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1 ation with astrocyte perivascular processes (endfeet).
2  contacting the vasculature with perisomatic endfeet.
3 ion in astrocyte free Ca(2+) in the soma and endfeet.
4 s to Plexin-expressing RGCs via their apical endfeet.
5  as mutants have unattached apical and basal endfeet.
6 r loss of pericytes, and retraction of glial endfeet.
7 ing of brain capillaries by tiled astrocytic endfeet.
8 with CHRFAM7A particularly enriched along RG endfeet.
9 microtubule-based transport, accumulating in endfeet.
10 eptors, and form aquaporin-4(+) perivascular endfeet.
11  of GLT-1 were also detected at perivascular endfeet.
12 both astrocytic cell bodies and perivascular endfeet.
13 nts, and pedicles, as well as to Muller cell endfeet.
14 PR56 is present in abundance in radial glial endfeet.
15  in the immediate vicinity of the glial cell endfeet.
16 , are less prominently concentrated in their endfeet.
17 gulator Cdc42 in radial glia regulates glial endfeet activities and inter-radial glial interactions.
18 er of dystrophin-associated protein in glial endfeet and a rare example of a glial protein with a rol
19 tion induced calcium transients in astrocyte endfeet and an associated dilation of upstream arteriole
20 more, harmonin was localized in the terminal endfeet and apical microvilli of MGCs, presynaptic regio
21 bination therapeutic strategy for hypoactive endfeet and astrocytic hyperactivity.
22 etween TRPV4 channels and IP3Rs in astrocyte endfeet and demonstrate that TRPV4 channels are engaged
23 ed aquaporin-4 polarization along astrocytic endfeet and dilated meningeal lymphatic vessels.
24  reduced expression of aquaporin 4 (AQP4) at endfeet and elevated expression of EAAT1/GLAST, with bot
25  disrupt the BBB, interfering with astrocyte endfeet and endothelial tight junction integrity, result
26 nteractions are mediated by growth cone-like endfeet and filopodia-like protrusions.
27  local TRPV4-mediated Ca(2+) oscillations in endfeet and further found that TRPV4 Ca(2+) signals are
28 ction, and pathological changes of astrocyte endfeet and highlights the gaps in our knowledge that fu
29 emoval of IP3R disrupts stress fibers in IOC endfeet and increases the basal retinal surface by ~40%,
30 s communicate with the adjacent radial glial endfeet and influence cortical development.
31 de of spontaneous Ca(2+) events in astrocyte endfeet and inversion of NVC from vasodilation to vasoco
32 pe mice, Kir4.1 is highly clustered in their endfeet and perivascular processes.
33 .1 was present and localized to the proximal endfeet and perivascular processes.
34 E-like immunoreactivity was localized to the endfeet and terminal process of Sus cells surrounding th
35 ward rectifying K(+) channel Kir4.1 at glial endfeet and that disruption of dystrophin and potassium
36 t membrane (BM) located between radial glial endfeet and the meninges during embryonic cerebellar dev
37 ng aquaporin 4 (a water channel in astrocyte endfeet) and integrin-alpha6 (a laminin receptor), are u
38 ociate with vascular areas void of astrocyte endfeet, and the developmental shift in microglial migra
39        Gap junctions (GJs) between astrocyte endfeet are composed of connexin 43 (Cx43) and Cx30, whi
40            We further show that radial glial endfeet are required for the basement-membrane anchoring
41 oepithelial endfeet demonstrating that these endfeet are the preferred site of basal lamina assembly.
42 a in dorsomedial cortex retract their apical endfeet at midneurogenesis and translocate to the overla
43 ayer of the retina and then spread into cell endfeet at the inner retinal surface.
44  KAB-2), which is highly concentrated at the endfeet at the vitreal border and to processes envelopin
45      In the CNS, it is enriched in astrocyte endfeet, at synapses, and at the glia limitans, where it
46 pecific Cre-mediated recombination disrupted endfeet BMs and led to hemorrhage in deep brain regions
47 We found no evidence of observable astrocyte endfeet Ca(2+) elevations following physiological visual
48                                    Astrocyte endfeet contacted the monolayer of BMVEC that formed tig
49 ion results in Ca(2+) increases in the glial endfeet contacting capillaries, but not arterioles, and
50                       Accordingly, astrocyte endfeet contain specialized organelles and proteins, inc
51                  Thus, channels in astrocyte endfeet could be targeted to reduce postischemic brain s
52                                   Astrocytic endfeet cover the entire cerebral vasculature and serve
53 tituting along the retracted neuroepithelial endfeet demonstrating that these endfeet are the preferr
54 e lateral membrane domain in the ventricular endfeet during interphase, mPar3 becomes dispersed and s
55  responsive to K(+) released from astrocytic endfeet during NVC, leading to impairment of this proces
56 nd Sema3F/Nrp1, exert dual effects on apical endfeet dynamics, nuclei positioning, and RGC progeny.
57     Photolysis of caged Ca(2+) in astrocytic endfeet ensheathing the vessel wall was associated with
58                                    Astrocyte endfeet enwrap the entire vascular tree within the centr
59                                   Astrocytic endfeet exhibited large-conductance, Ca2+-sensitive K+ (
60 scopy indicated that a similar proportion of endfeet exhibiting eHACSs also exhibited asymmetrical en
61                 Here we show that the apical endfeet express high levels of the adhesion and signalin
62 m channel (Kir), whereas the vessel-adjacent endfeet express Kir and BK potassium channels.
63 atter contains radial processes with subpial endfeet expressing vimentin (Vim).
64 el expression and polarization to astrocytic endfeet, facilitating interstitial solute clearance.
65 ter and Kir4.1 potassium channels from glial endfeet, formation of intracellular vacuoles in alpha-DB
66  of preexisting vessels, displace astrocytic endfeet from endothelial or vascular smooth muscle cells
67 paration to mechanically isolate radial glia endfeet from the soma, and we use photoconvertible prote
68 (eHACSs) after SAH that were not observed in endfeet from unoperated animals.
69                         A role for astrocyte endfeet has been demonstrated or postulated in many of t
70 lium, extending long basal processes to form endfeet hundreds of micrometers away from the soma.
71  of a collagen IV network underneath the IOC endfeet, implicating the extracellular matrix and its in
72 M and aquaporin-4, is expressed in astrocyte endfeet in human brain.
73 mon with the glia limitans that is formed by endfeet in other cortical areas.
74 ary processes, fine processes, and capillary endfeet in the brain of awake APP/PS1 transgenic mice (1
75 e B cells are anchored by specialized apical endfeet in the center of a pinwheel of ependymal cells.
76 tigated the coverage of vessels by astrocyte endfeet in the prefrontal cortex in MDD.
77  of radial glia and is concentrated at their endfeet in the stratum opticum, at the time retinal axon
78         The clustering of GLT-1 at astrocyte endfeet indicates that it might serve a specialized func
79 our understanding of how basal processes and endfeet influence neurogenesis.
80 ble channel aquaporin-4 (AQP4) to astrocytic endfeet is dependent on interactions between laminin and
81 n-associated protein scaffolds in astrocytic endfeet, is essential for the formation and functioning
82 ith) mice display detachment of radial glial endfeet, marginal zone heterotopias, and cortical dyslam
83 de that cytoskeletal and signaling events at endfeet may be controlled through translation of specifi
84 )-sensitive potassium channels in astrocytic endfeet mediated a majority of the dilation and the enti
85  expression is markedly reduced in astrocyte endfeet membranes adjacent to blood vessels in cerebellu
86                   Retraction of radial glial endfeet, midline fusion of brain hemispheres, and gliosi
87 ane (BM), abnormal anchorage of radial glial endfeet, mislocalized Cajal-Retzius cells, and neuronal
88  juxtavascular microglia ceases as astrocyte endfeet more fully ensheath the vasculature.
89 along vessels corresponded to when astrocyte endfeet more fully ensheath vessels.
90             The coverage of blood vessels by endfeet of AQP4-immunoreactive (IR) astrocytes was signi
91 ssion of AQP4 water channels in the vascular endfeet of astrocytes.
92 nel aquaporin-4 (AQP4) localized to vascular endfeet of astrocytes.
93 on in capillary endothelial cells and in the endfeet of capillary astrocytes, which suggests decrease
94 by contraction of actin stress fibers in the endfeet of interommatidial cells (IOCs).
95 (AQP)-4 channel clusters in the perivascular endfeet of mice in which astrocyte GJs (Cx30, Cx43) were
96 nd of the Na(+)/Ca(2+) exchanger NCX1 in the endfeet of perivascular astrocytes.
97               SARA is enriched at the apical endfeet of radial glia of the neocortex.
98 slation of the RhoGAP ARHGAP11A in the basal endfeet of RGCs control their morphology and mediate neu
99 l studies confirmed that the cell bodies and endfeet of Sus cells expressed high levels of apoE.
100 annel aquaporin-4 at astrocytic perivascular endfeet of the BBB.
101 s, such as an irreversible retraction of the endfeet of the neuroepithelial cells from the vitreal su
102 by cellular receptor proteins on the vitreal endfeet of the retinal neuroepithelial cells.
103 en the developmental formation of astrocytic endfeet on vessels and the termination of OPC perivascul
104 such as thin neuronal processes, astrocytes, endfeets, or protrusions.
105 ainly localized to Muller cell processes and endfeet, photoreceptor terminals, and photoreceptor oute
106 nd in vivo live imaging shows that astrocyte endfeet physically displace OPCs from vasculature, and g
107 atory substances from perivascular astrocyte endfeet, resulting in improved local blood flow.
108  composed of endothelial cells and astrocyte endfeet separated by a basal lamina at their interface.
109 usions along tanycyte processes and at their endfeet such as spines, swelling, en passant boutons, bo
110 comes restricted on the arrival of astrocyte endfeet such that juxtavascular microglia become highly
111 godendrocytes surrounding axons to astrocyte endfeet surrounding capillaries, the proposed panglial s
112 active agents (e.g., K(+)) from perivascular endfeet surrounding parenchymal arterioles.
113 ptor activity through an extensive astrocyte endfeet syncytium abutting subarachnoid and perivascular
114 perates the endothelial cells and astrocytic endfeet that comprise the blood-brain barrier is rich in
115                            Astrocytes extend endfeet that enwrap the vasculature, and disruptions to
116 l cells in the brain with fine processes and endfeet that intimately contact both neuronal synapses a
117 ted bipolar morphology with basal and apical endfeet that reside in distinct niches.
118 dothelial cells and pericytes, and astrocyte endfeet that surround the vessel but are separated from
119 es and capillaries, where they terminate in "endfeet" that encase the blood vessels.
120                 The hexagonal network of IOC endfeet tiling the retinal floor couples nearest-neighbo
121 cellular acidification measured beneath cell endfeet was 304% of the amplitude of the acidification b
122 munoprecipitation and microarray analyses of endfeet, we discover FMRP-bound transcripts, which encod
123 g is largely compartmentalized and capillary endfeet were as active as fine processes but hypoactive
124                     Additionally, astrocytic endfeet were hypertrophic and lacked appropriately polar
125  water homeostasis in perivascular astrocyte endfeet where it is concentrated.
126 eptide colocalizes in perivascular astrocyte endfeet where the 32 kDa polypeptide is abundantly expre
127 mRNA transport and localization to the basal endfeet, where ARHGAP11A is locally synthesized.
128 urn, stress fibers assemble in the IOC basal endfeet, where they contact grommets at anchorages media
129 al processes and is localized to radial glia endfeet, whereas vimentin mRNA is not localized in radia
130 artners influences organization of astrocyte endfeet which in turn impacts BBB integrity of the NVU.
131 s of N-cadherin concentrations at the apical endfeet, which can be partially restored by forced Yap e
132 quaporin 4 (AQP4) in perivascular astrocytic endfeet, which completely ensheathe the brain's capillar
133                                 Radial glial endfeet, which comprise glia limitans, grew out of the n
134 d assemble basement membranes (BMs) at their endfeet, which ensheath the cerebrovasculature.
135 itin mRNA is highly localized to radial glia endfeet, which is suggestive of its transport in these c
136 um signaling within the hypoactive capillary endfeet while reducing the hyperactivity of spontaneous
137 ndogenous calcium signaling within capillary endfeet, while surprisingly as active as astrocytic fine
138 glial network as mature astrocytes that form endfeet with blood vessels, couple electrically to neigh
139  Myh9 and Myh10 transcripts also localize to endfeet with distinct temporal dynamics.
140               Furthermore, the percentage of endfeet with eHACSs varied with time and paralleled the
141                                              Endfeet with eHACSs were present only around arterioles
142 that are significantly enriched in RGC basal endfeet, with MYH9 and MYH10 among the most abundant.
143  enclosing numerous fibroblast and astrocyte endfeet, with pouches of collagen fibrils at the interfa

 
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