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2 Enz-resistant CRPC cells and associated with enhanced transcription of a subset of tumor promoting ge
3 influences E2F-1 protein stability, and the enhanced transcription of a variety of downstream target
6 (NRF2) is orchestrated and amplified through enhanced transcription of antioxidant and antiinflammato
7 Genetic disruption of NELF in macrophages enhanced transcription of AP-1-encoding Fos and Jun and,
9 the unfolded protein response, manifested by enhanced transcription of ATF4 and C/EBP homologous prot
11 egions (-8.2 to -7.5 kb and -7.5 to -7.0 kb) enhanced transcription of both A-chain and heterologous
12 ely dependent on YvrI and is correlated with enhanced transcription of both the yvrI-yvrHa and the ox
15 G1 cyclins and CDKs and the other involving enhanced transcription of CDIs by the activated receptor
17 epithelial cells with PMA is associated with enhanced transcription of Cox-2 and increased production
18 t cellular transformation is associated with enhanced transcription of Cox-2 and increased production
20 of transcription factor, NF-kappaB; and (iv) enhanced transcription of cytochrome oxidase Vb (COX Vb)
21 the mouse dor promoter was required for the enhanced transcription of dor gene in phytohemagglutinin
22 lation induced by DNA damage correlates with enhanced transcription of downstream p53 target genes.
23 nally triggered CDNs lead to the CDNs-guided enhanced transcription of either the DFHBI aptamer or th
24 res despite the fact that neither polyphenol enhanced transcription of elastin mRNA or cellular proli
25 tion of AI-2 increased cell motility through enhanced transcription of five motility genes, we propos
27 f lamprey blood cells with rIL-17D.1 protein enhanced transcription of genes expressed by the B-like
28 e acetylation, chromatin reorganization, and enhanced transcription of genes, such as ET-1, enhancing
30 ives HIF-1alpha protein accumulation through enhanced transcription of HIF-1alpha mRNA, a process tha
32 p 3 innate lymphoid cells and Th17 cells and enhanced transcription of IL-22, Ab-mediated neutralizat
33 ich causes nuclear accumulation of TRX-1 and enhanced transcription of inflammatory mediators through
34 ed the inflammatory response, as measured by enhanced transcription of interleukin-6 and tumor necros
36 increased C-Jun transcriptional activity and enhanced transcription of matrix metalloproteinase 10 (M
39 d through an induction process that requires enhanced transcription of mtrCDE when gonococci are grow
40 ions are coupled with reduced phytotoxicity, enhanced transcription of MYB72 in roots, and a positive
43 lycosaminoglycan binding was associated with enhanced transcription of peptidoglycan recognition prot
44 that recognizes the inducers and signals the enhanced transcription of phase 2 genes does so by virtu
45 ERH1 results in salicylic acid accumulation, enhanced transcription of RPW8 and RPW8-dependent sponta
47 increased G4 accumulation is associated with enhanced transcription of signalling pathways previously
49 pendent interactions are required to achieve enhanced transcription of specific estrogen-receptor tar
50 ere with beta-catenin turnover, resulting in enhanced transcription of target genes through the incre
51 Jun, resulting in diminished degradation and enhanced transcription of the Bcl-2 homology 3 domain-on
52 tigen and beta-catenin in colon cancer cells enhanced transcription of the c-myc promoter, the downst
54 ncrease in Egr-1 protein and mRNA levels and enhanced transcription of the Egr-1 gene via serum respo
56 stasis by insulin is mediated in part by the enhanced transcription of the gene encoding SREBP-1c (st
57 stasis by insulin is mediated in part by the enhanced transcription of the gene encoding sterol regul
61 This increase in enzyme activity reflects E6-enhanced transcription of the human telomerase reverse t
62 dence that these mutations in SRC-3 promoted enhanced transcription of the IGFBP3 gene and globally i
63 we present evidence that anguibactin itself enhanced transcription of the iron-transport genes fatA
64 the activation of nuclear factor kappaB and enhanced transcription of the KC gene with equal potency
69 ases in RPE cells occurred primarily through enhanced transcription of the VEGF gene and via the IGF-
71 rd4 with growth factor-responsive genes, and enhanced transcription of these genes that could be atte
73 ription factors, and that is associated with enhanced transcription of this gene and increased enzyme
74 ha (HIF-1alpha) and HIF-2alpha, resulting in enhanced transcription of transforming growth factor-alp