ライフサイエンスコーパス: endocervix

1 y correlated with the secretory phase in the endocervix.

2 lations in the transition zone and adjoining endocervix.

3 the response to acute infection in the human endocervix.

4 positing Chlamydia muridarum directly on the endocervix.

5 tween the lower uterine corpus and the upper endocervix.

6 as infiltrating lymphocytes were seen in the endocervix.

7 ripheral blood mononuclear cells (PBMCs) and endocervix.

8 rachomatis and N. gonorrhoeae, respectively, endocervix, 65 and 40%; urine, 72 and 24%; and vagina, 8

9 ive and secretory phases of the cycle in the endocervix (adjusted p < 0.05).

10 ab specimens obtained by clinicians from the endocervix and by patients from their own vaginas.

11 ur goal was to analyze transcriptomes of the endocervix and ectocervix during the proliferative and s

12 EGs (score = log fold change/p value) in the endocervix and ectocervix revealed that (i) unstimulated

13 trual cycle phase-associated changes in both endocervix and ectocervix, which may modulate susceptibi

14 erized a human genital mucosa explant model (endocervix and ectocervix/vagina) to mimic genital herpe

15 e normally simple columnar epithelium of the endocervix and uterine cavity were monitored by keratin

16 the ileum, colon, rectosigmoid, endometrium, endocervix, and ectocervix.

17 and in women, cells from the ectocervix and endocervix are among the first host cells encountered by

18 , we investigated barrier dysfunction of the endocervix by host-derived factors, secreted in response

19 tivities by columnar epithelial cells in the endocervix can cause endogenous HIV-1 reactivation, whic

20 s and fibroblasts from the endometrium (EM), endocervix (CX) and ectocervix (ECX) significantly delay

21 In PBMCs and endocervix, E2 increased protein expression of total CFL

22 inct epithelial cell lines from normal human endocervix, ectocervix, and vagina to characterize gonoc

23 the RT (Fallopian tube, uterine endometrium, endocervix, ectocervix, and vaginal mucosa) and temporal

24 opian tube, but variably expressed by normal endocervix, endometrium, and fallopian tube (60, 64, and

25 In positive endocervix, endometrium, and fallopian tube specimens, H

26 ed from normal human vagina, ectocervix, and endocervix expressed mRNA for TLR1, -2, -3, -5, and -6.

27 Specimens from the endocervix had the highest proportions of lymphocytes, m

28 of actin-binding protein cofilin 1 (CFL1) in endocervix in the E2-dominated proliferative phase of th

29 l to initiate a synchronous infection in the endocervix in the first 24 h to evaluate the sequential

30 ype and restored infection in both PBMCs and endocervix; inhibited E2-induced expression of total CFL

31 These data suggest that the endocervix may have a particularly important role in het

32 a higher level of acute inflammation in the endocervix, oviduct, and mesosalpingeal tissues than in

33 hat the columnar epithelial cells lining the endocervix, the lower part of the uterus, are particular

34 sult from ascension of the bacteria from the endocervix to the oviduct, where an overly aggressive in

35 genital tract disease that ascended from the endocervix to the uterine horns, oviducts, and ovaries i

36 epithelial layers of the vagina, ectocervix, endocervix, uterus, and fallopian tubes in the female re

37 a 0.97), urethra/urine (83%, kappa=0.87) and endocervix/vagina (100%, kappa 1.0) (p<0.005 for all com

38 .97), urethra/urine (83%; kappa = 0.87), and endocervix/vagina (100%; kappa = 1.0; P .005 for all com

39 from the pharynx (n = 93), rectum (n = 88), endocervix/vagina (n = 89), and urethra/urine (n = 46).

40 mens from the pharynx (n=93), rectum (n=88), endocervix/vagina (n=89), and urethra/urine (46).

41 % in the pharynx, rectum, urethra/urine, and endocervix/vagina paired specimens, respectively.

42 s in the pharynx, rectum, urethra/urine, and endocervix/vagina, respectively.

43 Notably, Vdelta2 CD8(+) T cells of the endocervix were negatively correlated with chronic virem