ライフサイエンスコーパス: end-diastolic

1 remodeling at 6 months included decreases in end-diastolic (161 +/- 36 ml to 122 +/- 30 ml; p < 0.001

2 modeled with increase in LV left ventricular end-diastolic (175 mL +/- 35 to 201 mL +/- 40) and end-s

3 ic (51 +/- 13 vs 50 +/- 14 mm, p = 0.05) and end-diastolic (55 +/- 13 vs 52 +/- 14 mm, p = 0.003) dim

4 here was no difference in change in LVEF, LV end diastolic and end systolic diameters between the 2 g

5 d hearts revealed that all LV parameters (LV end-diastolic and -systolic dimensions, ejection fractio

6 The primary endpoint was change in LV end-diastolic and -systolic volume assessed by cardiac m

7 ther measures included 6-month changes in LV end-diastolic and -systolic volumes indexed to body surf

8 The outcomes included RV end-diastolic and end-systolic dimensions, RV ejection f

9 ith PA:A>1 had higher right ventricular (RV) end-diastolic and end-systolic volume indices accompanie

10 LV end-diastolic and end-systolic volumes and diastolic fun

11 The LV end-diastolic and end-systolic volumes and LV ejection f

12 gic pacing with either BiVP or HisBP, the LV end-diastolic and end-systolic volumes were significantl

13 gic pacing with either BiVP or HisBP, the LV end-diastolic and end-systolic volumes were significantl

14 Left ventricular end-diastolic and left atrial end-systolic volumes incre

15 Maximum end-diastolic aortic root diameters were measured in the

16 9 to 1.76; p < 0.001), and right ventricular end-diastolic area (HR: 1.04 per cm(2); 95% CI: 1.01 to

17 as associated with greater right ventricular end-diastolic area and worse right ventricular fractiona

18 In contrast, median RV end-diastolic area increased (P=0.001 versus baseline).

19 V function was assessed by echocardiographic end-diastolic area, end-systolic area, fractional area c

20 rsus 172+/-28 mL, P<0.001), right ventricle (end-diastolic area=27.0+/-4.8 versus 28.6+/-4.3 cm(2); P

21 elow tenth percentile and absent or reversed end-diastolic blood flow in the umbilical artery on Dopp

22 Only 2.1% of the stacks had an average end-diastolic cardiac image contrast below 30% of the dy

23 .0001]); and a reversal of LV remodeling (LV end diastolic diameter -2.49 mm [95% CI: -4.09 to -0.90;

24 py before implant (OR 2.2), left ventricular end-diastolic diameter <6.5 cm (OR 1.7), pulmonary systo

25 d reverse LV remodeling with reduction of LV end-diastolic diameter (-0.20 +/- 0.4 mm) and volume (-2

26 ARNi versus MI-vehicle demonstrated lower LV end-diastolic diameter (by echocardiography; 9.7+/-0.2 v

27 mary endpoint was change in left ventricular end-diastolic diameter (LVEDD) after 5 years, measured a

28 m (-2.9 to -0.6, p=0.0027), left ventricular end-diastolic diameter -1.3 mm, (-2.3 to 0.3, p=0.0128),

29 ion (LVEF), and remodeling (left ventricular end-diastolic diameter [LVEDD]) at presentation, were as

30 lts for cardiac (2 SNPs for left ventricular end-diastolic diameter and 5 SNPs for aortic root diamet

31 ion were a low LVEF, a high left ventricular end-diastolic diameter and a low heart rate after beta-b

32 ]=1.23 [1.1-1.37]; P<0.001) as was increased end-diastolic diameter at the apical third of the RV (mu

33 ween HIIT and MCT (P=0.45); left ventricular end-diastolic diameter changes compared with RRE were -2

34 Change in left ventricular end-diastolic diameter from baseline to 12 weeks was not

35 oup comparison of change in left ventricular end-diastolic diameter from baseline to 12 weeks.

36 ntricular ejection fraction was 14.5+/-5.3%, end-diastolic diameter was 7.33+/-0.89 cm, end-systolic

37 left ventricular ejection fraction, 57+/-8%; end-diastolic diameter, 4.81+/-0.58 cm; end-systolic dia

38 l diastolic thickness, left ventricular (LV) end-diastolic diameter, LV posterior wall thickness, LV

39 tion, left ventricular ejection fraction and end-diastolic diameter, sex, ischemic cardiomyopathy, at

40 Women with preeclampsia had smaller mean LV end-diastolic diameters (5.2 versus 6.0 cm; P=0.001), gr

41 RV end-diastolic diameters were significantly larger in pat

42 Left-ventricular end diastolic dimension was 179+/-65 mL at rest and incr

43 (LV) function (LV ejection fraction >50%, LV end-diastolic dimension </=70 mm, LV end-systolic dimens

44 ed as the composite of left ventricular (LV) end-diastolic dimension <33 mm/m(2) and absolute increas

45 =0.005) and a decrease of 0.7+/-0.2 cm in RV end-diastolic dimension (P<0.001) after intervention.

46 ction fraction z-score <-2) and LV dilation (end-diastolic dimension [LVEDD] z-score >2) at diagnosis

47 Baseline LV size was assessed by LV end-diastolic dimension and LV end-systolic dimension (L

48 Left ventricular end-diastolic dimension and mitral peak early filling ve

49 prominent in patients whose left ventricular end-diastolic dimension Z score before intervention is >

50 nd regression tree analysis identified an LV end-diastolic dimension z score less than -1.85 or the c

51 at patients with a baseline left ventricular end-diastolic dimension Z score of >2 exhibited a signif

52 less than -1.85 or the combination of an LV end-diastolic dimension z score of -1.85 or higher and a

53 3.98 versus -9.06+/-3.89, P<0.001) and lower end-diastolic dimension z scores (4.12+/-2.61 versus 4.9

54 wall thickness of 22+/-4 mm and nondilated (end-diastolic dimension, 39+/-7 mm).

55 assessed on measurements of left ventricular end-diastolic dimension, area, and volume.

56 ce, heart rate variability, left ventricular end-diastolic dimension, left ventricular ejection fract

57 parameters, including diastolic function, LV end-diastolic dimension, LV mass, and right ventricular

58 riables associated with reduction in MR were end-diastolic dimension, MR severity, clip location, and

59 lculated as the ratio of LV to ring size (LV end-diastolic dimension/ring size and LVESd/ring size).

60 End-diastolic elastance (Eed) was 0.14 (0.06-0.24) mm Hg

61 IUGR placentas displaying absent or reversed end-diastolic flow contained reduced myosin heavy chain,

62 t centile < 3rd/< 10th with absent umbilical end-diastolic flow, < 34 weeks).

63 cant overestimation of cardiac index, global end-diastolic index, extravascular lung water index, and

64 Global end-diastolic index, extravascular lung water index, and

65 95 near MTSS1 and rs10774625 in ATXN2 for LV end-diastolic internal dimension; rs806322 near KCNRG, r

66 On the basis of end-diastolic left ventricular pressure, 34 MI rats were

67 that adjusted for age, sex, height, weight, end-diastolic LV volume, augmentation index, end-systoli

68 model-based calculation of end-systolic and end-diastolic LV volumes.

69 reduced LV end-diastolic volume index and LV end-diastolic mass index in a large multiethnic populati

70 Irrespective of breath holding, LV end-diastolic mass was overestimated with SSIR (standard

71 ters (median: 41.2 versus 31.5; P=0.004) and end-diastolic mitral annular diameters (median: 35.5 ver

72 on LV end-diastolic pressure (LVEDP) or the end-diastolic P-V relationship (EDPVR) in sham rats.

73 4 different ways: the first was 1 gated bin (end-diastolic phase with 25% of the counts), the second

74 5 vs. 28+/-10 mmHg, P<0.001), and lowered LV end diastolic pressure (10+/-1 vs. 86+/-13 mmHg, P<0.001

75 cardiac hypertrophy, diastolic dysfunction (end diastolic pressure-volume relationship =0.051+/-0.00

76 mulation, exacerbated diastolic dysfunction (end diastolic pressure-volume relationship =0.11+/-0.004

77 ected against cardiac diastolic dysfunction (end diastolic pressure-volume relationship =0.110+/-0.00

78 ation and exacerbated diastolic dysfunction (end diastolic pressure-volume relationship =0.124+/-0.00

79 ased left ventricular developed pressure and end diastolic pressure.

80 ere RV dysfunction was indicated by elevated end-diastolic pressure (11.3+/-2.5 versus 5.7+/-2.0 mm H

81 90+/-80 versus 165+/-71 mL, P<0.0001) and LV end-diastolic pressure (14.3+/-10.2 versus 9.9+/-9.3 mm

82 5; P=0.02) and more likely to have higher RV end-diastolic pressure (HR, 1.07; 95% CI, 1.00-1.15; P=0

83 ine paradoxically decreased left ventricular end-diastolic pressure (LVEDP) and left ventricular end-

84 d diastolic volumes with little effect on LV end-diastolic pressure (LVEDP) or the end-diastolic P-V

85 nd sgridAND correlated with left ventricular end-diastolic pressure across both groups (average R(2)

86 onary artery pressure, high left ventricular end-diastolic pressure and a normal ejection fraction, s

87 ary cardiospheres decreased left ventricular end-diastolic pressure and increased cardiac output.

88 F as evidenced by increased left ventricular end-diastolic pressure and left ventricular volume index

89 ncluding heart rate, peak-systolic pressure, end-diastolic pressure and volume, end-systolic pressure

90 End-diastolic pressure correlated significantly with tis

91 Baseline noninvasive LV end-diastolic pressure correlated with invasive pulmonar

92 Although left ventricular end-diastolic pressure decreased in 45/10, it increased

93 ere further dichotomized by left ventricular end-diastolic pressure into postcapillary (left ventricu

94 l resection again blunted the increase in LV end-diastolic pressure secondary to volume expansion (+4

95 l approach would mitigate the increase in LV end-diastolic pressure that develops during volume loadi

96 th diastolic dysfunction or left ventricular end-diastolic pressure thresholds.

97 ction fraction, end-systolic volume, and the end-diastolic pressure volume relationship by Ang-(1-9)

98 ons revealed significant improvements in the end-diastolic pressure volume relationship, relaxation k

99 s, pericardiotomy blunted the increase in LV end-diastolic pressure with saline infusion, while enhan

100 (ejection fraction [EF] and left ventricular end-diastolic pressure) was assessed at days 28 and 56.

101 c pulmonary artery pressure-left ventricular end-diastolic pressure) was normal (<7 mm Hg) or elevate

102 fibrosis and left atrium diameter (marker of end-diastolic pressure), suggesting an improvement in di

103 ressure into postcapillary (left ventricular end-diastolic pressure, >15 mm Hg; n=269) and precapilla

104 9) and precapillary groups (left ventricular end-diastolic pressure, </=15 mm Hg; n=56).

105 us saturation, elevated systemic ventricular end-diastolic pressure, and elevated main pulmonary arte

106 orrelated with PH severity, left ventricular end-diastolic pressure, and left ventricular dilatation.

107 h HTN(+)HFpEF had increased left ventricular end-diastolic pressure, left atrial volume, N-terminal p

108 tion fraction) and hemodynamic variables (LV end-diastolic pressure, LV dP/dtmax, preload adjusted ma

109 TN(-)HFpEF had no change in left ventricular end-diastolic pressure, myocardial passive stiffness, co

110 Left ventricular end-diastolic pressure-guided fluid administration seems

111 equently in patients in the left ventricular end-diastolic pressure-guided group (6.7% [12/178]) than

112 action (p = 0.014) and improvement of the RV end-diastolic pressure-volume relationship in PH pigs tr

113 howed impaired relaxation and upward-shifted end-diastolic pressure-volume relationships despite pres

114 LV global longitudinal strain (LVGLS) and LV end-diastolic pressure.

115 ction, which subsequently led to elevated LV end-diastolic pressures and pulmonary hypertension.

116 n develop increases in left ventricular (LV) end-diastolic pressures during exercise that contribute

117 levant parameters, including RV systolic and end-diastolic pressures, cardiac output, RV size, and mo

118 s, had higher indexed right ventricular (RV) end-diastolic (range 85-326 mL/m(2), mean 148 mL/m(2)) v

119 Both end-systolic and end-diastolic RV volumes increased in SSc-PAH patients t

120 , in the HAART-exposed group, LV mass and LV end-diastolic septal thickness were lower whereas LV con

121 of four LV anatomic structures performed on end-diastolic short-axis cine cardiac MRI: LV trabeculat

122 Segments with end diastolic thickness >= 1.2 cm had a higher burden of

123 ameters, including mean flow velocity (MFV), end diastolic velocity (EDV), peak systolic velocity (PS

124 LV end-diastolic ventricular volumes were smaller (64 +/- 1

125 improvement in the indexed left ventricular end diastolic volume >=10% at 12 months.

126 jection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magnetic resonan

127 en by associations with baseline and DeltaLV end diastolic volume (P<0.0001 for each) and not wall th

128 increased LV weight/body weight ratio and LV end diastolic volume (WT, 50.8 mul; CatA-TG, 61.9 mul).

129 iated with higher (more adverse) LV mass, LV end diastolic volume and left atrial volume, but not wit

130 ed as the change in indexed left ventricular end diastolic volume and LVEF.

131 Right ventricular end diastolic volume increased by 49.4% after transannul

132 les of trastuzumab, indexed left ventricular end diastolic volume increased in patients treated with

133 nance to assess LV remodeling (LV mass-to-LV end diastolic volume ratio), function, tissue characteri

134 ciations of RV mass and, to a lesser extent, end diastolic volume with PM10-2.5 mass among susceptibl

135 A reduced left ventricular end diastolic volume, impaired early filling, and decrea

136 ed percentiles of LV end systolic volume, LV end diastolic volume, relative wall and septal thickness

137 n a 57% increase in LV mass (no change in LV end diastolic volume, resulting in an increase in the LV

138 s defined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

139 tein-losing enteropathy, ventricular indexed end-diastolic volume >125 mL/body surface area raised to

140 were predicted by a preoperative indexed RV end-diastolic volume </=158 mL/m(2) and RVESVi </=82 mL/

141 enhancing the saline-mediated increase in LV end-diastolic volume (+17+/-1 versus +10+/-2 mL; P=0.016

142 Fluid loading increased right ventricular end-diastolic volume (+31 +/- 13 mL; p = 0.004), right v

143 terms of end-systolic volume (0 +/- 3.3 ml), end-diastolic volume (- 0.4 +/- 2.0 ml) and ejection fra

144 , a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and end-systolic

145 o -3.80; P<0.00001), and tended to reduce LV end-diastolic volume (-2.26 mL; 95% confidence interval,

146 ssociated with a significant reduction of LV end-diastolic volume (-25.1 +/- 26.0 ml vs. -1.5 +/- 25.

147 s 87.0%) and improvement in left ventricular end-diastolic volume (-8.0 mL versus -12.7 mL), whereas

148 uretic treatment decreased right ventricular end-diastolic volume (-84 +/- 11 mL; p < 0.001), right v

149 grade was mild with significant decreases in end-diastolic volume (139 to 107 mL; P=0.03) and left at

150 Reduced pump speed caused increased LV end-diastolic volume (190+/-80 versus 165+/-71 mL, P<0.0

151 Patisiran also led to increased end-diastolic volume (8.3+/-3.9 mL, P=0.036), decreased

152 terol was causally associated with higher LV end-diastolic volume (beta = 1.85 ml; 95% confidence int

153 ss (beta=1.218; adjusted P=0.007), higher LV end-diastolic volume (beta=0.811; adjusted P=0.007), hig

154 4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95% confidence

155 LV end-diastolic volume (EDV) and stroke volume (SV) were d

156 analyzed to provide the reference limits for end-diastolic volume (EDV), end-systolic volume (ESV), e

157 End-diastolic volume (EDV), end-systolic volume (ESV), s

158 rs, including the end-systolic volume (ESV), end-diastolic volume (EDV), stroke volume (SV), and ejec

159 ic volume (ESV: Pearson r = 0.99, P < .001), end-diastolic volume (EDV: r = 0.97, P < .001), and ejec

160 g LV strain (HR, 1.63; P=0.005), exercise LV end-diastolic volume (HR, 1.38; P=0.048), and resting RV

161 r end-diastolic volume and right ventricular end-diastolic volume (left ventricular end-diastolic vol

162 lts were obtained with MRI: Left ventricular end-diastolic volume (LVDV) was 40 mL (LVDV per body sur

163 ut off values for change in left ventricular end-diastolic volume (LVEDV) and LV end-systolic volume

164 0% to 50% or greater, whose left ventricular end-diastolic volume (LVEDV) had normalised, and who had

165 t, among patients with available baseline LV end-diastolic volume (LVEDV) measures, 188 received bive

166 c performance according to the median PET LV end-diastolic volume (LVEDV), with smaller LVs defined a

167 CMR showed higher left ventricle end-diastolic volume (mean difference: 43+/-22.5 mL), hi

168 resonance (1.5 T) to measure RV mass (g), RV end-diastolic volume (mL), RV mass/volume ratio, and LV

169 on, and IMH correlated with the change in LV end-diastolic volume (Pearson's rho of 0.64, 0.59, and 0

170 stolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats, which was no

171 cardial lidocaine paradoxically decreased LV end-diastolic volume (preload) in CHF rats, which was no

172 rrected QRS area for heart size using the LV end-diastolic volume (QRSarea/LVEDV).

173 (PRF) nor PR volume (PRV) correlated with RV end-diastolic volume (r = 0.36; p = 0.15 and r = 0.37; p

174 = 0.95), end-systolic volume (r = 0.93), and end-diastolic volume (r = 0.90), and slightly lower corr

175 ed on LV mass index and the ratio of LV mass:end-diastolic volume (relative wall mass).

176 Right ventricular end-diastolic volume (RVDV) was 262 mL (RVDV/BSA, 164 mL

177 Right ventricular end-diastolic volume (RVEDV) was reduced in COPD compare

178 ed more favorable remodeling over 1 year (LV end-diastolic volume =157+/-34 to 150+/-38 mL) compared

179 assification based on LV dilatation (high LV end-diastolic volume [EDV] index) and concentricity (mas

180 scular events and a dilated LV (increased LV end-diastolic volume [EDV] indexed to body surface area)

181 volume [EDV] index) and concentricity (mass/end-diastolic volume [M/EDV](2/3)) in hypertensive patie

182 es were calculated from echocardiographic LV end-diastolic volume accounting for the integral of pump

183 rdiac function, measured by left ventricular end-diastolic volume and cardiac output.

184 of women were classified as having abnormal end-diastolic volume and internal diameter by ASE 2015 c

185 iminated the progressive deterioration in LV end-diastolic volume and LV end-systolic volume.

186 Indexed left ventricular end-diastolic volume and mass correlated with O2max (r=0

187 /m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular end-diastolic

188 During PEI, LV end-diastolic volume and stroke volume were increased in

189 s, there is a linear relationship between LV end-diastolic volume and the effective regurgitant orifi

190 ndent relationship between GLS groups and LV end-diastolic volume at 3 and 6 months (adjusted for cli

191 ty indices ([RA+aRV]/[fRV+LA+LV]) and fRV/LV end-diastolic volume corresponded only to some parameter

192 In both men and women, LV end-diastolic volume decreased (-9.8 and -13.3 mL per de

193 Left ventricular end-diastolic volume decreased (from 125.1 +/- 40.1 ml t

194 l adjustment for city attenuated the RV mass/end-diastolic volume findings.

195 mL [95% CI, 28.2-45.8]) and reduction in LV end-diastolic volume from 171.0 to 143.2 mL (difference,

196 ncrease and dilatation, but left ventricular end-diastolic volume improved because of reduced blood r

197 baseline to 12 months, left ventricular (LV) end-diastolic volume improved from 161 +/- 56 ml to 143

198 se; P<0.001), BMI was not correlated with RV end-diastolic volume in men (R=0.04; P=0.51).

199 (n = 167), and after replacing LV mass by LV end-diastolic volume in the regression models.

200 surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >

201 Patients with transplants had lower end-diastolic volume index (59.3+/-15.2 ml/m(2) vs. 71.4

202 0.0001), obesity (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001),

203 The left atrial end-diastolic volume index (LAEDVI), representing the mi

204 outcome was mean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

205 centrations and left ventricular (LV) EF, LV end-diastolic volume index (LVEDVI), LV end-systolic vol

206 small, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant

207 all stable increase in the right ventricular end-diastolic volume index (P<0.001).

208 olume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

209 se in change from baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) redu

210 Left ventricle end-diastolic volume index and end-systolic volume index

211 Greater ECE is associated with reduced LV end-diastolic volume index and LV end-diastolic mass ind

212 ces between groups in baseline values for LV end-diastolic volume index and LV end-systolic volume in

213 Empagliflozin reduced LV end-diastolic volume index by 8.2 (95% CI, -13.7 to -2.6

214 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m

215 ndex threshold of 227% or a left ventricular end-diastolic volume index of 58 ml/m(2) identified pati

216 CMR-derived end-diastolic volume index showed a weaker association w

217 had a greater extent of LGE and a higher LV end-diastolic volume index than other groups, but levels

218 as change from baseline in right ventricular end-diastolic volume index versus placebo.

219 ith improvements in LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume i

220 ET sympathetic denervation, left ventricular end-diastolic volume index, creatinine, and no angiotens

221 edictors (age, body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.0

222 rdiovascular magnetic resonance measures (LV end-diastolic volume index, LV ejection fraction), diure

223 and 87.3+/-18.7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volum

224 , left atrial volume index, left ventricular end-diastolic volume index, peak E wave, and the presenc

225 iuretic peptide, and larger left ventricular end-diastolic volume index.

226 al volume index, and larger left ventricular end-diastolic volume index.

227 maximal NC/C ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area

228 LV ejection fraction (LVEF) and left atrial end-diastolic volume indexed to body surface area, were

229 /e' ratio, left ventricular end-systolic and end-diastolic volume indexes (LVESVI and LVEDVI), left a

230 CMR-derived ventricular indexed end-diastolic volume is an independent predictor of tran

231 tal right/left-volume index was defined from end-diastolic volume measurements in CMR: total right/le

232 RV end-systolic volumes (RVESVi) (indexed RV end-diastolic volume pPVR versus immediately after PVR v

233 dditional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and trended toward i

234 (95% CI: 0.04, 0.36)] and a greater RV mass/end-diastolic volume ratio conditional on LV parameters.

235 associated with greater RV mass and RV mass/end-diastolic volume ratio conditional on the LV; howeve

236 associated with greater RV mass and RV mass/end-diastolic volume ratio relative to the LV.

237 measures of PM2.5, whereas those for RV mass/end-diastolic volume ratio were attenuated.

238 were directly associated with LV mass to LV end-diastolic volume ratio, a marker of cardiac remodell

239 V ejection fraction, LV mass, and LV mass to end-diastolic volume ratio.

240 lume, LV end-systolic volume, and LV mass to end-diastolic volume ratio; 4 loci for LV ejection fract

241 tion fraction, and indexed right ventricular end-diastolic volume resulted in significant improvement

242 fraction was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

243 (LV) ejection fraction was 31.3 +/- 9.3%, LV end-diastolic volume was 192.7 +/- 71 ml, and effective

244 LA end-diastolic volume was increased in patients with mitr

245 infarct size, LV end-systolic volume, and LV end-diastolic volume were analyzed with random-effects m

246 ssociated with greater RV mass and larger RV end-diastolic volume with or without further adjustment

247 ed normal values, left and right ventricular end-diastolic volume z scores were mildly enlarged (0.48

248 eas women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI point increase; P<

249 004) with stable LV chamber volumes (DeltaLV end-diastolic volume, 1+/-1 mL/m(2); P=0.48).

250 acterized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 172+/-28 mL, P<0.0

251 creased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher plasma volume,

252 28.6+/-4.3 cm(2); P=0.02), and left atrium (end-diastolic volume, 65+/-19 versus 72+/-19; P=0.02).

253 pertrophy was driven by LV dilation (DeltaLV end-diastolic volume, 9+/-3 mL/m(2); P=0.004) with stabl

254 in RV ejection fraction and left ventricular end-diastolic volume, although correlation coefficients

255 We quantified associations with RV mass, end-diastolic volume, and ejection fraction after contro

256 demographic data, risk factors for scar, LV end-diastolic volume, and LV mass.

257 rdiovascular risk factors, calcium score, LV end-diastolic volume, and mass in addition to resting he

258 associated with preserved stroke volume, LV end-diastolic volume, and mass/volume ratio as measured

259 al effective regurgitant orifice, indexed LV end-diastolic volume, and right ventricular systolic pre

260 dary efficacy end points included changes in end-diastolic volume, end-systolic volume, and ejection

261 Offline analysis generated 3-dimensional end-diastolic volume, ESV, SV, and free-wall RV longitud

262 periencing MACE showed higher left ventricle end-diastolic volume, higher left ventricle end-systolic

263 ts underwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-systolic volume

264 d improvements in LV end-systolic volume, LV end-diastolic volume, left ventricular ejection fraction

265 me-wide significant loci (3 loci each for LV end-diastolic volume, LV end-systolic volume, and LV mas

266 ined percent change in left ventricular (LV) end-diastolic volume, LV end-systolic volume, LV ejectio

267 e-wide association studies of 6 LV traits-LV end-diastolic volume, LV end-systolic volume, LV stroke

268 Automatic quantification of LV end-diastolic volume, LV myocardium mass, LV trabeculati

269 .96%; 95% confidence interval, -0.2 to 2.1), end-diastolic volume, or systolic volume were observed c

270 enhancing the saline-mediated increase in LV end-diastolic volume.

271 ter adjustment for baseline left ventricular end-diastolic volume.

272 rease of 15% or more in the left ventricular end-diastolic volume.

273 elated with the postoperative decrease in LV end-diastolic volume.

274 in left ventricular end-systolic volume and end-diastolic volume.

275 tion fraction, and indexed right ventricular end-diastolic volume.

276 End-systolic volume and end-diastolic volume/body surface area (BSA) were 62 (45

277 cular end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+/-13 and 69+

278 69+/-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+/-22 and 66+

279 Eed increased with increasing RV mass/BSA, end-diastolic volume/BSA, and T1 mapping and with decrea

280 Ees/Ea decreased with increasing RV end-diastolic volume/BSA, mass/BSA, and pulmonary arteri

281 olumes were significantly smaller in DM, (LV end-diastolic volume/m(2): -3.46 mL/m(2) [-5.8 to -1.2],

282 ) [-5.8 to -1.2], P=0.003, right ventricular end-diastolic volume/m(2): -4.2 mL/m(2) [-6.8 to -1.7],

283 lume: -4.3 [11.3] versus 7.4 [11.8], P=0.02; end-diastolic volume: -9.1 [14.9] versus 7.4 [15.8], P=0

284 size (Delta ejection fraction: P<0.04, Delta end-diastolic volume: P<0.02, Delta end-systolic volume:

285 remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P=0.004).

286 ge of 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x to 2.5x those

287 ths of exercise training (LA volumes 55%; LV end diastolic volumes 15% at 24 months versus baseline;

288 In the exercise group, both LA and LV end diastolic volumes increased proportionately (19% and

289 The LA:LV end diastolic volumes ratio did not change from baseline

290 ection fraction (P<0.01), reduced stroke and end-diastolic volumes (both P<0.001), decreased peak E'

291 mediate and midterm reductions in indexed RV end-diastolic volumes and RV end-systolic volumes (RVESV

292 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2), 108 +/- 2

293 End-systolic and end-diastolic volumes were calculated using blood pool i

294 creased significantly during exercise, while end-diastolic volumes were essentially unchanged.

295 ing demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes, stroke volu

296 (LV) ejection fraction, LV end-systolic and end-diastolic volumes, infarct size, and major adverse c

297 all thickening fraction, LV end-systolic and end-diastolic volumes, LV ejection fraction) and hemodyn

298 Significant decreases in LV end-diastolic volumes, LV end-systolic volumes, cardiac

299 As a result, LV end-diastolic wall thickness-to-chamber radius (h/R) rat

300 an age (1.6 vs. 1.7 years), left ventricular end-diastolic z-scores (+4.2 vs. +4.2), and left ventric