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1 al, mucous neck, and chief cells, but not to enterochromaffin-like-cell.
2 th parietal cells and histamine release from enterochromaffin-like cells.
3 ndirectly by releasing histamine from fundic enterochromaffin-like cells.
4                 It is synthesized by gastric enterochromaffin-like cells, a specific set of hypothala
5 e, with a decrease in number of parietal and enterochromaffin-like cells and an increase in number of
6  hormone-like hormone in histamine-secreting enterochromaffin-like cells and hepcidin in acid-secreti
7 irculating gastrin leads to proliferation of enterochromaffin-like cells and to the development of ga
8 tic atrophy were sustained while chief cell, enterochromaffin-like cell, and somatostatin cell popula
9 ance of the gastric mucosa, proliferation of enterochromaffin-like cells, and neoplastic transformati
10 ctivity was localized to the cell surface of enterochromaffin-like cells, and of myenteric and submuc
11 n, acting via cholecystokinin-2 receptors on enterochromaffin-like cells coupled to an increase in in
12 zymes and an increase in histamine-secreting enterochromaffin-like cells (ECLs).
13 y regulating the secretion of histamine from enterochromaffin-like cells, gastrin from G cells, and s
14 rboxylase, releasing histamine, and inducing enterochromaffin-like cell hypertrophy and hyperplasia.
15 t the difference between beta cells and awry enterochromaffin-like cells is a gradient of cell states
16 , parietal and zymogenic, but not for pit or enterochromaffin-like cell lineages in the oxyntic gastr
17 INS-GAS mice showed no evidence of increased enterochromaffin-like cell number, but instead exhibited
18 ine transport into secretory vesicles of the enterochromaffin-like cell of the gastric corpus.
19 MAT2 alone is expressed in histamine-storing enterochromaffin-like cells of the oxyntic mucosa of the
20 rstitial cells of Cajal of the intestine and enterochromaffin-like cells of the stomach.
21 s, including the role of hypergastrinemia on enterochromaffin-like cell proliferation and its relatio
22                                          All enterochromaffin-like cells respond to cholecystokinin-B
23 for VMAT2 in the transport of histamine into enterochromaffin-like cell secretory vesicles, and with
24                     Histamine, released from enterochromaffin-like cells stimulates the parietal cell
25  the stomach of mutant animals, parietal and enterochromaffin-like cells were decreased, providing a
26 ric neuroendocrine tumours (NETs) arise from enterochromaffin-like cells, which are located in oxynti
27               CDX2 emerged as a regulator of enterochromaffin-like cells, which we show resemble a tr