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1 ergy metabolism in the liver, intestine, and endocrine pancreas.
2 gh levels in the islets of Langerhans of the endocrine pancreas.
3 cellular punctate structures in cells of the endocrine pancreas.
4 s highly enriched for genes expressed in the endocrine pancreas.
5 epositing as cytotoxic amyloid fibers in the endocrine pancreas.
6 localized to the islets of Langerhans of the endocrine pancreas.
7 egral role in the regulatory pathways of the endocrine pancreas.
8 ular space surrounding the beta-cells of the endocrine pancreas.
9 ely localized to beta cells within the adult endocrine pancreas.
10 nical disease was evident in the exocrine or endocrine pancreas.
11 eters to assess the functional status of the endocrine pancreas.
12 gs of the pineal gland, adenohypophysis, and endocrine pancreas.
13 n the normal development and function of the endocrine pancreas.
14 ligands to morphogenetic events in the human endocrine pancreas.
15 nd insulin secretion in the beta-cell of the endocrine pancreas.
16  liver, adipose tissue, blood cells, and the endocrine pancreas.
17 mental processes in the brain as well as the endocrine pancreas.
18 nsulin and glucagon) was used to control the endocrine pancreas.
19 of the insulin gene in the beta-cells of the endocrine pancreas.
20  glucagon infusions) was used to control the endocrine pancreas.
21  glucagon infusions) was used to control the endocrine pancreas.
22 l, may serve as junction proteins within the endocrine pancreas.
23 regenerative medicine-inspired bioartificial endocrine pancreas.
24 d the exocrine pancreas while preserving the endocrine pancreas.
25 ibed for insulin-secreting beta-cells of the endocrine pancreas.
26 are spatially distinct from the exocrine and endocrine pancreas.
27 eneration, fully human-derived bioartificial endocrine pancreas.
28  adipose tissue regulates alpha-cells in the endocrine pancreas.
29 ve imaging of the serotonergic system in the endocrine pancreas.
30 ould therefore be used to estimate the human endocrine pancreas.
31 al nervous systems, olfactory epithelium and endocrine pancreas.
32 l noninvasive surrogate marker for the human endocrine pancreas.
33 ating insulin release from beta cells of the endocrine pancreas.
34 ring system that continuously reports on the endocrine pancreas.
35 an important player in the physiology of the endocrine pancreas.
36 he regulation of insulin secretion by the CF endocrine pancreas.
37 P1B and TCPTP in regulating ER stress in the endocrine pancreas.
38 rphological changes in both the exocrine and endocrine pancreas.
39         ERO1-beta is greatly enriched in the endocrine pancreas.
40 ocrine environment for MEN1 tumorigenesis in endocrine pancreas.
41 ors primarily in parathyroid, pituitary, and endocrine pancreas.
42 rograms that regulate the development of the endocrine pancreas.
43 y acting in peripheral tissues including the endocrine pancreas.
44 , we further explored the role of MCH in the endocrine pancreas.
45 derm that directly induces precursors of the endocrine pancreas.
46 inst the insulin-producing beta cells of the endocrine pancreas.
47 letal muscle, adipose tissue, brain, and the endocrine pancreas.
48 specific functions in the development of the endocrine pancreas.
49 -p48 in the development of both exocrine and endocrine pancreas.
50 opment and for proper differentiation of the endocrine pancreas.
51  rare transcripts expressed in the mammalian endocrine pancreas.
52  is the major site of K(ATP) channels of the endocrine pancreas.
53 ir causal agents: (1) alters function of the endocrine pancreas; (2) impairs function of adipose tiss
54                                       In the endocrine pancreas, ACE2 is expressed at low levels in b
55 its function in different organs such as the endocrine pancreas, adipose tissue, skeletal muscle, and
56                                       In the endocrine pancreas, alpha-cell-specific expression of th
57 -like environment guiding the genesis of the endocrine pancreas and advance current models for how di
58 PERK, are associated with dysfunction of the endocrine pancreas and diabetes.
59 ring gestation alters the development of the endocrine pancreas and favors the occurrence of T2D late
60 rentiation along new lineages, production of endocrine pancreas and insulin-secreting beta cells from
61 on of ATF3 in the pancreas leads to abnormal endocrine pancreas and reduced numbers of hormone-produc
62 esponse in promoting adaptive changes in the endocrine pancreas and suggests that enhancement of this
63 c development of the exocrine but not of the endocrine pancreas and that defects of Igf1r do not alte
64 le of NEUROD1 in both the development of the endocrine pancreas and the central nervous system in hum
65  established between growth of the embryonic endocrine pancreas and the islet cell replication that o
66  alone is sufficient to induce tumors in the endocrine pancreas and thymus.
67 y multiple tumors in the parathyroid glands, endocrine pancreas, and anterior pituitary.
68 so expressed in many tissues (eg, intestine, endocrine pancreas, and brain).
69 the stem cell layer of stratified epithelia, endocrine pancreas, and thymic medulla, with a pattern t
70 d Dnmt1, as differentiation of the liver and endocrine pancreas appeared normal.
71                             Neoplasms of the endocrine pancreas are extremely rare, and molecular mec
72 rotransmission, has an analogous role in the endocrine pancreas as a key control point of insulin sec
73   The islets of Langerhans reside within the endocrine pancreas as highly vascularized microorgans th
74 ation of interstitial fluid transport in the endocrine pancreas as well as for the pathophysiology of
75  Aqp7 only, not that of Aqp3 or Aqp9, in the endocrine pancreas at both the mRNA (by reverse transcri
76 ympathetic activity defines a nervous system/endocrine pancreas axis that is critical for beta cell m
77 dantly in adipose tissue, the brain, and the endocrine pancreas but scarcely in the exocrine pancreas
78 ntagonizing RA-mediated specification of the endocrine pancreas, but continues to promote differentia
79 effort was made to enrich for cDNAs from the endocrine pancreas by constructing libraries from isolat
80 monstrate light-activated stimulation of the endocrine pancreas by targeting parasympathetic (choline
81  depolarization, or insulin were assessed by endocrine pancreas cDNA microarrays.
82                                       Making endocrine pancreas cells at will is one of the major goa
83 tal development, cyclin D2 expression in the endocrine pancreas coincides with the replication of end
84                             Neoplasms of the endocrine pancreas, commonly referenced as pancreatic is
85                                          The Endocrine Pancreas Consortium was formed in late 1999 to
86                   Trans-differentiation into endocrine pancreas could have significant implications f
87  we demonstrate that senescent beta-cells of endocrine pancreas decide the onset and severity of chro
88 evidence for lineage plasticity in the human endocrine pancreas, demonstrating that alpha cells deriv
89                             The exocrine and endocrine pancreas developed normally in Perk-/- mice.
90 orally delineate the role of Hh in zebrafish endocrine pancreas development and investigate its relat
91  the expression of multiple genes related to endocrine pancreas development and islet function in the
92    We conclude that NEUROG3 is essential for endocrine pancreas development in humans and that as lit
93 plete loss of enteroendocrine cells, whereas endocrine pancreas development varies in an allele-speci
94 analyses implicate new signaling pathways in endocrine pancreas development, and identified sets of k
95 estion whether NEUROG3 is required for human endocrine pancreas development.
96 from four biologically significant stages of endocrine pancreas development: endoderm before pancreas
97 r cells that differentiate into exocrine and endocrine pancreas did not affect normal pancreas morpho
98 take of (11)C-5-HTP (e.g., the volume of the endocrine pancreas) did not decrease from T1D diagnosis
99 ly affecting the parathyroid, pituitary, and endocrine pancreas, due to the inactivation of the MEN1
100 density and pericyte distribution within the endocrine pancreas; expression of angiogenic factors was
101 urogenin3 functions as a master regulator of endocrine pancreas formation, and its deficiency leads t
102 netics for studying the neural regulation of endocrine pancreas function and suggest its therapeutic
103 ous studies have demonstrated stimulation of endocrine pancreas function by vagal nerve electrical st
104  humans with NEUROG3 mutations are born with endocrine pancreas function, calling into question wheth
105                     The incidence of delayed endocrine pancreas graft function and its impact on long
106 n order to decrease the incidence of delayed endocrine pancreas graft function and its negative impac
107                     The incidence of delayed endocrine pancreas graft function was 69%.
108                                      Delayed endocrine pancreas graft function was defined as total,
109 ilar for recipients without and with delayed endocrine pancreas graft function.
110 recognized evidence of a mechanism governing endocrine pancreas growth and function.
111 on of these isoforms within the exocrine and endocrine pancreas has not been explored in detail.
112                               The vertebrate endocrine pancreas has the crucial function of maintaini
113 t that some regenerative capabilities of the endocrine pancreas have been documented and recent resea
114 f the genes that regulate development of the endocrine pancreas have been identified, comparatively l
115         We report here that within the adult endocrine pancreas, Hhex is selectively expressed in the
116 etic regulation and tumor suppression in the endocrine pancreas; however, intriguing recent data sugg
117                     OXA is also found in the endocrine pancreas; however, little is known about its r
118 ustrating the regenerative capability of the endocrine pancreas in addition to advances in stem cell
119 id not discriminate between the exocrine and endocrine pancreas in control animals, whereas autoradio
120 ption factor required for development of the endocrine pancreas in mice.
121 o signal in differentiated beta-cells of the endocrine pancreas in regulating insulin production.
122                           Development of the endocrine pancreas includes a series of early events whe
123 that ARC protein is abundant in cells of the endocrine pancreas, including >99.5% of mouse and 73% of
124                             In addition, the endocrine pancreas, intrapancreatic nerves, and some ext
125      Early pro-inflammatory signaling in the endocrine pancreas involves activation of NF-kappaB, whi
126                                          The endocrine pancreas is comprised of beta and alpha cells
127                                          The endocrine pancreas is one of the most inaccessible organ
128                                          The endocrine pancreas is organized into clusters of cells c
129             A lineage tree of the developing endocrine pancreas is outlined to further illustrate thi
130                                          The endocrine pancreas is particularly sensitive to the type
131 s; however, the later differentiation of the endocrine pancreas is uniquely dependent upon high PDX1
132 aling functions in the fully developed adult endocrine pancreas is unknown.
133 ucagon is secreted by the alpha-cells of the endocrine pancreas (islets of Langerhans) during fasting
134 nic development and adult homeostasis of the endocrine pancreas, little is known about what regulates
135 mice fed VAD diets display remodeling of the endocrine pancreas, marked beta-cell apoptosis, shifts t
136 uced the expression of peptide YY, the early endocrine pancreas marker.
137 ulin metabolism and cell cycle regulation in endocrine pancreas (miR-15a and miR-17).
138 ical targets differentially activated in the endocrine pancreas of Men1 mice and highlight the need f
139  that both GLP-1 and CCK are produced in the endocrine pancreas of obese mice.
140 ystematic assessment of the pathology of the endocrine pancreas of patients with LADA and for compari
141 e that forms cytotoxic amyloid fibers in the endocrine pancreas of patients with type II diabetes (NI
142 also identified developmental changes in the endocrine pancreas of Snord116p-/m+ animals that persist
143  is markedly upregulated in the exocrine and endocrine pancreas of T1D and at-risk autoantibody-posit
144             Serotonergic biosynthesis in the endocrine pancreas, of which the islets of Langerhans is
145 doderm to control cell fate decisions in the endocrine pancreas progenitor lineage.
146 ols pancreas organogenesis, specification of endocrine pancreas progenitors, and the postnatal growth
147 genic mice revealed abnormalities within the endocrine pancreas, ranging from pancreatic islet hyperp
148 ional program also contained products of the endocrine pancreas (Reg-1 and insulin genes) and the exp
149 4a), resulting in limited capacity for adult endocrine pancreas regeneration.
150 was greater than in islets isolated from the endocrine pancreas (relative level 22 vs. 7%).
151 ynamic tissues in the body, the exocrine and endocrine pancreas relies heavily on the ISR to rapidly
152 noid contributions to the development of the endocrine pancreas remain unknown.
153 of the cardiovascular, immune, reproductive, endocrine pancreas, renal, and central nervous systems.
154                 In the absence of menin, the endocrine pancreas showed increase in cell proliferation
155 s that play a role in the development of the endocrine pancreas, such as insulin promoter factor-1 an
156 ne pancreas to a pathological process in the endocrine pancreas, suggesting pancreatitis might be a p
157 iated cell-intrinsic defense response in the endocrine pancreas that has evolved to enhance the fitne
158  recent advancements in research surrounding endocrine pancreas that hopefully will pave the way for
159                        In this bioartificial endocrine pancreas, the hardware will be represented by
160 nitor, because of the inaccessibility of the endocrine pancreas, the integrated relationship with ins
161           Parasympathetic innervation of the endocrine pancreas, the islets of Langerhans, has been s
162 ction of insulin secreting beta-cells in the endocrine pancreas to prevent and/or delay the onset or
163 was to determine whether the response of the endocrine pancreas to this mild hypoglycemia can occur i
164                                          The endocrine pancreas undergoes major remodeling during neo
165 he structure and function of the neonatal CF endocrine pancreas using a new CFTR-knockout ferret mode
166 tand the in vivo actions of cytokines in the endocrine pancreas, we explored the transcriptional resp
167 o investigate the role of GATA6 in the adult endocrine pancreas, we generated mice in which Gata6 is
168 ic pathways that regulate development of the endocrine pancreas, we generated transcriptional profile
169 two mouse models with Hhex deficiency in the endocrine pancreas, we show that Hhex is required for de
170 es toward release sites, particularly in the endocrine pancreas where secretagogin, SNAP-25, and synt
171     Nowhere is this more evident than in the endocrine pancreas, where disturbances in function or ma

 
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