ライフサイエンスコーパス: epidermoid carcinoma

1 -3 ovarian carcinoma, A375 melanoma, or A431 epidermoid carcinoma.

2 receptor expression is upregulated in human epidermoid carcinoma.

3 ) on Vero, human rhabdomyosarcoma, and human epidermoid carcinoma 2 cells.

4 e show that dense cultures of human salivary epidermoid carcinoma A253 cells exhibited elevated expre

5 Madin-Darby canine kidney (MDCK), and human epidermoid carcinoma (A253) cells contain the T-cell fac

6 filipin also inhibited CT activity in human epidermoid carcinoma A431 and Jurkat T lymphoma cells th

7 st two-hybrid interaction library from human epidermoid carcinoma A431 cells and identified four cell

8 ed/activated in response to insulin in human epidermoid carcinoma A431 cells as well as in mouse 3T3-

9 wth factor (EGF) receptor signaling in human epidermoid carcinoma A431 cells mediated by EGF.

10 We show that ultraviolet-B exposure of human epidermoid carcinoma A431 cells results in G1 cell cycle

11 Exposure of epidermoid carcinoma A431 cells to 0.1-1.0 mM ONOO- resu

12 ll-cycle deregulation and apoptosis of human epidermoid carcinoma A431 cells.

13 nd visible light, induces apoptosis in human epidermoid carcinoma A431 cells.

14 ) prevents completely the formation of human epidermoid carcinoma A431 xenografts in athymic mice.

15 In this study, employing human epidermoid carcinoma (A431) cells and silicon phthalocya

16 on of Bcl-2 in PDT apoptosis-sensitive human epidermoid carcinoma (A431) cells resulted in enhanced a

17 l cycle dysregulation and apoptosis of human epidermoid carcinoma (A431) cells.

18 ells and in PDT of apoptosis-sensitive human epidermoid carcinoma (A431) cells.

19 n to have excellent oral activity in a human epidermoid carcinoma (A431) xenograft model in nude mice

20 radiation- and chemotherapy-resistant human epidermoid carcinoma and a hormone-refractory prostate a

21 or regressions at 10 mg/kg in the A431 human epidermoid carcinoma and at 40 mg/kg for the SF767 human

22 ocotrienol would suppress the growth of A431 epidermoid carcinoma and B16-F10 melanoma in vitro and i

23 Nude mice bearing s.c. A431 epidermoid carcinoma and injected i.m. with 100 microg o

24 Subcutaneously implanted human A431 epidermoid carcinoma and U251 glioblastoma cells served

25 cumulated in KB-V-1, a vinblastine-resistant epidermoid carcinoma but not in KB-3-1, drug-sensitive w

26 n accordance with its false identity as oral epidermoid carcinoma, but only 57 articles that provided

27 e experimental treatment of established A431 epidermoid carcinoma, C33a cervix carcinoma, and LS174T

28 g a soluble Mr 42,000 sialidase into a human epidermoid carcinoma cell line (A431) provided an approa

29 In a human epidermoid carcinoma cell line (i.e. A431), 12(S)-HETE a

30 of IL-6 following UV irradiation of a human epidermoid carcinoma cell line (KB), and of normal human

31 rplastic parathyroid glands and in the human epidermoid carcinoma cell line A431, which mimics hyperp

32 cell lines (KB100 and KB300) from the human epidermoid carcinoma cell line KB by exposure to CPT.

33 A transfectant of A431 (a human epidermoid carcinoma cell line) expressing chloramphenic

34 n in human hepatoma cell lines and the human epidermoid carcinoma cell line, A431 which express TGFal

35 ce to toxin-mediated killing compared to the epidermoid carcinoma cell line, A431, despite succumbing

36 ed the phosphorylation of c-Met in the human epidermoid carcinoma cell line, A431.

37 ically inhibit in vivo metastasis of a human epidermoid carcinoma cell line, HEp-3.

38 rface expressed EGFRs in A431 cells, a human epidermoid carcinoma cell line, is composed of two subpo

39 lly expressed by the highly metastatic human epidermoid carcinoma cell line, M(+)HEp3.

40 vaded, to the same extent the human cervical epidermoid carcinoma cell line, ME180.

41 Treatment of apoptosis-sensitive human epidermoid carcinoma cells (A431) with PDT resulted in s

42 By blocking uPAR expression in human epidermoid carcinoma cells (HEp3), we have now identifie

43 growth factor receptor (EGFR)-overexpressing epidermoid carcinoma cells A431.

44 -phase arrest followed by apoptosis in human epidermoid carcinoma cells A431.

45 yanine (Pc4)-PDT-mediated apoptosis in human epidermoid carcinoma cells A431.

46 Here we show, using KB-V-1 MDR human epidermoid carcinoma cells and [3H]palmitic acid as meta

47 its degradation rate in infected A431 human epidermoid carcinoma cells and resulting in the alterati

48 f all tumor cells tested, only A431 (A431-V) epidermoid carcinoma cells developed partial resistance

49 Overexpression of EWI-2 in A431 epidermoid carcinoma cells did not alter cell adhesion o

50 y and cell uptake were studied in A431 human epidermoid carcinoma cells transfected with human CCK2R

51 tional regulator of VEGF expression, in A431 epidermoid carcinoma cells under both normoxic and hypox

52 ding of IRDye 800CW EGF to intact A431 human epidermoid carcinoma cells was quantified in a microplat

53 Human A431 epidermoid carcinoma cells were captured onto collagen-c

54 Human epidermoid carcinoma cells were incubated with 10 microg

55 Stimulation of A431 human epidermoid carcinoma cells with EGF resulted in a transi

56 CKI-responsive cell line (A431 human vulvar epidermoid carcinoma cells with functional Rb) and one C

57 we re-expressed a series of CD151 mutants in epidermoid carcinoma cells with near total, RNAi-mediate

58 IL-9 inhibited the migration of A-431 cells (epidermoid carcinoma cells) induced either by IFN-gamma

59 s, including WI-38 lung diploid cells, A-431 epidermoid carcinoma cells, and HeLa cervix epitheloid c

60 tyrosine in response to HGF-SF in A431 human epidermoid carcinoma cells, expressing the HGF/SF recept

61 is of loss-of-function mutants of A431 human epidermoid carcinoma cells, lacking protein kinase A, pr

62 zation when compared with nonmelanoma/KB-3-1 epidermoid carcinoma cells.

63 ogenous Brk and IRS-4 interact in A431 human epidermoid carcinoma cells.

64 e endogenous beta2AR expressed in A431 human epidermoid carcinoma cells.

65 uman alpha folate receptor in nasopharyngeal epidermoid carcinoma cells.

66 se 1B (PTP1B) was investigated in A431 human epidermoid carcinoma cells.

67 a highly disseminating variant of human HEp3 epidermoid carcinoma, HEp3-hi/diss.

68 We previously demonstrated in human epidermoid carcinoma KB cells that UVB irradiation activ

69 rine P388 leukemia, as well as against human epidermoid carcinoma KB/8.5 implanted sc in athymic mice

70 ed fractionation monitored with a human oral epidermoid carcinoma (KB) cell line.

71 oplasmic vesicles of HEp-2 cells (cells from epidermoid carcinoma larynx tissue).

72 adical surgery as the preferred treatment of epidermoid carcinoma of the anal canal.

73 a panel of tumor cell lines including human epidermoid carcinoma of the nasopharynx (KB) and its eto

74 east cancer (MCF-7), lung carcinoma (A-549), epidermoid carcinoma of the nasopharynx (KB), renal canc

75 idal action of ionizing radiation in a human epidermoid carcinoma xenograft (SQ-20B).

76 perior in vivo antitumor activity in a human epidermoid carcinoma xenograft model with no overt toxic

77 EGFR-overexpressing human epidermoid carcinoma xenografts in rats were used in all