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1 tes in 2015, of which 70% showed symptoms of envenomation.
2 phylaxis, most notably caused by Hymenoptera envenomation.
3 can protect against the damaging effects of envenomation.
4 be relevant to the pathology of brown spider envenomation.
5 arios, including hemostasis, thrombosis, and envenomation.
6 in vivo under conditions simulating scorpion envenomation.
7 tituent porins at doses equivalent to lethal envenomation.
8 thrombotic complications in Russell's viper envenomation.
9 ute to the pathology and mortality caused by envenomation.
10 ith clinically significant signs of scorpion envenomation.
11 g to the development of clinical symptoms of envenomation.
12 pecies worldwide cause medically significant envenomation.
13 ocal and systemic toxic effects of crotaline envenomation.
14 reduce the pain associated with the scorpion envenomation.
15 ate hosts or humans during oral ingestion or envenomation.
16 s and many of the other clinical symptoms of envenomation.
17 ernative and/or additional treatment against envenomation.
18 sistent with the severe pain associated with envenomation.
19 hiness, and muscle cramping after accidental envenomation.
20 defense function against helminths and snake envenomation.
21 pment of efficacious therapies against snake envenomation.
22 mmasome-dependent release and the outcome of envenomation.
23 ective therapeutic intervention for scorpion envenomation.
24 local inflammatory process caused by B atrox envenomations.
25 timated number of annual deaths due to snake envenomation (80,000 to 130,000) is similar to the estim
27 ms by which the innate immune system detects envenomation and initiates inflammatory and allergic res
28 e critical event following Chironex fleckeri envenomation and that rapid administration of zinc could
30 ancreatitis that develops following scorpion envenomation as both of these v-SNARE proteins are assoc
31 ration and necrosis due to Loxosceles spider envenomation ("bites") is among the best known sequelae
32 er diagnostic and therapeutic approaches for envenomation, but also to explore the clinical potential
34 ive and argue that, in the context of severe envenomation by arthropods or reptiles, anaphylaxis migh
37 efore are likely to cause pain after natural envenomations by D. vulnerans Our data reveal convergent
41 mportant to understand the spectrum of snake envenomation effects and approaches to management and to
44 nhibitors and repurposed drugs against snake envenomation from 2005 to date and proposed novel approa
45 at those rare individuals who receive severe envenomation from poisonous snakes, spiders, or scorpion
46 t their lethality and/or obnoxious effect of envenomation has been garnering greater attention owing
47 droctonus australis is primarily involved in envenomations in North Africa, notably in Tunisia and Al
48 children with neurotoxic effects of scorpion envenomation, intravenous administration of scorpion-spe
54 a (funnel-web spiders) cause rare but severe envenomation requiring medical intervention and sometime
55 enoms can decrease pathology associated with envenomation seems to be influenced by the type of venom
56 biology of venom and the management of snake envenomation since this topic was last addressed in the
58 by recruitment of neutrophils to the site or envenomation, the inflammasome is dispensable for the al
59 enom (CroFab) for the treatment of crotaline envenomation, the predominant venomous snakebite in the
60 sponsible for the long-lasting local pain of envenomation via activation of peripheral sensory neuron