ライフサイエンスコーパス: endplate potential

1 nstant of repolarisation of the conditioning endplate potential.

2 s determined by micro-electrode recording of endplate potentials.

3 frequency but not the amplitude of miniature endplate potentials.

4 aracterized by a reduction in both miniature endplate potential amplitude and AChR abundance accompan

5 Miniature endplate potential amplitude was reduced 3 d after SC ab

6 Miniature endplate potential amplitude, but not frequency, was red

7 s muscle biopsy revealed decreased miniature endplate potential amplitudes, reduced endplate size and

8 t decrease of the amplitude of the miniature endplate potential and no deficiency of the ACh receptor

9 the mean amplitude of spontaneous miniature endplate potentials and bungarotoxin binding.

10 fatigable muscle weakness, reduced miniature endplate potentials and endplate potentials, reduced mot

11 el kinetics, or endplate ultrastructure, but endplate potentials depolarizing the resting potential t

12 or DuP 697, prevents the delayed increase in endplate potential (EPP) amplitude normally produced by

13 n the kinetics of stretch-induced changes in endplate potential (EPP) amplitude or miniature EPP (mEP

14 Cyclohexanol (10-25 mM) reduced endplate potential (EPP) amplitudes by 10-40% and enhanc

15 ontrol solution), the quantal content of the endplate potential (EPP) depressed more rapidly (approxi

16 nse to nerve stimulation was determined from endplate potentials (EPPs) and endplate currents (EPCs)

17 Similarly, endplate potentials (EPPs) evoked at low frequency were

18 ality between staining/destaining and summed endplate potentials (EPPs) representing total transmitte

19 Spontaneous MEPPs and uniquantal endplate potentials (EPPs) were released over the same l

20 mplitude; extracellular stimulation elicited endplate potentials (EPPs) which resembled MEPPs.

21 uires cholinergic signaling, which generates endplate potentials (EPPs), and excitation, the amplific

22 ll partially or fully occupied and expressed endplate potentials (EPPs).

23 ological measurements of ACh secretion (i.e. endplate potentials, EPPs) and the component of the prej

24 ed acetylcholine (ACh) release (reflected as endplate potentials, EPPs) is well described by a simple

25 tosolic [Ca2+], and reduced the amplitude of endplate potentials evoked after the end of a stimulus t

26 oved synergistic in restoring suprathreshold endplate potentials in mouse diaphragms fully intoxicate

27 depressed nerve control, increased miniature endplate potential (MEPP) amplitude, decreased MEPP freq

28 monstrate an increased spontaneous miniature endplate potential (mEPP) frequency in Nedd4 mutants.

29 ncreases evoked quantal output and miniature endplate potential (MEPP) frequency, again by activating

30 The size of the miniature endplate potential (MEPP) increased 3- or 4-fold in prep

31 measuring the relative changes of miniature endplate potentials (mEPPs) and voltage responses to ste

32 Intervals of spontaneous miniature endplate potentials (MEPPs) are usually thought to follo

33 Boutons produced spontaneous miniature endplate potentials (MEPPs) of nearly normal amplitude;

34 C on neurosecretion in the form of miniature endplate potentials (MEPPs) were assessed.

35 Miniature endplate potentials (MEPPs) were more frequent near the

36 f spontaneous transmitter release (miniature endplate potentials (MEPPs)) from motor nerve terminals

37 tude or time course of spontaneous miniature endplate potentials (MEPPs).

38 tudy also showed a striking reduction of the endplate potential quantal content, consistent with addi

39 s, reduced miniature endplate potentials and endplate potentials, reduced motor endplate AChR number

40 Similar decreases in miniature endplate potential size ([integral]MEPP) followed repeti

41 ing continued, the fraction of the miniature endplate potential voltage-time integrals ( MEPPs) in th

42 the other hand, the mean amplitude of evoked endplate potentials was not decreased, due to an increas

43 The amplitude and rise time of miniature endplate potentials were also increased, but these chang

44 On average, 63% of the endplate potentials were also seen in both recordings.

45 The amplitude and frequency of miniature endplate potentials were reduced, indicating impaired ne