ライフサイエンスコーパス: enteritis

1 ases of CMV disease (one pneumonitis and one enteritis).

2 Two patients developed cytomegalovirus enteritis.

3 reptomycin to result in gut-restricted acute enteritis.

4 ammation and enterocyte apoptosis in toxin A enteritis.

5 Clostridium difficile toxin A (TxA)-induced enteritis.

6 ctivators are proinflammatory in TxA-induced enteritis.

7 e are markedly resistant to lethal radiation enteritis.

8 and one because of rejection after rotavirus enteritis.

9 inclusion body hepatitis, splenomegaly, and enteritis.

10 the pathogenesis of Clostridium perfringens enteritis.

11 with antibiotic treatment of E coli O157:H7 enteritis.

12 reas one dog was euthanized on day 17 due to enteritis.

13 elopment of S. flexneri-induced inflammatory enteritis.

14 vidence of inflammation in a rabbit model of enteritis.

15 n shown to be necessary for the induction of enteritis.

16 veloped bile duct and liver disease, but not enteritis.

17 of cattle results in a chronic granulomatous enteritis.

18 n and the pathogenesis of C. jejuni-mediated enteritis.

19 infiltration associated with toxin A-induced enteritis.

20 microbial product influx, driving Crohn-like enteritis.

21 nflammatory cell influx, fluid secretion and enteritis.

22 to neutrophil mucosal influx during toxin A enteritis.

23 ulence feature underlying Salmonella-induced enteritis.

24 ought to be essential in the pathogenesis of enteritis.

25 c enterocolitis with radiologically assessed enteritis.

26 ich are required for Salmonella invasion and enteritis.

27 ops in about 10% of patients with infectious enteritis.

28 g growth, and the household cost of invasive enteritis.

29 verned PUFA-induced chemokine production and enteritis.

30 distress and users of antibiotics during the enteritis.

31 rs for, and outcomes of IBS after infectious enteritis.

32 nce of IBS 3 months or more after infectious enteritis.

33 isolated enterectomy due to cytomegalovirus enteritis.

34 1 and SN2/SNAT5 by mast cells during chronic enteritis.

35 ith a well-established diagnosis of regional enteritis.

36 ress, Paneth cell impairment and spontaneous enteritis.

37 ell into two distinct groups, bacteremia and enteritis.

38 enic fever with symptoms of mucositis and/or enteritis.

39 worldwide leading cause of bacterial-induced enteritis.

40 d active colitis and mild-to-moderate active enteritis.

41 among random Campylobacter isolates causing enteritis, 275 enteritis-associated isolates, randomly c

42 among random Campylobacter isolates causing enteritis, 275 random enteritis-associated isolates of C

43 gellosis children than Campylobacter-induced enteritis ($4.27 vs. $3.49).

44 neumonia (69.2%), meningoencephalitis (50%), enteritis (46.2%), colitis (38.5%), syndrome (42.3%), vi

45 ree patients (FCL: 125) coincided with viral enteritis, 51 samples from 21 patients (FCL: 207) coinci

46 n (13.3% and 11.8%, respectively), norovirus enteritis (8.2% and 3%), cytomegalovirus disease or coli

47 flammation without rejection (group D: acute enteritis, 9; Helicobacter pylori, 1; Streptococcal phar

48 r role in the pathogenesis of avian necrotic enteritis, a disease that has emerged due to the removal

49 moderate, and severe rejections, nonspecific enteritis), although there was sufficient overlap to pro

50 n in individuals who did not have infectious enteritis, although there was heterogeneity among studie

51 s associated with Campylobacter and Shigella enteritis among under-5 children, the post-infection wor

52 reen 325 patients for inflammatory bacterial enteritis and a negative predictive value of 99.4% when

53 ARs can ameliorate C. difficile TcdA-induced enteritis and alter the outcome of C. difficile infectio

54 the duodenum was associated with nonspecific enteritis and CD8(+) T-cell activation.

55 immune-related diseases such as noninfective enteritis and colitis to be significantly associated wit

56 ion of S100a9 protected against experimental enteritis and colitis, and pharmacologic inhibition of S

57 A(2B)ARs mediate C. difficile toxin-induced enteritis and disease.

58 ch cause enteritis necroticans in humans and enteritis and enterotoxaemias of domestic animals, typic

59 Clostridium perfringens type B causes enteritis and enterotoxemia in domestic animals.

60 teritis necroticans in humans or necrotizing enteritis and enterotoxemia in domestic animals.

61 mportant toxins for C. perfringens diseases (enteritis and enterotoxemia) originating in the gastroin

62 trointestinal disorders in humans, including enteritis and enterotoxemia.

63 uxiliary virulence factor for C. perfringens enteritis and enterotoxemia.

64 C isolates cause both haemorrhagic necrotic enteritis and fatal enterotoxemias (where toxins produce

65 cells in the lamina propria and eosinophilic enteritis and fibrosis in the small intestine.

66 ous complications included a cytomegalovirus enteritis and four fungal infections (related to central

67 ed isolates from patients with uncomplicated enteritis and GBS, as well as isolates from animal sourc

68 obally distributed cause of human food-borne enteritis and has been linked to chronic joint and neuro

69 bacter jejuni is an important cause of human enteritis and has been linked to the development of auto

70 ct the sensitivity of individual patients to enteritis and have taken the first steps to develop such

71 Escherichia coli O157:H7 causes severe enteritis and hemolytic-uremic syndrome, mostly in young

72 cells on a BALB/c background succumbed with enteritis and hepatitis.

73 ithelial cells (IECs) results in spontaneous enteritis and increased susceptibility to induced coliti

74 Campylobacter fetus is a cause of enteritis and invasive extraintestinal disease in humans

75 e deletion after immune development leads to enteritis and lethality.

76 two graft losses: one because of adenoviral enteritis and one because of rejection after rotavirus e

77 bjected to systemic infections, peritonitis, enteritis and pneumonia induced by Staphylococcus aureus

78 s to have only short-term effects, bacterial enteritis and protozoan and helminth infections are foll

79 effective control measures against necrotic enteritis and providing potential new tools to the field

80 d possibly related to momelotinib (rotaviral enteritis and Staphylococcus pneumonia).

81 aninum has been associated with eosinophilic enteritis and suggested as a possible cause of diffuse u

82 ry but not sufficient for the development of enteritis and that C57BL/6 IL-10(-/-) mice can serve as

83 Escherichia coli O157 causes severe enteritis and the extraintestinal complication hemolytic

84 compared with individuals without infectious enteritis) and host- and enteritis-related risk factors.

85 GI toxicity (mucositis, enteritis, and diarrhea) appears to be the major combine

86 such as eosinophilic gastritis, eosinophilic enteritis, and eosinophilic colitis, herein referred to

87 nophilic esophagitis, eosinophilic gastritis/enteritis, and hypereosinophilia/hypereosinophilic syndr

88 inistered human immunoglobulin for norovirus enteritis, and it appeared to be an effective treatment

89 bjected to systemic infections, peritonitis, enteritis, and pneumonia induced by Staphylococcus aureu

90 for CN3685 to cause haemorrhagic necrotizing enteritis, apparently because the Agr-like system regula

91 Although the mechanism of C.jejuni-mediated enteritis appears to be multifactorial, flagella play co

92 Campylobacter- and Shigella-induced invasive enteritis are common in under-5 Bangladeshi children.

93 Women-particularly those with severe enteritis-are at increased risk for developing IBS, as a

94 oral candidiasis, nail dystrophy, gastritis, enteritis, arthritis, Sjogren's-like syndrome, urticaria

95 mucosal adenovirus infection associated with enteritis as well as parvovirus viremia in animals with

96 infectious, allergic, and inflammatory graft enteritis as well as post-transplant lymphoproliferative

97 n patients in both the extent of symptoms of enteritis as well as their duration.

98 ary point prevalence of IBS after infectious enteritis, as well as relative risk (compared with indiv

99 acter isolates causing enteritis, 275 random enteritis-associated isolates of Campylobacter jejuni we

100 ampylobacter isolates causing enteritis, 275 enteritis-associated isolates, randomly collected in the

101 After giardiasis enteritis at least 5% developed clinical characteristics

102 h Toxoplasma gondii results in dysbiosis and enteritis, both of which revert to normal during chronic

103 mia, cancer, and SBS-CIF caused by radiation enteritis, but fewer cases attributed to Crohn disease.

104 or inflammation, especially after infectious enteritis, but this has not yet resulted in changes in t

105 dii inhibits C. difficile toxin A-associated enteritis by blocking the activation of Erk1/2 MAP kinas

106 However, cytomegalovirus (CMV) enteritis can cause complications.

107 Norovirus enteritis can cause intractable diarrhea in solid organ

108 s, 41.9% developed IBS, and of patients with enteritis caused by bacterial infection, 13.8% developed

109 Enteritis caused by Clostridium difficile toxin (Tx) is

110 Johne's disease (JD), a chronic, infectious enteritis caused by Mycobacterium avium subsp.

111 uberculosis (PTB) is a chronic granulomatous enteritis caused by Mycobacterium avium subsp.

112 erizing the virulence mechanisms influencing enteritis caused by non-typhoid Salmonella spp.

113 Of patients with enteritis caused by protozoa or parasites, 41.9% develop

114 this period, two patients had granulomatous enteritis characteristic of Crohn's disease in multiple

115 y which this bacterium invades its host, the enteritis characteristically associated with salmonellos

116 In a Caco-2 cell model of enteritis, culture supernatants of the type B agrB null

117 s, we found >10% of patients with infectious enteritis develop IBS later; risk of IBS was 4-fold high

118 Radiation-induced enteritis develops in cancer patients treated with radio

119 Ten patients experienced 21 episodes of CMV enteritis, diagnosed by histopathology, virology, or bot

120 Rates of colonization and enteritis did not differ between male and female mice.

121 f the Salmonella virulence factors affecting enteritis do not appear to be required for infection of

122 that causes millions of cases of food-borne enteritis each year.

123 , in the absence of both, severe spontaneous enteritis emerges.

124 We phenotyped and mechanistically dissected enteritis evoked by a PUFA-enriched Western diet in 2 mo

125 studies, comprising 21,421 individuals with enteritis, followed for 3 months to 10 years for develop

126 Repeat CT showed improvement in enteritis; however, repeat push enteroscopy revealed mul

127 incidence, timing, and outcome of infectious enteritis (IE) after intestinal transplantation (ITx).

128 on included hypothyroidism in 5 subjects and enteritis in 1 subject.

129 specimens was used for the diagnosis of CMV enteritis in 21 patients.

130 pe C isolate CN3685 to cause bloody necrotic enteritis in a rabbit ileal loop model and also showed t

131 rombotic microangiopathy (iTMA) and ischemic enteritis in approximately 50% of infected human gut xen

132 phimurium mutants for their ability to cause enteritis in calves.

133 acteria that can cause chronic granulomatous enteritis in cattle, are difficult to distinguish on the

134 s one of the principal agents of acute viral enteritis in children.

135 tion is the most commonly notified bacterial enteritis in Germany.

136 ause attaching and effacing (AE) lesions and enteritis in humans and animals.

137 ens type C isolates cause enterotoxemias and enteritis in humans and livestock.

138 phimurium) infection, which causes foodborne enteritis in humans, remains elusive.

139 n serve as models for the study of C. jejuni enteritis in humans.

140 continues to be a leading cause of bacterial enteritis in humans.

141 ts a mechanism involved in Shigella-elicited enteritis in humans.

142 bacterial pathogen that is a common cause of enteritis in humans.

143 ngens type D strains cause enterotoxemia and enteritis in livestock via epsilon toxin production.

144 t triggers focal granuloma-like neutrophilic enteritis in mice that lack one allele of Gpx4 in IECs.

145 In this study, we induced radiation-induced enteritis in mice through abdominal irradiation, mimicki

146 tion.IMPORTANCE MEV causes fatal hemorrhagic enteritis in minks.

147 onomous parvovirus, causes acute hemorrhagic enteritis in minks.

148 e role of beta2-toxin in the pathogenesis of enteritis in neonatal pigs.

149 whether this protein plays the same role in enteritis in other animal species.

150 ases in domestic animals, including necrotic enteritis in piglets and typhlocolitis in horses.

151 perfringens, the causative agent of necrotic enteritis in poultry and humans, have the ability to deg

152 ssist with the rapid laboratory diagnosis of enteritis in puppies and highlight the need for continue

153 -type CN3685 caused haemorrhagic necrotizing enteritis in rabbit ileal loops.

154 ic agrB or luxS mutants to cause necrotizing enteritis in rabbit small intestinal loops or enterotoxe

155 protease in preventing C. difficile toxin A enteritis in rat ileum and determine whether it protects

156 t S. boulardii inhibits C. difficile toxin A enteritis in rats by releasing a 54-kDa protease which d

157 ter a large community outbreak of giardiasis enteritis in the city of Bergen, Norway were evaluated w

158 2-fold higher in patients who had infectious enteritis in the past 12 months than in those who had no

159 ern diet potently induced ER stress, driving enteritis in Xbp1(-/-IEC) and Gpx4(+/-IEC) mice.

160 omprise eosinophilic gastritis, eosinophilic enteritis (including eosinophilic duodenitis), and eosin

161 ether antibiotic treatment of E coli O157:H7 enteritis increases the risk of HUS.

162 with natural Treg, and in vivo they suppress enteritis induced by mouse effector T cells.

163 Enteritis induced by non-typhoid pathogenic Salmonella i

164 al inflammatory diseases, such as infectious enteritis, inflammatory bowel disease, and necrotizing e

165 Toxin A enteritis involves release of PGE(2), which activates th

166 Campylobacter enteritis is a food-borne or waterborne illness caused a

167 to the laboratory diagnosis of Campylobacter enteritis is based on the recovery of the organism from

168 patients with neutropenic enterocolitis when enteritis is considered.

169 While the likelihood of enteritis is dose dependent, there is also considerable

170 Severe isolated ICI-enteritis is rare and can lead to clinically significant

171 The type E animal enteritis isolates carrying these silent cpe sequences d

172 paratuberculosis results in a granulomatous enteritis (Johne's disease) that is often fatal.

173 ects of ICI are common, isolated ICI-induced enteritis leading to small bowel hemorrhage is rare.

174 at endothelial infection, iTMA, and ischemic enteritis might be central mechanisms underlying severe

175 In the enteritis model, TLR5KO mice had more severe gut patholo

176 tibiotic regimens commonly applied to murine enteritis models are used to examine the impact of antib

177 old higher in individuals who had infectious enteritis more than 12 months ago than in individuals wh

178 rodentium, a model pathogen for severe human enteritis, more rapidly than did WT mice.

179 s with the emerging infectious disease poult enteritis mortality syndrome.

180 iseases in dogs, including acute hemorrhagic enteritis, myocarditis, and cerebellar disease.

181 iseases in dogs, including acute hemorrhagic enteritis, myocarditis, and cerebellar disease.

182 evidence of acute rejection (n = 12), viral enteritis (n = 5), and nonspecific inflammation (n = 16)

183 e most common cause of bleeding, followed by enteritis (n=24), portal hypertensive lesions (n=15), Ro

184 he etiology of the poultry diseases necrotic enteritis (NE) and poultry gangrene (PG).

185 Necrotic enteritis (NE) caused by Clostridium perfringens infecti

186 Necrotic enteritis (NE) caused by Clostridium perfringens is one

187 lostridium perfringens type C to cause human enteritis necroticans (EN) is attributed to beta toxin (

188 Enteritis necroticans (pigbel), an often fatal illness c

189 In December 1998, enteritis necroticans developed in a 12-year-old boy wit

190 ium perfringens type C isolates, which cause enteritis necroticans in humans and enteritis and entero

191 lostridium perfringens type C isolates cause enteritis necroticans in humans or necrotizing enteritis

192 pidly fatal diseases in domestic animals and enteritis necroticans in humans, contain the genes for a

193 The causative organism of enteritis necroticans is Clostridium perfringens type C,

194 ing, gas gangrene (clostridial myonecrosis), enteritis necroticans, and non-foodborne gastrointestina

195 ding gas gangrene (clostridial myonecrosis), enteritis necroticans, antibiotic-associated diarrhea, a

196 athogen, is one of the most common causes of enteritis necroticans, gas gangrene and food poisoning.

197 e severe diseases, including myonecrosis and enteritis necroticans, in humans and animals.

198 They are responsible for enteritis necroticans, which was termed Darmbrand when o

199 nts--findings consistent with a diagnosis of enteritis necroticans.

200 he acute pathogenesis of type A FP or type C enteritis necroticans.

201 (CD), Irritable bowel syndrome (IBS), NSAIDs enteritis (NSAIDs), persistent NSE and no significant di

202 itis the findings of nonspecific small bowel enteritis (NSE) on capsule endoscopy (CE) poses a clinic

203 Radiologically assessed enteritis (odds ratio, 2.60; 95% CI, 1.32-7.56; p = 0.01

204 ), occurs worldwide as chronic granulomatous enteritis of domestic and wild ruminants.

205 E isolates, all associated with hemorrhagic enteritis of neonatal calves, were identified by multipl

206 Johne's disease is a chronic enteritis of ruminants associated with enormous worldwid

207 of Johne's Disease, a chronic granulomatous enteritis of ruminants.

208 A CT scan revealed enteritis of the duodenum and jejunum without colitis.

209 Patients with severe ICI-enteritis on endoscopy should be carefully monitored for

210 eases of the esophagus (15%), non-infectious enteritis or colitis (7.4%), functional dyspepsia (6%) a

211 n in conditions ranging from infective acute enteritis or colitis to inflammatory bowel disease is ac

212 of each branch of the UPR causes spontaneous enteritis or creates higher susceptibility for intestina

213 Since the lack of overt signs of enteritis or enterocolitis due to Salmonella infections

214 has been identified as a causative agent of enteritis or inapparent infections in turkeys.

215 y higher than those from patients with viral enteritis or normal biopsies [198 mg/kg compared with 7

216 Providers are encouraged to consider enteritis or proctocolitis in MSM as possibly having bee

217 the IFN-gamma knockouts either succumbed to enteritis or survived to develop marked triaditis, porta

218 amma knockout donors either developed severe enteritis or survived to develop triaditis, cholangitis,

219 ired for the development of lethal radiation enteritis or the microbiota-associated enhancement of en

220 the reliable detection of invasive bacterial enteritis or the reliable selection of specimens for cul

221 sumptive diagnosis of inflammatory bacterial enteritis or which can be used to determine the suitabil

222 , cholecystitis, duodenal ulcer, duodenitis, enteritis, or adnexal or testis pathologies.

223 uals with symptoms of acute gastroenteritis, enteritis, or colitis.

224 patients suspected of acute gastroenteritis, enteritis, or colitis.

225 a, F. nucleatum does not exacerbate colitis, enteritis, or inflammation-associated intestinal carcino

226 porcine sapelovirus (PSV) is known to cause enteritis, pneumonia, polioencephalomyelitis, and reprod

227 of food-poisoning and causes avian necrotic enteritis, posing a significant problem to both the poul

228 or gastrointestinal syndromes ("proctitis," "enteritis," "proctocolitis"), enteric pathogens or sexua

229 ainst TNF-alpha-induced injury and alleviate enteritis-related diarrhea.

230 without infectious enteritis) and host- and enteritis-related risk factors.

231 eases in mice and rats, including hepatitis, enteritis, respiratory diseases, and encephalomyelitis i

232 95% CI, 1.6-6.5), and clinical indicators of enteritis severity.

233 s of patients with documented E coli O157:H7 enteritis, some of whom developed HUS; had clear definit

234 ence genes tested, but 66% of nonbacteremic, enteritis strains also contained all the tested virulenc

235 an outbreak of inclusion body hepatitis and enteritis that affected neonatal Northern aplomado (Falc

236 hat activated LPMs secrete SP during toxin A enteritis that can lead to secretion of cytokines, sugge

237 Campylobacter jejuni can cause an enteritis that is associated with an acute inflammatory

238 erculosis results in chronic and progressive enteritis that traverses both subclinical and clinical s

239 rt of patients presenting with Campylobacter enteritis to be 1.17/1000 person-years, a rate 77 times

240 onal neuropathy (AMAN), Campylobacter jejuni enteritis triggers the production of anti-ganglioside Ab

241 Hemorrhagic enteritis virus (HEV), a type II avian adenovirus, cause

242 Mink enteritis virus (MEV) is associated with rapid, high-lev

243 Mink enteritis virus (MEV), an autonomous parvovirus, causes

244 valence of IBS at 12 months after infectious enteritis was 10.1% (95% confidence interval [CI], 7.2-1

245 and at more than 12 months after infectious enteritis was 14.5% (95% CI, 7.7-25.5).

246 Toxin A enteritis was accompanied by increased cellular infiltra

247 (2)-dependent Fas/FasL activation in toxin A enteritis was further assessed in either scid or FasL an

248 In contrast, CMV enteritis was identified in only one patient, who subseq

249 ting, diarrhea, and histologic gastritis and enteritis were commonly observed in dogs treated with th

250 ic neoplasm, and ulcerative colitis/regional enteritis were included.

251 rm of ocular adnexal involvement in regional enteritis, which affects the orbit far more frequently t

252 might reflect enhanced host defense against enteritis, which is more severe in those with acquired o

253 Older age was associated with NSAIDs enteritis, while more subjects without significant gastr

254 hat an individual who develops Campylobacter enteritis will also develop GBS during the subsequent 2-

255 s in humans ranging from mild to hemorrhagic enteritis with complications of hemolytic uremic syndrom

256 Pathologic findings revealed ischemic enteritis with patchy necrosis and fibrin thrombi in art

257 arasite Cryptosporidium parvum causes severe enteritis with substantial morbidity and mortality among

258 liary tract infection, abdominal abscess, or enteritis) with those who did not to identify clinical f

259 IDS patients can have episodes of Salmonella enteritis without dissemination.

260 uni is a major cause of bacterial food-borne enteritis worldwide, and invasion into intestinal epithe

261 ticus, a leading cause of seafood-associated enteritis worldwide, is dependent upon a type III secret