ライフサイエンスコーパス: enterotoxin

1 ge plasmids, and express both beta-toxin and enterotoxin.

2 duce additional toxins, e.g., beta2 toxin or enterotoxin.

3 eron response, and the first described viral enterotoxin.

4 pendent of the expression of the heat-labile enterotoxin.

5 of simultaneous exposure to these two potent enterotoxins.

6 ific IgE antibodies to Staphylococcus aureus enterotoxins.

7 ntestinal receptor for bacterial heat-stable enterotoxins.

8 viral virulence factor from other microbial enterotoxins.

9 ory therapy of diarrheas caused by bacterial enterotoxins.

10 etween asthma and exposure to staphylococcal enterotoxins.

11 rheas caused by cholera and Escherichia coli enterotoxins.

12 inhibitors, second messengers, and bacterial enterotoxins.

13 ificantly reduced in animals exposed to both enterotoxins.

14 jor virulence factor of the strain, Shigella enterotoxin 1, H4 flagellin, and O104 lipopolysaccharide

15 common pilus, flagellin and EAEC heat-stable enterotoxin 1.

16 3 decades, while detection of staphylococcal enterotoxin A (SEA) and toxic shock syndrome toxin (TSST

17 ated the effect of intranasal staphylococcal enterotoxin A (SEA) exposure on murine lung.

18 bacterial isolates containing staphylococcal enterotoxin A (SEA) from the affected skin of CTCL patie

19 reference materials (RMs) for Staphylococcal enterotoxin A (SEA) in cheese.

20 l agents; and PCR analysis of staphylococcal enterotoxin A (SEA) to SEH, toxic shock syndrome toxin 1

21 Ovalbumin (OVA)-specific, staphylococcal enterotoxin A (SEA)-nonreactive naive CD4 Tcon cells wer

22 enterotoxigenic Escherichia coli heat-stable enterotoxin A (ST) and evaluated under conditions of sta

23 , we establish Clostridium difficile and its enterotoxin A (TcdA) as Pyrin-activating agents and show

24 Staphylococcus aureus strains carrying enterotoxin A gene (sea) causes food poisoning and canno

25 response to the superantigen staphylococcal enterotoxin A on dendritic cells and a reduced number of

26 that after inhalation, Staphylococcus aureus enterotoxin A rapidly entered the bloodstream and induce

27 e model of SIRS that utilizes staphylococcal enterotoxin A specific for Vbeta3(+) T cells, we show th

28 terial toxins: cholera toxin, staphylococcal enterotoxin A, and toxic shock syndrome toxin.

29 re found to be 0.01 ng/mL for staphylococcal enterotoxin A, cholera toxin, botulinum toxin A, and ric

30 ying downstream of the TCR in staphylococcal enterotoxin A-specific CD8(+) T cells.

31 genic T cell stimulant Staphylococcus aureus enterotoxin A.

32 abile toxin, and three S. aureus toxins (the enterotoxins A and B and the toxic shock syndrome toxin)

33 , the limits of detection for staphylococcal enterotoxins A and B, cholera toxin, botulinum toxin A,

34 cation of six protein toxins (staphylococcal enterotoxins A and B, cholera toxin, ricin, botulinum to

35 allenges including new data on the rotavirus enterotoxin, a potential antiviral target.

36 Staphylococcus aureus, called staphylococcal enterotoxins (abbreviated SEA to SEU).

37 human and animal pathogen, and the cytotoxic enterotoxin (Act) is a crucial virulence factor of this

38 LTA1 protein is a novel, safe and effective enterotoxin adjuvant that improves protection of an intr

39 Since enterotoxin and beta toxin are produced in the intestine

40 omoted the cytotoxic activity and binding of enterotoxin and beta toxin more strongly than did full-l

41 toxicity-enhancing effects on C. perfringens enterotoxin and beta toxin, which are also important tox

42 mpared to that of an established V. cholerae enterotoxin and Escherichia coli heat-labile enterotoxin

43 Our findings define a role for B. fragilis enterotoxin and its activating protease in the pathogene

44 baseline conditions and with exposure to ST enterotoxin and suggests that further investigations of

45 hemolysin; (2) a group harboring the EAST-1 enterotoxin and the flagellar type H33 but no other prev

46 rhea, produce heat-stable and/or heat-labile enterotoxins and at least 25 different colonization fact

47 the first cells to respond to staphylococcal enterotoxins and contribute to the cytokine production a

48 orption capacity of Enterosgel for bacterial enterotoxins and endotoxin, bile acids and interaction w

49 s both heat-labile (LT) and heat-stable (ST) enterotoxins and is a major cause of diarrhea in infants

50 The genes encoding the enterotoxins and most of the colonization factors are lo

51 The role of enterotoxins and pathogenicity during repeat ETEC infect

52 with S. aureus led to decreased secretion of enterotoxins and phenotypic growth alterations consisten

53 lent strains that produce copious amounts of enterotoxins and spores.

54 (with or without genes encoding heat-labile enterotoxin), and Shigella spp.

55 with or without co-expression of heat-labile enterotoxin), and Shigella.

56 on of toxic shock syndrome toxin 1 (TSST-1), enterotoxin, and other superantigens by coagulase-negati

57 , GCC mediates diarrhea induced by bacterial enterotoxins, and an inverse relationship exists between

58 nd we establish that it is stable, expresses enterotoxins, and is not obviously transmissible by phag

59 e of absence of genes encoding for classical enterotoxins, and lack of plasmids encoding genes promot

60 Here, we verify that these enterotoxins are present in the human intestine during a

61 Both types of enterotoxins are regulated at the level of transcription

62 the heat-stable (ST) and/or heat-labile (LT) enterotoxins, as well as surface structures, known as co

63 versus 0.007%; p = 0.04) and staphylococcal enterotoxin B (Fo = 0.49% versus 0.26%; p = 0.04).

64 and IgA antibodies to Staphylococcus aureus enterotoxin B (SAEB) (P = 0.003) in nasal secretions fro

65 Detection of staphylococcal enterotoxin B (SEB) as a bacterial toxin causing severe

66 detection in various food of staphylococcal enterotoxin B (SEB) as a model up to 6 pg/mL at the dyna

67 Staphylococcal enterotoxin B (SEB) causes food poisoning in humans.

68 ere selected for detection of staphylococcal enterotoxin B (SEB) from 77 clinical Staphylococcus aure

69 Using anti-staphylococcal enterotoxin B (SEB) IgG as a "gate" and SEB as an "actua

70 Intranasal exposure to staphylococcal enterotoxin B (SEB) in C57BL/6 wild-type mice caused acu

71 resent the x-ray structure of staphylococcal enterotoxin B (SEB) in complex with its receptors, the T

72 d to improve the detection of Staphylococcal enterotoxin B (SEB) in food.

73 Staphylococcal enterotoxin B (SEB) is a bacterial superantigen that bin

74 Staphylococcal enterotoxin B (SEB) is a potent superantigen that contri

75 Staphylococcal enterotoxin B (SEB) is a potent toxin that can cause tox

76 Staphylococcal enterotoxin B (SEB) is a superantigen known to be a modu

77 Staphylococcal enterotoxin B (SEB) is a superantigen that cross-links t

78 -cell stimulating activity of Staphylococcal enterotoxin B (SEB) is an important factor in the pathog

79 f two recombinantly expressed Staphylococcal Enterotoxin B (SEB) mutants, a single point mutant (Y89A

80 Systemic exposure to staphylococcal enterotoxin B (SEB) rapidly and selectively recruited CD

81 se dust mite (HDM) and Staphylococcus aureus enterotoxin B (SEB) sensitization.

82 on (IFN) gamma in response to staphylococcal enterotoxin B (SEB) stimulation in 382 healthy infants a

83 When mice were injected with staphylococcal enterotoxin B (SEB) superantigen and H57-597 mAb, the ex

84 ogeneous detection method for staphylococcal enterotoxin B (SEB) utilizing core-shell-structured iron

85 or immunological detection of staphylococcal enterotoxin B (SEB) was designed, fabricated, and tested

86 id and sensitive detection of staphylococcal enterotoxin B (SEB) was developed using a novel acoustic

87 e (SPR) detection signal from staphylococcal enterotoxin B (SEB) was dramatically increased when the

88 Staphylococcal enterotoxin B (SEB), a potential biological warfare agen

89 body (Ab) therapeutic against staphylococcal enterotoxin B (SEB), a potential incapacitating bioterro

90 Staphylococcal enterotoxin B (SEB), a shock-inducing exotoxin synthesiz

91 fringens epsilon toxin (ETX), staphylococcal enterotoxin B (SEB), shiga toxin (STX), and plant toxin

92 role played by superantigens, staphylococcal enterotoxin B (SEB), staphylococcal enterotoxin C (SEC),

93 of a common food-borne toxin, Staphylococcal enterotoxin B (SEB).

94 icin, cholera toxin (CT), and staphylococcal enterotoxin B (SEB).

95 erparts for responsiveness to staphylococcal enterotoxin B (SEB).

96 cular emphasis on the role of staphylococcal enterotoxin B (SEB).

97 sponsive to SAgs, typified by staphylococcal enterotoxin B (SEB); ii) the human MAIT cell response to

98 e, heat-labile enterotoxin (LT), heat-stable enterotoxin b (STb), and enteroaggregative E. coli heat-

99 tinuously delivering the SAg, staphylococcal enterotoxin B (total of 10 mug/mouse), or PBS over 4 wk.

100 In this study, we show that staphylococcal enterotoxin B activates a Galphaq and PLCbeta2-dependent

101 +) T lymphocytes with soluble staphylococcal enterotoxin B and anti-CD28.

102 ton-Valentine leukocidin, and staphylococcal enterotoxin B and C negative, toxic shock syndrome toxin

103 r (PBS-BSA) and 0.1 ng/mL for staphylococcal enterotoxin B and LT.

104 fundamentally distinct SAgs, staphylococcal enterotoxin B and Mycoplasma arthritidis mitogen, on inf

105 cally important superantigens staphylococcal enterotoxin B and TSS toxin-1 with a single agent.

106 N-gamma(+) cells activated by staphylococcus enterotoxin B at 2 (P = .015) and 7 (P = .05) months of

107 e organic photodiode detected Staphylococcal enterotoxin B at concentrations as low as 0.5 ng/mL.

108 ity, and death in response to staphylococcal enterotoxin B challenge compared with wild-type mice.

109 a house dust mite extract and Staphylococcal enterotoxin B induced eczematous skin lesions.

110 MC were cultured for 7 d with staphylococcal enterotoxin B or IL-7 in the absence or presence of 100

111 on mitogenic stimulation with staphylococcal enterotoxin B or on antigenic stimulation with Candida a

112 Naive T cells primed by (staphylococcal enterotoxin B or tumor-associated protein-loaded) DC.Tbe

113 Choric exposure to staphylococcal enterotoxin B precipitated a lupus-like inflammatory dis

114 Chronic exposure to staphylococcal enterotoxin B resulted in a multisystem autoimmune infla

115 n-1-null mice challenged with staphylococcal enterotoxin B showed enhanced T cell accumulation in tis

116 ed constructs in an assay for staphylococcal enterotoxin B spiked into buffer showed the oriented dim

117 Staphylococcal enterotoxin B stimulated similar cytokine patterns in me

118 on-induced proliferation (via staphylococcal enterotoxin B stimulation) but inhibited homeostatic pro

119 In response to staphylococcal enterotoxin B stimulation, beta-cat(Tg) mice exhibited a

120 ction by cells in response to staphylococcal enterotoxin B stimulation.

121 imulation of the TCR with the staphylococcal enterotoxin B superantigen.

122 allenge with the staphylococcal superantigen enterotoxin B were comparable between WT and DKO mice.

123 T plus a polyclonal stimulus (staphylococcal enterotoxin B) or specific bacterial Ags, and effects on

124 demonstrated by superantigen (staphylococcal enterotoxin B)-induced deletion of Vbeta8(+) T cells.

125 phenotype was reproduced with staphylococcal enterotoxin B, a heterologous SAg that also targets Vbet

126 jected intraperitoneally with staphylococcal enterotoxin B, a pyrogenic superantigen, and their infla

127 l T cell-activating stimulus, staphylococcal enterotoxin B, Abs to CTLA-4 and PD-1 reversed HIV laten

128 ogical warfare agents, ricin, staphylococcal enterotoxin B, and epsilon toxin, in complex human biofl

129 or the toxins (cholera toxin, staphylococcal enterotoxin B, and ricin) were 1.6, 0.064, and 1.6 ng/mL

130 wild-type mice injected with staphylococcal enterotoxin B, and the administration of heparan sulfate

131 In the absence of staphylococcal enterotoxin B, only the combination of Abs to PD-1, CTLA

132 oligodendrocyte glycoprotein, Staphylococcal enterotoxin B, or in vitro with anti-CD3 anti-CD28 mAbs,

133 ted with anti-CD3 antibody or staphylococcal enterotoxin B, we found that chloramphenicol induces the

134 fferences in levels of Staphylococcus aureus enterotoxin B-induced cytokines between the two groups,

135 en human AD skin and allergen/staphylococcal enterotoxin B-induced mouse skin lesions, particularly i

136 ing a robust immune response (staphylococcal enterotoxin B-induced T cell activation).

137 ed pulmonary HIV-specific and staphylococcal enterotoxin B-reactive CD4(+) memory responses, includin

138 memory CD4+ T lymphocytes and staphylococcal enterotoxin B-stimulated cytokine production by total CD

139 Staphylococcal enterotoxin B-stimulated IL-2-producing cells were more

140 ther anti-CD3/28 antibody- or staphylococcal enterotoxin B-stimulated single-positive CD4(+) and CD8(

141 inhibitor U-73122 sensitizes staphylococcal enterotoxin B-treated mice to dexamethasone in vivo.

142 mmunized with a superantigen, staphylococcal enterotoxin B.

143 r cells (PBMCs) stimulated by staphylococcal enterotoxin B.

144 escribed for the detection of staphylococcal enterotoxin B.

145 spores or Pep(263) but not by staphylococcal enterotoxin B.

146 to the bacterial superantigen staphylococcal enterotoxin B.

147 atopic dermatitis-associated staphylococcal enterotoxin B.

148 superantigens (toxic shock syndrome toxin 1, enterotoxins B and C, and enterotoxin-like X) and cytoly

149 e two MRSA isolates, produced staphylococcal enterotoxins B, C, D, and E on overnight culture.

150 ith the bacterial enterotoxin staphylococcal enterotoxin-B (SEB), which naturally links a proportion

151 On Staphylococcus enterotoxin-B stimulation, which stimulates T cells thro

152 Enterotoxin-based adjuvants including cholera toxin and

153 lococcal enterotoxin B (SEB), staphylococcal enterotoxin C (SEC), and toxic shock syndrome toxin-1 (T

154 n Valentine leukocidin (PVL), staphylococcal enterotoxin C-1 (SEC-1), and phenol-soluble modulin alph

155 island encodes and expresses staphylococcal enterotoxin C3 (SEC3) and staphylococcal enterotoxin-lik

156 Rotavirus NSP4 is a viral enterotoxin capable of causing diarrhea in neonatal mice

157 Staphylococcus aureus enterotoxins cause debilitating systemic inflammatory re

158 Clostridium perfringens enterotoxin causes the gastrointestinal (GI) symptoms of

159 Domain I of Clostridium perfringens enterotoxin (cCPE) binds to the second extracellular loo

160 terminal fragment of Clostridium perfringens enterotoxin (cCPE) is a natural ligand for claudin-4.

161 Clostridium perfringens enterotoxin (CPE) action starts when the toxin binds to

162 gens type A strains producing C. perfringens enterotoxin (CPE) cause human food poisoning and antibio

163 tridium perfringens type A strains producing enterotoxin (CPE) cause one of the most common bacterial

164 Clostridium perfringens enterotoxin (CPE) causes food poisoning and antibiotic-a

165 Clostridium perfringens enterotoxin (CPE) causes the gastrointestinal symptoms o

166 Clostridium perfringens enterotoxin (CPE) causes the symptoms of a very common f

167 oisoning (FP) strains carrying a chromosomal enterotoxin (CPE) gene or the genetically related type C

168 belong to type C, carry beta-toxin (cpb) and enterotoxin (cpe) genes on large plasmids, and express b

169 Clostridium perfringens enterotoxin (CPE) is a major cause of food poisoning and

170 Clostridium perfringens enterotoxin (CPE) is a pore-forming toxin that causes th

171 Clostridium perfringens enterotoxin (CPE) is a pore-forming toxin with a unique,

172 Clostridium perfringens enterotoxin (CPE) is responsible for causing the gastroi

173 Clostridium perfringens enterotoxin (CPE) is the etiological agent of the third

174 al of food-associated stresses, and (ii) the enterotoxin (CPE) responsible for the symptoms of this f

175 ulate in the intestinal tract and produce an enterotoxin (CPE) that is responsible for the symptoms o

176 acterium Clostridium perfringens secretes an enterotoxin (CpE) that targets claudins through its C-te

177 many EN strains also express C. perfringens enterotoxin (CPE), suggesting that CPE could be another

178 -4) as receptors for Clostridium perfringens enterotoxin (CPE).

179 as the receptor for Clostridium perfringens enterotoxin (Cpe).

180 x [DI] = 0.924); generally positive only for enterotoxin D (74.5%); and resistant to clindamycin (98.

181 late and a high prevalence of staphylococcal enterotoxin D and the enterotoxin gene cluster.

182 the CT-related Escherichia coli heat-labile enterotoxin designated LT(R192G), or CpG oligodeoxynucle

183 bility of the astrovirus capsid to act as an enterotoxin, disrupting the gut epithelial barrier.

184 e-mutant of the Escherichia coli heat-labile enterotoxin (dmLT) adjuvant using microneedles.

185 with or without a double-mutant heat-labile enterotoxin (dmLT) as an adjuvant, in Bangladeshi childr

186 d variant of the superantigen staphylococcal enterotoxin E (SEA/E-120) linked to fragment antigen bin

187 GFP fusion) were activated by Staphylococcus enterotoxin E-coated Raji cells, NDE1 and dynein failed

188 tive type C isolates appeared to carry their enterotoxin-encoding cpe gene on a cpb plasmid.

189 elta act mutant (a T2SS-associated cytotoxic enterotoxin-encoding gene) and a Delta act Delta vasH mu

190 similarity between the iap/ibp plasmids and enterotoxin-encoding plasmids of type A isolates.

191 s have confirmed older observations that the enterotoxins enhance enteric bacterial colonization and

192 Staphylococcal food poisoning is caused by enterotoxins excreted into foods by strains of staphyloc

193 ults suggest that the host may also modulate enterotoxin expression because cells intoxicated with he

194 vel i.d. adjuvant of the type II heat-labile enterotoxin family, elicited strong systemic PspA-specif

195 pha1 and alpha2 is an important component of enterotoxin function and rotavirus pathogenesis, further

196 The enterotoxin gene cluster (egc) and the collagen adhesin

197 Genes of the enterotoxin gene cluster, seg, sei, sem, sen, and seo, w

198 ence of staphylococcal enterotoxin D and the enterotoxin gene cluster.

199 ar forms containing iap/ibp genes and silent enterotoxin gene sequences, with or without an IS1151-li

200 ious studies based on a design that involved enterotoxin genes cloned into a nontoxigenic fimbriated

201 ults in increased transcription of the major enterotoxin genes nhe, hbl, and cytK and the virulence r

202 d six distinct clones, which contained fewer enterotoxin genes than strains without lukF-PV.

203 taining open reading frames with homology to enterotoxin genes, restriction-modification systems, tra

204 ng heat-stable (estA) and heat-labile (eltA) enterotoxin genes.

205 IgE antibody concentrations in serum against enterotoxins, grass pollen (GP), and house dust mite all

206 Here, we identified a multicomponent enterotoxin, haemolysin BL (HBL), that engages activatio

207 on the mechanisms of action of the rotavirus enterotoxin highlight this pleiotropic protein as a good

208 Two families of bacterial heat-labile enterotoxins (HLTs) have been described: the type I HLTs

209 ere significantly increased in patients with enterotoxin IgE and nonatopic asthma.

210 Staphylococcal enterotoxin IgE antibodies, but not IgE against inhalant

211 We hypothesize that the presence of enterotoxin IgE in serum indicates the involvement of st

212 Enterotoxin IgE positivity was significantly greater in

213 a higher FEV(1) percent predicted value, and enterotoxin IgE was associated with a lower FEV(1) perce

214 percent of patients with severe asthma with enterotoxin IgE were considered nonatopic.

215 asthma (OR, 11.09; 95% CI, 4.1-29.6) versus enterotoxin IgE-negative subjects.

216 monstrated significantly increased risks for enterotoxin IgE-positive subjects to have any asthma (OR

217 Secretory diarrheas caused by bacterial enterotoxins, including cholera and traveler's diarrhea,

218 Adhesins and enterotoxins, including heat-labile (LT) and heat-stable

219 or colonization factor antigens (CFAs), and enterotoxins, including heat-labile enterotoxins (LT) an

220 as they are the final, rate-limiting step in enterotoxin-induced fluid secretion in the intestine.

221 n of the small intestine may determine which enterotoxin is maximally expressed.

222 CPE (Clostridium perfringens enterotoxin) is the major virulence determinant for C. p

223 Among them, NSP4 is the enterotoxin, known to disrupt cellular Ca(2+) homeostasi

224 own virulence genes which included those for enterotoxins, leukocidins, hemolysins, and surface prote

225 Staphylococcal enterotoxin-like K (SEl-K) is a potent mitogen that elic

226 cal enterotoxin C3 (SEC3) and staphylococcal enterotoxin-like toxin L (SElL), as confirmed by quantit

227 syndrome toxin 1, enterotoxins B and C, and enterotoxin-like X) and cytolysins (alpha-, beta-, and g

228 also had a high prevalence of staphylococcal enterotoxin-like X.

229 conditions for the optimal expression of one enterotoxin limit the expression of the other.

230 released by this pathogen is the heat-labile enterotoxin LT, which upsets the balance of electrolytes

231 (CFs) such as CFA/I fimbriae and heat-labile enterotoxin (LT) are important virulence factors and pro

232 wo highly homologous substrates: heat-labile enterotoxin (LT) from enterotoxigenic Escherichia coli (

233 in (CT) from Vibrio cholerae and heat-labile enterotoxin (LT) from enterotoxigenic Escherichia coli a

234 appears that toxins such as the heat-labile enterotoxin (LT) from Escherichia coli can help overcome

235 sibility of a vaccine containing heat-labile enterotoxin (LT) from ETEC delivered to the skin by patc

236 ent evidence suggesting that the heat-labile enterotoxin (LT) provides a colonization advantage for e

237 at express K88 (F4)(+) fimbriae, heat-labile enterotoxin (LT), heat-stable enterotoxin b (STb), and e

238 r effector molecule of ETEC, the heat-labile enterotoxin (LT), may enhance these interactions by stim

239 virulence factors, ETEC produces heat-labile enterotoxin (LT).

240 ibosyltransferase toxin known as heat-labile enterotoxin (LT).

241 meric B subunit of type IIb Escherichia coli enterotoxin (LT-IIb-B(5)), a doughnut-shaped oligomeric

242 toxic mutant of Escherichia coli heat-labile enterotoxin (LT-K63) and CpG1826 as model adjuvants coul

243 a 24 h period) in which either or both ETEC enterotoxins (LT and heat-stable toxin [ST]) were detect

244 As), and enterotoxins, including heat-labile enterotoxins (LT) and heat-stable enterotoxins (ST), are

245 ytes and their interaction with bacteria and enterotoxins may account for the noted increased suscept

246 Our data indicate that specialized, secreted enterotoxins may play a major role in one of these strat

247 stigate whether IgE to Staphylococcus aureus enterotoxins might be relevant to disease severity in ad

248 ce of an Escherichia coli mutant heat-labile enterotoxin [mLT(R192G)] or cholera toxin subunit B as a

249 Here, we show non-haemolytic enterotoxin (NHE) from the neglected human foodborne pat

250 e toxin B with no cross-reactions with other enterotoxins, nontoxigenic C. difficile, or other Clostr

251 this study reports on the mechanism by which enterotoxin NSP4 exerts cytotoxicity and the mechanism b

252 cAMP agonists, cholera toxin, or heat-stable enterotoxin of E. coli (STa toxin), with IC50 down to ap

253 hway and the effect of Staphylococcus aureus enterotoxins on the regulation of the pro-inflammatory n

254 Two types of enterotoxins, one heat labile and the other heat stable,

255 ucing adjuvants, such as type II heat-labile enterotoxin or cholera toxin, resulted in increased morb

256 taglandin E(2), Escherichia coli heat-stable enterotoxin, orexins, and carbonated beverages stimulate

257 strain carrying the gene for staphylococcal enterotoxin P (sep).

258 ne production associated with staphylococcal enterotoxin pathogenesis.

259 ds guanylin, uroguanylin, or the heat-stable enterotoxin peptide (ST).

260 acute lung response to Staphylococcus aureus enterotoxin, peripheral injection of poly(I:C) manifeste

261 However, some enterotoxin-positive type C isolates appeared to carry t

262 s cholera toxin or the heat-labile or stable enterotoxins produced by Escherichia coli) that invade c

263 o C coli (around two times), and heat-stable enterotoxin-producing E coli ([ST-ETEC] around 1.5 times

264 (OR: 1.46; 95% CI: 1.11, 1.91), heat-labile enterotoxin-producing E. coli (OR: 1.55; 95% CI: 1.04, 2

265 % CI, 24.4%-26.7%]), followed by heat-stable enterotoxin-producing Escherichia coli (AF, 18.4% [95% C

266 n 27.8% and 8.2%, respectively), heat-stable enterotoxin-producing Escherichia coli (in 21.2% and 8.5

267 ulence factors such as ystA, responsible for enterotoxin production, ail, attachment invasion locus g

268 SM101 reduced the levels of sporulation and enterotoxin production, supporting the involvement of Ab

269 cterized by distinct colonization factor and enterotoxin profiles.

270 f asthma to determine whether staphylococcal enterotoxins promote TH2 differentiation of allergen-spe

271 archaic plasmid that encoded staphylococcal enterotoxins R, J, and P.

272 s in various cell systems, and the bacterial enterotoxin receptor guanylyl cyclase C (GCC), the princ

273 tory diarrheas caused by bacterial and viral enterotoxins remain a significant cause of morbidity and

274 enterotoxin and Escherichia coli heat-labile enterotoxin reversed passive latex agglutination (VET-RP

275 IgE antibodies against Staphylococcus aureus enterotoxins (SAEs).

276 Labeled staphylococcal enterotoxin (SE) A, SED, and SEE were used to isolate si

277 e signaling that results from staphylococcal enterotoxin (SE) exposure.

278 Specific IgE to Staphylococcus aureus enterotoxins (SE-IgE) has been associated with asthma.

279 However, unlike the classical enterotoxins SEB and toxic shock syndrome toxin 1 (TSST-

280 eus) carriage and sensitization to S. aureus enterotoxins (SEs) have been associated with allergic di

281 es from involved skin express staphylococcal enterotoxins (SEs) that induce crosstalk between maligna

282 istance 26 A), cholera toxin and heat-labile enterotoxin (shortest distance 31 A), anti-HIV antibody

283 es as the receptor for bacterial heat-stable enterotoxin (ST) peptides and the guanylin family of gas

284 Oral delivery of the heat-stable enterotoxin (ST), an exogenous GUCY2C ligand, opposed RI

285 eat-labile enterotoxins (LT) and heat-stable enterotoxins (ST), are the key virulence factors in ETEC

286 Originally identified as a target of E. coli enterotoxin STa, GC-C is an important regulator of physi

287 enterotoxigenic Escherichia coli heat-stable enterotoxin STa, which deregulates this pathway and caus

288 bined CAR T cell transfer with the bacterial enterotoxin staphylococcal enterotoxin-B (SEB), which na

289 exit duct to drive efflux of antibiotics and enterotoxin STII out of the bacterial cell.

290 ux of macrolide antibiotics and secretion of enterotoxin STII.

291 es worldwide, often by producing heat-stable enterotoxins (STs), which are peptides structurally homo

292 Some bacteria produce secretory enterotoxins (such as cholera toxin or the heat-labile o

293 f inflammation and the presence of S. aureus enterotoxin (superantigen)-specific IgE in the nasal pol

294 i use OMVs to deliver bundles of heat labile enterotoxin to host cells.

295 HRV infection and RV enterotoxin treatment of HIEs caused physiological lumen

296 d the C. difficile toxin, in addition to the enterotoxins TsdA and TsdB.

297 clfB, cna, map/eap; P < .0001 for all) and 5 enterotoxins (tst, sea, sed, see, and sei; P </= .005 fo

298 E. coli-like colonies were screened for ETEC enterotoxins using a GM1 enzyme-linked immunosorbent ass

299 The distribution of ETEC enterotoxins varied between the symptomatic children (44

300 PE-induced cytotoxicity by preincubating the enterotoxin with soluble full-length recombinant claudin