ライフサイエンスコーパス: end-diastolic volume

1 to pump failure revealed a rapid decrease in end diastolic volume.

2 ter adjustment for baseline left ventricular end-diastolic volume.

3 rease of 15% or more in the left ventricular end-diastolic volume.

4 elated with the postoperative decrease in LV end-diastolic volume.

5 ssociated with greater RV mass and larger RV end-diastolic volume.

6 in left ventricular end-systolic volume and end-diastolic volume.

7 tion fraction, and indexed right ventricular end-diastolic volume.

8 enhancing the saline-mediated increase in LV end-diastolic volume.

9 Allogeneic MSCs reduced LV end-diastolic volumes.

10 T was associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.01), a lower left

11 e ratio of elevated left ventricular mass to end-diastolic volume (0.02 g/mL per unit).

12 terms of end-systolic volume (0 +/- 3.3 ml), end-diastolic volume (- 0.4 +/- 2.0 ml) and ejection fra

13 ned as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D implantation) an

14 004) with stable LV chamber volumes (DeltaLV end-diastolic volume, 1+/-1 mL/m(2); P=0.48).

15 eas women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI point increase; P<

16 n (45% vs. 56%; p < 0.001), increased median end-diastolic volume (100 ml/body surface area [BSA](1.3

17 dilated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with good systoli

18 /-4 and 65+/-4%; P<0.05), greater indexed LV end-diastolic volume (102+/-34 versus 84+/-14 and 85+/-1

19 s not associated with changes in LV volumes (end-diastolic volume 106 +/- 15 versus 110 +/- 22 mL; en

20 compared with patients with GLS</=-15.0% (LV end-diastolic volume 123+/-44 versus 106+/-36 mL and 121

21 4), and increased echocardiographic volumes (end-diastolic volume, 126+/-39 versus 112+/-33 mL/BSA(1.

22 grade was mild with significant decreases in end-diastolic volume (139 to 107 mL; P=0.03) and left at

23 A significant reduction of LV end-diastolic volume (149.7 +/- 41.4-140.1 +/- 43.9 mL;

24 ths of exercise training (LA volumes 55%; LV end diastolic volumes 15% at 24 months versus baseline;

25 e was a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence interval, 3-

26 acterized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 172+/-28 mL, P<0.0

27 ed more favorable remodeling over 1 year (LV end-diastolic volume =157+/-34 to 150+/-38 mL) compared

28 crease of 15 mL at >500 ng/mL, p=0.0026) and end-diastolic volumes (16 mL, p=0.0096) that might have

29 enhancing the saline-mediated increase in LV end-diastolic volume (+17+/-1 versus +10+/-2 mL; P=0.016

30 , a significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and end-systolic

31 Reduced pump speed caused increased LV end-diastolic volume (190+/-80 versus 165+/-71 mL, P<0.0

32 o -3.80; P<0.00001), and tended to reduce LV end-diastolic volume (-2.26 mL; 95% confidence interval,

33 creased E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher plasma volume,

34 CMR at 1 year demonstrated a decrease in end diastolic volume (208.7+/-20.4 versus 167.4+/-7.32 m

35 remodeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P=0.004).

36 (178 +/- 72 to 145 +/- 23 ml, p < 0.001), LV end-diastolic volume (242 +/- 85 ml to 212 +/- 63 ml, p

37 ssociated with a significant reduction of LV end-diastolic volume (-25.1 +/- 26.0 ml vs. -1.5 +/- 25.

38 ding 22 years were inversely associated with end-diastolic volume (-3.0 mL per unit mean hemoglobin A

39 Fluid loading increased right ventricular end-diastolic volume (+31 +/- 13 mL; p = 0.004), right v

40 ly related to reductions in left ventricular end-diastolic volume (-4.1 ml; 95% confidence interval [

41 nterval, -11.57 to -6.25; P<0.00001), and LV end-diastolic volume (-5.23 mL; 95% confidence interval,

42 Older age was associated with lower volumes (end-diastolic volume, -5 mLdecade; end-systolic volume,

43 Changes in end-diastolic volume (-6+/-14 versus -4+/-16 mL; P=0.67)

44 ignificant attenuation in the increase in LV end-diastolic volume (64.8 mL [60.7-71.3] to 83.5 mL [74

45 28.6+/-4.3 cm(2); P=0.02), and left atrium (end-diastolic volume, 65+/-19 versus 72+/-19; P=0.02).

46 jects in the highest quintile had smaller LV end-diastolic volumes (68+/-13 versus 58+/-12 mL/m(2); P

47 ts/min; p = 0.03), but no difference in peak end-diastolic volume (77 +/- 18 ml vs. 77 +/- 17 ml; p =

48 was associated with a small right ventricle (end diastolic volume, 79.8+/-13.2 versus 88.5+/-11.8 mL/

49 Patisiran also led to increased end-diastolic volume (8.3+/-3.9 mL, P=0.036), decreased

50 s 87.0%) and improvement in left ventricular end-diastolic volume (-8.0 mL versus -12.7 mL), whereas

51 had more than mild FMR despite increased LV end-diastolic volume (82 +/- 22, 86 +/- 23, and 51 +/- 1

52 uretic treatment decreased right ventricular end-diastolic volume (-84 +/- 11 mL; p < 0.001), right v

53 pertrophy was driven by LV dilation (DeltaLV end-diastolic volume, 9+/-3 mL/m(2); P=0.004) with stabl

54 lume: -4.3 [11.3] versus 7.4 [11.8], P=0.02; end-diastolic volume: -9.1 [14.9] versus 7.4 [15.8], P=0

55 es were calculated from echocardiographic LV end-diastolic volume accounting for the integral of pump

56 in RV ejection fraction and left ventricular end-diastolic volume, although correlation coefficients

57 LV end-diastolic volume, an index of LV remodeling, increas

58 iated with higher (more adverse) LV mass, LV end diastolic volume and left atrial volume, but not wit

59 ed as the change in indexed left ventricular end diastolic volume and LVEF.

60 ype and G6PDX mice but significantly greater end diastolic volume and wall thinning in G6PDX mice.

61 rdiac function, measured by left ventricular end-diastolic volume and cardiac output.

62 of the right ventricle were used to measure end-diastolic volume and ejection fraction.

63 MRI results showed a decrease of indexed RV end-diastolic volume and end-systolic volume (98 ml/m(2)

64 RV volumes (end-diastolic volume and end-systolic volume), stroke vo

65 of women were classified as having abnormal end-diastolic volume and internal diameter by ASE 2015 c

66 with placebo did not change left ventricular end-diastolic volume and left ventricular mass nor did i

67 iminated the progressive deterioration in LV end-diastolic volume and LV end-systolic volume.

68 Indexed left ventricular end-diastolic volume and mass correlated with O2max (r=0

69 /m(2); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular end-diastolic

70 During PEI, LV end-diastolic volume and stroke volume were increased in

71 s, there is a linear relationship between LV end-diastolic volume and the effective regurgitant orifi

72 mediate and midterm reductions in indexed RV end-diastolic volumes and RV end-systolic volumes (RVESV

73 We quantified associations with RV mass, end-diastolic volume, and ejection fraction after contro

74 54 +/- 13 years of age), noncontrast 3D EF, end-diastolic volume, and end-systolic volume had signif

75 re, 6-minute walk distance, left ventricular end-diastolic volume, and left ventricular ejection frac

76 demographic data, risk factors for scar, LV end-diastolic volume, and LV mass.

77 rdiovascular risk factors, calcium score, LV end-diastolic volume, and mass in addition to resting he

78 associated with preserved stroke volume, LV end-diastolic volume, and mass/volume ratio as measured

79 al effective regurgitant orifice, indexed LV end-diastolic volume, and right ventricular systolic pre

80 Improvement in end-diastolic volume at 1 year was predictive of subsequ

81 ndent relationship between GLS groups and LV end-diastolic volume at 3 and 6 months (adjusted for cli

82 independently of load/extrinsic forces, the end-diastolic volume at a common end-diastolic pressure

83 reased LV compliance, measured as reduced LV end-diastolic volume at an idealized LV end-diastolic pr

84 terol was causally associated with higher LV end-diastolic volume (beta = 1.85 ml; 95% confidence int

85 centric remodeling characterized by lower LV end-diastolic volume (beta=-0.21), higher concentricity

86 on (beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0.0001), and left

87 bcutaneous fat was associated with higher LV end-diastolic volume (beta=0.48), reduced concentricity

88 ss (beta=1.218; adjusted P=0.007), higher LV end-diastolic volume (beta=0.811; adjusted P=0.007), hig

89 End-systolic volume and end-diastolic volume/body surface area (BSA) were 62 (45

90 cular end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+/-13 and 69+

91 69+/-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+/-22 and 66+

92 endpoint variables for primary outcomes: LV end-diastolic volume/body surface area, LV ejection frac

93 ection fraction (P<0.01), reduced stroke and end-diastolic volumes (both P<0.001), decreased peak E'

94 7.9 [7.3-28.5], then decreased by 14 months (end-diastolic volume/BSA(1.3), end-systolic volume/BSA(1

95 Eed increased with increasing RV mass/BSA, end-diastolic volume/BSA, and T1 mapping and with decrea

96 Ees/Ea decreased with increasing RV end-diastolic volume/BSA, mass/BSA, and pulmonary arteri

97 tly higher ratio of left ventricular mass to end-diastolic volume (by 8%).

98 4.02 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95% confidence

99 ty indices ([RA+aRV]/[fRV+LA+LV]) and fRV/LV end-diastolic volume corresponded only to some parameter

100 In both men and women, LV end-diastolic volume decreased (-9.8 and -13.3 mL per de

101 Left ventricular end-diastolic volume decreased (from 125.1 +/- 40.1 ml t

102 Remodeling was defined as an increase in LV end-diastolic volume (DeltaEDV>0) between discharge and

103 The mean left ventricular end systolic and end diastolic volumes did not differ between the groups.

104 T intensive versus conventional treatment in end diastolic volume (EDV), end systolic volume, stroke

105 tolic (ePAD) pressures and an increase in LV end diastolic volume (EDV).

106 2.6 mL of CHAM (n=5) or saline (n=13) and LV end-diastolic volume (EDV) and MMP/TIMP profiles in the

107 LV end-diastolic volume (EDV) and stroke volume (SV) were d

108 +/- 13.0 mL; P < 0.0001), difference between end-diastolic volume (EDV) at rest and stress (DeltaEDV[

109 n left ventricular ejection fraction (LVEF), end-diastolic volume (EDV), and end-systolic volume (ESV

110 global function was assessed by measuring LV end-diastolic volume (EDV), end-systolic volume (ESV), a

111 e overall bias and limits of agreement of LV end-diastolic volume (EDV), end-systolic volume (ESV), a

112 analyzed to provide the reference limits for end-diastolic volume (EDV), end-systolic volume (ESV), e

113 End-diastolic volume (EDV), end-systolic volume (ESV), s

114 rs, including the end-systolic volume (ESV), end-diastolic volume (EDV), stroke volume (SV), and ejec

115 eft ventricular ejection fraction (LVEF) and end-diastolic volume (EDV).

116 ic volume (ESV: Pearson r = 0.99, P < .001), end-diastolic volume (EDV: r = 0.97, P < .001), and ejec

117 Filling was assessed by changes in LV end-diastolic volume (EDV; conductance catheter techniqu

118 LV end-diastolic volumes (EDV) were measured by echocardiog

119 assification based on LV dilatation (high LV end-diastolic volume [EDV] index) and concentricity (mas

120 scular events and a dilated LV (increased LV end-diastolic volume [EDV] indexed to body surface area)

121 ocardiographic measures of EF: cardiac size (end-diastolic volume [EDV]); contractile function (the e

122 dary efficacy end points included changes in end-diastolic volume, end-systolic volume, and ejection

123 However, changes in end-diastolic volume, end-systolic volume, and LVEF did

124 Biventricular end-diastolic volume, end-systolic volume, stroke volume

125 ing demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes, stroke volu

126 Offline analysis generated 3-dimensional end-diastolic volume, ESV, SV, and free-wall RV longitud

127 l adjustment for city attenuated the RV mass/end-diastolic volume findings.

128 greater than 20% relative increase in the LV end-diastolic volume from 1 to 12 wk (percentage injecte

129 mL [95% CI, 28.2-45.8]) and reduction in LV end-diastolic volume from 171.0 to 143.2 mL (difference,

130 - 5.8 mm Hg (p = 0.016) and a decrease in LV end-diastolic volume from 172 +/- 37 ml to 158 +/- 38 ml

131 follow-up documented a stepwise reduction in end-diastolic volume (from 147 mL [IQR, 95-191 mL] to 12

132 improvement in the indexed left ventricular end diastolic volume >=10% at 12 months.

133 tion >/=10 U, combined with a decrease in LV end-diastolic volume >/=10% at follow-up.

134 lization for heart failure, left ventricular end-diastolic volume >/=125 mL/m(2), and left atrial vol

135 s defined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

136 tein-losing enteropathy, ventricular indexed end-diastolic volume >125 mL/body surface area raised to

137 CMR-derived left ventricular end-diastolic volume >246 mL had good, although lower, d

138 ng tachycardia, low blood pressure, enlarged end-diastolic volume, high ejection fraction, and high c

139 periencing MACE showed higher left ventricle end-diastolic volume, higher left ventricle end-systolic

140 g LV strain (HR, 1.63; P=0.005), exercise LV end-diastolic volume (HR, 1.38; P=0.048), and resting RV

141 A reduced left ventricular end diastolic volume, impaired early filling, and decrea

142 ncrease and dilatation, but left ventricular end-diastolic volume improved because of reduced blood r

143 baseline to 12 months, left ventricular (LV) end-diastolic volume improved from 161 +/- 56 ml to 143

144 Left ventricular end-diastolic volume improved from 172 ml to 140 ml and

145 Cine MRI demonstrated a >40% increase in LV end-diastolic volume in both groups, consistent with a f

146 clusion initially induced an expansion of LV end-diastolic volume in IPAH (+7%; P < 0.05), whereas en

147 se; P<0.001), BMI was not correlated with RV end-diastolic volume in men (R=0.04; P=0.51).

148 (n = 167), and after replacing LV mass by LV end-diastolic volume in the regression models.

149 remental value of LVGLS for prediction of LV end-diastolic volume increase (0.14 [95% confidence inte

150 Right ventricular end diastolic volume increased by 49.4% after transannul

151 les of trastuzumab, indexed left ventricular end diastolic volume increased in patients treated with

152 In the exercise group, both LA and LV end diastolic volumes increased proportionately (19% and

153 After intervention, LV mass and end-diastolic volume increased and exercise capacity imp

154 control animals, LV end-systolic volume and end-diastolic volume increased from 6 to 30 weeks (media

155 Subsequently, RV end-systolic and end-diastolic volume increased more in both groups, resu

156 to the Frank-Starling relationship, greater end-diastolic volume increases ventricular output.

157 surgery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >

158 ravascular lung water (<10 mL/kg) and global end-diastolic volume index (<850 mL/m) in the transpulmo

159 had greater improvement in left ventricular end-diastolic volume index (-26.2 versus -7.4 mL/m(2)),

160 (66 +/- 5% to 54 +/- 9%, p < 0.0001) and LV end-diastolic volume index (108 +/- 28 ml/m(2) to 78 +/-

161 Patients with transplants had lower end-diastolic volume index (59.3+/-15.2 ml/m(2) vs. 71.4

162 tial shortening (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower

163 0.0001), obesity (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001),

164 3.4 vs 3.8 L/min/m(2); P = .001), and global end-diastolic volume index (GEDVI) (726 vs 775 mL/m(2);

165 The left atrial end-diastolic volume index (LAEDVI), representing the mi

166 outcome was mean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

167 centrations and left ventricular (LV) EF, LV end-diastolic volume index (LVEDVI), LV end-systolic vol

168 small, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant

169 all stable increase in the right ventricular end-diastolic volume index (P<0.001).

170 Stroke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during ex

171 olume index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

172 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak ex

173 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak ex

174 se in change from baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) redu

175 Left ventricle end-diastolic volume index and end-systolic volume index

176 Greater ECE is associated with reduced LV end-diastolic volume index and LV end-diastolic mass ind

177 ces between groups in baseline values for LV end-diastolic volume index and LV end-systolic volume in

178 Empagliflozin reduced LV end-diastolic volume index by 8.2 (95% CI, -13.7 to -2.6

179 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m

180 Resting left ventricular end-diastolic volume index increased (P=0.001) within th

181 Right ventricular end-diastolic volume index measured by means of cardiova

182 ndex threshold of 227% or a left ventricular end-diastolic volume index of 58 ml/m(2) identified pati

183 CMR-derived end-diastolic volume index showed a weaker association w

184 had a greater extent of LGE and a higher LV end-diastolic volume index than other groups, but levels

185 as change from baseline in right ventricular end-diastolic volume index versus placebo.

186 All patients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

187 ith improvements in LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume i

188 ET sympathetic denervation, left ventricular end-diastolic volume index, creatinine, and no angiotens

189 e were no significant differences in mean LV end-diastolic volume index, end-systolic volume index an

190 edictors (age, body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.0

191 rdiovascular magnetic resonance measures (LV end-diastolic volume index, LV ejection fraction), diure

192 and 87.3+/-18.7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volum

193 , left atrial volume index, left ventricular end-diastolic volume index, peak E wave, and the presenc

194 roke volume variation monitoring, and global end-diastolic volume index.

195 iuretic peptide, and larger left ventricular end-diastolic volume index.

196 al volume index, and larger left ventricular end-diastolic volume index.

197 ure (LV mass index: r = 0.35, p < 0.0001; LV end-diastolic volume index: r = 0.43, p < 0.0001) and LV

198 maximal NC/C ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area

199 LV ejection fraction (LVEF) and left atrial end-diastolic volume indexed to body surface area, were

200 /e' ratio, left ventricular end-systolic and end-diastolic volume indexes (LVESVI and LVEDVI), left a

201 (LV) ejection fraction, LV end-systolic and end-diastolic volumes, infarct size, and major adverse c

202 CMR-derived ventricular indexed end-diastolic volume is an independent predictor of tran

203 n was defined as (LA end-systolic volume--LA end-diastolic volume) / LA end-systolic volume.

204 r end-diastolic volume and right ventricular end-diastolic volume (left ventricular end-diastolic vol

205 ts underwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-systolic volume

206 d improvements in LV end-systolic volume, LV end-diastolic volume, left ventricular ejection fraction

207 s percentage of myocardium, left ventricular end-diastolic volume, left ventricular end-systolic volu

208 were predicted by a preoperative indexed RV end-diastolic volume </=158 mL/m(2) and RVESVi </=82 mL/

209 as a reduction in LV wall stress, reduced LV end-diastolic volume, LV end-systolic volume and increas

210 me-wide significant loci (3 loci each for LV end-diastolic volume, LV end-systolic volume, and LV mas

211 ined percent change in left ventricular (LV) end-diastolic volume, LV end-systolic volume, LV ejectio

212 e-wide association studies of 6 LV traits-LV end-diastolic volume, LV end-systolic volume, LV stroke

213 Automatic quantification of LV end-diastolic volume, LV myocardium mass, LV trabeculati

214 line LV function, who showed reduction in LV end-diastolic volume, LV wall stress, no change in LV en

215 all thickening fraction, LV end-systolic and end-diastolic volumes, LV ejection fraction) and hemodyn

216 Significant decreases in LV end-diastolic volumes, LV end-systolic volumes, cardiac

217 lts were obtained with MRI: Left ventricular end-diastolic volume (LVDV) was 40 mL (LVDV per body sur

218 jection fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magnetic resonan

219 ut off values for change in left ventricular end-diastolic volume (LVEDV) and LV end-systolic volume

220 0% to 50% or greater, whose left ventricular end-diastolic volume (LVEDV) had normalised, and who had

221 t, among patients with available baseline LV end-diastolic volume (LVEDV) measures, 188 received bive

222 efined as percent change in left ventricular end-diastolic volume (LVEDV), was analyzed in 3 prespeci

223 c performance according to the median PET LV end-diastolic volume (LVEDV), with smaller LVs defined a

224 volume [EDV] index) and concentricity (mass/end-diastolic volume [M/EDV](2/3)) in hypertensive patie

225 olumes were significantly smaller in DM, (LV end-diastolic volume/m(2): -3.46 mL/m(2) [-5.8 to -1.2],

226 ) [-5.8 to -1.2], P=0.003, right ventricular end-diastolic volume/m(2): -4.2 mL/m(2) [-6.8 to -1.7],

227 ite normalization of LV stroke volume and LV end-diastolic volume/mass ratio, there was a persistent

228 CMR showed higher left ventricle end-diastolic volume (mean difference: 43+/-22.5 mL), hi

229 nly decreased the temporal variability of LV end-diastolic volume measurements by the 2D biplane meth

230 tal right/left-volume index was defined from end-diastolic volume measurements in CMR: total right/le

231 increased from pre-Norwood to pre-stage II (end-diastolic volume [milliliters]/body surface area [BS

232 resonance (1.5 T) to measure RV mass (g), RV end-diastolic volume (mL), RV mass/volume ratio, and LV

233 P=0.002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04; P=0.026), exerci

234 left ventricular (LV) ejection fraction, LV end-diastolic volume, or New York Heart Association (NYH

235 .96%; 95% confidence interval, -0.2 to 2.1), end-diastolic volume, or systolic volume were observed c

236 en by associations with baseline and DeltaLV end diastolic volume (P<0.0001 for each) and not wall th

237 wer ejection fraction (p = 0.002), increased end-diastolic volume (p < 0.001), increased mass(i) (p <

238 G-but not CT-significantly overestimated the end-diastolic volume (p < 0.001), whereas 2D Echo and 3D

239 ly predictor of increase in left ventricular end-diastolic volume (p < 0.001).

240 ts were observed for the ratio of LV mass to end-diastolic volume (P=0.02) and with hyperinflation me

241 F23 on left ventricular (LV) mass (P=0.016), end-diastolic volume (P=0.029), and end-systolic volumes

242 0.0002), and increased ventricular dilation (end-diastolic volume, P=0.01).

243 size (Delta ejection fraction: P<0.04, Delta end-diastolic volume: P<0.02, Delta end-systolic volume:

244 on, and IMH correlated with the change in LV end-diastolic volume (Pearson's rho of 0.64, 0.59, and 0

245 Indexed RV end-diastolic volume plotted against neoaortic stroke vo

246 RV end-systolic volumes (RVESVi) (indexed RV end-diastolic volume pPVR versus immediately after PVR v

247 stolic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats, which was no

248 cardial lidocaine paradoxically decreased LV end-diastolic volume (preload) in CHF rats, which was no

249 rrected QRS area for heart size using the LV end-diastolic volume (QRSarea/LVEDV).

250 ntric geometry, defined by higher LV mass to end-diastolic volume quartiles, were associated with hig

251 t zone strongly correlated with reduction of end diastolic volume (r(2)=0.69, P=0.04) and end systoli

252 (PRF) nor PR volume (PRV) correlated with RV end-diastolic volume (r = 0.36; p = 0.15 and r = 0.37; p

253 e was a significant predictor of increase in end-diastolic volume (r = 0.51, P < 0.05).

254 fraction correlated closely with indexed RV end-diastolic volume (R = 0.79, p < 0.001) and modestly

255 = 0.95), end-systolic volume (r = 0.93), and end-diastolic volume (r = 0.90), and slightly lower corr

256 d mechanical dyssynchrony correlated with RV end-diastolic volume (r=0.39; P=0.03).

257 oved overall explained variance for volumes (end-diastolic volume, R(2)=0.43; end-systolic volume, R(

258 nance to assess LV remodeling (LV mass-to-LV end diastolic volume ratio), function, tissue characteri

259 The LA:LV end diastolic volumes ratio did not change from baseline

260 dditional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and trended toward i

261 (95% CI: 0.04, 0.36)] and a greater RV mass/end-diastolic volume ratio conditional on LV parameters.

262 associated with greater RV mass and RV mass/end-diastolic volume ratio conditional on the LV; howeve

263 associated with greater RV mass and RV mass/end-diastolic volume ratio relative to the LV.

264 measures of PM2.5, whereas those for RV mass/end-diastolic volume ratio were attenuated.

265 were directly associated with LV mass to LV end-diastolic volume ratio, a marker of cardiac remodell

266 nce of LVH and abnormal/normal LV mass to LV end-diastolic volume ratio.

267 V ejection fraction, LV mass, and LV mass to end-diastolic volume ratio.

268 lume, LV end-systolic volume, and LV mass to end-diastolic volume ratio; 4 loci for LV ejection fract

269 ed on LV mass index and the ratio of LV mass:end-diastolic volume (relative wall mass).

270 ed percentiles of LV end systolic volume, LV end diastolic volume, relative wall and septal thickness

271 2 models showed that the addition of indexed end-diastolic volume resulted in a significantly improve

272 tion fraction, and indexed right ventricular end-diastolic volume resulted in significant improvement

273 n a 57% increase in LV mass (no change in LV end diastolic volume, resulting in an increase in the LV

274 Right ventricular end-diastolic volume (RVDV) was 262 mL (RVDV/BSA, 164 mL

275 Right ventricular end-diastolic volume (RVEDV) was reduced in COPD compare

276 ge of 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x to 2.5x those

277 Systolic function (EF, end-systolic and end-diastolic volumes, stroke volumes) was not different

278 fraction was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

279 (LV) ejection fraction was 31.3 +/- 9.3%, LV end-diastolic volume was 192.7 +/- 71 ml, and effective

280 LV end-diastolic volume was 2-fold (P=0.005) higher in MR p

281 es and treatment group; each 10% decrease in end-diastolic volume was associated with a 40% reduction

282 LA end-diastolic volume was increased in patients with mitr

283 In CAP-treated dogs, end-diastolic volume was unchanged; ejection fraction in

284 infarct size, LV end-systolic volume, and LV end-diastolic volume were analyzed with random-effects m

285 ; stroke volume (SV), ejection fraction, and end-diastolic volume were assessed by echocardiography.

286 infarct size, LV end-systolic volume, and LV end-diastolic volume were estimated with random-effects

287 Pulmonary capillary wedge pressure and LV end-diastolic volume were measured at baseline, during d

288 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2), 108 +/- 2

289 End-systolic and end-diastolic volumes were calculated using blood pool i

290 creased significantly during exercise, while end-diastolic volumes were essentially unchanged.

291 Both the LV and RV end-systolic and LV end-diastolic volumes were increased compared with refer

292 Pulmonary capillary wedge pressures and LV end-diastolic volumes were measured at baseline, during

293 follow-up, left atrial and left ventricular end-diastolic volumes were significantly more reduced in

294 ciations of RV mass and, to a lesser extent, end diastolic volume with PM10-2.5 mass among susceptibl

295 ssociated with greater RV mass and larger RV end-diastolic volume with or without further adjustment

296 increased LV weight/body weight ratio and LV end diastolic volume (WT, 50.8 mul; CatA-TG, 61.9 mul).

297 echocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range, -1.3 to +8

298 ed normal values, left and right ventricular end-diastolic volume z scores were mildly enlarged (0.48

299 LV end-diastolic volume z-score increased (P=0.02), LV mass

300 The mean right ventricular (RV) end-diastolic volume z-score was 1.8 +/- 1.9; RV dilatio