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1 helium and stroma (e.g., edema and extensive eosinophil infiltration).
2 emokine genes expressed at stages of massive eosinophil infiltration.
3 creased IL-4, IL-5, and IL-13 production and eosinophil infiltration.
4 lammation characterized by TH2 cytokines and eosinophil infiltration.
5 ting C. neoformans-driven pulmonary IL-5 and eosinophil infiltration.
6 ry end point was the reduction in esophageal eosinophil infiltration.
7 tion of NMO-IgG and complement showed marked eosinophil infiltration.
8 and numbers were negatively correlated with eosinophil infiltration.
9 tly impaired, whereas there was no effect on eosinophil infiltration.
10 n in ear edema, inflammation, mast cell, and eosinophil infiltration.
11 in the reduction of cytokine production and eosinophil infiltration.
12 release from basophil/mast cells and induce eosinophil infiltration.
13 matory reactions and to look for evidence of eosinophil infiltration.
14 symptoms accompanied by a massive pulmonary eosinophil infiltration.
15 lergic airway inflammation, increased airway eosinophil infiltration (24-fold) correlated with secret
17 nd 11 (CCL11; or eotaxin-1) characterized by eosinophil infiltration and a subsequent T(H)2 immune re
19 tion between Th2 cytokine production, tissue eosinophil infiltration and activation, and, importantly
21 e PPD response to a type 2-like pattern with eosinophil infiltration and decreased TNF and RANTES, bu
22 At day 21, 16/16 test mice developed intense eosinophil infiltration and degranulation of the human m
24 athological changes, including inhibition of eosinophil infiltration and excess mucus production in t
26 ivo adduct-sensitized mice exhibited reduced eosinophil infiltration and IL-13 expression in the airw
27 cytokines is a central driver in responding eosinophil infiltration and increased airway hyperreacti
28 piratory failure, and death, associated with eosinophil infiltration and increased expression of eota
29 ostinfection, mice lacking SP-D have reduced eosinophil infiltration and interleukin-5 (IL-5) in lung
31 es of asthma, including airway inflammation, eosinophil infiltration and non-specific bronchial respo
32 during airway allergen challenge, inhibited eosinophil infiltration and prevented the development of
33 ter treatment, preceding the onset of dermal eosinophil infiltration and the development of clinicall
34 mmatory disorder of the esophagus defined by eosinophil infiltration and tissue remodeling with resul
35 vity, T(H)2 responses, mucus hypersecretion, eosinophil infiltration, and collagen deposition were no
36 cell, macrophage-like mononuclear cell, and eosinophil infiltration, and elevation of total serum Ig
37 ons exhibit epidermal and dermal thickening, eosinophil infiltration, and increased levels of the cys
38 markedly reduced epidermal thickening, lower eosinophil infiltration, and lower serum IgE levels comp
39 the gut microbiome induced IL-33, subsequent eosinophil infiltration, and mounting of Th2 immune resp
40 have drastically reduced lung inflammation, eosinophil infiltration, and mucous production following
41 ity to aerosolized methacholine, lung tissue eosinophil infiltration, and numbers of proliferating ce
42 sed allergen-induced airway hyperreactivity, eosinophil infiltration, and production of pro-inflammat
43 eosinophil-associated chemokines, eotaxins, eosinophil infiltration, and subsequent HILI were unclea
44 Staphylococcus aureus skin colonization and eosinophil infiltration are associated with many inflamm
45 onist decreases significantly lymphocyte and eosinophil infiltration as well as mRNA expression of eo
46 ier, microbial colonization, spongiosis, and eosinophil infiltration, but also aspects of psoriasis,
47 ng on the underlying disease and mechanisms, eosinophil infiltration can lead to organ dysfunction, c
48 .IL5tg mice, despite dramatic differences in eosinophil infiltration (CD45(+)CD11c(-)Gr1(-)MHC class
49 hibited increased lung ILC counts and tissue eosinophil infiltration compared with values in lean mic
51 tures of asthma such as airway constriction, eosinophil infiltration, edema, and mucus accumulation.
52 n-challenged null mice showed increased lung eosinophil infiltration, enhanced bone marrow and blood
53 Such metabolic improvements are mediated by eosinophil infiltration, enhanced type 2 cytokine signal
54 cytokine-associated disease characterized by eosinophil infiltration, epithelial cell hyperplasia, an
55 ced pulmonary inflammation, characterized by eosinophil infiltration, goblet cell hyperplasia with mu
58 F and lung immunohistology demonstrated that eosinophil infiltration in OVA-challenged Gal-3 KO mice
59 verall clinical findings appear to implicate eosinophil infiltration in proximal and distal dysmotili
61 ever, only perforin-sufficient CTL inhibited eosinophil infiltration in the airway, mucus production,
64 IgE, increased Th2 cytokine production, and eosinophil infiltration in the stomach-draining lymph no
65 me an attractive target in reducing unwanted eosinophil infiltration in various disorders including a
66 tures of allergic disease, including AHR and eosinophil infiltration, in uninfected OVA-sensitized/ch
67 developed mouse model exhibited substantial eosinophil infiltration, increased levels of inflammator
68 irway damage in asthmatic mice by decreasing eosinophil infiltration, inhibiting chemokines/cytokines
69 i-T1/ST2 monoclonal antibody (mAb) inhibited eosinophil infiltration, interleukin 5 secretion, and Ig
70 antibody treatment can effectively decrease eosinophil infiltration into hamster cutaneous healing w
71 bly, only dual IL-4/IL-13 blockade prevented eosinophil infiltration into lung tissue without affecti
72 ngly, DRD3 deficiency results in exacerbated eosinophil infiltration into the airways of mice undergo
73 e uncovered that immunosuppression increases eosinophil infiltration into the allograft in an IL-5-de
74 and challenge increased ocular symptoms and eosinophil infiltration into the conjunctiva over PBS co
80 ,696 correlated with up to a 97% decrease in eosinophil infiltration into the lower spinal cord as de
81 as shown by a clear inhibition of T cell and eosinophil infiltration into the lung tissue and airways
82 of 5 mg/kg Sephadex caused a time-dependent eosinophil infiltration into the lung, reaching a peak a
88 Sensitization in these neonates also induces eosinophil infiltration into the skin and elevates skin
91 e exhibited hallmark EoE features, including eosinophil infiltration, intraepithelial eosinophils, mi
92 ophin-deficient muscle; we show clearly that eosinophil infiltration is not a driving force behind ac
93 n was associated with epidermal hyperplasia, eosinophil infiltration, less large-cell transformation,
94 h included a significant reduction in airway eosinophil infiltration, mucus hypersecretion, and airwa
95 itor, PNRI-299, significantly reduced airway eosinophil infiltration, mucus hypersecretion, edema, an
96 montelukast significantly reduced the airway eosinophil infiltration, mucus plugging, smooth muscle h
97 pithelium were intensely immunoreactive, and eosinophil infiltration occurred at sites of eotaxin upr
100 ly, Th2 responses including, IgE production, eosinophil infiltration of the airway, subepithelial fib
101 on of a T helper 2-type cytokine response or eosinophil infiltration of the airways after allergic se
102 Airway sensitization was associated with eosinophil infiltration of the airways and increased pro
103 is an inflammatory disease characterized by eosinophil infiltration of the airways, actions of beta-
104 group of diseases characterized by selective eosinophil infiltration of the gastrointestinal (GI) tra
105 nable to develop airway hyperresponsiveness, eosinophil infiltration of the lung parenchyma, or IL-5
106 ype demonstrated by elevated levels of blood eosinophils, infiltration of eosinophils in the esophagu
108 obstruction by approximately 50%, inhibited eosinophil infiltration, reduced BALF concentrations of
109 a mechanism involving eotaxin to explain the eosinophil infiltration seen in a variety of human disea
111 racterized by increased CCL11 production and eosinophil infiltration such as Hodgkin's lymphoma, nasa
112 Breakthrough infection mice exhibit lung eosinophil infiltration that peaks at 7-10 days post-cha
113 ls of airway hyperreactivity and lung tissue eosinophil infiltration that were similar to those of th
114 CR1 deficiency did not inhibit neutrophil or eosinophil infiltration to the cornea or development of
115 g, as indicated by the increase of pulmonary eosinophil infiltration, type 2 cytokine production, and
116 e found enhanced type 2 cytokine production, eosinophil infiltration, vascular remodeling, and number
117 hase pulmonary inflammation, blocking airway eosinophil infiltration, VCAM-1 expression, and mucus hy
120 nocyte, lymphocyte, and, to a lesser degree, eosinophil infiltration was observed, peaking at 10-24 h
121 ic asthma is characterized mainly by massive eosinophil infiltration, which induces airway injury and