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1 al deficits and physiological changes to the enteric nervous system.
2 nd adaptive immunity, and dysfunction of the enteric nervous system.
3 HGF and MET are expressed in fetal and adult enteric nervous system.
4 n between the enteroendocrine system and the enteric nervous system.
5 e along with the drivers of their input, the enteric nervous system.
6 Little is known of how Tat affects the enteric nervous system.
7 ed during HIV infection are regulated by the enteric nervous system.
8 neuronal as well as glial progenitors in the enteric nervous system.
9 role of miRNAs in HFD-induced damage to the enteric nervous system.
10 motility disorders caused by diseases of the enteric nervous system.
11 eristalsis is significantly dependent on the enteric nervous system.
12 s show a significant increase in glia in the enteric nervous system.
13 (Hand2(+/-) mice) numbers of neurons in the enteric nervous system.
14 in contrast to the neurodegeneration in the enteric nervous system.
15 ural crest that colonize the gut to form the enteric nervous system.
16 role in the development and survival of the enteric nervous system.
17 eric neurons but also is latent in the human enteric nervous system.
18 es produced by enteroendocrine cells and the enteric nervous system.
19 neurite fasciculation necessary to form the enteric nervous system.
20 e most common congenital malformation of the enteric nervous system.
21 pin-releasing factor (CRF) in the guinea pig enteric nervous system.
22 nal development with a partial rescue of the enteric nervous system.
23 ory neurons (EP cells) within the developing enteric nervous system.
24 s of cells to the terminal bowel to form the enteric nervous system.
25 s a major inhibitory neurotransmitter in the enteric nervous system.
26 excitatory actions of CRF on neurons in the enteric nervous system.
27 n many inhibitory neurones of the vertebrate enteric nervous system.
28 ing factor (CRF) receptors in the guinea pig enteric nervous system.
29 noamine release in central neurons or in the enteric nervous system.
30 pes and glial cells present in the mammalian enteric nervous system.
31 ole of erbB2 in postnatal development of the enteric nervous system.
32 n myenteric and submucosal components of the enteric nervous system.
33 ing fast and slow synaptic excitation in the enteric nervous system.
34 ic and submucosal plexuses of the guinea pig enteric nervous system.
35 gically identified neurons in the guinea pig enteric nervous system.
36 ney organogenesis and the development of the enteric nervous system.
37 ng renal development and histogenesis of the enteric nervous system.
38 for neuronal replacement in disorders of the enteric nervous system.
39 iate into the neurons and glial cells of the enteric nervous system.
40 ty, suggesting that they may also affect the enteric nervous system.
41 neurotransmitter roles for NO and CO in the enteric nervous system.
42 l role in the development of the kidneys and enteric nervous system.
43 ecting the gut epithelium, immune system and enteric nervous system.
44 gulating the development and function of the enteric nervous system.
45 ng neurogenesis in the fully developed adult enteric nervous system.
46 odulating cholinergic neural reflexes in the enteric nervous system.
47 ant inhibitory motor neurotransmitter in the enteric nervous system.
48 inhibits cholinergic neural reflexes in the enteric nervous system.
49 nesyl-prelamin A are toxic to neurons of the enteric nervous system.
50 rs are localized to central, peripheral, and enteric nervous systems.
51 axonal number in the peripheral, central and enteric nervous systems.
52 hat which transports 5-HT in the central and enteric nervous systems.
53 eripheral, sympathetic, parasympathetic, and enteric nervous systems.
54 nce of cellular pathology in the central and enteric nervous systems.
55 differentiated neurons of the peripheral and enteric nervous systems.
58 t accumulation of 5-HITCA in the central and enteric nervous systems, along with data showing the deg
59 appear to be an intrinsic capability of the enteric nervous system and are not related to slow waves
60 inergic receptor subtype is expressed in the enteric nervous system and at intestinal neuromuscular j
61 the autonomic nervous systems, including the enteric nervous system and central autonomic network.
62 ion between these genes is restricted to the enteric nervous system and does not affect renal, coat c
63 pathway of adrenergic differentiation in the enteric nervous system and have defined a transient requ
64 ides a selective mechanism through which the enteric nervous system and innate immune system integrat
65 lionosis, is a developmental disorder of the enteric nervous system and is the most common cause of i
66 e involves an enterotoxin, activation of the enteric nervous system and malabsorption, suggesting tha
67 d carbon monoxide - that are produced by the enteric nervous system and share common molecular target
70 singly evident that interactions between the enteric nervous system and the immune system play an imp
71 des convey nutrient-regulated signals to the enteric nervous system and to distal organs, acting as c
72 he possible common origin of sympathetic and enteric nervous systems and provides new hope that we ma
73 e sensory, sympathetic, parasympathetic, and enteric nervous systems and the kidneys, as well as for
74 inhibitory nitrergic neural inputs from the enteric nervous system, and (iv) stretch receptors that
75 the immaturity of their intestinal barrier, enteric nervous system, and immune response to pathogens
76 unctions, the functional architecture of the enteric nervous system, and immune responses in the gut
77 ions between the central nervous system, the enteric nervous system, and the gastrointestinal tract.
78 the interactions among the vagus nerve, the enteric nervous system, and the intestinal epithelium ma
79 he genetic control of the development of the enteric nervous system, and the potential role of the in
81 iation of progenitor cells in the developing enteric nervous system are controlled by molecules such
82 tent of the sacral crest contribution to the enteric nervous system are not well established in roden
83 neuronal subtypes during development of the enteric nervous system are poorly understood despite its
84 es a viral enterotoxin and activation of the enteric nervous system, as well as malabsorption, sugges
85 on sequence variants in genes related to the enteric nervous system, as well as with monogenic and ch
86 nding and isolating early progenitors of the enteric nervous system based on their ability to form di
87 ardenburg-Shah syndrome combines the reduced enteric nervous system characteristic of Hirschsprung's
88 Vagal neural crest-derived precursors of the enteric nervous system colonize the bowel by descending
89 is sequence also modulates expression in the enteric nervous system consistent with its proposed role
91 ition to ionotropic glutamate receptors, the enteric nervous system contains functional group I metab
98 y recently to discuss the latest research on enteric nervous system development at a meeting organise
105 losion of interest in the development of the enteric nervous system driven by the need to understand
106 ation of immunity, changes that occur in the enteric nervous system during inflammation, the fundamen
107 nism by measuring colonization in hosts with enteric nervous system dysfunction due to a mutation in
108 cells (NCC) that ultimately give rise to the enteric nervous system (ENS) along the entire length of
109 Enteric glia are important components of the enteric nervous system (ENS) and also form an extensive
111 uding the gastrointestinal tract to form the enteric nervous system (ENS) and hematopoietic organs (b
113 the hypotheses that OT is endogenous to the enteric nervous system (ENS) and that OTR signaling may
114 ous system (CNS), there is evidence that the enteric nervous system (ENS) and the peripheral nervous
115 The majority of neurones and glia of the enteric nervous system (ENS) are derived from the vagal
117 t signaling is critical for formation of the enteric nervous system (ENS) because Ret activation prom
118 derived progenitors (ENPs) that generate the enteric nervous system (ENS) can lead to aganglionosis i
119 (HSCR) is a severe congenital anomaly of the enteric nervous system (ENS) characterized by functional
125 (5-HT) regulates central nervous system and enteric nervous system (ENS) development and long-term f
126 irschsprung disease is a serious disorder of enteric nervous system (ENS) development caused by the f
131 disease (HSCR), a multifactorial disorder of enteric nervous system (ENS) development, is associated
132 rify the role of Ret signaling components in enteric nervous system (ENS) development, we evaluated E
139 l gut function relies on the activity of the enteric nervous system (ENS) found within the wall of th
141 and A30P lines show robust abnormalities in enteric nervous system (ENS) function and synuclein-immu
143 sodium channel (VGSC) alpha subunits in the enteric nervous system (ENS) has not been described.
147 stinal obstruction due to the absence of the enteric nervous system (ENS) in the distal bowel and is
150 iets, the consumers' gut microbiota, and the enteric nervous system (ENS) interact to regulate gut mo
154 tanding the neurochemical composition of the enteric nervous system (ENS) is critical for elucidating
157 h the idea that phenotypic expression in the enteric nervous system (ENS) is determined, in part, by
163 ronal development in vitro; nevertheless, an enteric nervous system (ENS) is present in mice lacking
167 S: Neural stem and progenitor cells from the enteric nervous system (ENS) might serve as a source of
168 en have aggregated alpha-synuclein (aSyn) in enteric nervous system (ENS) neurons, which may be assoc
169 es in resident and inflammatory cells in the enteric nervous system (ENS) of macaques during the acut
173 ocate calcium (Ca2+) channel proteins in the enteric nervous system (ENS) of the rat and guinea pig.
175 ce and identity of neural progenitors in the enteric nervous system (ENS) of vertebrates is a matter
177 complex equilibrium, here, we show that the enteric nervous system (ENS) plays an essential and non-
178 ant oligogenic birth defect that occurs when enteric nervous system (ENS) precursors fail to colonize
182 tion of the bowel, results from a failure of enteric nervous system (ENS) progenitors to migrate, pro
189 Crohn's disease (CD) and the ability of the enteric nervous system (ENS) to produce PGD2 in inflamma
192 a balanced microbial community and that the enteric nervous system (ENS), a chief regulator of physi
193 array of functions, the bowel relies on the enteric nervous system (ENS), an intricate network of mo
194 estinal obstruction, striking defects in the enteric nervous system (ENS), and abnormal intestinal mo
195 rons are the major excitatory neurons of the enteric nervous system (ENS), and include intrinsic sens
196 function is controlled by its own intrinsic enteric nervous system (ENS), but it is additionally reg
197 of axonal tract configuration in the mature enteric nervous system (ENS), but profound abnormalities
198 ty alterations maybe due to an effect on the enteric nervous system (ENS), but the underlying mechani
199 ointestinal tract, and more specifically the enteric nervous system (ENS), in search of an early biom
201 e leads to degeneration and autophagy in the enteric nervous system (ENS), particularly in the submuc
202 the neural crest, impacts development of the enteric nervous system (ENS), possibly by regulating the
204 ium signaling plays an essential role in the enteric nervous system (ENS), the role of CaMKII in neur
205 pathway in the development of the mammalian enteric nervous system (ENS), very little is known regar
206 ointestinal physiology are controlled by the enteric nervous system (ENS), which is composed of neuro
226 ortant in the functional neurobiology of the enteric nervous system (ENS); nevertheless, details for
227 natide reduces food intake and activates the enteric nervous system (ENS; myenteric and submucosal pl
228 ord primary motor neurons (PMN), kidney, and enteric nervous systems (ENS) and have identified areas
229 , have been identified and several mammalian enteric nervous systems express CB1 receptors and produc
231 effect of this Gaucher mutation on motor and enteric nervous system function in these transgenic anim
232 otential of neural crest stem cells from the enteric nervous system (gut NCSCs) in vivo to evaluate t
235 (EGCs), the major cellular component of the enteric nervous system, have long been considered mere s
240 nteractions can influence development of the enteric nervous system in mouse models and suggests that
245 studies to describe the regeneration of the enteric nervous system in the sea cucumber Holothuria gl
247 isceral afferents (sensory) pathways and the enteric nervous system, including the interstitial cells
248 progenitor function in the dentate gyrus or enteric nervous system, indicating regional differences
250 ory detection in these processes, disordered enteric nervous system integration in diarrhea and const
251 es reveals that, in the absence of Pax3, the enteric nervous system is ablated from its inception.
252 flecting the current interest in the way the enteric nervous system is altered in disease and the sec
255 ibution of sacral crest-derived cells to the enteric nervous system is not affected by cocolonization
256 the requirement for erbB2 in maintaining the enteric nervous system is not cell autonomous, but rathe
262 eural crest cells fated to contribute to the enteric nervous system migrate ventrally away from the n
266 major changes in the smooth muscle layers or enteric nervous system occurred in the absence of these
267 processes of enteric glial cells within the enteric nervous system of CWD-infected Tg(CerPrP-E) mice
268 ade an important contribution to the ancient enteric nervous system of early jawless vertebrates, a r
270 ons of both brain stem emetic nuclei and the enteric nervous system of the gut; and 4) whether select
275 be selectively ablated by ganciclovir in the enteric nervous system, or in the injured forebrain or s
277 l neural crest to interrupt the migration of enteric nervous system precursor cells and thus create a
278 e expressed very early in the development of enteric nervous system precursors, and are already prese
279 und that control of differentiation of mouse enteric nervous system progenitor cells by EDN3 requires
282 l derived neurotrophic factor (GDNF) induces enteric nervous system regeneration in mouse models of H
286 recent advances in our understanding of the enteric nervous system, sensory physiology underlying pa
288 We describe neurochemical coding of the enteric nervous system, specifically the myenteric plexu
289 ating GDNF and ET-3 in the patterning of the enteric nervous system, suggest that specific pairing of
290 rity of neurons and glia that constitute the enteric nervous system, the intrinsic innervation of the
291 tite and energy balance by engagement of the enteric nervous system through cannabinoid receptors.
293 terface by demonstrating the capacity of the enteric nervous system to influence the microbiota.
296 year's issue on developmental aspects of the enteric nervous system, we have focused on several key t
297 nal progenitors capable of reconstituting an enteric nervous system when transplanted into a normal d
299 fore alter innervation and morphology of the enteric nervous system, which may contribute to colonic
300 hesized that Lewy pathology initiates in the enteric nervous system years prior to debut of clinical