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1    Lack of pendrin during this period led to endolymphatic acidification, loss of the endocochlear po
2  different ionic compositions, including the endolymphatic and perilymphatic spaces of the organ of C
3 d in an intact cochlear epithelium where the endolymphatic and perilymphatic surfaces could be separa
4  involving failure of the development of the endolymphatic appendage.
5 undles were exposed to the estimated in vivo endolymphatic Ca(2+) concentration (0.3 mm) present in t
6 eir hair bundles were exposed to the in vivo endolymphatic Ca(2+) concentration (40 microM), all mani
7 ivo may therefore be determined primarily by endolymphatic Ca(2+) concentration, repetitive acoustic
8 probability (P(o) ) in mature OHC is ~50% in endolymphatic [Ca(2+) ], resulting in a large standing d
9 owed for the first physiological estimate of endolymphatic Ca2+ near the MET channel (56 +/- 11 micro
10 encies (CF) and found that, at physiological endolymphatic calcium concentrations, approximately half
11 nificantly higher than those in the adjacent endolymphatic compartment and also higher than those of
12 sult in either collapse or distension of the endolymphatic compartment in the cochlea, with concomita
13 2X2 receptors on epithelial cells lining the endolymphatic compartment.
14 abel density significantly above that of the endolymphatic compartment.
15 her cells lining the cochlear and vestibular endolymphatic compartments express TRPML3.
16 mpairment, which is associated with enlarged endolymphatic compartments of the inner ear.
17 brates, we discovered that the inner ear and endolymphatic complex display a repertoire of previously
18 a P2X(2) receptors, which would regulate the endolymphatic concentration of the current-carrying ion
19 il embryonic day 15, after which time severe endolymphatic dilatation occurs, reminiscent of that see
20 ntire dorsal rim of the otic cup becomes the endolymphatic duct (ED), while the posteroventral rim be
21  ears often lack a distinct saccule, and the endolymphatic duct and common crus are invariably fused.
22 , the most common phenotype being failure of endolymphatic duct and common crus formation, accompanie
23  central vestibule, a coiled cochlea, and an endolymphatic duct and sac allowing fluid balance with t
24 e loss of dorsal otic structures such as the endolymphatic duct and sac is attributable to the downre
25 cular canals were the most sensitive and the endolymphatic duct and sac most resistant to exogenous N
26 cells in the thyroid gland as well as in the endolymphatic duct and sac of the inner ear, whereas pre
27 ed effects of VHL deficiency on "tumor-free" endolymphatic duct and sac of VHL patients.
28 alysis of clinically uninvolved "tumor-free" endolymphatic duct and sac tissues of VHL patients, we d
29 lar canals, utricle, saccule, cochlear duct, endolymphatic duct and sac, and neurons of the eighth cr
30   Pds expression was detected throughout the endolymphatic duct and sac, in distinct areas of the utr
31 2 normally promotes dorsal fates such as the endolymphatic duct and semicircular canals by positively
32                               Absence of the endolymphatic duct and swelling of the membranous labyri
33 e ventromedial otic vesicle, the base of the endolymphatic duct and the fusion plates of the semicirc
34 ivatives, including the semicircular canals, endolymphatic duct and utricle, are malformed or absent.
35 r work has shown that sensory organs and the endolymphatic duct each arise near the boundaries of bro
36 ing that leads to dorsal otic patterning and endolymphatic duct formation.
37 in the hindbrain from otic induction through endolymphatic duct outgrowth, and in the prospective neu
38 hat may be critical for the specification of endolymphatic duct outgrowth.
39 inner ears lack a non-sensory structure, the endolymphatic duct, and the membranous labyrinth is poor
40 selected topographical sites within the CNS, endolymphatic duct/sac epithelia are preferentially and
41 he primary effect of VHL deficiency on human endolymphatic duct/sac epithelium seems to be the genera
42 arise within the intraosseous portion of the endolymphatic duct/sac, the vestibular aqueduct.
43 be placed in the intraosseous portion of the endolymphatic duct/sac.
44 5 revealed that mtl and bsd homozygotes lack endolymphatic ducts and semicircular canals and have sho
45 (facial nerve exiting through jugular canal, endolymphatic ducts exiting posterior to the skull roof)
46 or growth and morphogenesis of the embryonic endolymphatic epithelium, a precursor of the endolymphat
47  and epithelial folding at the origin of the endolymphatic epithelium.
48 hought to be related to the shear motion and endolymphatic flow between the tectorial membrane (TM) a
49  in the larva, apparently owing to a loss of endolymphatic fluid in the ear, together with an over-in
50  supporting cells, secretory cells that make endolymphatic fluid or otolithic membranes, and simple e
51  several regions thought to be important for endolymphatic fluid resorption in the inner ear, consist
52  walls fuse together as normal; however, the endolymphatic fluid space in the semicircular canals is
53             Endolymphatic hydrops, increased endolymphatic fluid within the cochlea, is the key patho
54 p of patients with MD were observed: lack of endolymphatic hydrops (cases #1 and #7), various grades
55             Given the association of HI with endolymphatic hydrops (EH), imaging-based techniques for
56 symptoms to abnormal inner ear fluid buildup-endolymphatic hydrops (EH)-with pressure rise and repeti
57                                              Endolymphatic hydrops also can occur after noise trauma
58                               Thus, although endolymphatic hydrops and cochlear synaptopathy are both
59 re, future studies of reliable biomarkers of endolymphatic hydrops and real-time imaging are warrante
60 itative grading system may be easily used in endolymphatic hydrops assessment.
61                         In patients with MD, endolymphatic hydrops can be studied on MRI using 3D-FLA
62 f vestibular hydrops (cases #4, #5, and #6), endolymphatic hydrops herniation into the semi-circular
63                Although there is known to be endolymphatic hydrops involved in the pathological proce
64 determined by examining 3D T2 sequences, and endolymphatic hydrops was identified on delayed post-con
65  guinea-pig ears with experimentally-induced endolymphatic hydrops, a hallmark of Meniere's disease.
66 sociated with inner ear pathologies, such as endolymphatic hydrops, by changing the metabolite profil
67                                              Endolymphatic hydrops, increased endolymphatic fluid wit
68 place map is not fixed but can be altered by endolymphatic hydrops.
69 th OCT and demonstrate the ability to detect endolymphatic hydrops.
70  hemorrhage, or insidiously, consistent with endolymphatic hydrops.
71 e membranous labyrinth with the formation of endolymphatic hydrops.
72 mph and reduced perilymph, a disorder termed endolymphatic hydrops.
73 es glutamate release from hair cells but not endolymphatic hydrops.
74  the mammalian cochlea, responsible for both endolymphatic ion homeostasis and generation of the endo
75 hair bundles of mature OHCs are bathed in an endolymphatic-like Ca(2+) concentration (40 uM) in vitro
76 nt fibrocytes and a significant reduction of endolymphatic potential in high-frequency cochlear regio
77 ation selective stereociliary process or the endolymphatic potential, our data lend additional suppor
78 dle morphology, mechanotransduction and high endolymphatic potential, somatic electromotility for sou
79 ring loss associated with enlargement of the endolymphatic sac (EES).
80 endolymphatic epithelium, a precursor of the endolymphatic sac (ES) and duct (ED), which mediate endo
81                                          The endolymphatic sac (ES) is a cystic organ that is a part
82  = 0.04) and the MRI signal intensity of the endolymphatic sac (median signal intensity, affected vs.
83                 Here, we show that the mouse endolymphatic sac absorbs fluid in an SLC26A4-dependent
84 perior and a vertical duct that connects the endolymphatic sac and the labyrinth cavity.
85 otocyst and gradually becomes limited to the endolymphatic sac by stage 30.
86 (ELST) have been proposed to be derived from endolymphatic sac epithelium, but other possible structu
87   Slc26a4 expression in the developing mouse endolymphatic sac is required for acquisition of normal
88          Vasopressin hypersensitivity of the endolymphatic sac may be implicated in the pathogenesis
89 n = 71) with previously established distinct endolymphatic sac pathologies; i.e. the group MD-dg (ES
90 erapy, intratympanic gentamicin therapy, and endolymphatic sac surgery.
91                                              Endolymphatic sac tumors (ELST) have been proposed to be
92                                              Endolymphatic sac tumors (ELSTs) are associated with von
93                                              Endolymphatic sac tumors (ELSTs) occur sporadically or i
94 ed reports suggest a possible association of endolymphatic sac tumors (ELSTs), which are extremely ra
95 blastomas (the most common tumor in VHL) and endolymphatic sac tumors (ELSTs).
96                                    Papillary endolymphatic sac tumors are destructive, hypervascular
97 s) with histopathologically proved papillary endolymphatic sac tumors were retrospectively reviewed.
98 creatic cysts and tumors, pheochromocytomas, endolymphatic sac tumors, and epididymal cystadenomas.
99 araganglioma, cerebral hemangioblastoma, and endolymphatic sac tumors.
100 erplasia may compensate for cell loss in the endolymphatic sac, a key MD site.
101 drenal medullae and sympathetic paraganglia, endolymphatic sac, epididymis, and broad ligament.
102                       We showed that, in the endolymphatic sac, where fluid reabsorption occurs, SLC2
103 onstrate expression of ATP6B1 in cochlea and endolymphatic sac.
104 e crystals define the former position of the endolymphatic sac.
105  glands, epididymis, broad ligament, and the endolymphatic sac/petrous bone.
106 nd cysts of the pancreas, pheochromocytomas, endolymphatic-sac tumors, and papillary cystadenomas of
107 -cell RNA-seq analysis of pre- and postnatal endolymphatic sacs demonstrates two types of differentia
108                      Gross structures of the endolymphatic space and stria vascularis observed at the
109 e significantly prevented enlargement of the endolymphatic space in the inner ear areas; moreover, it
110 e cupula of each vertical canal occludes the endolymphatic space, its displacement should be proporti
111 ula extending only a small distance into the endolymphatic space.
112  well as a fusion of the utricle and saccule endolymphatic spaces into a common utriculosaccular cavi
113 roughout the forming semicircular canals and endolymphatic structures.

 
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