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1 eterminant of LPS that S Typhi shares with S Enteritidis.
2 e development of a SNP typing scheme with S. Enteritidis.
3 ia monocytogenes and Salmonella enterica sv. Enteritidis.
4 ribute to successful egg contamination by S. Enteritidis.
5 th Abs generated against S Typhimurium and S Enteritidis.
6 87/91 is a recently evolved descendent of S. Enteritidis.
7 VA) method for subtyping Salmonella serotype Enteritidis.
8 reaks caused by Salmonella enterica serotype Enteritidis.
9 n of outbreaks caused by Salmonella serotype Enteritidis.
10 with progeny response to S. enterica serovar Enteritidis.
11 t in the transmission of S. enterica serovar Enteritidis.
12 -transferase-LpfA fusion protein of serotype Enteritidis.
13 ge type (PT) reference strains of Salmonella enteritidis.
14 methods for the epidemiological typing of S. enteritidis.
15 ous than the flagella observed on Salmonella enteritidis.
16 he chicken ovaries by invasive strains of S. enteritidis.
17 ied nonpasteurized liquid eggs containing S. enteritidis.
18 rapid, and powerful subtyping method for S. enteritidis.
19 ogenes, Staphylococcus aureus and Salmonella enteritidis.
20 aradox surrounds Salmonella enterica serovar Enteritidis.
21 st OMPs isolated from S. Choleraesuis and S. Enteritidis.
22 sequence type 11 the major ST among serovar Enteritidis.
23 detection and characterization method for S. Enteritidis.
24 of the isolates, 3 of which were Salmonella Enteritidis.
25 Typhimurium and 1608 (15.8%) were Salmonella Enteritidis.
26 d ecological origins of S. enterica serotype Enteritidis.
27 did bacterial lipopolysaccharides Salmonella enteritidis (0.24 nmol/L, 5-fold) greater than Helicobac
29 f cross-immunizing infection with Salmonella Enteritidis; (3) an increase in the duration of infectio
30 of 85 Salmonella Typhimurium, 58 Salmonella Enteritidis, 32 other nontyphoidal Salmonella (NTS) sero
31 mologous (Typhimurium F98) and heterologous (Enteritidis 4973 and S. enterica O6,14,24: e,h-monophasi
32 proportion than Typhimurium (6%), including Enteritidis (7%), Heidelberg (13%), Choleraesuis (57%),
36 ubsequent challenge with virulent Salmonella enteritidis a selection against lpf phase-on variants wa
37 l showing the highest binding affinity to S. enteritidis, a DNA sequence of high affinity to the bact
38 e phenotypic response to S. enterica serovar Enteritidis, an F1 population of chickens was created by
39 lates serotyped, 160 (45.6%) were Salmonella Enteritidis and 152 (43.3%) were Salmonella Typhimurium.
40 ng NTS isolates, >/=80% (79.7% of Salmonella Enteritidis and 90.2% of Salmonella Typhimurium isolates
42 is of a patient with disseminated Salmonella enteritidis and a homozygous splice acceptor mutation in
43 mprises a positive selection step against S. enteritidis and a negative selection step against a mixt
52 ng Dice similarities for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains
53 enzyme combined PFGE for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains,
55 rs have the capacity to inhibit growth of S. enteritidis and S. typhimurium in bacterial cultures; th
56 amers, twelve rounds of selection to live S. enteritidis and S. typhimurium were performed, alternati
58 rain M5a1, Salmonella eastbourne, Salmonella enteritidis and Salmonella gelsenkirchen, respectively.
61 ing methods for Salmonella enterica serotype Enteritidis and survey the population structure of commo
63 Nontyphoidal Salmonella enterica serovars Enteritidis and Typhimurium are a common cause of gastro
66 Salmonella enterica serovar Enteritidis (S. enteritidis) and closely related serovars, suggesting th
67 Salmonella enterica serotype Enteritidis (S. enteritidis) and Escherichia coli O157:H7, has generated
68 m, 10% (10) were Salmonella enterica serovar Enteritidis, and 3% (3) were Salmonella enterica serovar
69 tions indicated that clinical isolates of S. enteritidis are highly heterogeneous in their ability to
75 med infection with the outbreak strains of S Enteritidis based on whole-genome sequencing (WGS), occu
76 dual strains of Salmonella enterica serotype Enteritidis beyond the phenotypic level; however, a cons
77 mbinant His-tagged flagellin from Salmonella enteritidis bound to TLR5 in detergent lysates from COS-
79 ears and case fatality was 20.3%; Salmonella Enteritidis case fatality (27.8%) was higher than for ot
81 ella (NTS), mainly serotypes Typhimurium and Enteritidis, cause invasive infections with high mortali
85 port the hypothesis that SPI-1 facilitates S Enteritidis colonization of the chicken and make SPI-1 a
87 t IgY from multiple animals infected with S. Enteritidis compared to those infected with S. Hadar.
89 Stanleyville (91% vaccine efficacy), and S. Enteritidis CVD 1944 protected mice against the group D
90 The vaccines S. Typhimurium CVD 1931 and S. Enteritidis CVD 1944 were immunogenic and protected BALB
91 t international travel by linking Salmonella Enteritidis data from the National Antimicrobial Resista
92 Typhimurium and Salmonella enterica serovar Enteritidis DeltaguaBA DeltaclpX live oral vaccines to p
93 xtensive analysis of clinical isolates of S. enteritidis, demonstrate the complex nature of Salmonell
94 hly selective and can successfully detect S. enteritidis down to 600 CFU mL(-1) (equivalent to 18 CFU
95 Salmonella enterica subsp. enterica serovar Enteritidis, drugs that are not appropriate for therapy
96 ction against lpf phase ON cells of serotype Enteritidis during a subsequent challenge, suggesting th
101 nterference/cross-reactivity from Salmonella enteritidis, Enterobacter agglomerans, Pseudomonas putid
102 ofiles of Listeria monocytogenes, Salmonella enteritidis, Escherichia coli, during growth in the pres
103 gainst some food-borne pathogens (Salmonella enteritidis, Escherichia coli, Listeria monocytogenes an
104 e of related pathogens, including Salmonella enteritidis, Escherichia coli, Staphylococcus aureus, Ps
105 Clinical isolates of S. enterica serovar Enteritidis exhibit a wide spectrum of virulence in mice
108 ave analyzed the pathway by which Salmonella enteritidis flagellin (FliC) activates murine and human
111 cs of Salmonella BSI in Blantyre; Salmonella Enteritidis from 1999 to 2002, Salmonella Typhimurium fr
112 nome sequence analysis of 675 isolates of S. Enteritidis from 45 countries, we show the existence of
115 of diversity within 104 isolates of serotype Enteritidis from eight unaffiliated poultry farms in Eng
116 on the molecular fingerprinting of serotype Enteritidis from poultry environments in the United King
117 tal of 192 NTS isolates (114 Typhimurium, 78 Enteritidis) from blood and stools from pediatric admiss
119 To investigate whether changes in serovar Enteritidis gene content contributed to this increased p
120 dentified in the Salmonella enterica serovar Enteritidis genome that is predicted to encode a protein
121 phism (SNP)-based cluster analysis of the S. Enteritidis genomes revealed well supported clades, with
123 Overall, MLVA typing of Salmonella serotype Enteritidis had enhanced resolution, good reproducibilit
128 The lipopolysaccharide (LPS) of Salmonella enteritidis has been implicated as a virulence factor of
130 from the Gram-negative bacterium Salmonella Enteritidis, identifying AdrA as the most potent inducer
131 injected intramuscularly 2 weeks later with Enteritidis, (ii) hens were contact infected with Kentuc
132 contact infected with Kentucky and then with Enteritidis, (iii) hens were injected with Enteritidis o
133 Salmonella enterica serovar typhimurium and enteritidis in blood samples without culture enrichment.
136 a survival advantage to S. enterica serovar Enteritidis in eggs by repairing DNA damage caused by eg
137 genetic analysis identified two strains of S Enteritidis in human cases that were subsequently identi
139 , confers colonization resistance against S. Enteritidis in neonatal chicks, phenocopying germ-free m
140 environments, which may ultimately assist S. Enteritidis in persistent and silent colonization of chi
142 try outbreak of Salmonella enterica serotype Enteritidis in the EU and European Economic Area (EEA).
143 uding two rare strains classified as serovar Enteritidis in the Salmonella reference collection B, on
144 ence that SEF14 fimbriae are expressed by S. enteritidis in vivo, previous studies showed that SEF14
145 The isolation rate of Salmonella serotype Enteritidis increased until 1996, whereas declines were
146 cted to evaluate whether NO production in S. Enteritidis-infected HD11 cells can be used as a biomark
148 demonstrate that NO-based screening using S. Enteritidis-infected HD11 cells is a viable tool to iden
150 e highly discriminatory for IgY following S. Enteritidis infection (p < 0.05) compared to infections
151 implications for Salmonella enterica serovar Enteritidis infection and transmission to eggs, along wi
152 We defined an outbreak-associated case of S. enteritidis infection as one in which S. enteritidis was
153 an both prevent the spread of AMR Salmonella Enteritidis infection in chickens and shift the bacteria
154 ure of ice cream associated with cases of S. enteritidis infection were compared with those of produc
158 acid-resistant Salmonella enterica serotype Enteritidis infections in the United States and recent i
160 ization after pathogenic S. enterica serovar Enteritidis inoculation and for circulating antibody lev
161 and transmission electron micrographs of S. enteritidis invasion of granulosa cells showed organisms
163 Salmonella enterica subsp. enterica serovar Enteritidis is a common food-borne pathogen, often assoc
171 ogical characteristic of S. enterica serovar Enteritidis is its association with chicken shell eggs,
172 Salmonella enterica serovar Typhimurium, S. Enteritidis is known to have pathobiology specific to ch
175 le each year and among pathogens, Salmonella Enteritidis is most widely found bacteria causing food b
176 y in recent decades, and S. enterica serovar Enteritidis is now one of the leading serovars of Salmon
179 Salmonella enterica serovar Enteritidis (S. Enteritidis) is a major etiologic agent of nontyphoid sa
180 Salmonella enterica serotype Enteritidis (S. enteritidis) is a major food-borne pathogen, and its inc
181 Salmonella enterica serovars Typhimurium and Enteritidis, is responsible for a major global burden of
182 type element with homology to the Salmonella enteritidis IS1351 element and Yersinia enterocolitica I
183 we conducted a retrospective analysis of S. Enteritidis isolates from seven epidemiologically confir
184 The resultant phylogeny allocated most S. Enteritidis isolates into two distinct clades (clades I
186 al and animal origins of Salmonella serotype Enteritidis isolates may have a considerable influence o
190 la Typhimurium and 30% (24/79) of Salmonella Enteritidis isolates tested were found to be multidrug r
195 d the effects of Salmonella enterica serovar Enteritidis lipopolysaccharide (LPS) and recombinant Sal
196 core polysaccharide-OPS (COPS) of Salmonella Enteritidis lipopolysaccharide (LPS) to flagellin protei
198 the published level of glucosylation for S. enteritidis LPS as well as for S. enteritidis LPS purcha
199 ion for S. enteritidis LPS as well as for S. enteritidis LPS purchased from Sigma Chemical Co., the 1
201 N-gamma +/+ mice following injection with S. enteritidis LPS, despite sustaining 11-fold reductions i
204 kers for the response to S. enterica serovar Enteritidis may result in the enhancement of the immune
205 fferent hosts, including S. enterica serovar Enteritidis (multiple hosts), S. Gallinarum (birds), and
207 riae on colonization of mice with a serotype Enteritidis mutant in which the lpf promoter region was
208 f the 687 NTS isolates, including Salmonella Enteritidis (n = 244 [35.5%]), Salmonella Typhimurium (n
210 0), Typhimurium (n = 163), Newport (n = 93), Enteritidis (n = 45), Dublin (n = 25), Pullorum (n = 9),
211 h Enteritidis, (iii) hens were injected with Enteritidis only, and (iv) hens were contact infected wi
213 ontaminated with Salmonella enterica serovar Enteritidis or Salmonella enterica serovar Typhimurium a
214 ety of strains, which suggests that serotype Enteritidis organisms representing different genomic gro
215 a single culture-proven foodborne Salmonella enteritidis outbreak in 1994, Salmonella-induced gastroe
216 of commonly encountered S. enterica serotype Enteritidis outbreak isolates in the United States.
217 a single culture-proven foodborne Salmonella enteritidis outbreak that involved 1811 patients (mostly
218 erefore, more information is needed about S. Enteritidis pathobiology in comparison to that of S. Typ
220 monella enterica subspecies enterica serovar Enteritidis phage type (PT) 4, which peaked in 1993.
221 arge outbreak of Salmonella enterica serovar Enteritidis phage type 14b affecting more than 30 patien
222 ection B, only eleven regions of the serovar Enteritidis phage type 4 (PT4) chromosome (sequenced at
224 determine the relationships between some S. Enteritidis phage types (PTs) commonly associated with f
225 confer protection against challenge with S. enteritidis, presumably because lpf phase-off variants w
226 falling since 1997, and levels of Salmonella Enteritidis PT4 have fallen to preepidemic levels and ha
227 host-promiscuous Salmonella enterica serovar Enteritidis PT4 isolate P125109 and a chicken-restricted
228 roarray comparisons of the sequenced serovar Enteritidis PT4 to isolates of the closely related serov
229 Whereas CVD 1931 did not protect against S. Enteritidis R11, CVD 1944 did mediate protection against
230 wild-type strains S. Typhimurium I77 and S. Enteritidis R11, respectively, were constructed by delet
233 sruption of this gene in S. enterica serovar Enteritidis rendered the organism more susceptible to eg
235 olonization with Salmonella enterica serovar Enteritidis requires a virulence-factor-dependent increa
236 U/mL and 94 CFU/mL for S. typhimurium and S. enteritidis, respectively, that could be attributed to t
239 re only found in Salmonella enterica serovar Enteritidis (S. enteritidis) and closely related serovar
240 hogens, such as Salmonella enterica serotype Enteritidis (S. enteritidis) and Escherichia coli O157:H
243 ent protein from Salmonella enterica serovar Enteritidis, S. enterica serovar Typhimurium, and Pseudo
244 genes of Salmonella typhimurium, Salmonella enteritidis, Salmonella arizonae, Shigella sonnei, and S
245 O157:H7, Salmonella typhimurium, Salmonella enteritidis, Salmonella arizonae, Shigella sonnei, Shige
246 uginosa, Pseudomonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escherichia coli).
248 onsin, reported Salmonella enterica serotype enteritidis (SE) infections during 1997 more than double
252 mbria genes were replaced both in Salmonella enteritidis (sefA, agfA and fimC) and Escherichia coli (
254 7.7%) from blood, 53 (30.8%) were Salmonella Enteritidis ST11 and 62 (36.0%) Salmonella Typhimurium S
255 human infection by some clades of Salmonella Enteritidis ST11 in East Africa, but not of human Salmon
256 identified cgMLST clusters within Salmonella Enteritidis ST11, Salmonella Heidelberg ST15, Salmonella
257 ctivity against Escherichia coli, Salmonella enteritidis, Staphylococcus aureus, and Mycobacterium bo
258 chicken macrophage HD11 cells, while dead S. Enteritidis stimulates a high level of NO production, su
259 osomal tlpA gene rendered a virulent serovar Enteritidis strain defective in intracellular survival a
260 for strain differentiation of more clonal S. Enteritidis strains and provides core genotypic markers
262 all this work shows that S. enterica serovar Enteritidis strains circulating in Uruguay have the same
263 -old and nineteen 10- to 20-year-old serovar Enteritidis strains from various hosts, using a Salmonel
266 Typhimurium and Salmonella enterica serovar Enteritidis strains that can serve as live oral vaccines
267 the subtyping of Salmonella enterica serovar Enteritidis strains to an epidemiologically relevant lev
271 allenge with nontyphoidal serovar Salmonella Enteritidis than with another nontyphoidal serovar, Salm
272 obal epidemic clade and two new clades of S. Enteritidis that are geographically restricted to distin
273 sed subtyping scheme for S. enterica serovar Enteritidis that relies on a single combined cluster ana
275 n a whole-gene scale were needed for serovar Enteritidis to become more prevalent in domestic fowl.
281 ggest that a bivalent (S. Typhimurium and S. Enteritidis) vaccine would provide broad protection agai
283 e used transposon mutagenesis to identify S. Enteritidis virulence genes by assay of invasiveness in
285 S. enteritidis infection as one in which S. enteritidis was cultured from a person who became ill in
289 an previously known, as a second clade of S. Enteritidis was revealed that is distinct from those com
291 e characteristics of clinical isolates of S. enteritidis, we determined the 50% lethal doses (LD(50))
292 he RNI/ROI resistance of S. enterica serovar Enteritidis, we transformed a genomic DNA library of SE2
294 Salmonella enterica serovars Typhimurium and Enteritidis, were assessed using lipopolysaccharide (LPS
295 used to examine a panel of 29 isolates of S. enteritidis which had been previously characterized by o
296 serovar Typhimurium and S. enterica serovar Enteritidis, which are usually linked to localized gastr
297 I as most concordant for S. enterica serovar Enteritidis, while XbaI, BlnI, and SpeI were most concor
298 tified a gene, yafD from S. enterica serovar Enteritidis, whose overexpression conferred upon S. ente
300 ned as laboratory-confirmed infection with S Enteritidis with the multiple-locus variable-number tand