ライフサイエンスコーパス: endothelial cell dysfunction

1 the SCS-groups, indicating a higher grade of endothelial cell dysfunction.

2 a possible mechanism of HHcy induced retinal endothelial cell dysfunction.

3 ant irradiation induces a sustained vascular endothelial cell dysfunction.

4 c signals are not well regulated and produce endothelial cell dysfunction.

5 tein CIII links hyperlipidemia with vascular endothelial cell dysfunction.

6 4, thrombomodulin, and eNOS mRNA, suggesting endothelial cell dysfunction.

7 itonavir does not appear to directly promote endothelial cell dysfunction.

8 ps because higher albumin excretion reflects endothelial cell dysfunction.

9 s to hyperglycemic conditions and monitoring endothelial cell dysfunction.

10 g inflammatory responses and contributing to endothelial cell dysfunction.

11 critical early events in fatty acid-mediated endothelial cell dysfunction.

12 actors in patients with COVID-19 also induce endothelial cell dysfunction.

13 ugh induction of oxidative stress leading to endothelial cell dysfunction.

14 ociated with poorly understood aging-related endothelial cell dysfunction.

15 blood samples induced human coronary artery endothelial cells dysfunction.

16 tly attenuated APAP-induced liver sinusoidal endothelial cell dysfunction and ameliorated hepatic mic

17 Endothelial cell dysfunction and apoptosis are critical

18 (CNV) and CC atrophy occur in the absence of endothelial cell dysfunction and are not necessarily sec

19 We hypothesized that IGF-1 causes endothelial cell dysfunction and expression of neutrophi

20 le of eliciting cell contact-dependent brain endothelial cell dysfunction and increased barrier perme

21 n demonstrated to promote pulmonary arterial endothelial cell dysfunction and induce pulmonary arteri

22 ty, migration, and proliferation, as well as endothelial cell dysfunction and inflammation.

23 lmonary arterial hypertension (PAH) includes endothelial cell dysfunction and proliferation and migra

24 ed soluble VEGF receptor 1 protein (sFlt-1), endothelial cell dysfunction and proteinuria.

25 Endothelial cell dysfunction and the proteolytic disorga

26 a progressive disease characterized by lung endothelial cell dysfunction and vascular remodeling.

27 aling will be useful targets for controlling endothelial cells dysfunction and consequently atheroscl

28 irculating IL-6 levels that can promote both endothelial cell dysfunction (and subsequent vascular dy

29 ent approaches to the clinical assessment of endothelial cell dysfunction; and outline some promising

30 Nitric oxide and endothelial cell dysfunction are also implicated in the

31 zeta has emerged as a pathologic mediator of endothelial cell dysfunction, based on its essential rol

32 tion and progression of arterial plaques and endothelial cell dysfunction, CAD is commonly viewed as

33 The likely mechanism is endothelial cell dysfunction due to increased MEK1 activ

34 elp preservation of hepatocyte function, but endothelial cell dysfunction during the cold preservatio

35 Diffuse endothelial cell dysfunction (ECD) documented by several

36 reduced PMN buildup and less lung epithelial/endothelial cell dysfunction (edema and hemorrhage).

37 and obliterative vasculopathy resulting from endothelial cell dysfunction, extensive fibrosis seconda

38 ew, we trace the evolution of the concept of endothelial cell dysfunction, focusing on recent insight

39 Endothelial cell dysfunction has been described in the c

40 Retinal endothelial cell dysfunction has been implicated in the

41 aspartate aminotransferase), and sinusoidal endothelial cell dysfunction (hyaluronic acid).

42 matomyositis and implicate monocyte-mediated endothelial cell dysfunction in dermatomyositis vasculop

43 othelial cells and thereby may contribute to endothelial cell dysfunction in diabetic vascular diseas

44 ion for understanding the pathophysiology of endothelial cell dysfunction in the multiple organ dysfu

45 is pressure transmission could contribute to endothelial cell dysfunction in various pathologies.

46 Endothelial cell dysfunction, in its broadest sense, enc

47 ion are hallmarks of early and late vascular endothelial cell dysfunction induced by diabetes.

48 derivatives, including medicinally important endothelial cell dysfunction inhibitor and anti-inflamma

49 Endothelial cell dysfunction is a crucial feature of dia

50 pelling evidence from animal models suggests endothelial cell dysfunction is a key initial trigger of

51 scribe the increasing evidence that cerebral endothelial cell dysfunction is a key mechanism of SVD.

52 Endothelial cell dysfunction is associated with bioavail

53 Endothelial cell dysfunction is pivotal to the pathophys

54 Vascular endothelial cell dysfunction mediated by antiphospholipi

55 s vascular smooth muscle cell proliferation, endothelial cell dysfunction, neovascularization, and th

56 Skeletal muscle atrophy and endothelial cell dysfunction occur in tandem in cardiova

57 encing revealed that cigarette smoke-induced endothelial cell dysfunction promoted monocyte recruitme

58 d circulating interleukin-6 levels), reduced endothelial cell dysfunction (reduced endothelial activa

59 ain pathological mechanisms: first, vascular endothelial cell dysfunction; second, a hyper-inflammato

60 On the other hand, endothelial cell dysfunction suggested by poor hyaluroni

61 dings offer insights into aging-related lung endothelial cell dysfunction that may contribute to defe

62 n healthy conditions and the contribution of endothelial cell dysfunction to cardiovascular disease.

63 kidney disease patients and the relation of endothelial cell dysfunction to cardiovascular risk in c

64 present study investigated how induction of endothelial cell dysfunction via high glucose treatment

65 Dasatinib treatment mediated endothelial cell dysfunction via increased production of

66 ing play a role in HS-induced saphenous vein endothelial cell dysfunction, which may contribute to pr

67 ions and was very likely secondary to severe endothelial cell dysfunction with hypoperfusion and exte