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1  via direct transcriptional up-regulation of enhancer RNA.
2  a super-enhancer element and its associated enhancer RNA.
3 lease, and elongation, while also processing enhancer RNAs.
4 irus-induced-TEs are either eRNAs or part of enhancer RNAs.
5 the human genome, are often transcribed into enhancer RNAs.
6 s, signaling pathways, and interactions with enhancer RNAs.
7 promoter upstream transcripts (PROMPTs), and enhancer RNAs.
8 for the biogenesis of small nuclear RNAs and enhancer RNAs.
9 n, thus defining a novel class of regulatory enhancer RNAs.
10 re not multiexonic, thus are more similar to enhancer RNAs.
11 s transcription units, generating non-coding enhancer RNAs.
12 tergenic lncRNAs, antisense transcripts, and enhancer RNAs.
13 and are required to produce their associated enhancer RNAs.
14 nhancer transcription process and non-coding enhancer RNAs.
15 for the biogenesis of small nuclear RNAs and enhancer RNAs.
16 er-enhancer interactions and the presence of enhancer RNAs.
17 rts, encompassing 33,321 long ncRNAs, 92,897 enhancer RNAs, 53,763 alternative polyadenylation events
18                                  We found an enhancer RNA, AANCR, that regulates the transcription an
19 , rs2280381 mediates IRF8 expression through enhancer RNA AC092723.1, which recruits TET1 to the IRF8
20 odels of genetically regulated expression of enhancer RNAs across 49 cell and tissue types, character
21 ociation studies, the model identified 7,775 enhancer RNAs and 3,548 long ncRNAs causally associated
22 timated genetically regulated expression for enhancer RNAs and canonical genes in a large-scale DNA b
23 re, we expand on current concepts to discuss enhancer RNAs and enhancer transcription, and how enhanc
24                 Structured (CRS overlapping) enhancer RNAs and extended 3' ends have significantly in
25 to target sites and by generating non-coding enhancer RNAs and localized regions of trimethylated his
26  (U snRNAs), PIWI-interacting RNAs (piRNAs), enhancer RNAs and nascent pre-mRNAs.
27 ons with enhancer RNAs emphasize the role of enhancer RNAs and the overall structural aspects of tran
28 th open chromatin maps to identify potential enhancer RNAs and their associated enhancer regions in t
29       The biological functions of extragenic enhancer RNAs and their impact on disease risk remain re
30 s to be mediated by long, rather than short, enhancer RNAs and to be more prominent in intragenic, ra
31 atin remodeling, transcribes a bidirectional enhancer RNA, and loops to physically interact with the
32 haracterized by PU.1 binding, an increase of enhancer RNA, and P300 recruitment.
33 arRNAs), including promoter-associated RNAs, enhancer RNAs, and repeat RNAs.
34 egulate RNAPII recruitment, the synthesis of enhancer RNAs, and the activation of tumor-promoting gen
35 calize with causal regulatory variation when enhancer RNAs are included.
36 cipitation of hnRNP H cross-linked to the N1 enhancer RNA, as well as gel mobility shift analysis of
37 l expression of regional genes and a retinal enhancer RNA at this locus was assessed by qPCR.
38 nstructed a user-friendly data portal, Human enhancer RNA Atlas.
39 enhancer RNAs independent of GWAS loci using enhancer RNA-based TWAS and determine the causal effects
40                                        Using enhancer RNA-based TWAS, we generate a comprehensive res
41 ly of distal enhancers expressing non-coding enhancer RNAs bi-directionally, reminiscent of "super en
42 ggests that enhancers can generate noncoding enhancer RNAs, but their (patho)biological functions rem
43  of lncRNA biology, such as the functions of enhancer RNAs, circular RNAs and chemical modifications
44 functional enhancers requires assembly of an enhancer RNA-dependent ribonucleoprotein (eRNP) complex
45 ection assay (GRO/PRO-cap) has advantages in enhancer RNA detection and active enhancer identificatio
46                                          The enhancer RNAs did not appear to contain a normal cap str
47                             We also describe enhancer RNA dynamics revealing transcriptional bursts i
48 uggest that MUNC is not a classic cis-acting enhancer RNA (e-RNA) acting exclusively by stimulating t
49                       We have identified new enhancer RNA elements and an extended stem-loop in the N
50                      Genetic modification of enhancer RNA elements can be utilized for designing atte
51       A complex of protein interactions with enhancer RNAs emphasize the role of enhancer RNAs and th
52 despread phenomenon which produces so-called enhancer RNA (eRNA) and occurs in an activity-dependent
53  thus, unstable transcripts such as putative enhancer RNA (eRNA) are dramatically increased.
54 ombination of DNase footprints and divergent enhancer RNA (eRNA) as markers for functional variants.
55                                        Using enhancer RNA (eRNA) expression to measure enhancer activ
56                                              Enhancer RNA (eRNA) is a type of long non-coding RNA tra
57                                              Enhancer RNA (eRNA) is a type of noncoding RNA transcrib
58                                           An enhancer RNA (eRNA) is transcribed in thyroid tissue fro
59 f divergent gene promoters and bidirectional enhancer RNA (eRNA) production at enhancers.
60 bal run-on (GRO) sequencing indicated robust enhancer RNA (eRNA) production from three of these GBSs
61 mapped p53-regulated enhancers by looking at enhancer RNA (eRNA) production.
62     Enhancer activities can be inferred from enhancer RNA (eRNA) signals, which requires enhancer tra
63 tor C/EBPa followed by chromatin opening and enhancer RNA (eRNA) synthesis from H3K4-monomethylated r
64 er-associated long noncoding RNA) is a novel enhancer RNA (eRNA) that amplifies oncogenic transcripti
65 rred via 40p53:WTp53-dependent inhibition of enhancer RNA (eRNA) transcription and subsequent failure
66 vation programs based on release of a shared enhancer RNA (eRNA) transcription checkpoint.
67                                Bidirectional enhancer RNA (eRNA) transcription is a widespread respon
68 r candidate compendium based on the detected enhancer RNA (eRNA) transcription start sites (TSSs) ava
69 her H3K27ac, both of which were required for enhancer RNA (eRNA) transcription.
70 upancy correlates with enhancer activity and enhancer RNA (eRNA) transcription.
71 onal enhancers together with their noncoding enhancer RNA (eRNA) transcripts.
72  uridine-rich small nuclear RNA (UsnRNA) and enhancer RNA (eRNA), and in the transcription of coding
73 lecular QTLs data, including mRNA, splicing, enhancer RNA (eRNA), and protein expression data from up
74                                              Enhancer RNAs (eRNA) are unstable non-coding RNAs, trans
75 lso transcribed into noncoding RNAs known as enhancer RNAs (eRNA).
76 calization, defining lncRNAs classes such as enhancer-RNAs (eRNA), bivalent-lncRNAs, and CTCF-associa
77   Over the last decade, the transcription of enhancer RNAs (eRNAs) - nascent RNAs transcribed from ac
78 diminishes the signal-dependent induction of enhancer RNAs (eRNAs) and abrogates stimulus-induced enh
79      Active enhancers produce bi-directional enhancer RNAs (eRNAs) and display CBP/p300-dependent his
80 at lack enhancer activity, were enriched for enhancer RNAs (eRNAs) and preferentially interacted in v
81  by Liang et al.(1) reveals that interacting enhancer RNAs (eRNAs) and promoter-transcribed upstream
82 acetylation and increased expression of both enhancer RNAs (eRNAs) and target genes.
83 based activator can induce the production of enhancer RNAs (eRNAs) and that this eRNA induction is po
84 re located in transcribed sequences encoding enhancer RNAs (eRNAs) and were shown to impair enhancer
85                                              Enhancer RNAs (eRNAs) are a class of long noncoding RNAs
86                                              Enhancer RNAs (eRNAs) are short noncoding RNAs transcrib
87 ription is pervasive at active enhancers and enhancer RNAs (eRNAs) are tightly coupled to regulated t
88                                              Enhancer RNAs (eRNAs) are transcribed by RNA polymerase
89 -on sequencing (Gro-seq) data found abundant enhancer RNAs (eRNAs) being transcribed at ESEs.
90                                              Enhancer RNAs (eRNAs) have emerged as an important compo
91                          Here we report that enhancer RNAs (eRNAs) identified by global nuclear run-o
92            We discuss the potential roles of enhancer RNAs (eRNAs) in early elongation and highlight
93 s encompass noncoding, actively transcribing enhancer RNAs (eRNAs) in the SCN, which in turn oscillat
94 riant, we investigated the potential role of enhancer RNAs (eRNAs) located near the GATA3 locus in re
95                Furthermore, the discovery of enhancer RNAs (eRNAs) provides an alternative approach t
96  understanding of enhancer transcription and enhancer RNAs (eRNAs) remains incomplete.
97                      Unbiased examination of enhancer RNAs (eRNAs) that cluster in specific circadian
98 number of long non-coding RNAs (lncRNAs) and enhancer RNAs (eRNAs) with emergent functionality.
99 ranscribes bi-directionally a novel class of enhancer RNAs (eRNAs) within enhancer domains defined by
100                               We identify 76 enhancer RNAs (eRNAs), 40 canonical lncRNAs, 65 antisens
101 , integrating transcription of coding genes, enhancer RNAs (eRNAs), and various other noncoding trans
102 miRNAs), long non-coding RNAs (lncRNAs), and enhancer RNAs (eRNAs), as well as yet undiscovered class
103 its upstream enhancers produce bidirectional enhancer RNAs (eRNAs), termed KLK3e.
104 selves also undergo transcription to produce enhancer RNAs (eRNAs), the significance of which is not
105    By overlapping virus-induced-TEs to human enhancer RNAs (eRNAs), we discovered that a proportion o
106 cluding promoter upstream antisense RNAs and enhancer RNAs (eRNAs), which positively correlates with
107 tive effect and both induce coding genes and enhancer RNAs (eRNAs).
108 ctional transcription to generate non-coding enhancer RNAs (eRNAs).
109 recruit transcription complexes and generate enhancer RNAs (eRNAs).
110 wn that enhancers are transcribed to produce enhancer RNAs (eRNAs).
111 nic cheRNA (icheRNA) and their similarity to enhancer RNAs (eRNAs).
112  are synthesized at active enhancers, called enhancer RNAs (eRNAs).
113 ng noncoding RNA (lncRNA) core promoters and enhancer RNAs (eRNAs).
114 ains (TADs) express noncoding RNAs, known as enhancer RNAs (eRNAs).
115 ed on enhancers, and are thus referred to as enhancer RNAs (eRNAs).
116 transcribed into long noncoding RNAs termed "enhancer RNAs" (eRNAs), their putative role in enhancer
117  most promising candidate, hypoxia-inducible enhancer RNA ( HERNA)1, was further examined by investig
118 ese findings demonstrate that AANCR is a key enhancer RNA in some cell types within the nervous syste
119 ate that ARIEL plays an oncogenic role as an enhancer RNA in T-ALL.
120 primary micro-RNAs, long noncoding RNAs, and enhancer RNAs in a large animal model of acute infarctio
121         We identify schizophrenia-associated enhancer RNAs independent of GWAS loci using enhancer RN
122         Here, we report that CTCF binding to enhancer RNAs is enriched when breast cancer cells are s
123 s the transcription of ncRNAs, especially of enhancer RNAs, is often low and cell type specific, how
124 Here we show that a conserved long noncoding enhancer RNA (lnc-eRNA), transcribed from an activity-se
125  histone-, promoter upstream transcript- and enhancer RNA-loci.
126 e in both structure and function and include enhancer RNAs, long ncRNAs, and microRNAs, all of which
127 hat enhancer transcription and the resulting enhancer RNAs may, in some cases, have functional roles,
128 ancers exhibiting differential expression of enhancer RNAs pointed a central role for Kruppel-like fa
129 ngaged RNA polymerase (Pol) II, transcribing enhancers, RNA Pol I and Pol III activities and potentia
130 s identified unstable transcripts resembling enhancer RNAs, pri-miRNAs, antisense transcripts, and pr
131  induction kinetics, chromosome looping, and enhancer RNA production to understand the distinct regul
132         Consequently, Brd4 deletion prevents enhancer RNA production, cell identity gene induction an
133  of engaged RNA polymerases showed a lack of enhancer RNAs, promoter-proximal pausing, and divergent
134 e use of genetically regulated expression of enhancer RNAs provides substantial tissue-specific predi
135                               Targeting this enhancer RNA reduced NRF1 levels and increased tumor cel
136                                          The enhancer RNAs remained in discrete spots within the nucl
137 rs with regulatory sites termed silencers or enhancers, RNA-RNA base-pairing interactions, or chromat
138                Competing endogenous RNAs and enhancer RNAs show interesting functional and regulatory
139 regulation, transcription factor binding and enhancer RNA signatures.
140 A Pol II transcription levels via impacts on enhancer RNA synthesis, and interacts with the rRNA prod
141 e II (RNAPII), including small nuclear RNAs, enhancer RNAs, telomeric RNAs, viral RNAs, and protein-c
142  -interesting when considering its impact on enhancer RNAs that are less prominent in single-celled o
143 evels in FFPE specimens while also detecting enhancer RNAs that arise from distal regulatory regions.
144 transcribed at CHPT1 enhancer (also known as enhancer RNA) that binds to the H3K27ac reader BRD4 and
145                 MYC induces transcription of enhancer RNA through recruitment of RNA polymerase II (R
146 ants plausibly altering the transcription of enhancer RNAs to influence the risks of Crohn's disease
147  enhancers and is required for activation of enhancer RNA transcription and recruitment of coactivato
148 ctivators, exemplified by p300, causing both enhancer RNA transcription and target gene activation.
149 st cancer cells, and loss of FOXA1 increases enhancer RNA transcription for a representative basal ge
150  coordinate induction of TNF, LTA, and hHS-8 enhancer RNA transcription occurs.
151 invokes changes in histone modifications and enhancer RNA transcription that correspond to altered ex
152 ancers exhibit reduced Mediator recruitment, enhancer RNA transcription, and enhancer-promoter intera
153  H3K27 acetylation and methylation and super-enhancer RNA transcription.
154                 Moreover, targeted depletion enhancer RNAs using antisense oligonucleotide silences B
155 AS and determine the causal effects of these enhancer RNAs using Mendelian randomization.
156 ndidate we named FIXER (fibrogenic LOX-locus enhancer RNA), we showed that its silencing limits fibro
157                Our studies demonstrated that enhancer RNAs were produced from a subset of the regulat
158                                          The enhancer RNAs were synthesized not from a defined site b
159 istically define svRNA as a small regulatory enhancer RNA, which functions to promote genome replicat
160 ion and within regulatory regions containing enhancer RNAs, while highlighting the conflict between e
161 ence of purifying selection on the resulting enhancer RNAs within the human population.

 
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