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1 ngle febrile, unprovoked seizures, or status epilepticus).
2 rotective actions in rodent models of status epilepticus.
3 was active throughout the episode of status epilepticus.
4 mpal neuronal death in vivo following status epilepticus.
5 however, it reduced susceptibility to status epilepticus.
6 pilocarpine- and kainic acid-induced status epilepticus.
7 process of epileptogenesis following status epilepticus.
8 llowing excitatory injury elicited by status epilepticus.
9 not predict progression to refractory status epilepticus.
10 resembles clinical EEG recordings of status epilepticus.
11 9%); 96 cases (54.2%) were refractory status epilepticus.
12 mortality, severity, and duration of status epilepticus.
13 pocampus of humans who died following status epilepticus.
14 ne drug for benzodiazepine-refractory status epilepticus.
15 er intra-amygdala kainic acid-induced status epilepticus.
16 outcomes in patients with convulsive status epilepticus.
17 auditory discrimination had incident status epilepticus.
18 are alone in patients with convulsive status epilepticus.
19 e context of a stroke-like episode or status epilepticus.
20 ts admitted to the ICU for convulsive status epilepticus.
21 epticus can be inhibited to terminate status epilepticus.
22 juries, including pilocarpine-induced status epilepticus.
23 tients with nonprincipal diagnoses of status epilepticus.
24 ts admitted to the ICU for convulsive status epilepticus.
25 ally in patients with complex partial status epilepticus.
26 red in the evaluation of the child in status epilepticus.
27 sions to be weaned with resolution of status epilepticus.
28 ered first-line therapy for pediatric status epilepticus.
29 picion for nonconvulsive seizures and status epilepticus.
30 ered in young children with recurrent status epilepticus.
31 iatric patients with super-refractory status epilepticus.
32 aesthetic liberation after refractory status epilepticus.
33 nosocomial infections diagnosed after status epilepticus.
34 hdrawal of anaesthetics in refractory status epilepticus.
35 raphic seizures, or 3) electrographic status epilepticus.
36 ll on our cohort of ICU patients with status epilepticus.
37 efinitions of seizures, epilepsy, and status epilepticus.
38 mortality, severity, and duration of status epilepticus.
39 mortality observed during focal onset status epilepticus.
40 ation in relation to the induction of status epilepticus.
41 he treatment of paediatric convulsive status epilepticus.
42 EEG pattern concerning for incipient status epilepticus.
43 olfactory system in the initial phase status epilepticus.
44 e management of paediatric convulsive status epilepticus.
45 r an early and transient increase poststatus epilepticus.
48 revalence was high in post convulsive status epilepticus (33.5%, 20.2%, and 32.9%), CNS infection (23
49 of patients with a second episode of status epilepticus accounted for 16 additional instances of ran
50 ctive review of adults with new-onset status epilepticus admitted to Mayo Clinic, Rochester MN betwee
51 B mice (n = 127) that had experienced status epilepticus after systemic treatment with pilocarpine 31
52 domisation to cessation of convulsive status epilepticus, analysed in the modified intention-to-treat
53 shifts in neuronal activity (kainate status epilepticus and CaMKIIa Gq DREADD activation) triggered
54 induced status epilepticus, and after status epilepticus and daily treatment beginning 24 hours later
55 iated with higher rates of refractory status epilepticus and death (with every 1mg/L: odds ratio 1.01
57 ogression between 2 and 5 months post-status epilepticus and drastically reduced the frequency of spo
58 emonstrate that blocking P2Y(1) after status epilepticus and during epilepsy has potent anticonvulsiv
59 rch to enable early identification of status epilepticus and efficacy of anti-epileptic drugs will be
61 for the treatment of drug-refractory status epilepticus and epilepsy.SIGNIFICANCE STATEMENT This is
62 The primary outcome of cessation of status epilepticus and improvement in the level of consciousnes
64 trographic seizures or electrographic status epilepticus and mortality or short-term neurologic outco
65 ondents continue to rely on myoclonic status epilepticus and neuroimaging, which were favored over me
66 n the contralateral hippocampus after status epilepticus and resulted in more frequent spontaneous se
68 to 5 weeks before pilocarpine-induced status epilepticus and these cells were then eliminated beginni
69 esthesia without developing recurrent status epilepticus, and a wean failure as either recurrent stat
71 maticus, alcohol withdrawal syndrome, status epilepticus, and acute behavioral psychologic disturbanc
72 mice 5 days after pilocarpine-induced status epilepticus, and after status epilepticus and daily trea
73 tients with encephalitis, seizures or status epilepticus, and antibodies to unknown neuropil antigens
74 tilation for more than 24 hours after status epilepticus, and arterial hypotension requiring vasopres
75 ed as a strategy for super-refractory status epilepticus, and better evidence for their use may becom
76 enytoin, and valproate in established status epilepticus, and here we describe our results after exte
77 common EEG seizure onset patterns and status epilepticus, and postulates a role for synaptic plastici
78 mendations regarding nutrition during status epilepticus are lacking, and it is unclear whether restr
79 truly refractory and super-refractory status epilepticus are seen infrequently at any given instituti
81 ing anaesthetic weaning in refractory status epilepticus as predictors of successful weaning from int
82 ardized and age-standardized rates of status epilepticus as the underlying cause of death in the Unit
83 HHV-7 primary infection with febrile status epilepticus as well as the role of reactivation of laten
84 vs. computed tomography in new-onset status epilepticus, as well as high rates of identification of
85 motor rhythmogenesis and touch-evoked status epilepticus associated with markedly impaired KCC2-depen
86 and midazolam significantly increased status epilepticus-associated neuronal injury in various brain
87 and critically, that the duration of status epilepticus at the time of treatment is an important pre
88 n vehicle-treated mice 2 months after status epilepticus, but remained at only 63% of controls in rap
89 ary efficacy outcome was cessation of status epilepticus by 10 minutes without recurrence within 30 m
91 to accurately predict the outcome of status epilepticus by measures of discrimination and calibratio
93 meable AMPA receptors modified during status epilepticus can be inhibited to terminate status epilept
95 al etiology, duration and severity of status epilepticus, Charlson comorbidity index, and treatment w
96 tatus Epilepticus Severity Score, and status epilepticus classification per the Status Epilepticus Se
102 ceived more calories and proteins per status epilepticus day, and increasing nutritional support was
103 e risk, 1.01; 95% CI, 1.001-1.01) per status epilepticus day, independent of potential confounders (i
104 dition characterized by recurrence of status epilepticus despite use of deep general anesthesia, and
105 f nonconvulsive seizure/nonconvulsive status epilepticus detected by continuous electroencephalograph
106 onconvulsive seizure or nonconvulsive status epilepticus detected by routine electroencephalography w
107 ons, likely reflecting an increase in status epilepticus diagnoses through improved diagnostic sensit
110 es at ICU admission and occurrence of status epilepticus during ICU stay were not associated with neu
111 d focal epilepsies with an electrical status epilepticus during slow sleep-like EEG pattern (six pati
113 nonconvulsive seizure, nonconvulsive status epilepticus, either nonconvulsive seizure or nonconvulsi
114 on, seizures including non-convulsive status epilepticus, endocrinopathy, or thiamine deficiency.
115 f nonconvulsive seizure/nonconvulsive status epilepticus/epileptiform activity and odds ratio of dete
116 de updates on identifying children in status epilepticus, etiologic considerations, and the rationale
118 8.85; 95% CI, 4.87-16.08), myoclonic status epilepticus (false-positive rate, 0.05; 95% CI, 0.02-0.1
119 prospectively demographics, clinical status epilepticus features, treatment, and outcome at discharg
123 ews protocols for treating convulsive status epilepticus from 33 emergency medical services systems i
124 long-standing hypothesis that febrile status epilepticus (FSE) can cause brain injury, particularly t
127 associated with poorer outcome after status epilepticus; furthermore, it portends higher infection r
128 febrile seizures; febrile or afebrile status epilepticus (>30 min); or with clusters of two or more f
129 g-resistant epilepsies and refractory status epilepticus has been further defined and is expected to
130 ons, which in their most severe form, status epilepticus, have a high mortality rate if not quickly t
131 mortality and the marked increase in status epilepticus hospitalizations, likely reflecting an incre
132 s are administered after the onset of status epilepticus, however, their effect on seizure severity i
133 synaptic transmission associated with status epilepticus; however, gamma-aminobutyric acidergic synap
136 is review discusses the management of status epilepticus in children, including both anticonvulsant m
141 ted P2X7R responses after focal-onset status epilepticus in mice, comparing changes in the damaged, i
143 ty of data on patients with new-onset status epilepticus in patients without a prior history of epile
145 Using a recently developed model of status epilepticus in postnatal day 7 rat pups that results in
146 -year study period, the prevalence of status epilepticus in primary admissions of septic patients inc
147 or treatment of paediatric convulsive status epilepticus in the UK; however, some evidence suggests t
148 ill present the current definition of status epilepticus, including a recently modified operational d
149 he definition and general approach to status epilepticus, including resource use, should evolve with
151 a potential role of Orai1 channels in status epilepticus induced by chemoconvulsants, we examined acu
152 n a well-characterized mouse model of status epilepticus-induced epilepsy (systemic pilocarpine).
153 rable and undesirable consequences of status epilepticus-induced TrkB activation are mediated by dist
155 across two different mouse models of status epilepticus (intra-amygdala kainic acid and intraperiton
171 that increased calorie intake during status epilepticus is independently associated with unfavorable
173 diazepine-refractory, or established, status epilepticus is thought to be of similar pathophysiology
174 n barrier pathology in rats following status epilepticus, late electrocorticography to identify epile
175 We then show that the persistent status epilepticus-like activity is associated with a reduction
177 had a similar exacerbating effect on status epilepticus-like activity, while a high concentration of
179 tion of GABAergic interneurons in the status epilepticus-like state, actually enhanced epileptiform a
180 not transplanted- pulmonary congestion with epilepticus (likely not related) versus transplanted inf
184 eizures progressed and at the peak of status epilepticus, motor and somatosensory cortex, retrospleni
187 encephalitis with seizures (one with status epilepticus needing pharmacologically induced coma; one
188 as described in patients with stroke, status epilepticus, neurodegenerative disorders, neurotrauma, a
190 firmed status epilepticus, refractory status epilepticus on day 1, "super-refractory" status epilepti
191 rate of progression to EEG-confirmed status epilepticus on the first day was lower in the hypothermi
192 ncreased C-reactive protein levels at status epilepticus onset were associated with higher rates of r
193 e male patients) developed refractory status epilepticus or epilepsia partialis continua along with e
194 nd a wean failure as either recurrent status epilepticus or the resumption of anaesthesia for the pur
198 y calorie intake and outcome in adult status epilepticus patients deriving from a 5-year cohort with
199 ion-standardized hospitalizations for status epilepticus per 100 000 persons increased by 56.4% (inci
200 younger than 18 years with convulsive status epilepticus presenting to 1 of 11 US academic pediatric
201 xcessive activation of TrkB caused by status epilepticus promotes development of temporal lobe epilep
202 ic regression analysis disclosed that status epilepticus, psychosis and cognitive dysfunction were st
203 is purpose, we used two distinct post-status epilepticus rat models, in which epilepsy was induced wi
204 for refractory generalised convulsive status epilepticus, rather than additional trials of second-lin
205 duced to 63% of controls 5 days after status epilepticus, recovered to 93% of controls in vehicle-tre
206 Pharmacologic P2Y(1) blockade during status epilepticus reduces also associated brain damage, delays
207 oencephalographically (EEG) confirmed status epilepticus, refractory status epilepticus on day 1, "su
208 s-sectional studies including 408 304 status epilepticus-related hospital visits using generalizable
209 d to estimate population-standardized status epilepticus-related hospitalization rates using Internat
210 imited data exist detailing trends in status epilepticus-related hospitalizations and mortality.
212 exists between the relatively stable status epilepticus-related mortality and the marked increase in
216 hs to under 18 years, with convulsive status epilepticus requiring second-line treatment, were random
217 lepticus on day 1, "super-refractory" status epilepticus (resistant to general anesthesia), and funct
218 ts, and older adults with established status epilepticus respond similarly to levetiracetam, fospheny
219 in approach in controlling refractory status epilepticus (RSE) and super-refractory status epilepticu
220 ing brain capillaries from rats after status epilepticus (SE) and in chronic epileptic (CE) rats.
221 set of severe acute seizures known as status epilepticus (SE) are crucial for the process of epilepto
223 levant question is whether early post-status epilepticus (SE) evoked chloride dysregulation is import
224 precursors into the hippocampus after status epilepticus (SE) greatly restrained SRS and alleviated c
225 r restraining seizures, cannot thwart status epilepticus (SE) induced neurodegeneration or down-strea
232 ed a rat model of pilocarpine-induced status epilepticus (SE) to investigate HIF-1alpha expression an
235 s in determining KCC2 activity during status epilepticus (SE) using knockin mice in which S940 is mut
238 in 50% of rats reduced threshold for status epilepticus (SE), accelerated epileptogenesis, and once
239 Transient brain insults, including status epilepticus (SE), can trigger a period of epileptogenesi
240 , such as trauma, stroke, anoxia, and status epilepticus (SE), cause multiple changes in synaptic fun
241 al inflammatory response ensues after status epilepticus (SE), driven partly by cyclooxygenase-2-medi
242 t-born DGCs after pilocarpine-induced status epilepticus (SE), whereas normotopic DGCs synapse onto b
249 rtile range, 42-76], 55% male, median Status Epilepticus Severity Score 3 [interquartile range, 2-4])
250 nation and calibration indicated that Status Epilepticus Severity Score performed reasonably well on
253 raphics, status epilepticus etiology, Status Epilepticus Severity Score, and status epilepticus class
254 thetic drugs, mechanical ventilation, Status Epilepticus Severity Score, refractoriness of seizures,
255 rolonged seizures in super-refractory status epilepticus (SRSE) have been shown to cause neuronal dea
258 ters resulted in immediate relapse of status epilepticus, suggesting a pivotal role of deep brain sti
260 frequency of complications following status epilepticus termination and return to premorbid function
262 isks of complications occurring after status epilepticus termination for no return to premorbid neuro
264 (SRSE) is a life-threatening form of status epilepticus that continues or recurs despite 24 hours or
265 boy with a prolonged super-refractory status epilepticus that eventually resolved after commencing de
266 The choice of drugs for patients with status epilepticus that is refractory to treatment with benzodi
267 n children and adults with convulsive status epilepticus that was unresponsive to treatment with benz
268 benzodiazepine-refractory convulsive status epilepticus, the anticonvulsant drugs levetiracetam, fos
269 d that, following pilocarpine-induced status epilepticus, there are two independent changes in HCN fu
270 successful anaesthetic liberation in status epilepticus; these findings are absent in patients with
271 idelines for management of refractory status epilepticus; this is, however, based on weak evidence.
272 are typically titrated in refractory status epilepticus to achieve either seizure suppression or bur
274 We examined nationwide variability in status epilepticus treatment by analyzing 33 publicly available
276 rials, such as the ESETT (Established Status Epilepticus Treatment Trial), compare effectiveness of a
277 ng pediatric patients with convulsive status epilepticus, treatment with lorazepam did not result in
279 domisation to cessation of convulsive status epilepticus was 35 min (IQR 20 to not assessable) in the
285 lizations were categorized by whether status epilepticus was the principal diagnosis, whether the pat
287 quired epilepsy induced by electrical status epilepticus, we show that oxidative stress occurs in bot
288 tability emerged around 6 weeks after status epilepticus, well after the onset of chronic seizures an
292 that 48% of children presenting with status epilepticus were unresponsive to benzodiazepine treatmen
294 treatment option in super-refractory status epilepticus when other treatment options have failed.
295 itically ill patients with convulsive status epilepticus who were receiving mechanical ventilation to
296 ith 11 episodes of serial seizures or status epilepticus, who underwent MRI and (18)F-FET PET, were s
297 diagnostic evaluation of the child in status epilepticus will help identify causes, which may require
298 and subsequently developed refractory status epilepticus, with dramatic electroclinical improvement u
299 m underlying benzodiazepine-resistant status epilepticus, with relevance to how this life-threatening
300 S), a subtype of new onset refractory status epilepticus, with the recombinant interleukin-1 (IL1) re