ライフサイエンスコーパス: endoneurial

1 The endoneurial and perineurial BLs of peripheral nerve also

2 of the blood-nerve barrier, proliferation of endoneurial and perineurial stromal cells, and entry of

3 development and is required to maintain the endoneurial architecture of peripheral nerves.SIGNIFICAN

4 nstrated a significant reduction in the mean endoneurial area in diabetic animals with 5 and 8 mm gap

5 anism for the deinhibition of laminin in the endoneurial basal lamina and may play an important role

6 ave shown that diabetes-induced reduction in endoneurial blood flow (EBF) and impaired endothelium-de

7 reversing the diabetes-induced impairment in endoneurial blood flow and MNCV than L-158809.

8 y prevented the diabetes-induced decrease in endoneurial blood flow and MNCV.

9 Endoneurial blood flow and motor nerve conduction veloci

10 s corrected the diabetes-induced decrease in endoneurial blood flow, significantly improved motor and

11 ropathy and drives inflammation and impaired endoneurial blood flow.

12 localized to Schwann cells, macrophages and endoneurial blood vessels.

13 ontrast to the endothelial cells forming the endoneurial blood-nerve barrier and the blood-brain barr

14 Schwann cells of degenerating efferents and endoneurial cells also incorporated label.

15 nt lateral plantar nerve increased slightly; endoneurial cells doubled.

16 Unexpectedly, Gli1 expression by endoneurial cells is independent of, and functions nonre

17 n axons and the spread of virus to glial and endoneurial cells of the nerve using EM immunocytochemis

18 xposure to 4 mM DOC, but access of K+ to the endoneurial compartment was more restricted after DOC th

19 cence indicated that Schwann cells and other endoneurial components of rat spinal nerve contain serin

20 Endoneurial density of p75-stained Schwann cells was inc

21 tions cooperatively with Dhh to drive normal endoneurial development.

22 early- and late-passage adult primary human endoneurial endothelial cells and laser-capture microdis

23 , which interactively control the privileged endoneurial environment and the pathogenesis of the pain

24 ells produced surface basal lamina; however, endoneurial, epineurial, and meningeal fibroblasts did n

25 During development, Gli1 also regulates endoneurial extracellular matrix production, nerve vascu

26 Axons, Schwann cells (SCs), and endoneurial fibroblasts (EFs) reside within the endoneur

27 This identified endoneurial fibroblasts as a novel neural crest derivati

28 Endoneurial fibroblasts, in addition to myelinating and

29 iopsies, CSF-1 is predominantly expressed by endoneurial fibroblasts, which are closely associated wi

30 ngoing hypoxia to microvascular dysfunction, endoneurial fibrosis, and increased metabolic requiremen

31 Comparison of epineurial and endoneurial findings suggested that colonization of epin

32 In EDN, endoneurial glucose, fructose, and sorbitol were signifi

33 Endoneurial glucose, fructose, sorbitol, and myo-inosito

34 Basal cAMP PDE activity in soluble endoneurial homogenates of normal nerve was 34.9 +/- 1.9

35 es were found to predominate in assays using endoneurial homogenates.

36 tudy, we observed the presence of persistent endoneurial hypoxia in a mouse model of traumatic periph

37 colonization of epineurial vessels preceded endoneurial infection.

38 xposing the sciatic nerve to MBP(84-104) via endoneurial injection produces robust mechanical hyperse

39 this calibration curve relating delta DC to endoneurial [K+] it was possible to calculate the change

40 ] it was possible to calculate the change in endoneurial [K+] occurring in intact preparations.

41 on with a Schwann cell-derived CSPG and that endoneurial laminin may be inhibited by this CSPG as wel

42 Identification of the endoneurial laminin-2 isoform and its receptor alpha-dys

43 Endoneurial laminins (Lms), beta1-integrins, and dystrog

44 n by sodium ions returning to their original endoneurial location, given that the axolemmal Na(+)-K(+

45 ent vessels may greatly increase the risk of endoneurial M. leprae bacteremia, and also enhance the r

46 growth factor receptor (p75) expression, and endoneurial macrophage apolipoprotein E (apo E) synthesi

47 owed a significant increase in the number of endoneurial macrophages (EMPhi) that lie around nerves i

48 elial cells and laser-capture microdissected endoneurial microvessels from four cryopreserved normal

49 ence controls, the proportion of TM-positive endoneurial microvessels was 15-fold lower (0.02 vs. 0.3

50 owever, flurbiprofen paradoxically prevented endoneurial NBF deficits but not MNCV slowing.

51 eductions in MNCV, Na,K-ATPase activity, and endoneurial NBF in flurbiprofen-treated ND and STZ-D rat

52 nerve conduction velocity (MNCV), total and endoneurial nerve blood flow (NBF), Na,K-ATPase activity

53 rve functional changes; that is, decrease in endoneurial nutritive blood flow, motor and sensory nerv

54 but not sciatic-tibial motor NCV, corrected endoneurial nutritive but not composite NBF, increased t

55 e pathway by which sodium ions return to the endoneurial space after they have entered the axon durin

56 signature genes of microglia, with only the endoneurial subset expressing a restricted number of the

57 The endoneurial T cells in the proximity of BB-1-positive Sc

58 AMP levels significantly after incubation of endoneurial tissue with various isozyme-specific inhibit

59 ructions regenerating axons encounter in the endoneurial tubes of old animals give rise to slower reg

60 riginal synaptic site by following SC-filled endoneurial tubes.

61 Both endoneurial vascular conductance and MNC velocity were d

62 m can permeate into the endoneurium from the endoneurial vasculature in a size- and stage-dependent m