ライフサイエンスコーパス: endothelium-dependent vasodilator

1 infusion of methacholine chloride (Mch), an endothelium-dependent vasodilator.

2 mulative administration of acetylcholine, an endothelium-dependent vasodilator.

3 itric oxide bioavailability at rest and with endothelium-dependent vasodilators.

4 t K(IR) channels function as 'amplifiers' of endothelium-dependent vasodilators.

5 ty and preserves renal vascular responses to endothelium-dependent vasodilators.

6 at rest, during exercise, and in response to endothelium-dependent vasodilators.

7 n produced by neocortical application of the endothelium-dependent vasodilator acetylcholine (10 micr

8 r changes were determined in response to the endothelium-dependent vasodilator acetylcholine (ACh, 10

9 nses to intra-arterial administration of the endothelium-dependent vasodilator acetylcholine (P=0.03)

10 n during intracoronary administration of the endothelium-dependent vasodilator acetylcholine and the

11 Coronary blood flow in response to the endothelium-dependent vasodilator acetylcholine as well

12 threshold dose of genistein potentiated the endothelium-dependent vasodilator acetylcholine but not

13 The endothelium-dependent vasodilator acetylcholine elicited

14 n but did not ameliorate the response to the endothelium-dependent vasodilator acetylcholine.

15 d by neural activity, hypercapnia, or by the endothelium-dependent vasodilator acetylcholine.

16 ndothelial cell function was assessed by the endothelium-dependent vasodilator acetylcholine.

17 cterized by vasoconstrictive response to the endothelium-dependent vasodilator acetylcholine.

18 coronary vasoconstriction in response to the endothelium-dependent vasodilator acetylcholine.

19 ) were calculated during (1) infusion of the endothelium-dependent vasodilators acetylcholine (ACh) a

20 008), Responses of resistance vessels to the endothelium-dependent vasodilators acetylcholine and ADP

21 Dose responses for an endothelium-dependent vasodilator, acetylcholine (ACh, v

22 Elevation of IP(3) by the endothelium-dependent vasodilator, acetylcholine, increa

23 absolute forearm blood-flow responses to the endothelium-dependent vasodilator, acetylcholine, increa

24 renergic receptor agonist, phenylephrine; b) endothelium-dependent vasodilator, acetylcholine; and c)

25 ents the impaired vascular relaxation to the endothelium-dependent vasodilator ACh, this agent may be

26 ion responses of precontracted arterioles to endothelium-dependent vasodilators adenosine 5'-diphosph

27 ter 2 hours of reperfusion, but those to the endothelium-dependent vasodilator ADP and the endotheliu

28 alpha(1) -agonist) during (i) infusion of an endothelium-dependent vasodilator alone (Protocol 1: ACh

29 tudies show that apelin receptor ligands are endothelium-dependent vasodilators and potent inotropes,

30 cetylcholine (7.5, 15 and 30 microg/min), an endothelium-dependent vasodilator, and sodium nitropruss

31 in) and bradykinin (BK, 2.5 micrograms/min), endothelium-dependent vasodilators, and sodium nitroprus

32 Although responses to acetylcholine, an endothelium-dependent vasodilator, are abnormal in patie

33 Responses to endothelium-dependent vasodilators, both in resistance v

34 /min), SLIGKV (160 to 800 nmol/min), and the endothelium-dependent vasodilator bradykinin (100 to 100

35 Relaxation to the endothelium-dependent vasodilator bradykinin was decreas

36 cid (LPA) has been recognized recently as an endothelium-dependent vasodilator, but several lines of

37 Similar to Ercc1(d/-) mice, age-related endothelium-dependent vasodilator dysfunction in Xpd(TTD

38 ate that blockade of ET-1 receptors improves endothelium-dependent vasodilator function in hypertensi

39 Since endothelium-dependent vasodilator function may be affect

40 n of increased ET-1 activity to the impaired endothelium-dependent vasodilator function of hypertensi

41 e and that this is associated with a reduced endothelium-dependent vasodilator function, endothelium-

42 s to acetylcholine and in vitro responses to endothelium-dependent vasodilators in angiotensin II-tre

43 sepiapterin or MH4 restores the response to endothelium-dependent vasodilators in pig coronary arter

44 ctors, indicating that the reduced effect of endothelium-dependent vasodilators in those with hyperte

45 blood flow elicited by neural activity or by endothelium-dependent vasodilators in WT mice but not in

46 Response of IP(DVP) to endothelium-dependent vasodilators may provide a measure

47 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

48 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

49 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

50 graded intrafemoral artery infusions of the endothelium-dependent vasodilator methacholine chloride

51 response to norepinephrine, and an impaired endothelium-dependent vasodilator response to acetylchol

52 erhomocyst(e)inemic mice restored the normal endothelium-dependent vasodilator response.

53 Endothelium-dependent vasodilator responses to acetychol

54 t with apo A-I(M)/PC prevented impairment of endothelium-dependent vasodilator responses to acetylcho

55 on and no impairments in vasoconstrictor and endothelium-dependent vasodilator responses, associated

56 amin E therapies similarly improved arterial endothelium-dependent vasodilator responsiveness consist

57 lipoprotein levels, specific improvement in endothelium-dependent vasodilator responsiveness is simi

58 ange in augmentation index in response to an endothelium-dependent vasodilator (salbutamol).

59 nd dilated in a dose-dependent manner to the endothelium-dependent vasodilators serotonin, ATP, and i

60 coronary artery constriction in response to endothelium-dependent vasodilator stimuli such as the co

61 w responses to intrabrachial infusion of the endothelium-dependent vasodilators substance P and acety

62 IK(Ca)/SK(Ca)), NS309 (10(-5) M), and to the endothelium-dependent vasodilators, substance P (10(-8)

63 Endothelium-dependent vasodilators, such as acetylcholin

64 roxide production and the response to either endothelium-dependent vasodilator used.

65 systemic vascular reactivity in response to endothelium-dependent vasodilators, which may be mediate

66 , we hypothesized that the responsiveness to endothelium-dependent vasodilators would be greater in t