ライフサイエンスコーパス: epidermal

1 Genome-wide mapping of epidermal 5-hmC in murine psoriasis revealed loci-specif

2 mplex pathogenesis involving immune cell and epidermal abnormalities.

3 morphological and cytological information in epidermal and dermal layers while reducing the need for

4 stic effect of these strategies in enhancing epidermal and dermal penetration.

5 Suction blistering captured epidermal and most immune cells equally well as biopsies

6 show that UV-B irradiation selectively kills epidermal and niche cells in the shoot apex.

7 mbrane and perinuclear space in both tobacco epidermal and soybean root cells.

8 It is a late-evolving epidermal appendage that has the primary function of pro

9 C3 as a hub coordinating multiple aspects of epidermal barrier acquisition.

10 The breakdown of the epidermal barrier and consequent loss of skin hydration

11 erapeutic opportunities to restore defective epidermal barrier conditions.

12 ings provide novel mechanistic insights into epidermal barrier formation and could open up new therap

13 strates the multitude of factors controlling epidermal barrier formation and homeostasis remains to b

14 of rapamycin complex 2 (mTORC2) signaling in epidermal barrier formation.

15 ureus application, impaired S1pr2(-/-) mouse epidermal barrier function allowed deeper bacterial pene

16 sms by which IDL improves skin hydration and epidermal barrier function, supporting IDL as an effecti

17 the skin microbiome, and the decrease in the epidermal barrier function.

18 ons were identified between Th2 measures and epidermal barrier gene-subsets and individual genes (FLG

19 llular signal transduction, is essential for epidermal barrier integrity.

20 nd filaggrin processing, a function vital to epidermal barrier integrity.

21 tabolism blocking agents, appears to protect epidermal barrier integrity.

22 idomics, and mouse genetics, we characterize epidermal barrier maintenance versus a newly established

23 ated with disease activity have functions in epidermal barrier properties and immune modulation.

24 nt with this, IDL increased the abundance of epidermal barrier proteins (FLG and involucrin) and prev

25 An effective epidermal barrier requires structural and functional int

26 Although the epidermal barrier typically excludes nucleic acid entry,

27 ve correlation between Treg-cell numbers and epidermal BMP7 expression in cutaneous psoriatic lesions

28 growth-related genes are highly activated by epidermal, but not by vascular, PIF4.

29 eld driving the persistence of the overlying epidermal cancer field.

30 s (DETC) concomitant with an accumulation of epidermal CD8(+)CD69(+)CD103(+) T(RM) cells in mice.

31 dy provides a molecular explanation for root epidermal cell fate switch in response to ribosomal defe

32 Moreci and Lechler follow the lifetime of an epidermal cell from its birth to its ultimate death, and

33 Our cell sorting-based study of the epidermal cell layer transcriptome confirms that core UV

34 interaction, E2F target gene expression, and epidermal cell number and shape in e2fb mutant and overe

35 Both epidermal cell optogenetic inhibition and interruption o

36 tin synthesis with cell wall development and epidermal cell size has been identified.

37 These PRPs are absent from root epidermal cell walls, and PRP accumulation is highly loc

38 Langerhans cells and inflammatory dendritic epidermal cells (IDEC) are located in the epidermis of A

39 rganized in nests and the surrounding larval epidermal cells (LECs).

40 ther than being randomly distributed, p53(+) epidermal cells are enriched only in areas overlying thi

41 CHCs are synthesised in sub-epidermal cells called oenocytes that are very difficult

42 However, the mutant had shorter epidermal cells in comparison with wild-type Hwayoung (H

43 All aerial epidermal cells in land plants are covered by the cuticl

44 Tip-growing rhizoid cells develop among flat epidermal cells in the epidermis of the early-diverging

45 rescent protein-tagged phyB (phyB-FP) in the epidermal cells of Arabidopsis hypocotyl and cotyledon.

46 Iron absorption in Arabidopsis root epidermal cells requires the IRT1 transporter that also

47 In leaf epidermal cells where a polygonal ER network can be reso

48 e it elongates invasive hyphae in underlying epidermal cells(5).

49 s of the stress sensor FoxO than neighboring epidermal cells, and hence exhibit no marked induction o

50 f silver precipitation were observed in some epidermal cells, indicating moderate cuticular uptake.

51 predominantly basally localized PIN1 in root epidermal cells, leading to agravitropic root growth.

52 the epidermis via small fenestrae in the sub-epidermal collagen fibril layer; most nerves exit abrupt

53 serve as crucial signaling hubs to maintain epidermal communication.

54 like phenotype characterized by dysregulated epidermal de novo lipid synthesis, altered lipid lamella

55 Its epidermal delivery and therapeutic efficacy were evaluat

56 dissolvable microneedle array (PLD-MNA) for epidermal delivery of powdered allergens and to evaluate

57 e factor 1 (FIH-1), we observed increases in epidermal DeltaNp63alpha expression along with prolifera

58 ere cultured on a topography that mimics the epidermal-dermal interface, spatial segregation of mean

59 n keratinocyte carcinoma, including enriched epidermal development and Wnt signaling pathways enriche

60 an important dataset for further studies on epidermal development in tomato and other species of the

61 nverge on transcription factors critical for epidermal development, p63 and Notch.

62 NCoR and SMRT, which function redundantly in epidermal development.

63 Our study identifies epidermal developmental processes required for digit sep

64 Single-cell RNA sequencing revealed that epidermal developmental progenitors form a more uniform

65 "core" signature characterized by disturbed epidermal differentiation and activation of IL-31/IL-1 s

66 cient skin that reflect an altered course of epidermal differentiation and enhanced inflammatory resp

67 g SMARCA complex components, SOX6 suppresses epidermal differentiation and epigenetically silences cr

68 This enables vitamin D to regulate epidermal differentiation and hair follicle cycling and,

69 These TGM1 mutations trigger the abnormal epidermal differentiation and impaired cutaneous barrier

70 control five subclusters located within the epidermal differentiation complex on chromosome 25; (2)

71 lumab led to a stronger increase in level of epidermal differentiation markers.

72 se to dithranol belonged to keratinocyte and epidermal differentiation pathways and IL-1 family membe

73 ers has been pivotal to the understanding of epidermal differentiation, function, and renewal.

74 nd previously unrecognized genes controlling epidermal differentiation.

75 entified two genes that on knockdown induced epidermal differentiation: ELOVL1, encoding elongation o

76 ced VZV propagation in culture and prevented epidermal disruption in skin explants.

77 nd involves a strong genetic predisposition, epidermal dysfunction, and T-cell driven inflammation.

78 Here, we present the first large-area epidermal electronic system (L-EES) and demonstrate its

79 PAR-6 and PKC-3 are required in the epidermal epithelium for animal growth, molting, and the

80 emerging roles of adipose FABP (A-FABP) and epidermal FABP (E-FABP) in the fields of obesity, chroni

81 and showed no overt signs of shifting to an epidermal fate.

82 encing, we identify molecular changes in the epidermal, fibroblast, and immune cells of Ovol1-deficie

83 al epidermal repair response, the protective epidermal function was impaired in Ric(EKO) mice, as rev

84 and detail how this journey is necessary for epidermal function.

85 ions with AD severity, barrier function, and epidermal gene expression in the first US early-life coh

86 vailable, retrovirally transduced autologous epidermal grafts and intradermal lentivirally engineered

87 ed with MDA-MB-231 cells expressing miR-149, epidermal growth factor (EGF) and amphiregulin expressio

88 g for two physiologically important ligands, epidermal growth factor (EGF) and hepatocyte growth fact

89 e crypt-villus axis and is the source of the epidermal growth factor (EGF) family member NEUREGULIN1

90 In PC12 cells, epidermal growth factor (EGF) induces transient ERK acti

91 Epidermal growth factor (EGF) maintains intestinal stem

92 Reduced concentrations of epidermal growth factor (EGF) of maternal origin within

93 Systemic administration of epidermal growth factor (EGF) promoted HSC DNA repair an

94 cell equilibrium is feedback control of the epidermal growth factor (EGF) protease Rhomboid (Rho).

95 rylated in vitro by the Tyr kinase domain of epidermal growth factor (EGF) receptor.

96 We have found that epidermal growth factor (EGF) triggered an enrichment of

97 estigate the molecular mechanism, we utilize epidermal growth factor (EGF)-inducible immediate early

98 his site was predicted to separate the first epidermal growth factor (EGF)-like domain from the remai

99 ll as in animals following overexpression of epidermal growth factor (EGF).

100 transforming growth factor-beta [TGF-beta1], epidermal growth factor [EGF], platelet-derived growth f

101 Apoptotic neutrophils further release epidermal growth factor and promote the differentiation

102 The epidermal growth factor and the basic fibroblast growth

103 The presence of rigid epidermal growth factor domains in protein C as opposed

104 amin K-dependent clotting factors containing epidermal growth factor domains, such as factors VII, IX

105 yrosine kinase with immunoglobulin-like loop epidermal growth factor homology domain 2, vascular endo

106 We analysed internalization of epidermal growth factor in patient cells and identified

107 selectivity of ADAM17 toward Heparin-binding epidermal growth factor like growth factor (HB-EGF), a c

108 The resultant anti-epidermal growth factor receptor (anti-EGFR) AEC worked

109 Here, a six-arginine-tailed anti-epidermal growth factor receptor (EGFR) affibody was emp

110 Epidermal growth factor receptor (EGFR) and human epider

111 B and gH directly bind and activate cellular epidermal growth factor receptor (EGFR) and integrin bet

112 transcripts coding for oncoproteins such as epidermal growth factor receptor (EGFR) and MYC can supp

113 t FCHSD2 loss impacts recycling of the RTKs, epidermal growth factor receptor (EGFR) and proto-oncoge

114 We apply this model to epidermal growth factor receptor (EGFR) antibodies and f

115 tyrosine kinases, including signaling by the epidermal growth factor receptor (EGFR) family (EGFR1-4

116 identified that the combination of afatinib (epidermal growth factor receptor (EGFR) inhibitor) and Y

117 Epidermal growth factor receptor (EGFR) is a prototype r

118 Asymmetric dimer formation of epidermal growth factor receptor (EGFR) is crucial for E

119 a specific tyrosine residue in STING by the epidermal growth factor receptor (EGFR) is required for

120 Acquired drug resistance in epidermal growth factor receptor (EGFR) mutant non-small

121 r proportion of adenocarcinoma histology and epidermal growth factor receptor (EGFR) mutations.

122 rized by mutually exclusive mutations in the epidermal growth factor receptor (EGFR) or the guanosine

123 We report that epidermal growth factor receptor (EGFR) regulates DNA re

124 The epidermal growth factor receptor (EGFR) signaling cascad

125 tome of RIS and discovered that genes of the epidermal growth factor receptor (EGFR) signaling pathwa

126 Falpha), interleukin 6 receptor (IL-6R), and epidermal growth factor receptor (EGFR) signaling.

127 Targeting epidermal growth factor receptor (EGFR) through an allos

128 Several epidermal growth factor receptor (EGFR) tyrosine kinase

129 I's role in EMT, we examined the activity of epidermal growth factor receptor (EGFR), a known EMT dri

130 kinases (RTKs) on host cells, including the epidermal growth factor receptor (EGFR), and activates c

131 RTK hepatocyte growth factor receptor (MET), epidermal growth factor receptor (EGFR), and human epide

132 eceptor tyrosine kinases (RTKs), such as the epidermal growth factor receptor (EGFR), locally increas

133 such as poly (ADP-ribose) polymerase (PARP), epidermal growth factor receptor (EGFR), Vascular endoth

134 ty specifically in cells over-expressing the epidermal growth factor receptor (EGFR), with over 99% r

135 gnificant attenuation of the pro-tumorigenic Epidermal Growth Factor Receptor (EGFR)-Akt axis, and fi

136 ced non-small-cell lung cancer (NSCLC) is an epidermal growth factor receptor (EGFR)-directed oral ty

137 nsformation in a Drosophila genetic model of epidermal growth factor receptor (EGFR)-driven tumorigen

138 monstrated a connection that upregulation of epidermal growth factor receptor (EGFR)-leukemia inhibit

139 ade radiation-mediated apoptosis by p53- and epidermal growth factor receptor (EGFR)-mediated DNA rep

140 utic strategy for tumours overexpressing the epidermal growth factor receptor (EGFR).

141 es to enhancing KSHV infectivity through the epidermal growth factor receptor (EGFR).

142 also depended on the transactivation of the epidermal growth factor receptor (EGFR).

143 model antibody cetuximab and its target, the epidermal growth factor receptor (EGFR).

144 ERBB4 is a member of the epidermal growth factor receptor (EGFR)/ERBB subfamily o

145 The human epidermal growth factor receptor (EGFR/ERBB1) is a recep

146 01 assessed whether dual versus single human epidermal growth factor receptor 2 (HER2) -targeting dru

147 mal growth factor receptor (EGFR), and human epidermal growth factor receptor 2 (HER2) are involved i

148 Amplification and/or overexpression of human epidermal growth factor receptor 2 (HER2) are observed i

149 eptor (ER), progesterone receptor, and human epidermal growth factor receptor 2 (HER2) expression, is

150 rosine kinase inhibitor neratinib is a human epidermal growth factor receptor 2 (HER2) inhibitor appr

151 Resistance of breast cancer to human epidermal growth factor receptor 2 (HER2) inhibitors inv

152 Human epidermal growth factor receptor 2 (HER2) is overexpress

153 esophageal adenocarcinomas (EACs) are human epidermal growth factor receptor 2 (HER2) positive.

154 r (ER), progesterone receptor (PR) and human epidermal growth factor receptor 2 (HER2), are absent is

155 Human epidermal growth factor receptor 2 (HER2)-amplified brea

156 The TEXT/SOFT HR-positive/human epidermal growth factor receptor 2 (HER2)-negative analy

157 (AC) in BRCA carriers with stage I-III human epidermal growth factor receptor 2 (HER2)-negative breas

158 Estrogen receptor-positive/human epidermal growth factor receptor 2 (HER2)-negative cases

159 tors for hormone receptor-positive and human epidermal growth factor receptor 2 (HER2)-negative metas

160 Methods: Patients with ER+/human epidermal growth factor receptor 2 (HER2)-negative metas

161 germline BRCA1/2 mutation (gBRCAm) and human epidermal growth factor receptor 2 (HER2)-negative metas

162 In the HER2CLIMB study, patients with human epidermal growth factor receptor 2 (HER2)-positive breas

163 NE trial, patients with stage I to III human epidermal growth factor receptor 2 (HER2)-positive breas

164 Patients with human epidermal growth factor receptor 2 (HER2)-positive metas

165 Conclusion Human epidermal growth factor receptor 2 (HER2)-targeted imagi

166 Background Human epidermal growth factor receptor 2 (HER2)-targeted thera

167 labeled forms of EGFR and its paralog, human epidermal growth factor receptor 2 (HER2/ERBB2) in vesic

168 Injection site and the tumor markers human epidermal growth factor receptor 2 and estrogen receptor

169 While targeted therapies exist for human epidermal growth factor receptor 2 positive (HER2 +) bre

170 en receptor, progesterone receptor and human epidermal growth factor receptor 2 status and tumor grad

171 rmal growth factor receptor (EGFR) and human epidermal growth factor receptor 3 (HER3) have been inve

172 ly show that p-Ezrin (T567) is controlled by epidermal growth factor receptor and MET.

173 nbasal, according to cytokeratins 5/6 and/or epidermal growth factor receptor positivity by immunohis

174 ry identified EHD1 (Eps15 [endocytic adaptor epidermal growth factor receptor substrate 15] homology

175 ging as biomarkers of acquired resistance to Epidermal Growth Factor Receptor therapy.

176 Radionuclide molecular imaging of human epidermal growth factor receptor type 2 (HER2) expressio

177 d-generation, CNS-active, irreversible, oral epidermal growth factor receptor tyrosine kinase inhibit

178 The oncogene epidermal growth factor receptor variant III (EGFRvIII)

179 Expression of insulin receptor and epidermal growth factor receptor was reduced in hippocam

180 eceptor tyrosine kinase family members EGFR (epidermal growth factor receptor) and Her2 are among the

181 i ASPP2 signaling by inhibitors of the EGFR (epidermal growth factor receptor) signaling pathway-iden

182 evant proteins, including apolipoprotein E4, epidermal growth factor receptor, CD71 and programmed de

183 (ER), progesterone receptor (PR), and human epidermal growth factor receptor-2 (HER2) are the three

184 oss of RHAMM function or expression promoted epidermal growth factor receptor-regulated MMP-9 express

185 ) were analyzed to map the safety profile of epidermal growth factor receptor-tyrosine kinase inhibit

186 ting loci has been characterized as a mutant epidermal growth factor receptor.

187 nding lectin]; and 3 with lower risk: ErbB1 [epidermal growth factor receptor], GDF-11/8 [growth diff

188 ctivated Rac1 in membrane ruffles induced by epidermal growth factor stimulation.

189 Interleukin-17 and epidermal growth factor were identified as important inf

190 ancient and similarly sized domains, such as Epidermal Growth Factor, Fibronectin Type 3, Immunoglobu

191 decreased EGFR degradation when activated by epidermal growth factor, increased EGFR protein expressi

192 ses the hydroxylation of Asp/Asn-residues in epidermal growth factor-like domains (EGFDs).

193 cacy of locally administered heparin-binding epidermal growth factor-like growth factor (HB-EGF), a p

194 ls and identified a defect in endocytosis of epidermal growth factor.

195 Most angiosperms produce trichomes-epidermal hairs that have protective or more specialized

196 data identifies NIPP1 as a key regulator of epidermal homeostasis and as a potential target for the

197 how that PRANCR is a novel lncRNA regulating epidermal homeostasis and identify other lncRNA candidat

198 ivalents, validating their essential role in epidermal homeostasis and reinforcing the critical natur

199 y skin signature genes linked to the loss of epidermal homeostasis and uniquely repressed an IFN-indu

200 Histologic lesions consisted of epidermal hydropic change leading to vesiculation and er

201 a]anthracene treatment alone but reduced the epidermal hyperplasia during 12-O-tetradecanoylphorbol-1

202 utophagy, attenuates imiquimod (IMQ)-induced epidermal hyperplasia in adult mice as well as naturally

203 Mutant embryos display epidermal hyperplasia, but also apical cell extrusions,

204 erized by intense inflammation and prominent epidermal hyperplasia.

205 tion of NIPP1 in mouse epidermis resulted in epidermal hyperproliferation, a reduced adherence of bas

206 inoglycan still induced nodule primordia and epidermal infections, but further progression of the sym

207 Desmosomes govern epidermal integrity while GJs facilitate small molecule

208 component of anchoring fibrils at the dermal-epidermal junction.

209 the cell cycle, in mouse epidermis and human epidermal keratinocytes (HEKs).

210 ific keratins K1, K10 and K2 in normal human epidermal keratinocytes (NHEK) and two important cell li

211 melanin effectively prevented neonatal human epidermal keratinocytes (NHEK) from G2/M phase arrest un

212 Normal human epidermal keratinocytes and 3D raft treatment with SR-A

213 and MARCO reduced SNA uptake in normal human epidermal keratinocytes and 3D rafts after topical appli

214 us (VZV) is a skin-tropic virus that infects epidermal keratinocytes and causes chickenpox.

215 sions, and migration and that differentiated epidermal keratinocytes are a component of the stem cell

216 Incubation of normal human epidermal keratinocytes at 4(o)C or with sodium azide pr

217 s cell carcinoma (SCC) are both derived from epidermal keratinocytes but are phenotypically diverse.

218 Epidermal keratinocytes dialogue with sensory neurons th

219 ts on matched primary dermal fibroblasts and epidermal keratinocytes from human donors on three bioma

220 tips, where they are transferred to adjacent epidermal keratinocytes through pathways that involve mi

221 f CXCL14 was significantly down-regulated in epidermal keratinocytes with age.

222 We found that in epidermal keratinocytes, soy PG inhibited TLR2 and TLR4

223 g-term ex vivo expansion of functional human epidermal keratinocytes.

224 e also attenuated by chrysin pretreatment of epidermal keratinocytes.

225 Here we characterize the dermal T cell and epidermal Langerhans cell components of the skin immune

226 several cell types, including keratinocytes, epidermal Langerhans cells (LC), and dermal dendritic ce

227 , we investigated the age-related changes in epidermal Langerhans cells (LCs), which play a sentinel

228 l structural and functional integrity of the epidermal layer in developing Xenopus embryos.

229 ggrin, a critical component of the uppermost epidermal layer.

230 und a significant reduction in the number of epidermal LCs in sun-protected skin with age.

231 es in human skin results in the reduction of epidermal LCs with age, and CXCL14 may provide a therape

232 ransient expression in Nicotiana benthamiana epidermal leaf cells demonstrated localization of this f

233 or suppressor can promote early premalignant epidermal lesion formation.

234 as most prominently time-of-day dependent in epidermal leukocytes, suggesting that these cell types p

235 or severe burns and, recently, diseases with epidermal loss, such as junctional epidermolysis bullosa

236 We reveal in mouse interfollicular epidermal, mammary and hair follicle epithelia that geno

237 modulator of melanosome movements in primary epidermal melanocytes and identifies ILK and GSK-3 as im

238 x disease in which autoimmune destruction of epidermal melanocytes results in patches of depigmented

239 We previously showed that in human epidermal melanocytes UVA activates a G-protein coupled

240 Moreover, the results suggest that epidermal metabolite profiling may be useful for other a

241 As a possible application of epidermal metabolite profiling, we asked whether metabol

242 Accordingly, here, a programmable epidermal microfluidic valving system is devised, which

243 skin-impermeable drugs by creating transient epidermal micropores, and micropore lifetime directly af

244 heral neuropathy in mammals, indicating that epidermal mitochondrial H(2)O(2) and its effectors could

245 contained UV/oxidative-signature disruptive epidermal mutations that manifested positive selection i

246 de life-threatening conditions such as toxic epidermal necrolysis, Stevens-Johnson syndrome, and hype

247 tiforme, Stevens-Johnson syndrome, and toxic epidermal necrolysis.

248 istence of antigen-specific Trm cells in the epidermal niche.

249 It shares variable stomatal complexes and epidermal oil cells with angiosperm leaves from the lowe

250 re differentiated into ESPCs and PSC-derived epidermal organotypic rafts (PSC-EORs).

251 t that CED-3 caspase negatively regulates an epidermal p38 stress-responsive MAPK pathway to promote

252 Stomatal patterning is regulated by the EPIDERMAL PATTERING FACTOR (EPF) family of secreted pept

253 des: EPF1 enforces stomatal spacing, whereas EPIDERMAL PATTERNING FACTOR-LIKE9 (EPFL9), also known as

254 grations that precede and succeed ACD during epidermal patterning.

255 These pHSCs showed a psoriatic epidermal phenotype and characteristic cytokine profiles

256 F4 or auxin signaling, and overexpression of epidermal phyB suppresses thermoresponsive growth, indic

257 se both epidermal PIF4 transcription and the epidermal PIF4 DNA-binding ability.

258 Additionally, inactivation of epidermal PIF4 or auxin signaling, and overexpression of

259 we show that high temperatures increase both epidermal PIF4 transcription and the epidermal PIF4 DNA-

260 ses thermoresponsive growth, indicating that epidermal PIF4-auxin pathways are essential for the temp

261 n addition, DX314-induced keratinization and epidermal proliferation effects are observed in a rhino

262 However, the role of autophagy in epidermal proliferation, particularly under conditions w

263 ectively, autophagy plays a positive role in epidermal proliferation, which is in part via regulating

264 Key epidermal psoriatic features, including T-cell and dendr

265 or HF-SC migration and their contribution to epidermal regeneration.

266 biquitin ligase FBXO32 specifically inhibits epidermal renewal without affecting overall proliferatio

267 Despite a compensatory transcriptional epidermal repair response, the protective epidermal func

268 om endogenous retroelements were detected in epidermal RNA, which suggests that double-stranded RNA d

269 in as a mechanism to maintain the aggressive epidermal SCC phenotype.

270 We found overexpression of TC-PTP increased epidermal sensitivity to DMBA-induced apoptosis and it d

271 eostatic TM epidermis is distinct from other epidermal sites and has discrete proliferative zones wit

272 Epidermal squamous cell carcinoma (SCC) is a common and

273 subpopulations, three recapitulating normal epidermal states, and a tumor-specific keratinocyte (TSK

274 a global public health burden originating in epidermal stem and progenitor cells (ESPCs) of the skin

275 cells persist stochastically throughout the epidermal stem cell population.

276 We use here the seam cells, a population of epidermal stem cells in Caenorhabditis elegans, to study

277 In dividing epidermal stem cells, we found that cell growth is coupl

278 ed spatiotemporal ERK MAPK dynamics in human epidermal stem cells.

279 Phase transitions during epidermal stratification crowded cellular spaces with li

280 cytoplasm vacuolization and partial loss of epidermal stratification.

281 nalyses of these genes showed enrichment for epidermal stress-responsive factors, including the fatty

282 Epidermal structure and barrier function were investigat

283 Thus, epidermal structure and function are driven by phase-sep

284 rsed the effects of TC-PTP overexpression on epidermal survival and proliferation.

285 lacement of the normally occurring dendritic epidermal T cells (DETC) concomitant with an accumulatio

286 ed corneocyte fragility, decreased dendritic epidermal T cells, and an exaggerated percutaneous immun

287 eaction correlates with the number of CD8(+) epidermal T(RM) cells, which again correlates with aller

288 However, the composition and dynamics of the epidermal T-cell subsets during ACD are not known.

289 ed PASI by 74% versus controls and decreased epidermal thickness by 56%.

290 Epidermal thickness did not differ with obesity but the

291 hyperproliferation, coinciding with reduced epidermal thickness.

292 ed for predicted binding motifs for critical epidermal transcription factors including AP1, GRHL, and

293 res and accurately predicts future overlying epidermal tumor formation.

294 The underlying causes are unclear, but epidermal, unmyelinated axons have been shown to be the

295 transfer to keratinocytes, a key process for epidermal UV photoprotection.

296 Stomata are epidermal valves that facilitate gas exchange between pl

297 To discover epidermal vulnerabilities, patient-derived pluripotent s

298 nized and non-cutinized domains of the outer epidermal wall, and affects cell size.

299 k within the inner cell walls, not the outer epidermal wall, in guiding organ flattening of organ pri

300 s their ability to invade and migrate during epidermal wound healing.