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1 nella), IpgB1 and IpgB2 (Shigella), and Map (enteropathogenic Escherichia coli).
2 osure of calpastatin-overexpressing cells to enteropathogenic Escherichia coli.
3 and were reported to contain the eae gene of enteropathogenic Escherichia coli.
4 and one is similar to the AIDA-I adhesin of enteropathogenic Escherichia coli.
5 o gastric epithelial cells resembles that of enteropathogenic Escherichia coli.
6 causes disease similar to the human pathogen enteropathogenic Escherichia coli.
7 rmation of attaching and effacing lesions by enteropathogenic Escherichia coli.
8 uptake as well as Cdc42-dependent uptake of enteropathogenic Escherichia coli.
9 shigella, salmonella, Yersinia species, and enteropathogenic Escherichia coli.
10 he attachment and effacement associated with enteropathogenic Escherichia coli.
11 iated with the type IV pilus gene cluster of enteropathogenic Escherichia coli, a transposase from Vi
14 -to-severe diarrhoea associated with typical enteropathogenic Escherichia coli among children aged 6-
15 nding cis-complemented derivatives of rabbit enteropathogenic Escherichia coli and compared their abi
17 related clinically important human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
19 s homologous to those of the human pathogens enteropathogenic Escherichia coli and enterohemorrhagic
20 derstanding of the molecular pathogenesis of enteropathogenic Escherichia coli and enterohemorrhagic
21 n secretion and translocation from wild-type enteropathogenic Escherichia coli and hypersecretion fro
22 athogens such as Mycobacterium tuberculosis, enteropathogenic Escherichia coli and Pseudomonas aerugi
24 homology to type III secreted proteins from enteropathogenic Escherichia coli and Yersinia and, base
25 perone-delivered to the translocase, EscV in enteropathogenic Escherichia coli, and cross it in stric
26 bacter rodentium is the rodent equivalent of enteropathogenic Escherichia coli, and it causes colitis
31 angstrom resolution cryo-EM structure of the enteropathogenic Escherichia coli ATPase EscN in complex
32 We purified the PulE homologue BfpD of the enteropathogenic Escherichia coli bundle-forming pilus (
35 scriptional regulator of a defence island in enteropathogenic Escherichia coli composed of a toxin-an
36 ity island' from the prototype AE bacterium, enteropathogenic Escherichia coli, containing all previo
37 gens such as Yersinia pseudotuberculosis and enteropathogenic Escherichia coli disarm host cells by i
40 intimate attachment of enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC) to mam
41 The human pathogens enterohemorrhagic and enteropathogenic Escherichia coli (EHEC and EPEC), as we
42 ith SadA from Salmonella enterica, EhaG from enteropathogenic Escherichia coli (EHEC), and UpaG from
44 cellular (S. Typhimurium) and extracellular (enteropathogenic Escherichia coli) enteric pathogens, vi
47 n 24 months with MSD, infection with typical enteropathogenic Escherichia coli, enterotoxigenic E col
48 /GII, rotavirus A, Plesiomonas shigelloides, enteropathogenic Escherichia coli, enterotoxigenic E. co
51 eotide sequence was determined for pMAR7, an enteropathogenic Escherichia coli (EPEC) adherence facto
53 of attaching and effacing pathogens such as enteropathogenic Escherichia coli (EPEC) and Citrobacter
54 l invasion by pathogenic bacteria, including enteropathogenic Escherichia coli (EPEC) and Citrobacter
60 Regulation of virulence gene expression in enteropathogenic Escherichia coli (EPEC) and enterohemor
63 It had been suggested that the flagella of enteropathogenic Escherichia coli (EPEC) and enterohemor
64 on of several important virulence factors in enteropathogenic Escherichia coli (EPEC) and reduced EPE
65 ues and fluids in response to infection with enteropathogenic Escherichia coli (EPEC) and Shiga-toxig
71 The type IV bundle-forming pili (BFP) of enteropathogenic Escherichia coli (EPEC) are required fo
73 ude two hydrophobic proteins, represented in enteropathogenic Escherichia coli (EPEC) by EspB and Esp
83 lifA, for lymphocyte inhibitory factor A) in enteropathogenic Escherichia coli (EPEC) encoding a prot
84 The attaching and effacing (A/E) pathogen enteropathogenic Escherichia coli (EPEC) forms character
87 yte effacement (LEE) pathogenicity island of enteropathogenic Escherichia coli (EPEC) has not been de
91 rs were performed to determine whether prior enteropathogenic Escherichia coli (EPEC) infection confe
119 The plasmid-encoded Per regulatory locus of enteropathogenic Escherichia coli (EPEC) is generally co
121 diarrhea induced by the food-borne pathogen enteropathogenic Escherichia coli (EPEC) is not known.
122 ression of the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli (EPEC) is regulated at
127 rial pathogens like Salmonella, Shigella and enteropathogenic Escherichia coli (EPEC) is the transloc
128 initial steps in biofilm development, and in enteropathogenic Escherichia coli (EPEC) it is mediated
131 Although the bundle-forming pilus (BFP) of enteropathogenic Escherichia coli (EPEC) mediates microc
132 ttens that were presumptively diagnosed with enteropathogenic Escherichia coli (EPEC) on the basis of
139 Production of type IV bundle-forming pili by enteropathogenic Escherichia coli (EPEC) requires BfpB,
140 cing (AE) lesion formation on enterocytes by enteropathogenic Escherichia coli (EPEC) requires the EP
141 tion of type IV bundle-forming pili (BFP) by enteropathogenic Escherichia coli (EPEC) requires the pr
145 on the enteroadherent factor plasmid of the enteropathogenic Escherichia coli (EPEC) strain B171-8 (
146 and virulence-associated -components in the enteropathogenic Escherichia coli (EPEC) strain E2348/69
147 gative transfer system identified in O119:H2 enteropathogenic Escherichia coli (EPEC) strain MB80 by
154 n, CesT, serves a chaperone function for the enteropathogenic Escherichia coli (EPEC) translocated in
160 Here, we report that the bacterial pathogen enteropathogenic Escherichia coli (EPEC) uses the type I
161 Outer membrane intimin directs attachment of enteropathogenic Escherichia coli (EPEC) via its Tir rec
163 to inhibit attachment of microcolony-forming enteropathogenic Escherichia coli (EPEC) was investigate
166 ive proteins are secreted extracellularly by enteropathogenic Escherichia coli (EPEC), a leading caus
173 ns and actin polymerization, the hallmark of enteropathogenic Escherichia coli (EPEC), enterohemorrha
174 es, collected semimonthly, were screened for enteropathogenic Escherichia coli (EPEC), enterotoxigeni
175 ded type IV bundle-forming pilus produced by enteropathogenic Escherichia coli (EPEC), has recently b
176 creen outer membrane proteins from 50 rabbit enteropathogenic Escherichia coli (EPEC), human EPEC, an
180 thogens, including Citrobacter rodentium and enteropathogenic Escherichia coli (EPEC), sharing only 2
181 processes as well as actin-based motility of enteropathogenic Escherichia coli (EPEC), vaccinia, and
182 e factor in two groups of enteric pathogens: enteropathogenic Escherichia coli (EPEC), which is a maj
183 E) lesions is central to the pathogenesis of enteropathogenic Escherichia coli (EPEC)-mediated diseas
193 mmon organisms detected by the GI panel were enteropathogenic Escherichia coli (EPEC, n = 21), norovi
194 zyme in the interaction between the host and enteropathogenic Escherichia coli(EPEC) and Shiga-toxige
197 rt the 1.9 A resolution crystal structure of enteropathogenic Escherichia coli GfcC, a periplasmic pr
199 and Campylobacter coli, Cryptosporidium spp, enteropathogenic Escherichia coli, heat-stable enterotox
200 cted with Citrobacter rodentium, a model for enteropathogenic Escherichia coli infection in humans, t
201 acter rodentium infection, a mouse model for enteropathogenic Escherichia coli infection, Hvem-/- mic
202 eal pathogens in the post-GI PCR cohort were enteropathogenic Escherichia coli (n = 14, 8%), noroviru
203 eal pathogens in the post-GI PCR cohort were enteropathogenic Escherichia coli (n=14, 8%), norovirus
204 secretion system effector protein NleE from enteropathogenic Escherichia coli plays a key role in th
205 rodentium, a murine model pathogen for human enteropathogenic Escherichia coli, predominantly coloniz
206 s an attaching and effacing strain of rabbit enteropathogenic Escherichia coli (REPEC) that causes di
207 how that important other pathogens including enteropathogenic Escherichia coli, Shigella flexneri, an
208 orum sensing to TTS in enterohemorrhagic and enteropathogenic Escherichia coli show that quorum sensi
210 enterocyte effacement (LEE) is necessary for enteropathogenic Escherichia coli to cause characteristi
211 entium uses virulence factors similar to the enteropathogenic Escherichia coli to produce attaching a
215 y, this mechanism closely mirrors that of an enteropathogenic Escherichia coli virulence factor, MAP,
216 mip for Legionella pneumophila, and eaeA for enteropathogenic Escherichia coli) were not detected, va