戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 dicating that the virus may use endosomes to enter host cells.
2 that use different cell surface receptors to enter host cells.
3 t binds up to five GM1 glycosphingolipids to enter host cells.
4  which receptor is used by a viral strain to enter host cells.
5 d viruses exploit the endomembrane system to enter host cells.
6 ay utilize clathrin-dependent endocytosis to enter host cells.
7  unclear whether TrkC is used by T. cruzi to enter host cells.
8 ifferent pathways that animal viruses use to enter host cells.
9  often hijacked by intracellular microbes to enter host cells.
10 ACE2 from a broad range of animal species to enter host cells.
11  three proteins--sigma1, mu1, and sigma3--to enter host cells.
12 pilus and the products of tolQRA in order to enter host cells.
13 involved in the ability of L. pneumophila to enter host cells.
14 ium significantly impaired in its ability to enter host cells.
15  drug was removed the parasites were able to enter host cells.
16  gliding, to move on solid substrates and to enter host cells.
17 ses COVID-19, uses its spike glycoprotein to enter host cells.
18 revolving door mechanism exploited by IAV to enter host cells.
19 e protein's receptor binding domain (RBD) to enter host cells.
20 c effectors, including PiSCR74, which do not enter host cells.
21 ycoprotein anchored in the viral membrane to enter host cells.
22 es two envelope glycoproteins, Gn and Gc, to enter host cells.
23 ession of ACE2 which SARS- CoV-2 exploits to enter host cells.
24 l species, including bats but not humans, to enter host cells.
25  gene ssaH, are transcribed after Salmonella enters host cells.
26 ersists regarding the mechanism by which LPS enters host cells.
27                      Influenza A virus (IAV) enters host cells after attachment of its hemagglutinin
28 e respiratory syndrome coronavirus 2 uses to enter host cells, along with the host protease TMPRSS2.
29       Many viruses and toxins disassemble to enter host cells and cause disease.
30 al and cellular factors that allows HSV-1 to enter host cells and establish infection.
31 2) is the pivotal receptor for SARS-CoV-2 to enter host cells and provides thus a link between COVID-
32 antly increases the ability of Salmonella to enter host cells and required interaction with both acti
33                                      To both enter host cells and spread from cell to cell within inf
34 ic bacteria interact with human integrins to enter host cells and to augment host colonization.
35 sing by host proprotein convertases (PCs) to enter host cells and to cause disease.
36                The anthrax lethal toxin (LT) enters host cells and enzymatically cleaves MAPKKs or ME
37 ine encephalomyelitis virus (TMEV) binds and enters host cells and the molecules involved are not com
38 ensin-converting enzyme 2 (ACE2) receptor to enter host cells, and neutralizing antibodies are effect
39 ytic pathway, to NPC1(+) LE/Lys, in order to enter host cells, and that they do so to access high lev
40                                        After entering host cells, ArPEC25 localizes to the nucleus an
41 vide new information about how the BoNTs can enter host cells as well as a basis for understanding th
42          The JCPyV-HD5 complexes bind to and enter host cells but are reduced in their ability to rea
43  mutant could produce normal appressoria and enter host cells but failed to develop, indicating the a
44 8-kDa mutant protein retains the capacity to enter host cells but lacks detectable toxic activity.
45              Members of this phylum actively enter host cells by a multistep process with the help of
46 nica multiple nucleopolyhedrovirus (AcMNPV), enter host cells by clathrin-mediated endocytosis.
47                                         HNVs enter host cells by fusing the viral and cellular membra
48                        Parainfluenza viruses enter host cells by fusing the viral and target cell mem
49                              Paramyxoviruses enter host cells by fusing the viral envelope with a hos
50 viral infections.IMPORTANCEEnveloped viruses enter host cells by fusing their envelope with a cellula
51                    Diverse enveloped viruses enter host cells by fusing their envelopes with cell mem
52 d pathogen human parainfluenza virus type 3, enter host cells by fusion of the viral and target cell
53    Ebola and Marburg viruses are believed to enter host cells by receptor-mediated endocytosis.
54 s organism not only is able to attach to and enter host cells by using its cell surface-associated fa
55     Together, these results suggest that HSV enters host cells by an intracellular route independent
56 ry syndrome coronavirus 2 (SARS-CoV-2) virus enters host cells by binding to the angiotensin-converti
57 prevented BDV infection, indicating that BDV enters host cells by endocytosis and requires an acidic
58                              Influenza virus enters host cells by endocytosis.
59 al RNA-containing viral particles, and (iii) enter host cells, complete reverse transcription, enter
60 evidence that the ability of streptococci to enter host cells contributes to the frequent failure of
61                            Enveloped viruses enter host cells either through endocytosis, or by direc
62 fs were required for the fungal effectors to enter host cells in the absence of any pathogen-encoded
63 rphology improves the ability of a virion to enter host cells in the presence of neutralizing antibod
64 lectively, these data suggest that C. jejuni enters host cells in a targeted and tightly controlled p
65  the molecular mechanisms utilized by PRV to enter host cells is an important step in identifying nov
66  late blight pathogen Phytophthora infestans enter host cells is unknown.
67                                  This strain enters host cells less effectively than does the wild-ty
68 oviruses of different species are assumed to enter host cells mainly by interactions between the knob
69  better understanding of how influenza virus enters host cells may lead to the development of more-ef
70                      Influenza A virus (IAV) enters host cells mostly through clathrin-dependent rece
71 (ACE2) is crucial for SARS-CoV-2 to bind and enter host cells, no study has systematically assessed t
72 er, the receptor(s) that the PSV utilizes to enter host cells remains largely unknown.
73 by which these fungal sRNAs are secreted and enter host cells remains unclear.
74 of the mechanism by which HPV16 binds to and enters host cells remains fragmented.
75                                           To enter host cells, the viral spike protein (S-protein) bi
76      Both viruses have evolved an ability to enter host cells through direct interaction with the ang
77                    Diverse enveloped viruses enter host cells through endocytosis and fuse with endos
78                          Influenza A viruses enter host cells through endosomes, where acidification
79     While the spherical virions are known to enter host cells through exploitation of clathrin-mediat
80 e prelimary results suggest that filoviruses enter host cells through receptor-mediated endocytosis v
81 The single-stranded DNA (ssDNA) parvoviruses enter host cells through receptor-mediated endocytosis,
82 es (IAV) and several other viruses that also enter host cells through the endocytic pathway.
83                                    The virus enters host cells through binding of a receptor-binding
84                    Here, we reveal that HKU5 enters host cells utilizing Pipistrellus abramus (P.abr)
85                       Human papillomaviruses enter host cells via a clathrin-independent endocytic pa
86                                   Poxviruses enter host cells via a conserved entry-fusion complex (E
87 Human coronaviruses HCoV-NL63 and SARS-CoV-2 enter host cells via ACE2 and utilize the protease TMPRS
88        Influenza virus has been described to enter host cells via clathrin-mediated endocytosis.
89                                Bacteriophage enter host cells via membrane-bound surface receptors of
90                                 Alphaviruses enter host cells via receptor-mediated endocytosis, usin
91 protein is the main handle for SARS-CoV-2 to enter host cells via surface angiotensin-converting enzy
92                                         hMPV enters host cells via a unique mechanism, in that viral
93 ry syndrome coronavirus 2 (SARS-CoV-2) virus enters host cells via an interaction between its Spike p
94 e and fungal pathogens have been inferred to enter host cells, where they interact with host resistan