ライフサイエンスコーパス: end systole

1 erformed separately at both end diastole and end systole.

2 as the dicrotic notch and retrograde flow at end systole.

3 ht atrial volume ratio (LARA) was defined at end systole.

4 ains occurring from control to ring state at end-systole.

5 postoperative MR was LV sphericity index at end-systole.

6 nulus and leaflets were computed at mid- and end-systole.

7 astole, isovolumic systole, peak-systole and end-systole.

8 re calculated from 3-D marker coordinates at end-systole.

9 LV systole with minimum MA area occurring at end-systole.

10 d fit quality if the trigger delay is set to end-systole.

11 trol, Duran, and Physio; ANOVA=0.005) and at end systole (14.5+/-6.2, 10.5+/-5.5, and 5.8+/-2.5 mm, r

12 .5 +/- 2.7 to 13.7 +/- 2.4 mm, p = 0.03) and end systole (16.1 +/- 2.9 to 18.5 +/- 1.8 mm, p = 0.03),

13 er at the endocardium than the epicardium at end systole (24+/-5% versus 16+/-3%; P<0.05, n=8), consi

14 se-apex length increased at end-diastole and end-systole (all +1 mm, P<0.05).

15 ains occurring from control to ring state at end-systole along the annulus were calculated.

16 Image series reconstructed at end systole and end diastole were used to measure LV-LAS

17 the origins of the mitral valve leaflets at end systole and end diastole.

18 1.5 T, with images aquired simultaneously at end systole and middiastole.

19 periods of cardiac motion identified during end systole and middiastole.

20 scans were between those with triggering to end-systole and diastasis.

21 reases in LV volumes and sphericity index at end-systole and end-diastole.

22 Volume and mass were calculated at end-systole and end-diastole.

23 d [Formula: see text]-ATP were higher during end-systole and lower during diastasis than in untrigger

24 ormal strain pattern with distinct peaks pre-end-systole and post-end-systole in inferior-lateral wal

25 tip (10.3 versus 6.4 mm, MR versus no-MR, at end-systole) and increased r of the anterior papillary m

26 MR severity, LV volumes at end-diastole and end-systole, and LA volumes were measured at baseline, d

27 illary blood volume between end diastole and end systole at baseline.

28 y and serial changes in regional geometry at end systole.Beginning as a narrow band of fully perfused

29 inded, independent readers on cine images in end systole by using a freely available software package

30 tance (left ventricular maximum elastance at end systole), cardiac output, circumflex artery blood fl

31 real time (32-64 Hertz) or triggered 1:1 at end systole during a 20% C3 or C3C4 droplet infusion.

32 with the LOX group, whereas left ventricular end-systole elastance was preserved at baseline levels.

33 Right ventricular end-systole elastance was significantly improved in the

34 marker coordinates at end diastole (ED) and end systole (ES) were computed.

35 of the annulus increased only 9.2+/-6.3% at end systole (ES).

36 End-systole (ES) radial and longitudinal strain images w

37 ted from 3-dimensional marker coordinates at end-systole (ES).

38 ange in the cavity area from end diastole to end systole (fractional area change [FAC]), was related

39 Ejection after end systole has a positive effect on ventricular perform

40 astole and LA volumes, but not LV volumes at end-systole in degenerative MR, is consistent with corre

41 with distinct peaks pre-end-systole and post-end-systole in inferior-lateral wall was frequent in pat

42 both continuously and intermittently (every end systole) in the fundamental (2 MHz) and harmonic (tr

43 tment, left ventricular maximum elastance at end systole increased and was unchanged in controls (30

44 We conclude that triggering to end-systole is superior to triggering to diastasis.

45 levated border zone fiber stresses from mean end-systole levels of 28.2 kPa (control) to 23.3 kPa (tr

46 ic contours were automatically propagated to end systole, mean differences were 2.0 g +/- 3.6 (P =.05

47 ngth, referring to true MAD, was measured at end systole on pre- and post-operative transthoracic ech

48 tion between diameter and volume was good at end-systole (r = 0.91, p < 0.0001) and end-diastole (r =

49 tole and 42+/-17%, 37+/-17%, and 21+/-10% at end systole, respectively, for Control, Duran, and Physi

50 ring acute ischemic mitral regurgitation, at end systole, the anterolateral edge of the central scall

51 d 2.5+/-0.12 mm toward the mitral annulus at end systole; the posterior papillary muscle geometry was

52 PV data measured at normalized time (tN) and end systole (tmax) to predict intercept: Vo(SB) = [EN(tN

53 jected onto the MA plane at end diastole and end systole to assess PM dynamics.

54 nd a correspondence between end diastole and end systole was computed with a novel algorithm.

55 LV volumes at end-systole was significantly reduced in functional MR b

56 ular free wall curvature (C(FW)) measured at end systole were used to derive the curvature ratio (C(I

57 Formula: see text]-ATP peaks was best during end-systole when blood contamination of ATP and Pi signa

58 te left ventricular volume reduction (VR) at end systole, with EDV kept constant.