ライフサイエンスコーパス: endothelial cell

1 t commonly caused by MAP2K1 mutations in the endothelial cell.

2 motility and altered pericyte association to endothelial cells.

3 ayer coating the luminal surface of vascular endothelial cells.

4 urface P-selectin levels in IL-13-stimulated endothelial cells.

5 non-cytotoxic effect for HIMEC non-malignant endothelial cells.

6 creased by antagomiR-144 in cultured cardiac endothelial cells.

7 sponsible for NE gene expression in infected endothelial cells.

8 m of proteins targeted by H(2)S(n)) in human endothelial cells.

9 rial DNA (mtDNA) release into the cytosol of endothelial cells.

10 the apical side of polarized epithelial and endothelial cells.

11 l of several cell lines such as platelets or endothelial cells.

12 ses neutrophil transmigration through aortic endothelial cells.

13 n with the consequent functional recovery of endothelial cells.

14 he secretome of primary human umbilical vein endothelial cells.

15 hain and subsequent shrinkage of human brain endothelial cells.

16 ration are attenuated in human BBS4 silenced endothelial cells.

17 ich lipoproteins on the surface of capillary endothelial cells.

18 eostatic signaling to a subset of arteriolar endothelial cells.

19 loading group, promoted the angiogenesis of endothelial cells.

20 FB is released by neighboring epithelial and endothelial cells.

21 els through localized insult to the vascular endothelial cells.

22 ts by increasing autophagic flux in vascular endothelial cells.

23 tors, and its downstream target molecules in endothelial cells.

24 tion; PLAT promotes angiogenesis via LRP1 in endothelial cells.

25 of alpha(4)beta(7) that is expressed on gut endothelial cells.

26 sels by limiting the plasticity of committed endothelial cells.

27 l transition, similar to TGFbeta1-stimulated endothelial cells.

28 creased vascular resistance damages vascular endothelial cells-a marker of which is increased platele

29 ptome analysis of mouse PVEC vs. adult brain endothelial cells (ABEC) in mono-culture or NPC co-cultu

30 tivity, alters NO production, which leads to endothelial cell activation, monocyte accumulation, foam

31 ngioblast markers (Flk1, Tal1/Scl1, platelet endothelial cell adhesion molecule 1, vascular endotheli

32 icant increase in the expression of platelet endothelial cell adhesion molecule-1 (PECAM-1) and a dec

33 lent (mAb) affinity ligands specific for the endothelial cell adhesion molecules, PECAM-1 (CD31) and

34 Functional studies included endothelial cell adhesion, shear stress-induced cell ali

35 Endothelial cells adopt tissue-specific characteristics

36 ted how preventing Wnt ligand secretion from endothelial cells affects zonation patterns under homeos

37 ed that rhIGF-1/BP3 treatment increased lung endothelial cell and alveolar type 2 cell proliferation.

38 gomyelinase (ASM) are secreted by irradiated endothelial cells and act as bystander factors to enhanc

39 Using primary human retinal vascular endothelial cells and an established human endothelial c

40 4 as miR-122 inhibition decreases miR-204 in endothelial cells and aorta.

41 promotes cancer cell interactions with brain endothelial cells and astrocytes, blood-brain barrier ex

42 onents of the blood-brain barrier, including endothelial cells and astrocytes.

43 The basement membrane that seperates the endothelial cells and astrocytic endfeet that comprise t

44 its role in mediating leukocyte adhesion to endothelial cells and guiding leukocytes across the vasc

45 version in TGFbeta1-exposed human and murine endothelial cells and improved venous thrombus resolutio

46 ion-depolarised filopodia dynamics in motile endothelial cells and induced mispatterning of blood ves

47 elium dysfunction using coculture of primary endothelial cells and intestinal T84 cells.

48 Endothelial cells and macrophages are likely targets as

49 CTEPH-endothelial cells and murine endothelial cells lacking T

50 o decipher the crosstalk between organotypic endothelial cells and parenchymal cells for identificati

51 contrast, in the bloodstream, iMPs activated endothelial cells and platelets and induced epithelial-t

52 Calcium is an essential second messenger in endothelial cells and plays a pivotal role in regulating

53 t govern hemogenic specification of vascular endothelial cells and the generation of multilineage HSP

54 th an increased pro-inflammatory response in endothelial cells and the subsequent damage of the epith

55 aB-dependent gene transcription in recipient endothelial cells and this effect was partly attenuated

56 c and attractive to phagocytes and activated endothelial cells and thus contributes to the anaemic an

57 scriptional programs in vascular cells, like endothelial cells and vascular smooth muscle cells, card

58 onset and/or progression of HHV-8-associated endothelial-cell and B-cell pathologies, including AIDS-

59 er or stromal cells, including immune cells, endothelial cells, and fibroblasts.

60 alpha) is expressed in retinal Muller cells, endothelial cells, and in retinal pigment epithelium; ag

61 hannels are a major Ca(2+) influx pathway in endothelial cells, and regulatory protein AKAP150 (A-kin

62 We found that LINC00313 can repress endothelial cell angiogenesis-related properties potenti

63 eliorated the ability of MC(TC)LUVA to alter endothelial cell angiogenic activities in vitro and ex v

64 Ceramide-induced endothelial cell apoptosis boosts intestinal stem cell r

65 irculating and lung myelomonocytic cells and endothelial cells are a major source of PTX3, and PTX3 p

66 Endothelial cells are an integral part in lung fibrosis.

67 lity were significantly decreased, while the endothelial cell area and the variation in cell size wer

68 serve as markers for KSHV-infected KS lesion endothelial cells as well as novel therapeutic targets t

69 stic human primary cell line (lung lymphatic endothelial cells) as a typical normal host cell from th

70 with normal non-neoplastic cells, including endothelial cells, astrocytes and immune cells, constitu

71 Cardiomyocytes, fibroblasts, and endothelial cells attached well to eADF4(C16)-RGD coatin

72 evant cell types including epithelial cells, endothelial cells, B cells, T cells and hepatocytes.

73 VEGF quickly relaxes the endothelial cell barrier by triggering signaling events

74 tors by which VEGF and anti-VEGF control the endothelial cell barrier in cells that were chronically

75 nor-derived human dermal blood and lymphatic endothelial cells (BEC and LEC, respectively) to show th

76 Importantly, this interaction may occur on endothelial cells because binding of MBL to beta2-GPI wa

77 These interactions change when the endothelial cells become dysfunctional and have an impac

78 Blood vascular endothelial cells (BECs) control the immune response by

79 We previously showed that AVM-brain endothelial cells (BECs) secreted higher VEGF (vascular

80 microvascular network morphology as well as endothelial cell biology.

81 Autologous sets of blood outgrowth endothelial cells (BOECs), smooth muscle cells (BO-SMCs)

82 ithelial cells and bovine pulmonary arterial endothelial cells (BPAECs).

83 an, influences various signaling pathways in endothelial cells by binding to VEGFR2.

84 ory conditions, costimulation of human brain endothelial cells by NMDA agonists (NMDA or glycine) and

85 fected cells was recapitulated in uninfected endothelial cells by the exogenous expression of ORFK12

86 genes infection among primary human vascular endothelial cells can be attributed entirely to robust,

87 idal endothelial cells (LSEC) and continuous endothelial cells (CEC) through histomorphological and m

88 -seq was performed on microglia and cerebral endothelial cells (CECs).

89 ression of junctional proteins at sinusoidal endothelial cell-cell contacts, switching capillaries fr

90 on-neuronal (astrocytes, microglia, vascular endothelial cells) cells of cortical (medial prefrontal

91 establishment and expansion of human corneal endothelial cells (CEnC) has provided a source of transp

92 alers, were mainly expressed by the vascular endothelial cell cluster almost exclusively in DFU, indi

93 s, we demonstrate that pericytes rather than endothelial cells colocalize with these bridges.

94 ated recruitment and activation of lymphatic endothelial cells.Conclusions: A unique population of LA

95 Cocultures of human NK cells and endothelial cells confirmed that addition of missing sel

96 ) had a positive serology, 22 (22.5%) had an endothelial cell count < 2000 cells/mm(2) and 6 (6.1%) a

97 fest refractive spherical equivalent (MRSE), endothelial cell count (ECC), and corneal thickness.

98 Main outcomes measured were visual acuity, endothelial cell count (ECC), rates of secondary graft f

99 a younger donor cornea age and higher donor endothelial cell count are associated with better long-t

100 IOP, corneal status, and endothelial cell count values were in the normal range.

101 gmatic vector changes, contrast sensitivity, endothelial cell count, and possible adverse events were

102 a discovery cohort of HLA antibody-negative, endothelial cell crossmatch-positive sera obtained from

103 ation, compared to classical two-dimensional endothelial cell cultures.

104 In vascular endothelial cells, cysteine metabolism by the cystathion

105 with extensive loss of primary processes and endothelial cell damage had the highest rates of the com

106 rophil degranulation and neutrophil-mediated endothelial cell damage in patients with ANCA-associated

107 e levels, and attenuated neutrophil-mediated endothelial cell damage.

108 were done on the second day and at 3 months: endothelial cell density (ECD in cells/mm), corneal tran

109 r mesopic conditions), intraocular pressure, endothelial cell density (ECD) and patient impairment.

110 Endothelial cell density (ECD) was measured without any

111 ed included central corneal thickness (CCT), endothelial cell density (ECD), coefficient of variation

112 acuity (BSCVA), refractive astigmatism (RA), endothelial cell density, immunologic rejection, herpeti

113 , spherical equivalent, hyperopic shift, and endothelial cell density.

114 When the corneal endothelial cells density (ECD) drops, the HCEC may deco

115 lesions and extensive plasticity of SMC- and endothelial cell-derived cells including 7 distinct clus

116 Collectively, these data demonstrate that endothelial cell-derived IL-6 enhances the self-renewal

117 review, we provide an overview of hemogenic endothelial cell development and highlight the molecular

118 egulation of total retinal cells and retinal endothelial cells during non-infectious uveitis.

119 newly established in vitro three-dimensional endothelial cell (EC) and PC co-culture model, transform

120 Here, we investigate the role of ORP2 in endothelial cell (EC) cholesterol and PI(4,5)P(2) distri

121 quently caused by capillary nonperfusion and endothelial cell (EC) loss.

122 om the epithelium is required for a distinct endothelial cell (EC) population in the mouse lung.

123 ication, we dissected murine and human liver endothelial cell (EC) populations into liver sinusoidal

124 impact of cross-talk between tumor cell- and endothelial cell (EC)-secreted IL6 on HNSCC growth and t

125 Pulmonary endothelial cells (ECs) are an essential component of th

126 Endothelial cells (ECs) are highly glycolytic and genera

127 Endothelial cells (ECs) are widely heterogenous dependin

128 Endothelial cells (ECs) display considerable functional

129 Endothelial cells (ECs) display remarkable plasticity du

130 injectable alginate-RGD hydrogel laden with endothelial cells (ECs) for further analysis.

131 Contact between inflammatory cells and endothelial cells (ECs) is a crucial step in vascular in

132 RNA-seq), we have identified a population of endothelial cells (ECs) present early in HIO differentia

133 Human endothelial cells (ECs) synthesize, store, and secrete v

134 Cyp26b1 is highly enriched in lung endothelial cells (ECs) throughout development.

135 rehensively characterize subclasses of brain endothelial cells (ECs) under both normal conditions and

136 Endothelial cells (ECs) within the BRB/BBB are tightly c

137 chemokine via its primary receptor CCR10 in endothelial cells (ECs).

138 postnatal inactivation of Kif11 in vascular endothelial cells (ECs).

139 s regulated by a microRNA (miR), miR-15a, in endothelial cells (ECs).

140 tivated PI3K/Akt/mTOR and VEGFR2 pathways in endothelial cells (ECs).

141 sms (ie, vascular permeability controlled by endothelial cells [ECs]).

142 RNA sequencing) were investigated in retinal endothelial cells exposed to high glucose.

143 We found that endothelial cells express genes typically found in the s

144 ous cancer cells and actively dividing tumor-endothelial cells express the thyrointegrin alphavbeta3

145 zed phospholipid content, activates arterial endothelial cells, facilitating increased transendotheli

146 ardiac cell types, including cardiomyocytes, endothelial cells, fibroblasts, and macrophages, as well

147 of the TME, such as targeting pericytes and endothelial cells for vascular normalization, are provin

148 APT-1 enzyme activity was decreased in endothelial cells from db/db mice.

149 Total retinal cells and retinal endothelial cells from naive and EAU mice were sorted an

150 tanding of the factors that adversely affect endothelial cell function could contribute to the develo

151 matic loss of osteoblasts and alterations in endothelial cell function.

152 Furthermore, we also show that endothelial cell functionality is impaired, and when com

153 (SOC) activation leads to formation of inter-endothelial cell gaps and barrier disruption.

154 Glomerular endothelial cells (GEC) are a crucial component of the g

155 he response of immortalized human glomerular endothelial cells (GEnC) to ionizing radiation (IR).

156 r injury, indicating hypoxic like damage and endothelial cell growth and proliferation.

157 previously showed that FECH is required for endothelial cell growth in vitro and choroidal neovascul

158 ABC-treated endothelial cells had higher levels of ICAM (intercellul

159 sed inhibition studies, and NET/human aortic endothelial cell (HAEC) cocultures.

160 The propagation and expansion of corneal endothelial cells has been widely reported.

161 rsistently infects human brain microvascular endothelial cells (hBMECs), the primary barrier that res

162 Generally, the loss rate of human endothelial cells (HCEC) in routine cataract surgery is

163 Human corneal endothelial cells (HCEnCs) were exposed to various tempe

164 e from the transdifferentiation of hemogenic endothelial cells (hemECs).

165 cells for identification of determinants of endothelial cell heterogeneity, and could lead to advanc

166 human non-malignant intestine microvascular endothelial cells (HIMEC) was assessed.

167 In line with these data, human microvascular endothelial cells (HMEC-1) displayed an OMV-associated,

168 In addition, periventricular endothelial cells house a GABA signaling pathway with di

169 ession system and in human primary pulmonary endothelial cells (hPPECs).

170 lature, while in human retinal microvascular endothelial cells (HRMECs), TNF-alpha stimulation causes

171 siRNA-mediated CRBN knock down in human endothelial cells (HUVEC and HMVEC-L), did not affect en

172 lyRad were evaluated on human umbilical vein endothelial cells (HUVEC) in vitro.

173 helial cells (MLEC) and human umbilical vein endothelial cells (HUVEC) with SHIP-1 knockdown were ana

174 eptor 1 [C5aR1]) on human umbilical vascular endothelial cells (HUVECs) and examined how C3a or C5a a

175 inished in YY1-depleted human umbilical vein endothelial cells (HUVECs).

176 In pulmonary endothelial cells, I(SOC) activation leads to formation

177 eq profiling of flow-sorted malignant cells, endothelial cells, immune cells, fibroblasts, and bulk c

178 unction are characteristics of dysfunctional endothelial cells in diabetic patients.

179 populations with neighbouring epithelial and endothelial cells in health, and across a range of kidne

180 ave a natural capacity to differentiate into endothelial cells in infarcted hearts.

181 y plays critical roles in the maintenance of endothelial cells in response to cellular stress caused

182 lmonary arterial and lung MV (microvascular) endothelial cells in response to DNA damage and oxidant

183 We observed that dermal endothelial cells in rosacea had an increased expression

184 on of bradykinin 1 receptor (B1R) and B2R on endothelial cells in the lungs.

185 Using zebrafish and human endothelial cells in vitro, we show ECs deficient in CDP

186 obiological characteristics of KSHV-infected endothelial cells, including a potential mechanism of es

187 lack of cortactin in cultured primary brain endothelial cells inhibited leukocyte transmigration.

188 Here, we tested the hypothesis that endothelial cell-initiated signaling is necessary to mai

189 These findings indicate that OPC-endothelial cell interactions regulate neonatal white ma

190 entiated fetal human pulmonary microvascular endothelial cell interactions, inhibited tube stability,

191 reduced invasion, and impaired melanoma cell-endothelial cell interactions.

192 modulating molecular assembly the at T cell-endothelial cell interface.

193 led in vitro by placing human umbilical vein endothelial cells into a hypoxic incubator (1% O2) for 2

194 ated regulation of podosomes is critical for endothelial cell invasion associated with angiogenesis.

195 We differentiated these iPSCs into endothelial cells (iPSC-ECs).

196 fference between MEndoT-derived and coronary endothelial cells is essential for understanding the rev

197 pulation, observed in LECs, but not in blood endothelial cells, is dependent on the spontaneous virus

198 t cancer but also on the membrane ERalpha of endothelial cells, it accelerates endothelial healing th

199 Here, we generated induced vascular endothelial cells (iVECs) and smooth muscle cells (iSMCs

200 ons, inhibited tube stability, and disrupted endothelial cell junctions.

201 trated, (6) cytoplasmic vesicles in vascular endothelial cells known to stain for NADPH diaphorase we

202 Endothelial cells lacking Flvcr2 had altered expression

203 CTEPH-endothelial cells and murine endothelial cells lacking TGFbetaRII simultaneously expr

204 alysis in the present study demonstrate that endothelial cells latently infected with KSHV express se

205 n previously reported that KSHV infection of endothelial cells leads to an increase of integrin alpha

206 n and is thought to originate from lymphatic endothelial cells (LEC).

207 d a new infection model of primary lymphatic endothelial cells (LECs) infected with a lytically repli

208 Breast cancer cells 'educate' lymphatic endothelial cells (LECs) to support tumor vascularizatio

209 n of the VEGF-C receptor VEGFR3 in lymphatic endothelial cells (LECs).

210 r endothelial cells and an established human endothelial cell line, we investigated the role of AMP-a

211 Endothelial cell lineage tracing showed that BNP directl

212 If cell cycle is blocked, haemogenic endothelial cells lose their EHT potential and adopt a n

213 Mean endothelial cell loss as compared to preoperative donor

214 Endothelial cell loss at 6 months postoperatively was si

215 Endothelial cell loss did not differ significantly betwe

216 Endothelial cell loss in the study group was 12%-22% hig

217 f +0.36 diopters (P < .001) was observed and endothelial cell loss measured 33%.

218 Median 6-month central endothelial cell loss was 31% versus 29% with netarsudil

219 comes included rates of detachment/rebubble, endothelial cell loss, best spectacle-corrected visual a

220 cell (EC) populations into liver sinusoidal endothelial cells (LSEC) and continuous endothelial cell

221 plasmic lipid-binding protein found in brain endothelial cells, makes protein-protein contact with th

222 d also that propranolol-induced apoptosis in endothelial cells may occur via mitochondrial stress.

223 lity of the corneae depends on the number of endothelial cells (mean: 2109 +/- 67 cells/mm(2) (range:

224 a3 integrin S-sulfhydration was required for endothelial cell mechanotransduction in vitro as well as

225 ctions, but whether PPARbeta/delta modulates endothelial cell metabolism to support the dynamic pheno

226 six brain cell types: Astrocytes, pericytes, endothelial cells, microglia cells, oligodendrocytes, an

227 on, compounds HT C1, C2, C4 and C6 inhibited endothelial cell migration and formation of tubular-like

228 Primary mouse lung endothelial cells (MLEC) and human umbilical vein endoth

229 Confluent endothelial cell monolayers infected with either W83 or

230 c activity and morphology of human pulmonary endothelial cell monolayers.

231 a the S1P receptor 1 (S1PR1) in the vascular endothelial cells of lung and kidney.

232 We have previously shown that endothelial cells of the periventricular vascular networ

233 y factors (eg, ICAM-1, E-selectin, MCP-1) in endothelial cells or vascular smooth muscle cells and de

234 For DS endothelial cells, our analysis revealed most down-regul

235 nd spatial gradients-emanating from vascular endothelial cells outwards-in fibroblasts.

236 We obtain pulmonary arterial endothelial cells (PAECs) from lungs of patients and con

237 llular transdifferentiation program in which endothelial cells partially lose their endothelial ident

238 igh TEER primary porcine brain microvascular endothelial cell (PBMEC) culture to assess the impact of

239 cally connected cells that include capillary endothelial cells, pericytes, and vascular smooth muscle

240 ase-neutralizing mAb on human umbilical vein endothelial cell permeability were assayed using a Trans

241 not non-MetS-lEVs, increased Rap1-dependent endothelial cell permeability.

242 ase (NOS) expression in primary human aortic endothelial cells (pHAECs).

243 Investigate the endothelial cell phenotype (s) that causes Shock-Induced

244 the TNF-alpha-induced osteoclast formation, endothelial cell phenotypic changes, foam cell formation

245 we discuss how calcium signaling pathways in endothelial cells play an essential role in affecting ba

246 Endothelial cells play an important role in maintenance

247 In endothelial cells, PLXND1 is a direct force sensor and f

248 signaling may modulate peripheral immune and endothelial cell populations in a highly context-depende

249 howed reduced retinal neovascularization and endothelial cell proliferation in OIR.

250 al cells (HUVEC and HMVEC-L), did not affect endothelial cell proliferation, migration, or tube forma

251 eract each other in regulating KSHV-infected endothelial cell proliferation.

252 ith marked changes in the S-sulfhydration of endothelial cell proteins involved in mediating response

253 , with direct cell-cell contact, TME-derived endothelial cells provide the Notch ligand Jagged1 (Jag1

254 Brain embryonic periventricular endothelial cells (PVEC) crosstalk with neural progenito

255 eptor activin-like kinase 1 (ALK-1) promotes endothelial cell quiescence.

256 g family member A) and impaired flow-induced endothelial cell realignment.

257 cific deletion of Fn-EDA in SMCs, but not in endothelial cells, reduced intimal hyperplasia and suppr

258 siRNA depletion of ITGB3 in vIL-6-expressing endothelial cells resulted in a decrease in adhesion to

259 dothelial-mesenchymal transition in valvular endothelial cells, resulting in increased proteoglycan s

260 Here we show that Akt3 depletion in primary endothelial cells results in decreased uncoupled oxygen

261 distinct human cell lines and primary human endothelial cells results in reduced TcsL sensitivity, w

262 The endothelial cell "S-sulfhydrome" consisted of 3446 indiv

263 her, these data reveal that liver sinusoidal endothelial cells sense the microbiome, actively orchest

264 tivin receptor-like kinase 1 (ALK1)-mediated endothelial cell signalling in response to bone morphoge

265 parative transcriptome analysis with retinal endothelial cells sorted from Tie2-GFP mice, which expre

266 Retinal endothelial cell sorting in wild type C57BL/6 mice was v

267 urements, and flow-induced vasodilatation in endothelial cell-specific CSE knockout mice and in a sma

268 In sharp contrast, mice harboring endothelial cell-specific deletion of IDO show an improv

269 ome maintenance and lytic cycle in lymphatic endothelial cells, supporting Kaposi sarcoma tumorigenes

270 verting enzyme 2), which is expressed on the endothelial cell surface.

271 iated macrophages, as well as more lymphatic endothelial cells than tumors from PyMT mice.

272 ate that vasodilation is largely mediated by endothelial cells that actively relay signals from the c

273 racellular matrix and supporting stromal and endothelial cells that both maintain stem cell pluripote

274 sh a previously undescribed mechanosensor in endothelial cells that regulates cardiovascular pathophy

275 r of ion channels in smooth muscle cells and endothelial cells-the two major classes of vascular cell

276 ecause ASM/ceramide were secreted by primary endothelial cells, their contribution was studied in int

277 cosylated APOE induced apoptosis in cultured endothelial cells through lectin-like oxidized LDL recep

278 tive that enables preservation of the host's endothelial cells to eliminate risks of endothelial reje

279 nistic basis for the similarity of KS lesion endothelial cells to neuroendocrine tumors remains unkno

280 hem find blood and lymphatic vessels, and to endothelial cells to stabilize the vasculature.

281 t an 18 h exposure of human pulmonary artery endothelial cells to the different nanoparticles modestl

282 nd in soluble Pfn1's involvement in vascular endothelial cell tumor cell cross-talk.

283 oligodendrocytes, microglia, astrocytes, and endothelial) cell types.

284 Sequencing of RNA from the endothelial cells uncovered the activation of Gene Ontol

285 lveolar epithelial cells, myofibroblasts and endothelial cells undergo coordinated morphogenesis to g

286 lying mechanisms of HIV Tat in KSHV-infected endothelial cells undergoing viral lytic reactivation re

287 We generated human periventricular endothelial cells, using human pluripotent stem cell tec

288 eminal ganglion sensory neurons and vascular endothelial cells (VEC) and found that neurons isolated

289 e due to the ability of SARS-CoV-2 to invade endothelial cells via ACE-2 (angiotensin-converting enzy

290 TNF induces R-Ras upregulation in endothelial cells via JNK and p38 mitogen-activated prot

291 tly stimulated the proliferation of resident endothelial cells via NPR-A binding and p38 MAP kinase a

292 r results showed that all compounds affected endothelial cell viability in vitro at low micromolar do

293 arietal epithelial cells, and three types of endothelial cells were identified.

294 Retinal endothelial cells were isolated by flow cytometry either

295 In these embryos, all endothelial cells were of human origin.

296 alk between EphA4-Tie2 signaling in vascular endothelial cells, which is mediated through p-Akt regul

297 coverage by OCT which was defined as luminal endothelial cells with 2 abluminal layers of smooth musc

298 Treatment of endothelial cells with BMP-9 triggers the extensive endo

299 Similarly, treatment of endothelial cells with LDL reduces BMP-9-induced SMAD1/5

300 ome induced by thrombin and APC signaling in endothelial cells with the quantification of 11,266 uniq