ライフサイエンスコーパス: end-diastolic dimension

1 proved fractional shortening and ventricular end-diastolic dimensions.

2 was documented by long-term normalization of end-diastolic dimensions.

3 B6 mice had significantly larger end-diastolic dimension (3.31+/-0.42 mm versus 2.83+/-0.

4 wall thickness of 22+/-4 mm and nondilated (end-diastolic dimension, 39+/-7 mm).

5 nventional measures of LV systolic function (end-diastolic dimension, 42 +/- 6 mm; ejection fraction,

6 ved (CSQ vs. CSQ/betaARKct, left ventricular end diastolic dimension 5.60 +/- 0.17 mm vs. 4.19 +/- 0.

7 <0.0001) and increased left-ventricular (LV) end-diastolic dimension (9.7+/-0.2 mm versus 7.0+/-0.4 m

8 e for ejection fraction and left ventricular end-diastolic dimension [95% each]; the lowest proportio

9 onal area change), preload (left ventricular end-diastolic dimension), afterload (end-systolic wall s

10 , congestive heart failure, and increased LV end-diastolic dimension among those with myocarditis (n=

11 o predict postoperative systolic stress from end-diastolic dimension and EF.

12 Baseline LV size was assessed by LV end-diastolic dimension and LV end-systolic dimension (L

13 Left ventricular end-diastolic dimension and mitral peak early filling ve

14 produced decreases in left ventricular (LV) end-diastolic dimension and MR at 2 years (p < 0.001); C

15 After 7 days of SVT, LV end-diastolic dimension and myocyte length both increase

16 hange in loading conditions as defined by LV end-diastolic dimension and total peripheral resistance

17 group II, both p < 0.05), but the changes in end-diastolic dimension and wall stress differed.

18 cantly smaller increases in end-systolic and end-diastolic dimensions and areas at both midpapillary

19 nuated the increase in left ventricular (LV) end-diastolic dimensions and impairment in LV systolic p

20 Left ventricular end-systolic and end-diastolic dimensions and left ventricular end-systol

21 related significantly with right ventricular end-diastolic dimensions and severity of pulmonary valve

22 % increase in LV/BW, a 0.2 mm decrease in LV end diastolic dimension, and no change in fractional sho

23 In linear regression models, LV mass, end-diastolic dimension, and septal and posterior wall t

24 assessed on measurements of left ventricular end-diastolic dimension, area, and volume.

25 2%; AC6-KO: 52+/-4%; p<0.001) and reduced LV end-diastolic dimension (CON: 4.6+/-0.1 mm; AC6-KO: 3.6+

26 mean left ventricular fractional shortening, end-diastolic dimension, contractility, or mass in eithe

27 Patients were categorized as those in whom end-diastolic dimension declined after the operation (gr

28 nal shortening was increased (33 +/- 6%) and end-diastolic dimension decreased (2.02 +/- 0.30 cm) com

29 In group Ib, end-diastolic dimension decreased and systolic wall stre

30 End-diastolic dimension decreased only in NCX.

31 as a trend toward increased left ventricular end-diastolic dimension during ITD use.

32 rt rate, blood pressure, or left ventricular end-diastolic dimension, each of which had a coefficient

33 cluded left ventricular mass, contractility, end-diastolic dimension, fractional shortening, blood pr

34 The patient's left ventricular end-diastolic dimension, frequency of aortic valve openi

35 shown by reductions in the left ventricular end-diastolic dimension from 59+/-8 to 52+/-6 mm (P</=0.

36 The increase in end-diastolic dimension from day 1 to week 4 was 3.1+/-0

37 ed by any of four methods), left ventricular end-diastolic dimension greater than 6.5 to 7 cm, a rest

38 up I, n = 15) and those with no reduction in end-diastolic dimension (group II, n = 6).

39 heart failure, QRS duration > or =120 ms, LV end-diastolic dimension > or =55 mm, and LV ejection fra

40 ccompanied by a decrease in left ventricular end-diastolic dimension >/=10% at 12 months of follow-up

41 ecause of a reduction in LV end-systolic and end-diastolic dimensions in the stentless group.

42 ed after RVP (17 +/- 5 versus 42 +/- 3%) and end-diastolic dimension increased (2.36 +/- 0.44 versus

43 ing was reduced (19+/-1 versus 45+/-1%), and end-diastolic dimension increased (5.67+/-0.11 versus 3.

44 Left ventricular end-diastolic dimension increased 20% as plasma Ang II l

45 ft ventricular enlargement (left ventricular end-diastolic dimension increased from 1.43+/-0.03 to 1.

46 cardiography decreased (-23.6+/-2.0%) and LV end-diastolic dimension) increased (+10.9+/-1.0%), where

47 (n=161; 48%) was associated with pre-CRT LV end-diastolic dimension index <3.1 cm/m(2), global longi

48 In particular, end-diastolic dimension indexed to BSA was greater in wo

49 an increment of 1 SD in the left ventricular end-diastolic dimension, indexed for height.

50 ce, heart rate variability, left ventricular end-diastolic dimension, left ventricular ejection fract

51 (LV) function (LV ejection fraction >50%, LV end-diastolic dimension </=70 mm, LV end-systolic dimens

52 ed as the composite of left ventricular (LV) end-diastolic dimension <33 mm/m(2) and absolute increas

53 inol attenuated decreased FS and elevated LV end-diastolic dimension, LV end-systolic dimension, and

54 parameters, including diastolic function, LV end-diastolic dimension, LV mass, and right ventricular

55 S) fell (13.4+/-1.4% versus 39.1+/-1.0%) and end-diastolic dimension (LVEDD) increased (5.61+/-0.11 v

56 not significantly improve cardiac output, LV end-diastolic dimension (LVEDD) or LVEDD/wall thickness

57 At 2 days after ACF, LV end-diastolic dimension (LVEDD)/wall thickness was incre

58 ction fraction z-score <-2) and LV dilation (end-diastolic dimension [LVEDD] z-score >2) at diagnosis

59 [4.2%]; P = .003), smaller left ventricular end-diastolic dimension (median [IQR], 48 [46-52] vs 51

60 riables associated with reduction in MR were end-diastolic dimension, MR severity, clip location, and

61 ients experienced a substantial reduction in end-diastolic dimension, no change in EF and a reduction

62 DHF patients had EF of 58+/-8%, end-diastolic dimension of 50+/-10 mm, estimated resting

63 n contrast, SHF patients had EF of 24+/-10%, end-diastolic dimension of 68+/-11 mm, ePAD of 18+/-7 mm

64 ); Coapsys provided a greater decrease in LV end-diastolic dimension (p = 0.021).

65 =0.005) and a decrease of 0.7+/-0.2 cm in RV end-diastolic dimension (P<0.001) after intervention.

66 rrelated significantly with left ventricular end-diastolic dimension (p=0.0092), and inversely with e

67 significant reduction in LV end-systolic and end-diastolic dimensions (P<0.001).

68 linear function slopes for left ventricular end-diastolic dimension, pulsatility index, and power we

69 ears, and the LV posterior wall thickness to end-diastolic dimension ratio <0.14.

70 lculated as the ratio of LV to ring size (LV end-diastolic dimension/ring size and LVESd/ring size).

71 he remaining patients had a left ventricular end-diastolic dimension slope >-0.16.

72 was used to define the relations between LV end-diastolic dimension, systolic wall stress and EF.

73 e predicted from a mathematic model relating end-diastolic dimension to EF and systolic wall stress.

74 terial elastance), L-NMMA increased preload (end-diastolic dimension) to a lesser degree (3.8%+/-1.5%

75 significant improvements in left ventricular end-diastolic dimension (vehicle, 4.7+/-0.1 mm; IDN-1965

76 Left-ventricular end diastolic dimension was 179+/-65 mL at rest and incr

77 n fraction was 0.50+/-0.16, left ventricular end-diastolic dimension was 5.0+/-0.9 cm, and left atria

78 The average left ventricular end-diastolic dimension was 61.4 mm for affected and 48.

79 ually with both end points, but increased LV end-diastolic dimension was associated only with transpl

80 For idiopathic DCM, increased LV end-diastolic dimension was associated with increased tr

81 At 8 weeks after MI, LV end-diastolic dimension was lower with MMPi than in the

82 nsistently, the increase in left ventricular end-diastolic dimension was of lesser magnitude (+0.47 v

83 rdial shortening increased (25+/-2%) and the end-diastolic dimension was reduced (4.92+/-0.17 cm) com

84 olic wall stress was unchanged; in group II, end-diastolic dimension was unchanged and wall stress in

85 II; c) the decrease rate of left ventricular end-diastolic dimensions was greater in Group III than i

86 age, body surface area, preoperative FSz and end-diastolic dimension were not.

87 ed but not in G-CSF-treated mice, whereas LV end-diastolic dimensions were smaller in all three group

88 onal shortening, end-systolic dimension, and end-diastolic dimension with local PDGF delivery (P < 0.

89 ace (P<0.0001), and a lower left ventricular end-diastolic dimension z score at presentation (P=0.04)

90 prominent in patients whose left ventricular end-diastolic dimension Z score before intervention is >

91 nd regression tree analysis identified an LV end-diastolic dimension z score less than -1.85 or the c

92 at patients with a baseline left ventricular end-diastolic dimension Z score of >2 exhibited a signif

93 less than -1.85 or the combination of an LV end-diastolic dimension z score of -1.85 or higher and a

94 In contrast, increased LV end-diastolic dimension z score was associated only with

95 monstrated progressive increases in their LV end-diastolic dimension Z score within the first 3 years

96 3.98 versus -9.06+/-3.89, P<0.001) and lower end-diastolic dimension z scores (4.12+/-2.61 versus 4.9

97 Similarly, left ventricular (LV) end-diastolic dimension z scores, which ranged from -5 t