ライフサイエンスコーパス: endothelial

1 2-photon and intravital microscopy to study endothelial activation and leukocyte-endothelial interac

2 Endothelial activation and stress index (EASIX) was show

3 reated mice showed reduced expression of the endothelial activation molecule VCAM-1 but increased exp

4 nd demonstrating that disturbed flow-induced endothelial activation required IRAK-1.

5 adhesion molecule (ICAM)-1 are biomarkers of endothelial activation, which has been implicated in the

6 Furthermore, it is appreciated that endothelial adhesion molecules are heavily N-glycosylate

7 as well as resistin, which augments monocyte-endothelial adhesion.

8 high-fat diet-fed mice to assess the role of endothelial AKAP150-TRPV4 signaling in blood pressure re

9 Using lung microvascular endothelial and alveolar epithelial cells, we demonstrat

10 nd pro-death pathways in malignant, stromal, endothelial and immune cells, hence causing a profound c

11 lacking TGFbetaRII simultaneously expressed endothelial and mesenchymal markers and transcription fa

12 tivation of the NADPH oxidase, uncoupling of endothelial and neuronal nitric oxide synthase, and vasc

13 rticularly enriched in lesional macrophages, endothelial, and smooth muscle cells.

14 hepatic coagulation factors and extrahepatic endothelial anticoagulant protein S, required for thromb

15 KBP51 axis to inhibit I(SOC) and protect the endothelial barrier against calcium entry-induced disrup

16 everity by damaging the pulmonary epithelial-endothelial barrier and increasing pulmonary oedema.

17 apeutic targets for diseases associated with endothelial barrier dysfunction.

18 ) BMMCs, indicating that Fn14 is crucial for endothelial barrier function.

19 ependent manner, induced a rapid drop in the endothelial barrier integrity of HLMVECs.

20 nti-inflammatory responses, prosurvival, and endothelial barrier stabilization.

21 that correlates with increased leakiness of endothelial barriers.

22 regulated so that the parasite decreases its endothelial binding capacity, allowing increased splenic

23 distinct early and sustained immunologic and endothelial biomarker signatures and decreased long-term

24 ion of CD54, MHC Class I and II molecules in endothelial but not epithelial cells, which exhibited co

25 Restricting expression of Akt1E17K to endothelial, cardiac or smooth muscle cells resulted in

26 quently caused by capillary nonperfusion and endothelial cell (EC) loss.

27 om the epithelium is required for a distinct endothelial cell (EC) population in the mouse lung.

28 impact of cross-talk between tumor cell- and endothelial cell (EC)-secreted IL6 on HNSCC growth and t

29 icant increase in the expression of platelet endothelial cell adhesion molecule-1 (PECAM-1) and a dec

30 lent (mAb) affinity ligands specific for the endothelial cell adhesion molecules, PECAM-1 (CD31) and

31 ed that rhIGF-1/BP3 treatment increased lung endothelial cell and alveolar type 2 cell proliferation.

32 Ceramide-induced endothelial cell apoptosis boosts intestinal stem cell r

33 VEGF quickly relaxes the endothelial cell barrier by triggering signaling events

34 microvascular network morphology as well as endothelial cell biology.

35 alers, were mainly expressed by the vascular endothelial cell cluster almost exclusively in DFU, indi

36 ) had a positive serology, 22 (22.5%) had an endothelial cell count < 2000 cells/mm(2) and 6 (6.1%) a

37 Main outcomes measured were visual acuity, endothelial cell count (ECC), rates of secondary graft f

38 IOP, corneal status, and endothelial cell count values were in the normal range.

39 gmatic vector changes, contrast sensitivity, endothelial cell count, and possible adverse events were

40 a discovery cohort of HLA antibody-negative, endothelial cell crossmatch-positive sera obtained from

41 ation, compared to classical two-dimensional endothelial cell cultures.

42 e levels, and attenuated neutrophil-mediated endothelial cell damage.

43 were done on the second day and at 3 months: endothelial cell density (ECD in cells/mm), corneal tran

44 r mesopic conditions), intraocular pressure, endothelial cell density (ECD) and patient impairment.

45 , spherical equivalent, hyperopic shift, and endothelial cell density.

46 review, we provide an overview of hemogenic endothelial cell development and highlight the molecular

47 entiated fetal human pulmonary microvascular endothelial cell interactions, inhibited tube stability,

48 ons, inhibited tube stability, and disrupted endothelial cell junctions.

49 r endothelial cells and an established human endothelial cell line, we investigated the role of AMP-a

50 Endothelial cell loss at 6 months postoperatively was si

51 Endothelial cell loss did not differ significantly betwe

52 Endothelial cell loss in the study group was 12%-22% hig

53 f +0.36 diopters (P < .001) was observed and endothelial cell loss measured 33%.

54 comes included rates of detachment/rebubble, endothelial cell loss, best spectacle-corrected visual a

55 ctions, but whether PPARbeta/delta modulates endothelial cell metabolism to support the dynamic pheno

56 c activity and morphology of human pulmonary endothelial cell monolayers.

57 ase-neutralizing mAb on human umbilical vein endothelial cell permeability were assayed using a Trans

58 Investigate the endothelial cell phenotype (s) that causes Shock-Induced

59 the TNF-alpha-induced osteoclast formation, endothelial cell phenotypic changes, foam cell formation

60 howed reduced retinal neovascularization and endothelial cell proliferation in OIR.

61 eptor activin-like kinase 1 (ALK-1) promotes endothelial cell quiescence.

62 Retinal endothelial cell sorting in wild type C57BL/6 mice was v

63 verting enzyme 2), which is expressed on the endothelial cell surface.

64 nd in soluble Pfn1's involvement in vascular endothelial cell tumor cell cross-talk.

65 r results showed that all compounds affected endothelial cell viability in vitro at low micromolar do

66 ression of junctional proteins at sinusoidal endothelial cell-cell contacts, switching capillaries fr

67 Collectively, these data demonstrate that endothelial cell-derived IL-6 enhances the self-renewal

68 Here, we tested the hypothesis that endothelial cell-initiated signaling is necessary to mai

69 oligodendrocytes, microglia, astrocytes, and endothelial) cell types.

70 nor-derived human dermal blood and lymphatic endothelial cells (BEC and LEC, respectively) to show th

71 We previously showed that AVM-brain endothelial cells (BECs) secreted higher VEGF (vascular

72 Autologous sets of blood outgrowth endothelial cells (BOECs), smooth muscle cells (BO-SMCs)

73 Endothelial cells (ECs) are highly glycolytic and genera

74 Endothelial cells (ECs) are widely heterogenous dependin

75 Endothelial cells (ECs) display remarkable plasticity du

76 Contact between inflammatory cells and endothelial cells (ECs) is a crucial step in vascular in

77 RNA-seq), we have identified a population of endothelial cells (ECs) present early in HIO differentia

78 Cyp26b1 is highly enriched in lung endothelial cells (ECs) throughout development.

79 rehensively characterize subclasses of brain endothelial cells (ECs) under both normal conditions and

80 Endothelial cells (ECs) within the BRB/BBB are tightly c

81 chemokine via its primary receptor CCR10 in endothelial cells (ECs).

82 postnatal inactivation of Kif11 in vascular endothelial cells (ECs).

83 Glomerular endothelial cells (GEC) are a crucial component of the g

84 Human corneal endothelial cells (HCEnCs) were exposed to various tempe

85 e from the transdifferentiation of hemogenic endothelial cells (hemECs).

86 human non-malignant intestine microvascular endothelial cells (HIMEC) was assessed.

87 helial cells (MLEC) and human umbilical vein endothelial cells (HUVEC) with SHIP-1 knockdown were ana

88 d a new infection model of primary lymphatic endothelial cells (LECs) infected with a lytically repli

89 Breast cancer cells 'educate' lymphatic endothelial cells (LECs) to support tumor vascularizatio

90 n of the VEGF-C receptor VEGFR3 in lymphatic endothelial cells (LECs).

91 lity of the corneae depends on the number of endothelial cells (mean: 2109 +/- 67 cells/mm(2) (range:

92 Primary mouse lung endothelial cells (MLEC) and human umbilical vein endoth

93 eminal ganglion sensory neurons and vascular endothelial cells (VEC) and found that neurons isolated

94 sms (ie, vascular permeability controlled by endothelial cells [ECs]).

95 ted how preventing Wnt ligand secretion from endothelial cells affects zonation patterns under homeos

96 Using primary human retinal vascular endothelial cells and an established human endothelial c

97 4 as miR-122 inhibition decreases miR-204 in endothelial cells and aorta.

98 onents of the blood-brain barrier, including endothelial cells and astrocytes.

99 version in TGFbeta1-exposed human and murine endothelial cells and improved venous thrombus resolutio

100 ion-depolarised filopodia dynamics in motile endothelial cells and induced mispatterning of blood ves

101 Endothelial cells and macrophages are likely targets as

102 CTEPH-endothelial cells and murine endothelial cells lacking T

103 t govern hemogenic specification of vascular endothelial cells and the generation of multilineage HSP

104 c and attractive to phagocytes and activated endothelial cells and thus contributes to the anaemic an

105 scriptional programs in vascular cells, like endothelial cells and vascular smooth muscle cells, card

106 Endothelial cells are an integral part in lung fibrosis.

107 Cardiomyocytes, fibroblasts, and endothelial cells attached well to eADF4(C16)-RGD coatin

108 These interactions change when the endothelial cells become dysfunctional and have an impac

109 ory conditions, costimulation of human brain endothelial cells by NMDA agonists (NMDA or glycine) and

110 fected cells was recapitulated in uninfected endothelial cells by the exogenous expression of ORFK12

111 s, we demonstrate that pericytes rather than endothelial cells colocalize with these bridges.

112 egulation of total retinal cells and retinal endothelial cells during non-infectious uveitis.

113 ous cancer cells and actively dividing tumor-endothelial cells express the thyrointegrin alphavbeta3

114 of the TME, such as targeting pericytes and endothelial cells for vascular normalization, are provin

115 Total retinal cells and retinal endothelial cells from naive and EAU mice were sorted an

116 ABC-treated endothelial cells had higher levels of ICAM (intercellul

117 The propagation and expansion of corneal endothelial cells has been widely reported.

118 In addition, periventricular endothelial cells house a GABA signaling pathway with di

119 lmonary arterial and lung MV (microvascular) endothelial cells in response to DNA damage and oxidant

120 Using zebrafish and human endothelial cells in vitro, we show ECs deficient in CDP

121 led in vitro by placing human umbilical vein endothelial cells into a hypoxic incubator (1% O2) for 2

122 trated, (6) cytoplasmic vesicles in vascular endothelial cells known to stain for NADPH diaphorase we

123 Endothelial cells lacking Flvcr2 had altered expression

124 CTEPH-endothelial cells and murine endothelial cells lacking TGFbetaRII simultaneously expr

125 alysis in the present study demonstrate that endothelial cells latently infected with KSHV express se

126 a the S1P receptor 1 (S1PR1) in the vascular endothelial cells of lung and kidney.

127 y factors (eg, ICAM-1, E-selectin, MCP-1) in endothelial cells or vascular smooth muscle cells and de

128 nd spatial gradients-emanating from vascular endothelial cells outwards-in fibroblasts.

129 Endothelial cells play an important role in maintenance

130 Here we show that Akt3 depletion in primary endothelial cells results in decreased uncoupled oxygen

131 her, these data reveal that liver sinusoidal endothelial cells sense the microbiome, actively orchest

132 parative transcriptome analysis with retinal endothelial cells sorted from Tie2-GFP mice, which expre

133 iated macrophages, as well as more lymphatic endothelial cells than tumors from PyMT mice.

134 nistic basis for the similarity of KS lesion endothelial cells to neuroendocrine tumors remains unkno

135 t an 18 h exposure of human pulmonary artery endothelial cells to the different nanoparticles modestl

136 Sequencing of RNA from the endothelial cells uncovered the activation of Gene Ontol

137 lying mechanisms of HIV Tat in KSHV-infected endothelial cells undergoing viral lytic reactivation re

138 e due to the ability of SARS-CoV-2 to invade endothelial cells via ACE-2 (angiotensin-converting enzy

139 TNF induces R-Ras upregulation in endothelial cells via JNK and p38 mitogen-activated prot

140 Retinal endothelial cells were isolated by flow cytometry either

141 Similarly, treatment of endothelial cells with LDL reduces BMP-9-induced SMAD1/5

142 stic human primary cell line (lung lymphatic endothelial cells) as a typical normal host cell from th

143 alpha) is expressed in retinal Muller cells, endothelial cells, and in retinal pigment epithelium; ag

144 hannels are a major Ca(2+) influx pathway in endothelial cells, and regulatory protein AKAP150 (A-kin

145 with normal non-neoplastic cells, including endothelial cells, astrocytes and immune cells, constitu

146 evant cell types including epithelial cells, endothelial cells, B cells, T cells and hepatocytes.

147 In vascular endothelial cells, cysteine metabolism by the cystathion

148 zed phospholipid content, activates arterial endothelial cells, facilitating increased transendotheli

149 plasmic lipid-binding protein found in brain endothelial cells, makes protein-protein contact with th

150 In endothelial cells, PLXND1 is a direct force sensor and f

151 alk between EphA4-Tie2 signaling in vascular endothelial cells, which is mediated through p-Akt regul

152 r of ion channels in smooth muscle cells and endothelial cells-the two major classes of vascular cell

153 sels by limiting the plasticity of committed endothelial cells.

154 l transition, similar to TGFbeta1-stimulated endothelial cells.

155 motility and altered pericyte association to endothelial cells.

156 ayer coating the luminal surface of vascular endothelial cells.

157 of alpha(4)beta(7) that is expressed on gut endothelial cells.

158 ated recruitment and activation of lymphatic endothelial cells.Conclusions: A unique population of LA

159 ells is a minimal signal capable of inducing endothelial contraction and transient microvascular leak

160 38 patients) underwent DMEK mainly for Fuchs endothelial corneal dystrophy (FECD; 85.3%) or bullous k

161 nderwent DMEK for various indications (Fuchs endothelial corneal dystrophy [FECD]: n = 111; bullous k

162 Fuchs' endothelial corneal dystrophy was classified clinically

163 fect is due to synergic modifications of the endothelial cytoskeleton and junctional remodeling.

164 apid uterine growth, changes associated with endothelial damage (preeclampsia, eclampsia, and HELLP s

165 In addition, endothelial damage and thromboinflammation, dysregulatio

166 atory milieu in the circulation and vascular endothelial damage markers within patients with COVID-19

167 that SARS-CoV-2 triggers complement-mediated endothelial damage, but the mechanism is unclear.

168 sequent excessive inflammatory responses and endothelial damage.

169 Endothelial differentiation and function were enhanced b

170 This approach identified Endothelial differentiation-related factor 1 (EDF1) as a

171 ypoxia, proangiogenic factors production and endothelial differentiation.

172 w approach to treat diseases associated with endothelial dysfunction and vascular hyperpermeability.

173 se life-threatening disease characterized by endothelial dysfunction and vascular leakage.

174 of therapeutic benefit in the prevention of endothelial dysfunction associated with preeclampsia.

175 Concentrations of inflammatory and endothelial dysfunction biomarkers were determined at cl

176 Endothelial dysfunction is a hallmark of preeclampsia, a

177 d to inflammation (inflammatory response and endothelial dysfunction) were related to the severity of

178 is typical in people with diabetes, reflects endothelial dysfunction, and increases the risk of end-o

179 now considered a cornerstone in I/R-related endothelial dysfunction, which further impairs local mic

180 ctive platelets, hypercoagulable status, and endothelial dysfunction.

181 .g. plasma [ATP]) as opposed to an intrinsic endothelial dysfunction.

182 (PAR1) to promote inflammatory responses and endothelial dysfunction.

183 d outcomes of primary transplants for Fuchs' endothelial dystrophy (FED) were analyzed using Kaplan-M

184 Constitutively, compared with normal endothelial (Endo-N) cells, Endo-T cells differentially

185 Endothelial failure and immunological graft rejection re

186 , thus restricting these cells to a vascular endothelial fate.

187 describe a neural mechanism that increases endothelial fenestrations and enhances the hypothalamic

188 ox, inflammatory profiles, and biomarkers of endothelial function (NO(2)(-) and ADMA).

189 determined the effects of AT, RT, and CT on endothelial function and systolic (SBP)/diastolic blood

190 It is vital for endothelial function as it participates in microvascular

191 lux may be a therapeutic strategy to protect endothelial function from dyslipidemia and diabetic comp

192 ent a novel therapeutic modality to preserve endothelial function in diabetes.

193 Improvements in endothelial function in eNOS(-/-) mice were abrogated in

194 rst time MA pharmacokinetics, and effects on endothelial function in vivo, have been reported in huma

195 Endothelial function was assessed by forearm blood flow

196 Changes at 3 and 6 mo in endothelial function, arterial stiffness, systemic-systo

197 ancing KLF2 expression and activity improves endothelial function, controls multiple genes critical f

198 lic triterpenes was associated with improved endothelial function.

199 nd specifically the effect of triterpenes on endothelial function.

200 Aerobic exercise training (AT) improves endothelial function.

201 that GPR4, an endothelial H(+) receptor, and endothelial Galpha(q/11) proteins mediate the CO(2)/H(+)

202 paradigm, we identified a core set of brain endothelial genes whose expression is regulated by neuro

203 ra also prevented NS1-induced degradation of endothelial glycocalyx components.

204 vel is becoming recognized as a biomarker of endothelial glycocalyx damage in select pathologies.

205 eview summarizes the metabolic regulation of endothelial glycocalyx HA and its potential as a therape

206 There were no changes in measures of endothelial glycocalyx or microvascular function.

207 r responders had increased baseline vascular endothelial growth factor (VEGF) (880.0 pg/mL vs 245.4 p

208 Vascular endothelial growth factor (VEGF) and semaphorin-binding

209 (SIS) immobilized with heparin and vascular endothelial growth factor (VEGF) could be implanted into

210 of anti-angiogenic drugs targeting vascular endothelial growth factor (VEGF) has transformed therapy

211 are uniquely sensitive to increased vascular endothelial growth factor (VEGF) stimulation due to a re

212 Anti-vascular endothelial growth factor (VEGF) treatment of neovascula

213 OPT-302 is a novel inhibitor of vascular endothelial growth factor (VEGF)-C and VEGF-D.

214 Herein, we identified that vascular endothelial growth factor (VEGF)-C, a potent lymphangiog

215 , is driven by chronic elevation of vascular endothelial growth factor (VEGF).

216 Vascular endothelial growth factor A (VEGF-A) and its binding to

217 targeting 2 proangiogenic factors, vascular endothelial growth factor A (VEGFA) and angiopoietin 2 (

218 Blockade of vascular endothelial growth factor A (VEGFA) and angiopoietin-2 (

219 Anti-vascular endothelial growth factor acts faster than laser therapy

220 ntial differential efficacy of anti-vascular endothelial growth factor agents in the treatment of DME

221 Treatment with anti-vascular endothelial growth factor agents.

222 l bone level and the expressions of vascular endothelial growth factor and core-binding factor subuni

223 Adeno-associated virus 8-vascular endothelial growth factor C (AAV8-VEGF-C) was injected i

224 In the adult brain, vascular endothelial growth factor D (VEGFD) is required for stru

225 n: Eyes receiving intravitreal anti-vascular endothelial growth factor injections from July 1, 2013,

226 sion in metastatic RCC treated with vascular endothelial growth factor receptor (VEGFR) tyrosine kina

227 cells (BECs) secreted higher VEGF (vascular endothelial growth factor) and lower TSP-1 (thrombospond

228 associated with reduced hippocampal vascular endothelial growth factor-A (VEGF-A) in both male and fe

229 ifically whether macrophage-derived vascular endothelial growth factor-A (Vegf-A) is crucial to estab

230 nsforming growth factor-beta(1) and vascular endothelial growth factor-A secretion was measured in se

231 ected with adeno-associated virus 1-vascular endothelial growth factor-A165 under control of a hypoxi

232 d a feedback loop between PROX1 and vascular endothelial growth factor-C (VEGF-C) signaling.

233 Modulation of vascular endothelial growth factor-mediated immune suppression vi

234 Here, we found that GPR4, an endothelial H(+) receptor, and endothelial Galpha(q/11)

235 ted with other glycosaminoglycans, including endothelial heparan sulfate and chondroitin sulfate E, b

236 trate a unique protective role for pulmonary endothelial HIF-2alpha and how decreased expression of t

237 at is known to play an essential role in the endothelial homeostasis and the binding of BMP-9 to the

238 h factor (PDGFB) in human pulmonary arterial endothelial (HPAE) cells.

239 Functional expression of endothelial ICAM-1 and VCAM-1 was confirmed by T-cell in

240 Our study reveals that endothelial identity and function is critically controll

241 sh revealed a role for the ETS factor FEV in endothelial identity downstream of ETV2 (Etsrp in zebraf

242 Our findings demonstrate that chronic endothelial inflammation adversely impacts niche activit

243 2 shRNA that the inhibitory effect of NMP on endothelial inflammation and subsequent monocyte adhesio

244 Endothelial integrity is vital for homeostasis and adjus

245 and functional level and disrupted astrocyte-endothelial interactions in both animal models and human

246 o study endothelial activation and leukocyte-endothelial interactions.

247 es VEGFA production and reciprocally induces endothelial Jag1 in a paracrine manner.

248 cued hepatic vascular cavernoma formation in endothelial KRASG12D- or BRAFV600E-expressing mice.

249 Therefore MEndoT-derived cells are an endothelial-like cell population that can be regulated t

250 As shown by immunostaining of endothelial makers, renal vascular densities were decrea

251 diac function by improving the expression of endothelial markers in MEndoT-derived cells.

252 ata reveal a context-dependent regulation of endothelial metabolism by GW0742, where metabolic activi

253 Restoration of endothelial MHC I rendered MHC I-deficient mice suscepti

254 Endothelial miR-1 regulates eosinophil trafficking in th

255 age on central corneal thickness and corneal endothelial morphology as well as to identify the relati

256 required Nck1 in vitro and in vivo, showing endothelial Nck1 and IRAK-1 staining in early human athe

257 ions primes plaque inflammation by enhancing endothelial NF-kappaB signaling.

258 lease inflammatory mediators that upregulate endothelial niche E-selectin expression.

259 density lipoprotein (HDL), which signals via endothelial niche S1PR1 to spur regeneration over fibros

260 ll-conductance K(+) (IK and SK) channels and endothelial nitric oxide synthase (eNOS) are present in

261 diated contractile inhibition via the enzyme endothelial nitric oxide synthase.

262 om Jak2V617F mice resulting from a disturbed endothelial NO pathway and increased endothelial oxidati

263 mitogen-activated protein kinase (MAPK) and endothelial NO synthase (eNOS) in EA.hy926 cells treated

264 A-285222 treatment enhanced dermis endothelial NO synthase expression and plasma NO levels

265 proposed strategy to enhance delivery across endothelial or epithelial monolayers is conjugation to c

266 global and conditional deletion of Vegfc in endothelial or leptin receptor-positive (LepR+) cells le

267 to-mesenchymal transition indicates possible endothelial origin and could be the hallmark for future

268 sturbed endothelial NO pathway and increased endothelial oxidative stress.

269 (HIF) genes, HIF1A (encoding HIF-1alpha) and endothelial PAS domain protein 1 (EPAS1 encoding HIF-2al

270 an on-chip electrochemical method to measure endothelial permeability in a 3D hydrogel-based vascular

271 n the anti-inflammatory effects generated by endothelial PHD2/HIF-1 signaling.

272 atment led to an inflammatory, prothrombotic endothelial phenotype that promoted platelet activation.

273 that LYVE-1 may have limited mobility in the endothelial plasma membrane, but no biophysical investig

274 ECs to pharyngeal arches, the remodeling of endothelial plexus into the PAAs, and the remodeling of

275 venous capillary ECs and respective resident endothelial progenitors appear to be at the origin of CC

276 recovery after transplantation by decreasing endothelial proliferation and LepR+ cell regeneration.

277 morphogenetic protein) are known to regulate endothelial quiescence after secretion from the liver an

278 Pharmacological or genetic inhibition of the endothelial RAS-MAPK1 signaling pathway rescued hepatic

279 rate a critical vascular protective role for endothelial S1P(1) in the mouse brain.

280 The degree of endothelial selectivity varied inversely with the FUS pr

281 Endothelial SHIP-1 is essential in controlling fibrotic

282 The role of vascular endothelial signals in tailoring the phenotype and funct

283 demonstrate that the combined correction of endothelial Smad1/5/8, mTOR, and VEGFR2 pathways opposes

284 Conditional endothelial-specific disruption of Piezo1 in adult mice

285 as embryonic zebrafish, we demonstrate that endothelial-specific gain of function mutations in Kras

286 , which express GFP under the control of the endothelial-specific receptor tyrosine kinase promoter T

287 e constant change between a soft and a stiff endothelial surface is imperative for proper functioning

288 o discuss how regulating N-glycoforms on the endothelial surface may allow for the recruitment of spe

289 n of protein C (PC) to activated PC (aPC) on endothelial surfaces.

290 ecule VCAM-1 but increased expression of the endothelial tight junction proteins ZO-1 and occludin, k

291 HE cells undergo an endothelial to hematopoietic cell transition, giving ris

292 The mechanisms of this endothelial-to-hematopoietic transition (EHT) are poorly

293 Positivity for markers of an endothelial-to-mesenchymal transition indicates possible

294 markers and transcription factors regulating endothelial-to-mesenchymal transition, similar to TGFbet

295 angiogenesis, the most commonly used is the "Endothelial Tube Formation Assay" (ETFA).

296 Chemical genetic screening of endothelial tube formation provides a robust approach fo

297 Arterial, endothelial, venous, angiogenic, and mural cell markers

298 R-Ras, in the functional adaptation of high endothelial venules to increase naive T cell trafficking

299 uman mammary duct in proximity to a perfused endothelial vessel.

300 PAR-1 deficiency was associated with reduced endothelial von Willebrand factor expression, which has