コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 arteries and the role played by caveolae in endothelium-dependent relaxation.
2 nd estradiol plus progesterone do not affect endothelium-dependent relaxation.
3 sic coronary artery disease risk factors and endothelium-dependent relaxation.
4 stradiol plus progesterone did not influence endothelium-dependent relaxation.
5 ha release, and improved coronary arteriolar endothelium-dependent relaxation.
6 ae structure and integrity are essential for endothelium-dependent relaxation.
7 howed increased aortic stiffness and reduced endothelium-dependent relaxation.
8 oxidase, decreased O2*- levels, and improved endothelium-dependent relaxation.
9 hereas, in pregnant rats, PVAT-media reduced endothelium-dependent relaxation.
10 gs had no deterioration in contraction or in endothelium-dependent relaxation.
11 ed with decreased NO production and impaired endothelium-dependent relaxation.
12 dothelial cell hydrogen peroxide release, or endothelium-dependent relaxation.
13 er the onset of sepsis improves or maintains endothelium-dependent relaxation.
14 ial O2-. levels is not sufficient to improve endothelium-dependent relaxation.
15 of superoxide dismutase (SOD) would improve endothelium-dependent relaxation.
16 ed SOD, or cis-vaccenic acid did not augment endothelium-dependent relaxations.
17 ar function was measured via contraction and endothelium-dependent relaxation after stimulation with
18 d preservation of vascular smooth muscle and endothelium-dependent relaxations after prolonged hypoth
19 evels, resulting in reduced O2- and improved endothelium-dependent relaxation and basal NO release in
20 hyperpolarization, because in many arteries endothelium-dependent relaxation and hyperpolarization r
23 38 prevented NADP(H) depletion and preserved endothelium-dependent relaxation and NO generation with
24 tly (P < .05) enhanced acetylcholine-induced endothelium-dependent relaxation and NO, and reduced con
25 t to diabetes mellitus-induced impairment in endothelium-dependent relaxation and reendothelializatio
26 cy due to ET-1-induced O2- leads to impaired endothelium-dependent relaxation and that gene transfer
27 during diabetes by suppressing impairment of endothelium-dependent relaxation and that protection by
28 ich restored NO bioavailability and improved endothelium-dependent relaxations and HDL endothelium-pr
29 LPS or iNOS expression has little effect on endothelium-dependent relaxation, and eNOS activity does
30 gonist SR141716A (10 microM) also attenuated endothelium-dependent relaxations but this inhibition wa
31 yme A (HMG CoA) reductase inhibitors improve endothelium-dependent relaxation by increasing ecNOS act
32 cids and GLP-1 were associated with improved endothelium-dependent relaxation compared with sham-oper
33 In addition, impaired acetylcholine-induced endothelium-dependent relaxation could be related to dec
34 n human internal mammary arteries, depressed endothelium-dependent relaxations could not be attribute
35 2-CreNox2KO mice, along with preservation of endothelium-dependent relaxation during angiotensin II s
36 whether this agent attenuates the depressed endothelium-dependent relaxation during early sepsis.
38 ic tone was significantly potentiated, while endothelium-dependent relaxation (EDR) was impaired in s
40 P<.001), which was associated with impaired endothelium-dependent relaxations (EDRs) in aortic rings
41 vinblastine, their arteries maintained full endothelium-dependent relaxation, even after very high t
42 endothelium and smooth muscle contributes to endothelium-dependent relaxations evoked by both recepto
43 however, fibrinogen (0-2 microM) affected an endothelium-dependent relaxation, followed by recontract
44 significantly improved acetylcholine-induced endothelium-dependent relaxation in AMPKalpha2(-/-) mice
45 s demonstrated a significant preservation of endothelium-dependent relaxation in animals treated with
46 ion of l-sepiapterin normalized the impaired endothelium-dependent relaxation in aortas isolated from
48 ease in STIM1 protein expression, attenuates endothelium-dependent relaxation in diabetic coronary ar
49 of antioxidants, which are known to improve endothelium-dependent relaxation in HC, smooth muscle SE
50 osure to lucigenin resulted in inhibition of endothelium-dependent relaxation in isolated aortic ring
51 stress and SERCA oxidation and improved the endothelium-dependent relaxation in isolated mouse aorta
52 lose inspection reveals a specific effect on endothelium-dependent relaxation in mesenteric resistanc
56 e control, exhibited striking improvement in endothelium-dependent relaxation in response to acetylch
58 ed after 2 months exhibited markedly reduced endothelium-dependent relaxation in response to acetylch
60 ivity of the vasculature and to the impaired endothelium-dependent relaxation in the diseased kidney.
61 KATP channel mechanism, whereas PC preserves endothelium-dependent relaxation in the subepicardium th
62 at this time point, diabetes did not impair endothelium-dependent relaxation in vessels in wild-type
63 VHF rats (VHF vs. VC, P < 0.05) and blunted endothelium-dependent relaxation in VHF and PHF rats (VH
64 rom E also revealed a better preservation of endothelium-dependent relaxation in vitro (maximum relax
65 ed peptides (SFLLRN and SLIGRL) both induced endothelium-dependent relaxations in PGF2alpha-contracte
68 mice exhibited an accelerated impairment of endothelium-dependent relaxations in response to in vitr
70 finding that both trypsin and SLIGRL induced endothelium-dependent relaxations indicates the presence
73 heterozygout mice (db/m) mice and effects on endothelium-dependent relaxation, insulin sensitivity, a
75 pothesis that hypoxia-induced attenuation of endothelium-dependent relaxation is mediated by alterati
77 exhibit augmented contraction and diminished endothelium-dependent relaxation, most likely due to dec
84 reduced ejection fraction, mitral E/A ratio, endothelium-dependent relaxation of coronary arteries, t
85 with preserved left ventricular function and endothelium-dependent relaxation of coronary microvessel
86 st-stimulated production of nitric oxide and endothelium-dependent relaxation of isolated blood vesse
90 emporal profile of Ca(2+) dynamics underlies endothelium-dependent relaxation of swine coronary arter
91 re no acute changes in BP or the NO-mediated endothelium-dependent relaxation of the brachial artery
94 ived hyperpolarizing factor (EDHF)-mediated, endothelium-dependent relaxations of small mesenteric ar
97 to HIV-associated hypertension and impaired endothelium-dependent relaxation remains ill defined.
98 the Ca(2+) concentration in the ER, and (3) endothelium-dependent relaxation that was attenuated in
99 l stimuli acetylcholine or bradykinin evoked endothelium-dependent relaxation that was similar in con
100 endothelium and is associated with impaired endothelium-dependent relaxation, the effects of micromo
101 nses to cyclopiazonic acid, which stimulates endothelium-dependent relaxation through a receptor-inde
104 abetes caused a 25% reduction in NO-mediated endothelium-dependent relaxation to acetylcholine for ao
107 ed contractile responses to UTP and enhanced endothelium-dependent relaxation to calcium ionophore A2
109 grity of the endothelium was assessed by the endothelium-dependent relaxation to substance P in a pai
110 with superoxide dismutase plus sepiapterin, endothelium-dependent relaxations to A23187, as well as
112 on endothelium of pigs selectively augments endothelium-dependent relaxations to bradykinin by incre
114 ls, (2) angiotensin receptors do not mediate endothelium-dependent relaxations to the heptapeptide, a
115 (2) to amplify the ROS-induced impairment of endothelium-dependent relaxation via reduction of nitric
117 postexperimental coronary arteries, maximal endothelium-dependent relaxation was greater in CP+GSH t
120 ared to the wild type, acetylcholine-induced endothelium-dependent relaxation was significantly impai
122 reendothelialization of the damaged vessel, endothelium-dependent relaxation was tested in isolated
123 eotide regulatory (Gi) protein can result in endothelium-dependent relaxation, we tested the hypothes
126 helium-independent and prostacyclin-mediated endothelium-dependent relaxations were not changed.
127 els also demonstrated significantly impaired endothelium-dependent relaxation whereas the E1/E4-AV ve
128 choline and bradykinin both led to dose- and endothelium-dependent relaxation which was unaffected by
129 of WT aortas to Tg26 CD4(+) T cells impaired endothelium-dependent relaxation, which was blocked by I
130 gh glucose in vitro induced an impairment of endothelium-dependent relaxations, which was prevented b
131 sels from apoE(-/-) mice, uric acid improved endothelium-dependent relaxation while having no effect
132 cant time- and titer-dependent impairment in endothelium-dependent relaxation, with no effect on cont