advanced glycation end-product signalling.

263 lls and expression of advanced glycosylation end-product specific receptor (AGER).

264 heir receptors, including advanced glycation end product-specific receptor (RAGE), trigger various in

265 and soluble-receptor for advanced glycation end products (sRAGE) in bronchoalveolar lavage(BAL) and

266 the soluble receptor for advanced glycation end products (sRAGE).

267 form of the receptor for advanced glycation end-products (sRAGE) are elevated during acute respirato

268 erum soluble receptor for advanced glycation end-products (sRAGE) with acute and chronic morbidities

269 AGE (soluble receptor for advanced glycation end products) strongly associate with ARDS risk.

270 a concomitant increase of advanced-glycation end-products suggesting that allysine may be involved in

271 -lysine, one of the major advanced glycation end products, suggesting the prominent role of carbamyla

272 observed in receptor for advanced glycation end products, surfactant protein D, angiopoietin-2, inte

273 que substrates and when combined, synthesize end products that precisely mimic those in vivo, demonst

274 oreover, the receptor for advanced glycation end products that recognizes MG and GO adducts and glyox

275 he C nucleoside Psi to form stable bisulfite end products that yield signatures for next-generation s

276 nguages has mostly consisted of studying the end product; the process by which ad hoc signs are trans

277 d as shelf-stable precursors to a variety of end products through simple functional group transformat

278 ants such as trichloroethene (TCE) to benign end-products through aqueous phase reactions.

279 Culture contamination, end-product toxicity, and energy efficient product recov

280 ceptor RAGE (receptor for advanced glycation end-products) transmits proinflammatory signals in sever

281 he increased formation of advanced glycation end products under certain pathological conditions.

282 ey can convert CO2 directly into the desired end product using solar energy.

283 rade could not be distinguished across other end product variables such as axillary vein damage (P =

284 F-alpha, the receptor for advanced glycation end products, vascular endothelial growth factor, perios

285 athways converge to the desired biosynthetic end product via the (SAM-dependent) retro-Claisen rearra

286 orm as transient intermediates or unreactive end-products via e(aq)(-) reactions with precursor struc

287 The pattern of end products was altered, and lactate production was tot

288 exquisite biological sensor, the fibrin clot end-product was replaced with a synthetic material under

289 Under humid conditions (50% RH), the major end products were 4-oxopentanal, 4-oxobutanoic acid, and

290 Advanced glycation end products were assessed with a fluorometric assay.

291 n m/z 300 and 350, the major condensed-phase end products were levulinic acid (LLA) and succinic acid

292 duct soluble receptor for advanced glycation end products were significantly attenuated in fat-fed mi

293 non-pigmented durum wheat genotypes, and the end-products were compared with commercial pasta.

294 lites, the precursors for advanced glycation end products, were significantly elevated in plasma and

295 a, TLR4, and receptor for advanced glycation end products, whereas mRNA levels of anti-inflammatory T

296 ave been used to pair pathway precursors and end products with cognate biosynthetic genes.

297 ducing sugars and include advanced glycation end products with deleterious health effects.

298 tools towards the production of high quality end-product with increased bioactive properties without

299 should be considered for alternative uses of end-products with high bioactivity.

300 We proposed that advanced glycation end-products would induce limitations to mitochondrial f